The NYTimes' Crimes, "Trojan Horse," "Stealth Disabler" - it's the same thing
Mort Zuckerman
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Subject: [SpinLyme] The NYTimes' Crimes, "Trojan Horse," "Stealth
Disabler" - it's the same
thing
Date: Aug 29, 2008 10:04 AM
ARTICLE BELOW
=====================
http://groups.google.com/group/scilyme2/browse_thread/thread/727b3a6b408d2c88?hl=en
Immune Suppression from Fungi (mentioned in the article below):
http://www.actionlyme.org/BIOWEAPONEERS_CORIXA_YALE_TLRS.htm
What you want to do is come up with a sturdy bacterium that can
cause plant rot and damage to livestock, especially the kind that
does not show up until after the grain has been stored for half
a year - like fungi - and slowly reduces fertility in livestock
such that it is not noticed initially. That's a "stealth disabler."
http://www.actionlyme.org/JohnDunn_Brookhaven.htm
"It's the perfect stealth pathogen," says John Dunn at the
Department of Energy and Department of Defense at the Brookhaven
cyclotron where they make medical nuclides...
=================================================
So, I am more inclined to believe that Lyme was an accidental
release from Plum Island, than anything deliberate, because what
if the world found out?
What if the world figured out that it is next to impossible that
Lyme, Connecticut (Plum Island) was not the outbreak area?
http://www.actionlyme.org/BIOWEAPONEERS_CORIXA_YALE_TLRS.htm
VIII. Ed Bosler and outbreak areas
http://www.ncbi.nlm.nih.gov/sites/entrez?Db=pubmed&Cmd=DetailsSearch&Term=3577493[uid]
(This supports the assertion that Plum Island was the original
outbreak area.)
Guess what?
Too late.
Plus, the whole thing is made to appear worse due to the NYT's Holc
Noble
episode. And guess what? I was *there* at the Lyme Foundation's
office
in Hartford www.lyme.org that day that Holc Noble was there.
Ie., I know what some of the Lyme cryme data was that was given to
Holc
Noble, since Karen asked me to go retrieve it, LOL.
Thus, the NYTimes' cover up of the Lyme crymes is real and not a joke.
I was there.
KMDickson
==========================================
http://ftrsupplemental.wordpress.com/category/nato/
February 15, 2007
History of Lyme Disease as a Bioweapon
Posted by ftrsummary under 9/11, AIDS, Bio Weapons, Iraq, NATO, Nazi
No Comments
Lyme is a Biowarfare Issue
A BRIEF HISTORY BY ELENA COOK
Introduction
The world of Lyme disease medicine is split into two camps – the US
government-backed
“Steere camp”, which maintains the disease is hard-to-catch, easily
cured, and rarely
causes chronic neurological damage, and the “ILADS camp”, which
maintains the opposite.
The Steere camp is intricately bound up with the American biowarfare
establishment,
as well as with giant insurance and other corporate interests with a
stake in the
issue. The ILADS doctors lack such connections, but are supported
instead by tens
of thousands of patients rallying behind them.
Because the Steere camp has been massively funded and promoted by
federal agencies,
its view has dominated Lyme medicine not just in the US, but across
much of the
world. The result has been suffering on a grand scale. Below is a
concise history
of the military aspects of this cover-up.
- - - -
Weapons of Mass Infection
The development of biological weapons has never been confined to
dictatorships or
“rogue” regimes. During the Second World War America, Britain and
Canada collaborated
closely on developing offensive bioweapons, and offensive research
continued as
an openly acknowledged activity of the US scientific establishment
during the Cold
War. Only in 1972 was this work banned by international treaty.
Meanwhile the Maryland-based
labs at Fort Detrick, for example, had produced millions of
mosquitoes, ticks and
other vectors for the purpose of spreading lethal germs.[i] The island
of Gruinard,
off the coast of Scotland, was only declared habitable again in 1990,
nearly fifty
years after the British first contaminated it during anthrax
experiments. [ii]
Ticks, which vector Lyme disease, have been studied as biowarfare
instruments for
decades. Such well-known biowar agents as tularaemia and Q-fever are
tick-borne.
