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[Kerby Reynolds]

Youve probably heard the claims by now: Here's a diet that's delicious, easy to stick with, and guaranteed to help you lose weight effortlessly. Or, perhaps it's supposed to build muscle, protect your joints or prevent Alzheimer's. Whatever the diet and whatever the claim, there's a good chance that it is, indeed, too good to be true.

In recent years, high protein diets are among the most popular, whether the protein is consumed as a supplement (protein shakes for body builders!) or simply a larger than usual portion of a balanced diet (such as The Zone, Atkins or Paleo Diets).

So, consuming enough protein is required to stave off malnutrition; it may also be important to preserve muscle mass and strength as we age. And, in recent years, some have advocated a higher protein diet to rev up metabolism to make it easier to lose excess weight, though success in this regard is highly variable.

But some experts suggest that these recommendations are all wrong and that we should be consuming more protein, up to twice the standard recommendations. Still others claim that the average American diet already contains too much protein.

The short answer is yes. As with most things in life, there can be too much of a good thing and if you eat too much protein, there may be a price to pay. For example, people that eat very high protein diets have a higher risk of kidney stones. Also a high protein diet that contains lots of red meat and higher amounts of saturated fat might lead to a higher risk of heart disease and colon cancer, while another high protein diet rich in plant-based proteins may not carry similar risks.

It's hard to provide a specific answer since so much is still uncertain and the experts themselves don't agree. However, for the average healthy person (who is not an elite athlete or heavily involved in body building) it's probably best to keep total protein intake to no more than 2 gm/kg of ideal body weight; that would be about 125 grams/day for a 140-pound person with a normal body mass index (BMI).

Devisulphur 800 WG is a potent fungicide and acaricide formulated as water-dispersible granules, with 800 gm/kg of Sulphur as its active ingredient. This product offers a broad-spectrum solution for controlling various diseases like downy and powdery mildew, leaf spots, rusts, and scabs, and it is effective on crops such as tomatoes, potatoes, onions, cucurbits, and roses. Additionally, Devisulphur provides essential sulfur nutrients to address sulfur deficiency in crops

Safety precautions include wearing protective gear such as rubber gloves, boots, and a face shield during application, avoiding inhalation of the spray mist, and ensuring the product does not come into contact with skin or eyes. In case of ingestion or skin/eye contact, immediate first aid measures are specified, emphasizing the importance of seeking medical advice for poisoning

Usage instructions detail the dilution process for application, with specific dosages per arpent, hectare, and per liter of water, ensuring compatibility and effectiveness while cautioning against mixing with alkaline products.

Manufactured by UPL Limited and distributed by The Mauritius Co-operative Agricultural Federation Ltd, Devisulphur 800 WG is marked with a WHO Class III rating, indicating moderate toxicity. The product has a re-entry period of 10 hours and a pre-harvest interval ranging from 4 to 5 days, depending on the crop. It allows for up to 8 applications per crop cycle, with intervals of 10-14 days between applications.

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Autoimmune hemolytic anemia of childhood (AIHA) is often refractory to 2 mg/kg of prednisone. We report 2 infants with AIHA treated with initial doses of 5 gm/kg IVGG followed by maintenance doses of 2 gm/kg/wk. Infant #1 had required greater than 20 mg of prednisone/day for more than 6 months and had previously received 2 gm/kg of IVGG without apparent effect. After 5 gm/kg IVGG treatment his hemoglobin rose slowly over 4 weeks from 7.7 to 10.9, despite tapering of prednisone, and remains stable. Infant #2 had his second relapse of immune hemolytic anemia with the hemoglobin decreasing to 7 gm/dl. Two weeks of IVGG treatment stabilized his hemoglobin; 1 weeks after treatment was stopped his hemoglobin fell to 4.5 gm/dl. Reinduction with 5 gm/kg IVGG and continued maintenance restabilized his hemoglobin at 7. Both patients had IgG antibodies which failed to bind to rH null cells after elution and abnormally decreased in vitro nonspecific antibody synthesis. Recent work by Salama et al demonstrates that a part of the RES Fc receptor blockade mediating an increased platelet count during IVGG treatment of ITP is due to low grade red cell immune hemolytic; therefore, they state that AIHA cannot be treated by IVGG. We demonstrate that AIHA can be treated by IVGG albeit at higher dosage than even in ITP (5 gm/kg vs 2 gm/kg). Other possible reasons for difficulty of treatment of AIHA by IVGG or steroids may include larger cell mass, lack of red cell Fc receptors or a more avid RES.

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