Wormser sounds very bioweaponeery
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Mort Zuckerman
Jul 5, 2008, 11:26:37 AM7/5/08
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Subject: Wormser sounds very bioweaponeery
Date: Jul 5, 2008 11:22 AM
I never really looked too closely at the IDSA 2006 regurgitation of
their 2000 batch
of excrement, having known what went on in 2000, and knowing all along
they were
doing a 180 on their own previously published data.
However, before too many days go by (ActionLyme activity is pretty hot
of its own,
via referral), all Lyme victims and their worthy attendants should
keep a copy of
this (the webpage I link below) to their harddrives, and better yet,
also email
it to your foreign terrorist friends in other nations (I'm a
"terrorist,"
according to Yale and the State of Corrupticut for having exposed
their crimes-
that being the object of the Bushie/Mossad illegal wiretapping).
The best outcome is Russia infiltrates Canadian and Mexican business,
such that
when NAFTA happens, they can take American Fraud Corporations (who are
at present
being protected by the USDOJ KarlRovesque US Attorneygate Soap Opera
Digest US Attorneys
and the distracted FBI porn-watchers) for all they've got.
Kathleen M. Dickson
http://www.actionlyme.org/BRAIN_PERMANENT.htm
CHAPTER 7, Lyme Crooks Report Treatment Failure about 20 times
(This chapter is still working, however the reference below
are obviously
available to the interested- 080705, 10:00 AM EST KMDickson)
This is data (20+ reports of persistence past treatment and in
the brain,
by the crooks, themselves), the Lyme crooks (Yale/NYMC/ALDF/IDSA/
Kaiser cabal) refused
to turn over to the Connecticut Attorney General for a year and a half
before settling
out of court.
SAY THE IDSA GUIDELINES (GARY WORMSER, et al, or the Original
ALDF.com "entrepreneurial
trio" gang of:
"The entrepreneurial trio are Durland Fish, Ph.D.,
former director
of the College's Lyme Disease Center and now a research scientist at
Yale; Gary
P. Wormser, M.D., still professor of medicine and pharmacology and
chief of the
Division of Infectious Diseases at the College; and John J. Connolly,
Ed.D., former
College president and current chairman of the board of the American
Lyme Disease
Foundation, Inc., which had its genesis on the Valhalla campus in
1990."
http://www.journals.uchicago.edu/doi/full/10.1086/508667 ◄ "The
Guidelines"
"The notion that symptomatic, chronic B. burgdorferi infection
can
exist despite recommended
treatment courses of antibiotics (tables 2 and 3) in the
absence of objective
clinical signs of
disease, is highly implausible as evidenced by (1) the lack of
antibiotic
resistance in this genus
[39, 40, 310], (2) the lack of correlation of persistent
symptoms with laboratory
evidence of
inflammation or with the eventual development of objective
physical signs
[223, 257, 288, 289],
and (3) the lack of precedent for such a phenomenon in other
spirochetal
infections [315-317].
[Lyme is a "STEALTH DISABLER" and as shown in the Yale
Congenital
Lyme Autopsy report, there was "NO INFLAMMATION." SEE THE BIOMARKERS
and the MARK KLEMPNER CHAPTER; NOT CAPTURING THEIR OWN IDENTIFIED
BIOMARKERS of
disease and instead, deploying invalid mumbo-jumbos is a research
fraud crime.]
Additional compelling evidence against the hypothesis that
persistent symptoms
are the result of
persistent infection is the fact that the concentrations of
antibodies against
B. burgdorferi in
many of these patients diminish to undetectable levels [257,
286, 288, 318].
The panel is unaware
of any chronic infection in which antibody titers diminish
despite persistence
of the causative
organism. [VERY DAMNING statement. This phenomenon is related
to the "stealth
disabler" mechanisms of fungal or mycoplasmal or mycobacterial
antigens- no
antibodies are made against the infection due to TLR2 tolerization and
the downregulation
of HLA or antigen-presenting molecules Wormser is aware of the
immune suppression
aspects of OspA vaccination, since he reported about it:
Gary Wormser reporting the blunting of the immune
response in
vaccinated animals:
http://www.ncbi.nlm.nih.gov/pubmed/10865170?ordinalpos=5&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_RVDocSum
"OspA interferes with the response of lymphocytes
to proliferative
stimuli including a blocking of cell cycle phase progression."