The Borrelia genus of bacteria, which encompasses the Borrelia
burgdorferi species-group
(to which Lyme disease is attributed), was studied by the infamous WW2
Japanese
biowar Unit 731, who carried out horrific experiments on prisoners in
Manchuria,
including dissection of live human beings. [iii] Unit 731 also worked
on a number
of other tick-borne pathogens.
After the war, the butchers of Unit 731 were shielded from prosecution
by the US
authorities, who wanted their expertise for the Cold War. [iv] The US
government
also protected and recruited German Nazi bioweaponeers under the aegis
of the top-secret
Operation Paperclip. [v]
Borreliosis, or infection with microbes belonging to the borrelia
genus, had been
dreaded during the Second World War as a cause of the often-fatal
disease relapsing
fever. The new post-war era of penicillin meant that many bacterial
infections could
now be easily cured. However, borrelia were known for their ability to
adopt different
forms under conditions of stress (such as exposure to antibiotics).
Shedding their
outer wall, (which is the target of penicillin and related drugs),
they could ward
off attack and continue to exist in the body.
Lyme disease is not usually fatal, and it is sometimes argued that,
with rapidly
lethal agents like smallpox and plague available, an army would have
no interest
in it. However, what is important to understand here is that
incapacitating or “non-lethal”
bioweapons are a major part of biowarfare R&D[vi], and have been for
decades.
For example, during the Second World War, brucellosis, chronically
disabling but
not usually fatal, was a major preoccupation. Military strategists
understand that
disabling an enemy’s soldiers can sometimes cause more damage than
killing them,
as large amount of resources are then tied up in caring for the
casualties. An efficient
incapacitating weapon dispersed over a civilian population could
destroy a country’s
economy and infrastructure without firing a shot. People would either
be too sick
to work, or too busy looking after those who were.
The EIS and the “Discovery” of Lyme
Modern Lyme history begins in 1975 when a mother in the town of Old
Lyme, Connecticut
reported the outbreak of a strange, multi-system disease. The town
lies directly
opposite the Plum Island biowarfare research lab where, according to
former Justice
official John Loftus, Nazi scientists brought to the US after WW2 may
have test-dropped
“poison ticks”. [vii] It should be noted that Loftus’ reputation for
gathering accurate,
hard-hitting information is strong – strong enough to bring down in
disgrace the
former Chancellor of Austria and Secretary-General of the UN Kurt
Waldheim, after
the latter’s wartime SS record was revealed.
While it’s not yet known if Plum Island experimented on Lyme-causing
borrelia, the
lab’s directors openly admitted to Michael Carroll, author of a
recently-published
book which is endorsed by two former State Governors, that they kept
“tick colonies”.
The “hard tick” Amblyomma americanum, a known carrier of Borrelia
burgdorferi, was
one of the subjects of the Island’s experiments. [viii]This tick is
not the one
most commonly associated with transmitting Borrelia burgdorferi, but
it is implicated
in harbouring Borrelia lonestari, believed to be the cause of a “Lyme-
like illness”
in the American south. [ix]
Carroll’s book reveals a shocking disregard for safety, in this lab
handling some
of the most dangerous germs on earth. Eyewitnesses described how
infected animals
were kept in open-air pens. Birds swooping down into the pens could
have picked
up and spread infected ticks worldwide.
When Polly Murray made her now-famous call to the Connecticut health
department
to report the strange epidemic among children and adults in her town,
her initial
reception was lukewarm. However, some weeks later, she got an
unexpected call from
a Dr David Snydman, of the Epidemic Intelligence Service (EIS), who
was very interested.
He arranged for fellow EIS officer Dr Allen Steere to get involved. By
the time
Mrs. Murray turned up for her appointment at Yale, the doctor she had
expected to
see had been relegated to the role of an onlooker. Allen Steere had
taken charge
– and his views were to shape the course of Lyme medicine for the next
thirty years,
up till today. [x]
To understand the significance of all this, we need a closer look at
the Epidemic
Intelligence Service, the EIS.
