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Hunger in the type-2 diabetic.

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Andrew B. Chung, MD/PhD

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Feb 9, 2007, 9:26:10 PM2/9/07
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convicted neighbor Kumar wrote:

> http://groups.google.com/group/sci.med.cardiology/msg/6d728ff9844134d2?

"Why such overeating can't be right eating of one nutrient but
overeating of others?"

It is the hungry person who falsely believes that "hunger is bad" who
overeats.

A hungry person is healthy and is therefore not deficient in any
nutrients so that subsequent overeating does automatically mean
overnourishment:

http://HeartMDPhD.com/HeartMDPhD.com/overweight.asp

Andrew <><
--
Andrew B. Chung, MD/PhD
http://EmoryCardiology.com

Kumar

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Feb 10, 2007, 3:12:42 AM2/10/07
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Andrew B. Chung, MD/PhD wrote:
> Kumar wrote:
>
> > http://groups.google.com/group/sci.med.cardiology/msg/6d728ff9844134d2?
>
> "Why such overeating can't be right eating of one nutrient but
> overeating of others?"
>
> It is the hungry person who falsely believes that "hunger is bad" who
> overeats.
>
> A hungry person is healthy and is therefore not deficient in any
> nutrients so that subsequent overeating does automatically mean
> overnourishment:
To be healthy or to maintain homeostatis and survival, why hunger for
used or excessively excreted nutrients(to maintain survival
indirectly) can't be stimulated? Problem may be in that, we take
complex nutrients composed foods instead of specific nutrient/s based
food, which may cause intake of unrequired nutrient along with whole
food. Choice, palatability, taste, likings, quantity etc....i.e.
specific food for specific/needed requirements can be important.
Hunger is good therefore/because we get it of reqired nutrients, not
of all nutrients.

Glocose/Salt or otherwise related hypertonicity or acidosis
extravascularily, may effect shift of ICF nutrients to ECF resulting
their lower quantities in ICF, compensatory hunger and food intake AND
excess quantity in ECF, blood and in urine to maintain normal blood
levels till body can maintain otherwise hypoemias, if body can't
maintain in chronic long term persisting conditions. Probably, kidney
tries to maintain such abnormailties till it can, but collapse on long
term chronic maintaining. Probably, glucose movement into cells or
insulin's normal senstivity may be effected due to hypertonicity or
acidosis extravascularily(conentration pull towards ECF) showing IR?

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