'In syphilis, patients who are regarded as having treatment
failure
typically have persistent or rising titers of antibodies [319]. [THE
IMMUNE SUPPRESSING
PlumIsland PAM3CYS LIPOPEPTIDE OSPA IS NOT FOUND IN TREPONEMA
PALLIDUM]
"Finally, Lyme disease lacks characteristics of other
infections that
justify longer treatment courses, such as infections in
immunodeficient hosts, [YOU
ARE REALLY GONNA BE SHOCKED when you see the Plum Island Mycoplasmal-
immune suppressing
data, the activation of latent viruses data (auto-kill turned off) the
HIV Pam3Cys
immune depression data, and the Fungal Vaccines data] infections in
which a pathogen
is inhibited but not killed by antimicrobial therapy or in which
available antimicrobials
are minimally active in vitro [JOHNSON CULTURING FROM SPHEROPLAST
COULD TAKE MONTHS],
infections caused by an intracellular pathogen [KLEMPNER- "cef
fails"],
infections involving a biofilm, infections on a heart valve, or
infections involving
a clinical site in which there is ischemia, a foreign body, a
sequestrum***, or
frank pus [170]. The “cystic” forms of B. burgdorferi that have been
seen under
certain growth conditions in vitro have not been shown to have any
clinical significance
[320]. [INTRACELLULAR CYSTS are the KEY TO LATENCY, well-known
historically, and
this is an application of the NEGATIVE DATA RULE, which is another
research fraud
crime indictment point; THE MOUSE INFECTIVITY TEST HAS NOT BEEN
PERFORMED ON TREATED
MICE OR MONKEYS]
CDC and NIH Rocky Mountain antibody-gold sphere method
as applied
to borreliosis:
The Method Developed:
http://www.pubmedcentral.nih.gov/picrender.fcgi?artid=261539&blobtype=pdf
*** Applied to Borreliosis by NIH Rocky Mountain Lab:
http://www.pubmedcentral.nih.gov/picrender.fcgi?artid=269963&blobtype=pdf
The Dorward/Garon Gold Test Apparently detects plenty
of shed antigen.
And it looks pretty sequestery to me, not to mention we could never
determine sequestery
sites in a human unless we had a lot of brains to biopsy, although the
dentists
did a pretty good job detecting oral treponemes sequestering in the
brains of Alzheimer's
patients- See Item 23, below.
1975 at an international spirochetal diseases conference
for which Willy
Burgdorfer was present; "The ability of the borrelia, especially tick-
borne
strains to persist in the brain and in the eye after treatment with
arsenic or with
penicillin or even after apparent cure is well known (1). The
persistence of treponemes
after treatment of syphilis is a major area which currently requires
additional
study (3,5,10,11)." -Jay Sanford, US Military Hospital, Bethesda,
MD
As you can see, Gary Wormser, ad IDSA et al, are trying to
deny everything
that Lyme actually is. Lyme is stealth (no antibodies late in the
disease), latent,
intracellular, is resistant to antibiotics (goes into a self-
protective spheroplast
form upon exposure to antibiotics which is not an "end stage" as the
crooks claim), it goes into a "dormant" or serum-starved or
spheroplast
mode (known to the crooks themselves as a "Trojan Horse"), Lyme
belongs
to the class of diseases where there is typically no inflammation. It
does not
belong to NIAID or Anthony Fauci's division, but the parasitic
diseases division
- but it's not going to belong to either the CDC or the NIH at all
after not
too long, because when they're done being investigated we will find
that that
NIH and the CDC are the same as the NEJM "peer-reviewers" - not a one
of them isn't interest conflicted. In fact, we will find out what is
the exact
relationship between the CDC and Kaiser or else the NAFTA-eers of
Canada and Mexico
will discover it and prosecute it.
At this time I recommend that Russia and China infiltrate
Canadian and
Mexican business structures such that when the NAFTA NationRape of the
North American
Continent takes place, Russia can return the favor for the Israeli
Oligarchic NationRape
Escapade under Yeltsin.
More of these clear rebuttals below and in the
RICOCHRON.htm page.