The EIS is an elite, quasi-military unit of Infectious Disease experts
set up in
the 1950’s to develop an offensive biowarfare capability. Despite the
banning of
offensive biowar in the 1970’s, the crack troops of the EIS continue
to exist, ostensibly
for non-offensive research into “emerging disease” threats, a blanket
phrase covering
both bioweapon attacks and natural epidemics at the same time.
Graduates of the
EIS training program are sent in to occupy strategic positions in the
US health
infrastructure, taking leadership at federal and state health
agencies, in academia,
industry and the media. The organisation also extends its influence
abroad, training
officers for public health agencies in Britain, France, the
Netherlands etc. [xi]
[xii]
In fact a high proportion of Steere camp Lyme experts are involved
with the EIS.
Given that the EIS is a small, elite force, (in 2001 the CDC revealed
there were
less than 2500 EIS officers in existence since the unit was first
created in 1951
[xiii]), it seems incredible that so many of America’s top Infectious
Disease experts
would devote their careers to what they themselves claim is a “hard-to-
catch, easily-cured”
disease.
Within a few years of Steere’s “discovery” of Lyme disease (the unique
Lyme rash,
and certain associated symptoms, had been recognised in Europe nearly
a century
before), it was announced that its bacterial cause had been
identified. The microbe
was accidentally found by biowarfare scientist Willy Burgdorfer and
was subsequently
named for him. Burgdorfer has championed the Lyme patients’ movement
and is not
suspected of any wrongdoing. However it is not impossible that he was
unwittingly
caught up in a chain of events that were not as random as they might
have seemed.
Burgdorfer was a Swiss scientist who had been recruited by the US
Public Health
Service in the 1950’s. He was highly experienced with both ticks and
borrelia, but
after being told that the government were not interesting in funding
work with the
latter, he switched to work with Rickettsia and other pathogens. [xiv]
In 1981,
Burgdorfer was sent a batch of deer ticks by a team studying Rocky
Mountain Spotted
Fever on the East Coast. In charge of the team was one Dr Jorge
Benach. [xv] Benach
subsequently spent much of his career as a Steere camp Lyme
researcher. In 2004
he was chosen as recipient for a $3 million biowarfare research grant.
[xvi]
Cutting open some of Benach’ ticks, Burgdorfer noticed microfilaria
(microscopic
worm young). This was a subject he had been studying recently, only
these microfilaria
were different. They were exceptionally large, large enough to be seen
with the
naked eye.[xvii] His curiosity naturally piqued, he opened up several
more ticks.
There he was surprised to find the spiral-shaped germs of borrelia.
Cultivation is necessary in order to isolate bacteria for study, so
that diagnostic
tests, vaccines or cures can be developed. Borrelia are very difficult
to grow in
culture. However, by “lucky coincidence”, another scientist had
recently joined
the lab where he worked, and had apparently been involved in an
amazing breakthrough
in this area. So naturally Burgdorfer handed the infected ticks over
to him. [xviii]
That scientist was Dr. Alan Barbour, an officer, like Steere and
Snydman, of the
Epidemic Intelligence Service, with a background in work on anthrax,
one of the
most terrifying biowarfare agents known. [xix]
EIS man Barbour therefore became the first to isolate the prototype
organism on
which all subsequent Lyme disease blood tests would be based. [xx]
This is very
significant, as a huge body of evidence [xxi] indicates the
unreliability of these
tests, which are routinely used to rule out the disease. Additionally,
all DNA detection
of the Lyme agent in ticks and animals is ultimately based, directly
or indirectly,
on the genetic profile of the strain first isolated by Barbour.
Shortly after Barbour’s discovery, other species and strains of the
Lyme-causing
bacteria were isolated, especially in Europe. They were all classified
based on
their resemblance to Barbour’s organism, and have been grouped into a
category called
Borrelia burgdorferi sensu lato or “Bbsl” for short. However, a
borrelia was subsequently
found in the southern US (referred to briefly above) which appears not
to be a member
of Barbour’s Bbsl group at all.[xxii]
The bacteria, named Borrelia lonestari, often evades detection on Lyme
blood tests,
is not found using DNA tests, and does not grow in Barbour’s culture
medium which
is used world-wide for lab study. [xxiii]And yet, it appears to cause
an illness
identical to Lyme – down to the “bullseye rash”, which, though not
present in all
patients, is considered unique to Lyme disease.
