Subject: Fallon on Lyme Brain (Mollicutes and Tripalmitoyl Cysteine)
Date: Dec 2, 2009 6:55 PM
FALLON ARTICLE BELOW ABOUT SYSTEMIC
LYME BRAIN
===============
Once one has Chronic Lyme, one then has
stealth bacteria, especially if treated with
antibiotics. You know, L-forms and of course
the ever-infamous downstream Pam3Cys tolerance
and incompetence to mycoplasmal infections:
http://www.actionlyme.org/Pam3Cys_Version15.htm
Shows up like this:
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2632055/?tool=pubmed
which is why we always feel so
goshbalmed sh*tty.
However, these mycoplasma-attached-to-
erythrocytes and all the other downstream
mechanisms which have been recorded as associated
with Pam3Cys tolerance
http://www.actionlyme.org/Pam3Cys_Version15.htm
is really, in fact, a function of penises.
How do we know?
Because nothing is Lyme, and therefore
what is nothing must be something, which
means psychiatric. And psychiatric means
"has to do with penises." And "in between
is the Twilight Zone:"
http://groups.google.com/group/sci.med.diseases.lyme/msg/771ff9a0de47ca88?dmode=source
Tsk, you really should take a course in
Logic, Bri, so you can unnastands these things.
Kathleen M. Dickson
http://www.actionlyme.org
PS: Mollicutes Hijacking Metabolism (Fatiggy
anyone??):
http://www.ncbi.nlm.nih.gov/pubmed?term=1021090[uid]&cmd=DetailsSearch&log$=details
Aust J Exp Biol Med Sci. 1976 Oct;54(5):449-58.
Eperythrozoon ovis: the difference in carbohydrate metabolism between
infected and uninfected sheep erythrocytes.
Sutton RH.
The glucose utilization lactic and pyruvic acid production and oxygen
uptake of normal and Eperythrozoon ovis infected sheep erythrocytes
were measured under aerobic conditions. Infected cells showed marded
increases in both glucose utilization and acid production as compared
with controls. Uninfected erythocyte samples which included a
percentage of reticulocytes comparable to that found in E. ovis
infection showed no apparent difference in glucose utilization and
lactic acid production form the normal control erythrocytes, although
considerable increases in the oxygen uptake were recorded.
http://www.ncbi.nlm.nih.gov/sites/entrez?db=pubmed&cmd=link&linkname=pubmed_pubmed&uid=1021090&ordinalpos=1&log$=relatedarticles&logdbfrom=pubmed
PSS: Sweeg went Borneo on Lisa Masterson
when she started talking about Dark Field.
She then ended up in the nuthouse for
saying such TRUE things about real
science...
Tully at Fort Detrick:
http://ijs.sgmjournals.org/cgi/reprint/40/2/160
Awww, aren't ^^^ they cute?
===================================
FALLON
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2632055/?tool=pubmed
Arch Gen Psychiatry. 2009 May;66(5):554-63.
Regional cerebral blood flow and metabolic rate in persistent Lyme
encephalopathy.
Fallon BA, Lipkin RB, Corbera KM, Yu S, Nobler MS, Keilp JG, Petkova
E, Lisanby SH, Moeller JR, Slavov I, Van Heertum R, Mensh BD, Sackeim
HA.
Department of Psychiatry, College of Physicians and Surgeons of
Columbia University, New York, New York 10032, USA. ba...@columbia.edu
CONTEXT: There is controversy regarding whether objective
neurobiological abnormalities exist after intensive antibiotic
treatment for Lyme disease. OBJECTIVES: To determine whether patients
with a history of well-characterized Lyme disease and persistent
cognitive deficit show abnormalities in global or topographic
distributions of regional cerebral blood flow (rCBF) or cerebral
metabolic rate (rCMR). DESIGN: Case-controlled study. SETTING: A
university medical center. PARTICIPANTS: A total of 35 patients and 17
healthy volunteers (controls). Patients had well-documented prior Lyme
disease, a currently reactive IgG Western blot, prior treatment with
at least 3 weeks of intravenous cephalosporin, and objective memory
impairment. MAIN OUTCOME MEASURES: Patients with persistent Lyme
encephalopathy were compared with age-, sex-, and education-matched
controls. Fully quantified assessments of rCBF and rCMR for glucose
were obtained while subjects were medication-free using positron
emission tomography. The CBF was assessed in 2 resting room air
conditions (without snorkel and with snorkel) and 1 challenge
condition (room air enhanced with carbon dioxide, ie, hypercapnia).
RESULTS: Statistical parametric mapping analyses revealed regional
abnormalities in all rCBF and rCMR measurements that were consistent
in location across imaging methods and primarily reflected
hypoactivity. Deficits were noted in bilateral gray and white matter
regions, primarily in the temporal, parietal, and limbic areas.
Although diminished global hypercapnic CBF reactivity (P < .02) was
suggestive of a component of vascular compromise, ***the close
coupling between CBF and CMR suggests that the regional abnormalities
are primarily metabolically driven.*** Patients did not differ from
controls on global resting CBF and CMR measurements.
CONCLUSIONS: Patients with persistent Lyme encephalopathy have
objectively quantifiable topographic abnormalities in functional brain
activity. These CBF and CMR reductions were observed in all
measurement conditions. Future research should address whether this
pattern is also seen in acute neurologic Lyme disease.
PMID: 19414715 [PubMed - indexed for MEDLINE]
"[Real] scientists are *fiercely* independent. That's the good
news."-- NIH's Top Fool, Anthony Fauci
GOOD ARTICLE----------------
>
> "[Real] scientists are *fiercely* independent. �That's the good
> news."-- NIH's Top Fool, Anthony Fauci