Remember, the IDSA keywords: "INFLAMMATION" and "OBJECTIVE
SIGNS" (meaning they are still referring to "Lyme Disease" as the
Dearborn definition of "autoimmune bad knee" as seen in the RICO
graphics),
because that later comes in in Congenital Lyme ("lack of
inflammation")
and the Biomarkers chapter as regards the description of the immune
suppression
outcomes of Lyme, which is the key to all of their crimes, especially
as regards
this fungal vaccine, LYMErix or ImmuLyme, or rOspA and the activation
of latent
viruses of all kinds.
The crooks' own treatment failure or persistence articles
are below.
The IDSA statement about the cysts or the spheroplasts is
yet another
criminal matter, since they all referenced Dave Nelson's "reversion
to
intact spirochete form from spheroplast within one minute of the
addition of rabbit
blood" report, Russell Johnson said it could take months in culture
for the
cyst form to regenerate in media, and the Mouse Infectivity Test has
never been
tried as a treatment efficacy trial, ever, to my knowledge in lab
animals. They
cannot make the claim that the cyst form hasn't any clinical
significance, since
here we have again the Crooks' Negative Data Rule.
At the present time, there are a whole slew of Russian
scientists now
studying these intracellular serum-starvation ("Stringent Response")
forms
at New York Medical College.
http://iai.asm.org/cgi/reprint/70/6/3061
The difficulty in testing for congenital syphilis- lest we
forget, syphilis
testing was always terrible, despite Wormser's attempt to re-write
history
http://www.pubmedcentral.nih.gov/picrender.fcgi?artid=262629&blobtype=pdf
It's pretty hard to find a cohort nowadays of non-HIV
infected syphilis
patients.
COMPREHENSIVE ARTICLE ON SYPHILIS:
http://www.pubmedcentral.nih.gov/articlerender.fcgi?tool=pubmed&pubmedid=16418521
25 PERSISTENCE ARTICLES (brain, intracellular, spheroplast) by the
CROOKS and/or
THEIR ASSOCIATES
BSK is not the only media these spirochetes grow in. The gold
standard
of persistence is still the Mouse Infectivity Test (MIT), although it
should probably
be the Hamster or some other rodent Infectivity Test, which is the
transfer of SPINAL
FLUID (not synovial fluid, since who cares about knees) from one
infected rodent
to an uninfected one and allow the infection to disseminate elsewhere,
and look
for whole spirochetes by staining, etc or re-ear puncture transfer,
etc., to determine
treatments' efficacies..
CDC's Alan Barbour published in 1986 that rodent brains was
the actual
former storage media for spirochetes.
Yes.
http://mmbr.asm.org/cgi/reprint/50/4/381?view=long&pmid=3540570
"Borrelia spirochetes so reliably infected rodent brains that
we used
to use rodent brains as the main storage media."
For a clarification, hamster/guinea pig/rat/whatever brains
are better than
mouse brains, which is why the crooks use only mouse brains for their
treatment
studies, since they don't want the truth about Lyme revealed,
apparently, ever:
http://www.pubmedcentral.nih.gov/picrender.fcgi?artid=441260&blobtype=pdf
"Borrelia do not survive long in mouse brains."
1944, relapsing fever during WWII, Lancet, Sept 30, 1944, page
436
NEUROLOGICAL COMPLICATIONS OF relapsing fever
Ronald Bodley Scott, DM, OXFD, FRCP
LIEUT.-COLONEL RAMC; OFFICER I/C A MEDICAL DIVISION
THE campaigns in the Lybyan Desert yielded cases of
relapsing fever
of a type not previously known in Egypt. They were due to a infection
with Treponema
recurrentis, whose immunological characteristics have not yet been
determined, transmitted
by the bite of an unnamed argasid tick of the genus ornithodoros,
bearing a close
resemblence to O. erraticus, the vector of the Tunisand strain of
treponema (Adler,
1942). This tick probably inhabited the burrows of desert rodents and
infection
was commonly acquired in caves, slit trenches and tombs.
Characteristic of this fever was the high proportion of
cases in which
the central nervous system was invaded. Most descriptions of the
disease include
the comment that nervous system sequelae occur and that they are more
common in
the tick-botne than in the louse-borne fever; but theirt frequency and
variety are
not generally recognized. This paper is concerned with a small series
of cases
seen in 1941 and 1942; its observations consequently apply to the
disease of the
north-west Africa coast. It is likely, however, that this reservation
is not absolute"
the characteristics of Tr. recurrentus are so labile that strains from
the same
locality, and even from different relapses in the same case, may show
immunological
variations (Ashbel 1943). This the clinician is probably justified in
discounting
the importance of differences of strain and in regarding the tick-
borne relapsing
fever as an entity, however heretical the immunologist may consider
this view.