In 2005 Barbour, who spent much of his career studying the “hard-to-
catch, easy-to-cure”
Lyme disease, was placed in charge of the multi-million new biowarfare
mega-complex
based at University of California at Irvine (UCI). [xxiv] Barbour is
joined there
by his close colleague and fellow Steerite Jonas Bunikis, author of
recent papers
calling for a restrictive approach to Lyme diagnosis.
The Spread and the Spin
By the late 1980’s it was realised that Lyme disease was rapidly
spreading out of
control. Cases were reported across America, Europe and Asia. Federal
health agencies
launched a major propaganda effort to limit diagnosis and so
artificially “contain”
the epidemic. The National Institute of Health (NIH) appointed
biowarfare expert
Edward McSweegan as Lyme Program officer. [xxv] Under his leadership
the diagnostic
criteria was skewed to exclude most sufferers, especially those with
chronic neurological
illness. McSweegan’s successor at NIH, Dr Phil Baker, is an anthrax
expert [xxvi],
and has continued his policies.
The Center for Disease Control (CDC) is another federal body which has
had a major
impact on how Lyme is diagnosed and treated. Its influence extends
abroad, with
European public health departments drawing up policies based on CDC
guidelines.
It should be remembered that it is the CDC which trains the Epidemic
Intelligence
Service, and much of the leadership of CDC has traditionally been
drawn from EIS
ranks. Therefore it comes as no surprise to learn that David Dennis,
the head of
vector-borne diseases at CDC, with massive influence over Lyme issues,
was involved
with the EIS. However, we could legitimately wonder why, at lower
levels of the
CDC hierarchy, EIS officers - the nation’s heavyweight infectious
disease experts
- continue to play such a major role in investigating the supposedly
“hard-to-catch,
easily cured” Lyme. (For example, EIS officers Martin Schriefer and
Captain Paul
Mead.) [xxvii].[xxviii]
In 2001, responding to the protest of thousands of patients that
standard two or
three-week antibiotic courses were not sufficient, the NIH
commissioned biowarfare
scientist Mark Klempner to study persistence of Lyme infection. ILADS
doctors had
found that patients left untreated in the early phase often needed
long courses
of antibiotics, [xxix] sometimes for years. Klempner, however,
concluded that persistent
Lyme infection did not exist. In 2003 Klempner was appointed head of
the new $1.6
billion biowarfare top-security facility being developed at Boston
University. Shortly
after, the news emerged that there had been an escape of the deadly
bug tularaemia
which was not properly reported to the authorities. [xxx]
In 2005 the author discovered a document on the NIH website listing
Lyme as one
of the potential bioterrorism agents studied in BSL-4 (top security)
labs. After
this was publicised, the NIH announced they had made a “mistake”, and
removed the
words “Lyme disease” from the page. (At the time of writing, the
original is still
available in cached Internet archives. [xxxi]) However, at around the
same time,
a CDC source leaked the identical information to the Associated Press.
[xxxii] Moreover,
the Science Coalition, comprising entities as prestigious as the
American Medical
Association, Yale University, and the American Red Cross, maintain a
website which,
at the time of writing, also lists Lyme as a disease studied for its
biowarfare
potential. [xxxiii] Could these three major organisations all have, co-
incidentally,
made the same “mistake”?
In 2004 the UK government denied that Lyme was a threat in Britain and
told Parliament
that no Lyme research had been conducted since 1999. [xxxiv] Yet the
report of the
official UK delegation to an international conference on the
prevention of bioterrorism
revealed that Lyme was being studied at Porton Down, Britain’s top
biowarfare facility.