NEUROTROPIC CHARACTER OF Tr. recurrentis
In the animal the neurotropism of this treponema is well-
established.
Ashbel (1943), investigating 17 strains of Tr. persica, found that
organisms could
be isolated from the brains of guinea pigs 117-398 days after apparent
recovery
from the infection. In some cases this cerebral invasion proved fatal
and post-mortem
examination showed small perivascular hemorrhages and infiltrations
with lymphocytes,
monocytes and macrophages. The neurotropism of various strains has
been shown to
be equally great in other animals (Heronimus 1928).
The predilection of the treponema for nervous tissue in
the animals
raises the question of whether it is similarly neurotropic in man.
Data are not plentiful; but as long ago as 1874 Ponfick
reported petechial
hemorrhages [LIEGNER] in the brains of cases dying in the Berlin
epidemic. Belezky
and Umanskaja (1930) have recorded the findings in 8 fatal cases:
clinical observations
are scanty in their paper but only one had symptoms of disease of the
nervous system.
In all instances microscopy showed a patchy perivascular infiltration
of the pia
with monocytes, lymphocytes and plasma cells, and in places the
cerebral vessels
were encircled by a similar cellular halo. In 3 cases treponemata
were found in
the brain substance, diffusely distributed and in no constant relation
to vessels.
More recently, Ungar has described the case of a woman
dying in the
puerperium with relapsing fever and cerebral symptoms. The post-
mortem findings
included a cholesteatoma of the lateral recess edema of the cisternal
pia-arachnoid
and hemorrhages in the caudal part of the pons and the floor of the
4th ventricle.
Sections showed the Virchow-Robin spaces distended with erythrocytes,
lymphocytes
and monocytes; treponemata were recovered from the cerebrospinal fluid
and from
the tumor.
Evidence of a less convincing nature is provided by
Spielmeyer and Jahnel
(1926) from examination of the brains of paretics in whom therapeutics
infection
with Tr. duttoni had proved lethal. These authors describe a
perivascular infiltration
which differed from that seen in general paresis by virtue of the
scanty plasma
cells and the numerous macrophages.
These observations show that Tr. recurrentis has
neurotropic characters
in man as well as in the animal, and they furnish a morbid anatomical
foundation
for the varied neurological guises the disease may assume...
Treatment.-Details of treatment have been omitted from the
case reports;
but in each instance an organic arsenical was given. There was no
evidence that
these druigs reduced the number of relapses or the ability to invasion
of the nervous
system. Adler, Theodor and Schieber (1936) found that untreated cases
had fewer
relapses than those that received intravenous arsphenamine. Cooper
(1942), while
admitting that the dosage was insufficient, noted a relapse in 41 of
57 cases after
arsenic had been gievn; in 17 Tr. recurrentis was again present in
blood films.
Arsphenamine often appears to cut short a bout of fever and it is
perhaps possible
that more energetic treatment was required. More recently
tryparsamide and bismuith
have been found effective (McAlpine 1943).
Prognosis.- It is not easy to assess prognosis in a
disease of long
duration under the conditions necessarily operative in military
practice; so many
patients are transferred to other hospitals before full recovery.
None of this
series died; indeed it is rarre for tick-borne relapsing fever to be
fatal. The
majority appeared to recover completely but case 9 illustrates the
frequency of
repeated relapses and the chronic ill heatlh which may result from
this disorder.
http://www.actionlyme.org/1944,%20Relapsing%20Fever%20During%20WWII.htm
So, it is a little hard to believe that we don't have the same
disease
they had during World War II and so on and so forth and arsenic as a
treatment for
spirochetes since the time of Paul Ehrlich and so on and that all of a
sudden spirochetes
behave better now and will only attack knees for a month and then roll
over and
die. Were we Lyme victims not really sick, we would have no reason to
search for
the evidence that we knew would exist to prove we are. It's that
simple.
We absolutely knew the data would exist that proves we are as
sick as we
say we are. Think about it. We absolutely knew we could prove these
bastards at
Yale were lying,
Get the whole picture in your mind:
Borreliae are permanent, intracellular brain and nerve
infections
which go dormant in a spheroplast form, and these facts are all
acknowledged and/or
researched and reported, and/or described, and/or referenced by the
Lyme criminals
themselves.
The shed Osps suppress the immune response due to
tolerization,
and..
...the shed Osps suppress the immune system by
anchoring the auto-kill
kinases, or, the immune suppression caused by these Osps results in
the activation
of latent viruses of all kinds.
Lyme Borreliae are usually delivered by a microscopic
tick (US Army:
"ticks could be used to spread diseases"), resulting in chronic,
ongoing
neurologic damage,
But the testing for Lyme only allows for a person to
be diagnosed
with the KNEE-Disease or LYMErix Disease, or the hypersensitivity
reaction to OspA,
which was Steere's new, Dearborn Definition...
Lyme Borreliosis is a triple stealth disabler:
1) The worst cases are not detectable
(deliberately)
2) It's permanent and incurable, so in currently
healthy
people it can be like a Trojan Horse, emerging later as any of the
Great Imitator's
outcomes (ALS, Lupus, MS, etc)
3) Is delivered by microscopic, stealth ticks
Worse yet, the Lyme criminals literally stalk,
wiretap, harass,
order the false arrests of, sic the CPS on, report falsely to medical
boards, and
otherwise attack Lyme victims and their treaters, constantly
persecuting them, especially
the smarter and louder ones and especially as regards congenital Lyme.
as if the
medical community can't figure out that the same thing happens in
congenital
syphilis.
Well, what am I saying? The "medical community"
can't figure anything out.
All of this would have been believable as either a
natural development
"evolutionary bottleneck" or an accidental release from Plum Island,
but
for the harassment.
Why the harassment? Why is the CDC allowing for
this harassment?
Is it because they're protecting their own criminal CDC officers, Alan
Steere
and Allen Barbour, and their own patents with SmithKline in Europe, or
is this bioweapons
information that they don't want to get out?
Or is it because all these secret outcomes have
some meaning
as regards all childhood vaccines damage?
Or is it because the childhood vaccines damage is
some sort
of a deliberate campaign to get rid of the genetically weak, since it
has been tried
before by this same clique of British and American Bankers?
Since the CDC is the CIA of bioweapons and the CIA
works for
the Bigs (Rockefeller), and the Mossad works for the British Bigs
(Rothschild),
it does not matter, since we the people are not the Bigs. We don't
have any
aristocrats in this country, and even if we did, we don't see or know
any intelligent
ones, so they can't claim brains as the essence of their superiority.
==================================================================
*1)
In 1975 there was apparently an international conference on
spirochetal diseases
and a Dr. Jay Sanford of the Uniformed Services Military Hospital in
Bethesda, Maryland
reported:
"The ability of the borrelia, especially tick-borne strains to
persist
in the brain and in the eye after treatment with arsenic or with
penicillin or even
after apparent cure is well known (1). The persistence of treponemes
after treatment
of syphilis is a major area which currently requires additional study
(3,5,10,11)."
-Jay Sanford, US Military Hospital, Bethesda, MD
This textbook was edited by Russell Johnson who was an advisor to
the criminal
gang of the ALDF.com
Russell Johnson later published for the Infectious Diseases
Society of America
that: "Although spirochetes can often be detected in culture media
after 3
weeks of culture, some isolates may not be visible for several
months."
IDSA says we need to culture these suckers for several months.
Did Mark Klempner
do that in his bogus long term treatment "study?"
NO.
=================================================================
2)
http://www.pubmedcentral.nih.gov/picrender.fcgi?artid=373079&blobtype=pdf
By CDC Officer Alan Barbour, in 1986, The Biology of Borrelia Species:
"The propensity for borrelia to go to the brain of infected
mammals suggests
that the relationship between these spirochetes and neural tissues is
not trivial.
Further study of this attraction and the interaction that follows may
reveal the
basis for the significant nerve and brain involvement in Lyme
borreliosis"--
This is self-explanatory. We have previously seen that rodent
brains so reliably
were a home for spirochetes that they were actually the culture
media. We also
acknowledge that ceftriaxone for brain diseases was a treatment IDSA
themselves
used and offered and still use, so Lyme must not be a self-limiting
knee disease
as Allen Steere and Yale's Steven Malawista claim.