[xxxv] Britain, and many other European countries, take their lead on
Lyme from
a body called EUCALB, rooted in Steere camp methodology. NATO has also
been directly
involved in moves to “harmonise” European Lyme diagnosis along
Steerite lines
A Bug of Many Talents
Lyme’s ability to evade detection on routine medical tests, its myriad
presentations
which can baffle doctors by mimicking 100 different diseases, its
amazing abilities
to evade the immune system and antibiotic treatment, would make it an
attractive
choice to bioweaponeers looking for an incapacitating agent. Lyme’s
abilities as
“the great imitator” might mean that an attack could be misinterpreted
as simply
a rise in the incidence of different, naturally-occurring diseases
such as autism,
MS, lupus and chronic fatigue syndrome (M.E.). Borrelia’s inherent
ability to swap
outer surface proteins, which may also vary widely from strain to
strain, would
make the production of an effective vaccine extremely difficult. (A
vaccine developed
for the public by the Steere camp in collaboration with Glaxo
Smithkline was pulled
from the market a few years ago amid class action lawsuits [xxxvi].)
Finally, the
delay before the appearance of the most incapacita
ting symptoms would allow plenty of time for an attacker to move away
from the scene,
as well as preventing people in a contaminated zone from realising
they had been
infected and seeking treatment. Often in the early period there is no
rash, only
vague flu-like or other non-specific symptoms which might be dismissed
by GP’s,
or ignored by the patient.
The 2003 proposal for a rapid-detection method for biowarfare by Dr JJ
Dunn of Brookhaven
National Lab seems to add further grounds for suspicion. It is based
on the use
of two “sentinel” germs – plague and Lyme. [xxxvii]
In 1999 Lyme patient advocacy leader Pat Smith was amazed to find, on
visiting an
Army base at an old biowar testing ground in Maryland, that the US
Dept. of Defence
has developed a satellite-linked system that enables soldiers to read,
in real-time,
off a display on their helmet’s visor, information about the rate of
Lyme-infected
ticks wherever they may be on earth. Unit commanders could update the
database using
state-of-the-art portable PCR machines, which test for Lyme DNA in
soldiers bitten
by ticks. [xxxviii] The use of such cutting-edge technology for a
supposedly “hard-to-catch,
easy-to-cure” illness seems odd, to say the least!
Lyme is often complicated by the presence of co-infecting diseases in
the same tick,
e.g. those caused by the microbes of babesia, bartonella, mycoplasma
(believed by
some researchers to be the cause of Gulf War illness), ehrlichia,
microfilaria and
encephalitis viruses. Investigations into some of these, too, have
been led by American
biowar experts.
It could be argued that some of these Lyme researchers have been
awarded biowar-related
grants simply because they are Infectious Disease specialists, which
is a natural
terrain from which to recruit. After all, research budgets for biowar
have ballooned
massively since the anthrax attacks of 2001; there is a demand for
large numbers
of personnel to work on such projects.
Well, there are two things that could be said here. First, researchers
who have
spent much or most of their careers studying a “hard-to-catch, easily-
cured” disease
would not appear to be the best choice as recipients of this type of
grant, unless
the “easily-cured” disease had some relation to biowarfare. Second,
while some infectious
disease specialists began to study biowarfare organisms for the first
time after
2001, this is not necessarily the case with the Steerites. Klempner,
for example,
was studying ways to increase the virulence of Yersinia pestis, the
causative agent
of plague, over 20 years ago [xxxix]; Barbour researched anthrax for
the Army in
the 1970’s. [xl]
The defeat of Saddam Hussein in the 1991 Gulf War was followed by the
drawing up,
by the UN Special Commission (UNSCOM) of a list of microbes to be
monitored in Iraq.
Among them - the borrelia genus in general, and Borrelia burgdorferi
in particular.
[xli] UNSCOM also included organisms such as ehrlichia and babesia,
which are often
present in Lyme-infected ticks, and are acquired as concurrent
illnesses when a
person is bitten.
There are other organisms on the UN list not generally associated, in
the public
mind, with biowarfare, and it could be argued that the UN was simply
being extra
cautious by casting a wide net. However, whether Lyme bacteria were
present in Iraq
at that time or not, they certainly are today, and US Army manuals
warn soldiers
to protect themselves from the disease [xlii] If we are to accept the
traditional
Steerite explanation for the rise of Lyme – that it is a natural
consequence of
a recent population explosion of deer due to reforestation, combined
suburbanisation,
bringing humans into contact with forests – then the presence of Lyme
in the dusty
sand dunes of Iraq seems perverse.