And here we have in the same CDC officer's 1986 report that there
are, gasp,
"gemma:"
[You want to GO HERE for other older references published
before the
Great Whore of Medicine, Allen Steere emerged onto the scene]
=================================================================
3) Russell Johnson's patent for the very first Lyme vaccine
"The chronic forms of the disease such as arthritis (joint
involvement),
acrodermatitis chronica atrophicans (skin involvement), and Bannwart's
syndrome
(neurological involvement) may last for months to years and are
associated with
the persistence of the spirochete. A case of maternal-fetal
transmission of B. burgdorferi
resulting in neonatal death has been reported. Domestic animals such
as the dog
also develop arthritis and lameness to this tick-borne infection. For
every symptomatic
infection, there is at least one asymptomatic infection. Lyme disease
is presently
the most commonly reported tick-borne disease in the United States."
--
The patent also says:
"The infection may be treated at any time with antibiotics
such as
penicillin, erythromycin, tetracycline, and ceftriaxone. Once
infection has occurred,
however, the drugs may not purge the host of the spirochete but may
only act to
control the chronic forms of the disease. Complications such as
arthritis and fatigue
may continue for several years after diagnosis and treatment."
================================================================
4) At the time of the writing of this book, CDC officer Alan
Barbour has on
his website, the following statement:
"We are taking a multi-discipline approach, including methods
of genetics,
cell biology, and immunology, to study in depth two spirochetal
diseases: Lyme disease
and relapsing fever. These tick-borne infections are notable for
multiphasic antigenic
variation through DNA recombinations in the case of relapsing fever,
the occurrence
of chronic arthritis in the case of Lyme disease, and invasion of and
persistence
in the brain in the case of both diseases. ---Alan Barbour
============================================================
*5) In 1994 Alan Steere and other published a report entitled:
http://www.annals.org/cgi/content/full/121/8/560
The Long-Term Clinical Outcomes of Lyme Disease: A Population-
based Retrospective
Cohort Study
right arrow Nancy A. Shadick; Charlotte B. Phillips; Eric L.
Logigian; Allen
C. Steere; Richard F. Kaplan; Victor P. Berardi; Paul H. Duray; Martin
G. Larson;
Elizabeth A. Wright; Katherine S. Ginsburg*; Jeffrey N. Katz; and
Matthew H. Liang
15 October 1994 | Volume 121 Issue 8 | Pages 560-567
In that report:
"Patient 12 had had high fever, meningeal symptoms, and
subsequent
arthritis in 1982. She was noted to have a positive serologic test
result for Lyme
disease 4 years later and was treated with 2 weeks of parenteral
penicillin. She
later developed a progressive speech disorder, bradykinesia, and
abnormal ocular
motor function. Magnetic resonance imaging of the brain showed
scattered white matter
lesions in the hemispheres and pons, and she was diagnosed with
supranuclear palsy.
Lumbar puncture showed no selective concentration of antibody in the
spinal fluid.
Nevertheless, she was re-treated with 2 weeks of parenteral
ceftriaxone in 1989
that had no effect on her neurologic symptoms. During the time of
observation, this
patient died. At autopsy, lymphoid mononuclear cells were observed
surrounding the
intracerebral vessels in one section. Using Dieterle silver stain, a
spirochete
was present in the cortex and another was exterior to a leptomeningeal
vessel."
One of Steere's multiply-treated patients died anyway with
spirochetes in
her brain.
How amazing that that suddenly doesn't happen any more, despite no
new breakthroughs
in antibiotic treatments or their delivery.
The Primer's Shell Game is where these Lyme crooks use the
wrong DNA
(or RNA) primers (they use the variable Osp primers instead of the non-
variable
chromosomally encoded) to test Lyme victims. These same crooks use
the correct
DNA primers when they want to find Lyme or borrelia in ticks (to
patent). It's
a shell game. They all know that all that they can use for treatment
outcomes is
non-variable DNA or RNA.
Mark Klempner never reported his primers in his Chronic Lyme
"study,"
and he would not tell me, either, in an email correspondence I had
with him. Therefore,
the Lyme crooks have never validly reported on the presence of
borrelia in humans,
with the exception of Gary Wormser's study of EMs in which he found
2/9 patients
who had EM, who had been treated with antibiotics still had
spirochetes in their
tissues. This data can be found on the HOW RICO WILL BE CHARGED PAGE
===============================================================
*6, 7) In 1994 and in 1996, Steere and Nocton published a reports
of treatment
failure and brain invasion using RNA and DNA methods and they found
treatment failed
in a third of the patients with their OspA primer sets. In the first
report, in
1994, published in the New England Journal of Medicine, that they used
4 primer
sets. Three were for the OspA gene (which we know undergoes antigenic
variation
and therefore these are the wrong probes). The fourth set was for 16S
RNA intragenic
spacer (remember now, for Borrelia burgdorferi, ignoring all the other
possibilities).