And what of the doctors of the opposing camp, those associated with
ILADS? ILADS
doctors and researchers increasingly find themselves persecuted,
victims of spurious
charges made against them to Medical Boards, and are hounded out of
their professions.
At the time of writing, paediatrician Dr Charles Ray Jones, credited
by thousands
of parents with restoring the health of their disabled children, is
under trial,
accused of misconduct. A few years ago, Dr Lida Mattman, a Nobel Prize
nominee who
worked on an alternative culture medium for Lyme, was ordered to shut
down operations
by police who arrived at her lab with handcuffs. Dozens of doctors who
had been
treating Lyme successfully according to their clinical judgement,
rather than relying
on insensitive blood tests or arbitrary limits on antibiotic duration,
have been
forced to stop. The president of ILADS, Dr Raymond Stricker, has told
the press
he believes Lyme disease is a bioweapon.
Summary
Lyme disease is the subject of hot controversy, with the “Steere camp”
claiming
it is an easily cured ailment, while the ILADS camp views it as a
severely disabling,
multi-symptom neurological disease.
The number of Steere camp Lyme researchers with a background in the
Epidemic Intelligence
Service (EIS) and/or biowarfare research is too numerous to be pure co-
incidence.
Two scientists who have played a central role in the Lyme story,
Barbour and Klempner,
have been placed in charge of new biowar super-labs set up in the
aftermath of 9-11,
where they are aided by some of their Steerite colleagues. Others,
while not in
charge of super-labs, are nevertheless in receipt of substantial
grants for biowarfare
research.
The United States and some of its NATO allies have a long and sordid
history of
experimentation into biological weapons of mass destruction and mass
incapacitation.
The Borrelia genus and ticks as biowar vectors have been studied for
decades, and
recent revelations about the Plum Island disease lab, across the water
from old
Lyme, Connecticut are worrying. The development of the so-called “non-
lethal weapons”
has been a major part of biowar science for decades.
Suspicion is further fuelled by the declaration by America’s National
Institute
of Health that a document on their website listing Lyme as a microbe
studied for
bioterrorism potential was a “mistake”, just at the time that a CDC
source leaked
the same “mistake” to the Associated Press. British delegates at an
international
conference on the prevention of bioterrorism revealed that intense
work on Lyme
and other tick-borne disease is conducted at the UK’s top biowar lab
at Porton Down.
Lyme has been chosen as a “sentinel organism” in a method of rapidly
detecting bioweapons,
and the whole genus, or category, of borrelia was included among those
to be monitored
by the UN in Iraq after the first Gulf War. US soldiers in Iraq today
are warned
by the military to protect themselves against the disease.
It’s possible to see the modern history of Lyme as a string of events
with an EIS
member at every crucial node. The discovery of new Lyme-causing
borrelia, genetically
distinct from the Borrelia burgdorferi group first cultured by EIS
officer Alan
Barbour, throws up the question as to whether the Bbsl organisms he
introduced to
medicine was the only, (or even the most) relevant borrelia. The
testing and diagnostic
regimens based on the views of Barbour, Steere, etc and backed by
federal health
agencies such as CDC and NIH currently condemn huge numbers of Lyme
patients to
a medical limbo, without treatment or recognition for their disease.
The cost in
human suffering may be unimaginable.
- - - -
[i] Williams P, and Wallace D, “Unit 731, the Japanese Army’s Secret
of Secrets”,
Hodder and Stoughton 1989, p284
[ii] BBC news website http://news.bbc.co.uk/1/hi/scotland/1457035.stm
[iii] Howard Cole, Chief of Intelligence at America’s Chemical Warfare
Service,
reported in “Unit 731” , p105
[iv] “Unit 731”. Op cit.
[v] Carroll, Michael “Lab 257-The Disturbing Story of the Government’s
Secret Germ
Laboratory”, Harper Collins 2004
[vi] Joint Non-Lethal Weapons Directorate> https://www.jnlwd.usmc.mil/
vii] “Lab 257” op cit.
[viii] ibid.
[ix] Bacon et al, “Glycerophosphodiester phosphodiesterase gene (glpQ)
of Borrelia
lonestari identified as a target for differentiating Borrelia species
associated
with hard ticks”, J Clin Microbiol 2004 May;42(5):2326-8.