These were rather valuable reports, overall, since they prove that
Allen Steere
and the ALDF cabal is saying something much different today about the
what the disease
actually is (brain infection) and about treatment outcomes.
http://content.nejm.org/cgi/content/full/330/4/229
Detection of Borrelia burgdorferi DNA by polymerase chain reaction
in cerebrospinal
fluid in Lyme neuroborreliosis.
"of 73 patients with Lyme arthritis who were untreated or
treated with
short courses of oral antibiotics before testing, 70 (96 percent) had
positive PCR
results. In contrast, of 19 patients who received either parenteral
antibiotics
or long courses of oral antibiotics, only 7 (37 percent) had positive
test results
after treatment (P<0.001). In the 29 patients for whom serial samples
were available,
all pretreatment samples were positive."
Self-explanatory.
About a third of the patients still had spirochetes in their knees
after antibiotic
treatment.
- - - -
The following is not a treatment outcomes assessment, but merely
demonstrates
that Allen Steere acknowledges the brain disease called "chronic
neuroborreliosis"
and that he previously had proven with the synovial fluid DNA post-
treatment analysis,
that treatment failed in a third of the cases. As I do not have the
full text of
this report, and the probes were not reported, I can't comment on
whether or
not they were the correct ones. As the correct ones by this time had
been used
by Gary Wormser and Robert Schoen when assessing tick bite treatment
outcomes and
whether or not the spirochete missing the OspA-B plasmid belonged to
this new New
England deer tick relapsing fever group, respectively, we can guess
that Allen Steere
maybe knew which primer probes to use: 16S and 23S RNA, but better
from more from
the genera:
http://www.ncbi.nlm.nih.gov/pubmed/8769624
Detection of Borrelia burgdorferi DNA by polymerase chain
reaction in cerebrospinal
fluid in Lyme neuroborreliosis.
: J Infect Dis. 1996 Sep;174(3):623-7.
Nocton JJ, Bloom BJ, Rutledge BJ, Persing DH, Logigian EL, Schmid
CH, Steere
AC.
Division of Rheumatology/Immunology, New England Medical Center,
Boston, Massachusetts02111,
USA.
A polymerase chain reaction (PCR) assay that detects Borrelia
burgdorferi DNA
in cerebrospinal fluid (CSF) was evaluated as a diagnostic test for
acute or chronic
Lyme neuroborreliosis. In one laboratory, 102 samples were tested
blindly, and 40
samples were retested in a second laboratory. In the first laboratory,
B. burgdorferi
DNA was detected in CSF samples in 6 (38%) of 16 patients with acute
neuroborreliosis,
11 (25%) of 44 with chronic neuroborreliosis, and none of 42 samples
from patients
with other illnesses. There was a significant correlation between PCR
results and
the duration of previous intravenous antibiotic therapy. The overall
frequency of
positive results was similar in the second laboratory, but concordance
between the
laboratories and among primer-probe sets was limited because many
samples were positive
with only one primer-probe set. Thus, PCR testing can sometimes detect
B. burgdorferi
DNA in CSF in patients with acute or chronic neuroborreliosis, but
with current
methods, the sensitivity of the test is limited.
PMID: 8769624 [PubMed - indexed for MEDLINE]
Perhaps the best way to prove antibiotic efficacy is with monkeys
treated with
ceftriaxone and then subject to the Mouse (monkey) Infectivity Test.
=======================================================================
*8, 9) As reported in the previous chapter, and in the
introduction of this
one, Mark Klempner says the IV drug ceftriaxone, which is used for
meningitis (and
not knee-only diseases), does not kill all the spirochetes (click here
for full
text journal article)
"Fibroblasts protected B. burgdorferi for at least 14 days of
exposure
to ceftriaxone. Mouse keratinocytes, HEp-2 cells, and Vero cells but
not Caco-2
cells showed the same protective effect. Thus, several eukaryotic cell
types provide
the Lyme disease spirochete with a protective environment contributing
to its long-term
survival." HERE on Medline (same report as above)
Mark Klempner here describes a special nerve and brain
degrading enzyme,
Matrix-metalloproteinase 130 that he found in chronic Lyme patients.