[x] Murray, Polly, “The Widening Circle”, St Martin’s Press 1996
[xi] http://www.cste.org/PS/2006pdfs/PSFINAL2006/06-EC-01FINAL.pdf.
[xii] Center for Disease Control website> www.cdc.gov/eis;
http://www.cdc.gov/od/oc/media/pressrel/r010720.htm
[xiii] ibid.
[xiv] Barbour, Alan “Lyme Disease: the Cause, the Cure, the
Controversy”, The John
Hopkins University Press 1996, p 29.
[xv] “The Widening Circle”, op cit. p.174
[xvi]> > New York State Office of> Science, Technology and Academic
Research,
NYStar News http://www.nystar.state.ny.us/nl/archives2004/longislandA08-04.htm
[xvii]Beaver, PC and Burgdorfer, W “A microfilaria of exceptional size
from the
ixodid tick, Ixodes dammini, from Shelter Island, New York” J
Parasitol 1984 Dec;70(6):963-6
[xviii] Barbour, Alan “Lyme Disease: the Cause, the Cure, the
Controversy”, The
John Hopkins University Press 1996 p30.
[xix] University of California at Irvine website
http://today.uci.edu/news/media_advisory_detail.asp?key=80
[xx] Barbour, Alan op cit.
[xxi] See 17 pages of citations from peer-reviewed medical literature
archived at
http://www.lymeinfo.net/medical/LDSeronegativity.pdf
[xxii] Bacon et al, op cit.
[xxiii] Varela et al, “First Culture Isolation of Borrelia lonestari,
Putative Agent
of Southern Tick-Associated Rash Illness “, J Clin Microbiol. 2004
March; 42(3):
1163–1169
[xxiv] UCI Medical Centre http://www.ucihealth.com/News/Releases/06-05BiodefenseResearch.htm
[xxv] McSweegan biography http://advance.uri.edu/quadangles/spr2004/story9.htm
-
profile6
[xxvi] NIH News> http://www.nih.gov/news/pr/apr2006/niaid-23.htm
[xxvii] CDC http://www.cdc.gov/eis/conference/archives/2003ProgramAbstracts.pdf
[xxviii] NY Academy of Sciences http://www.nyas.org/biodef/speakers.asp
[xxix] ILADS Treatment Guidelines> http://www.ilads.org/guidelines.html
[xxx] http://www.washingtonpost.com/wp-dyn/articles/A27646-2005Jan21.html
[xxxi] For example, this one at:
http://web.archive.org/web/20050208095246/http:/www.niaid.nih.gov/factsheets/detrick_qa.htm
[xxxii] See MSNBC article featured on http://www.ctlymedisease.org/featurearticle02.htm
[xxxiii] http://www.sciencecoalition.org/glossary/glossary_main.htm
[xxxiv] http://www.theyworkforyou.com/wrans/?id=2004-12-15a.203881.h&s=”lyme+disease”
[xxxv] http://www.opbw.org/new_process/mx2004/bwc_msp.2004_mx_wp51_rev.1_E.pdf
[xxxvi] http://business.guardian.co.uk/story/0,3604,663032,00.html
[xxxvii]> Sherr, V comment in the Lancet
http://www.thehumansideoflyme.net/viewarticle.php?aid=60&PHPSESSID=109a322bd7e9529b2e74c151aface839
[xxxviii] http://www.lymediseaseassociation.org/EICSconference.doc
[xxxix] Pollack C, Straley SC, Klempner MS, “Probing the
phagolysosomal environment
of human macrophages with a Ca2+-responsive operon fusion in Yersinia
pestis” Nature.
1986 Aug 28-Sep 3;322(6082):834-6.
[xl] Rees et al, “Epidemiologic and laboratory investigations of
bovine anthrax
in two Utah counties in 1975” Public Health Rep, 1977 Mar-Apr;92(2):
176-86.
[xli] “Note by the Executive Chairman of the Special Commission
established pursuant
to paragraph 9 (b) (i) of Security Council resolution 687 (1991)”
Document date:
17 March 1995 Ref- S/1995/208> http://www.fas.org/news/un/iraq/s/s1995-0208.htm
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