Be sure to
read why he performed this study.
Reports 8 and 9 here are the Klempner reports. There are more
that 20 studies
either by the crooks or their associates which proves treatment fails,
or rather,
that IDSA is reporting falsely that "there is no evidence of
persisting infection
after antibiotic treatment." This is their own evidence.
=======================================================================
10) In 1999, Mark Klempner reported with Denise Huber at Tufts,
"Autoimmunity;
Is is Me or Thee?"
http://www.nature.com/nm/journal/v5/n12/abs/nm1299_1346.html;jsessionid=47122A0C58C911613E8BDE31ED7C9609
Klempner reported that OspA or LYMErix could cause brain
disease as if that
needs an explainer.
"T cells that react to OspA, OspC and p 22 epitopes also
recognize
myelin basic protein, SST-R1m and IL-1R, respectively, possibly
leading to encephalopathy
and radiculopathy..."
We're not too impressed with the T cell data to support that
hypothesis,
but we are impressed with Mark Klempner's non-reporting the possible
association
between OspA vaccination and brain damage, since in 1999, LYMErix had
come onto
the market. We're also impressed with the fact that in 2003 Mark
Klempner said
there was no such thing as cognitive impairment associated with Lyme
infection.
We claim that myelin has something to do with brains and nerves, and
that the spinal
fluid was from whence Mark discovered the very special MMP-130.
NINDS' Roland Martin did not prove T cell autoimmunity, and so he
went home
to Germany. Martin failed to prove that the Multiple Sclerosis
version of "Lyme
Disease" is caused by T cell autoimmunity, just as Allen Steere failed
to find
T cell autoimmunity was the cause of the knee kind of Lyme. If Lyme-
Knee and Lyme-Brain
are not really T cell autoimmunity diseases, then what are they ya
think?
Read more about that on this page: 080515.htm
See Mark Klempner's nice flow chart (more detailed version,
HERE):
==========================================================================
*11) Yale Pathology and the Congenital Brain Infection of Newborn
Resulting
in Death "The death of the newborn was probably due to respiratory
failure
as a consequence of perinatal brain damage."
The child and mother were seronegative, they were treated, and
there was remarkably
"no inflammation." Yale's Eugene Shapiro claimed in PBS' Life
on Earth Series that the only way you can have a disease is if you
have inflammation.
The baby died of congenital Lyme brain damage, anyway. This is
not the only
such report, and they report on several babies who died from
congenital Lyme.
MORE HERE
==========================================================================
12) When trying to strike fear into the hearts of the New England
Munchausers,
hypochondriacs, trophy-wives who co-suffer Viagra-deficit syndrome,
Fibrofemzalgiacs,
"what I call Lyme Paranoia, " antibiomaniacs, members of the "Lyme
internet cult," and other delusional newsgroup posters, Yale's Robert
Schoen
said about the LYMErix vaccine:
http://www.annals.org/cgi/reprint/132/8/661.pdf
In late-stage disease [a disease now called Munchausen's by proxy,
etc],
the central nervous system [which we know to be a complicating
variable and should
be thrown out] may be involved. A new diagnostic test [not ever to be
used, since
it is a scientifically valid marker of real illness and we Lyme crooks
never go
there, preferring the services of the professional, perpetual pee-pee
whacking Voo-Dooists
of medicine, psychiatry] measuring glial fibrillary acidic protein in
the cerebrospinal
fluid may prove to be a useful tool in measuring such involvement.
"Oh," we say.
"My, my. A new disease for us bored housewives to wear. GFAp-
itis. We
can talk about it at cocktail parties..."
==========================================================================
*13) Having had the nearly dead dumped on him repeatedly, the very
famous and
brilliant (truly) Kenneth B. Liegner of Armonk, New York, and having
had the experience
and training to go with the bodies, decided to send samples of
multiply-treated
human bodies to the crooks, to have THEM determine whether or not Lyme
infection
persists past treatment and VIOLA!
CDC participates in (Liegner) autopsy and identifies persisting
DNA in samples
of patients treated many times for Lyme, which therefore proves
antibiotic treatment
does not completely eradicate the Lyme infection.
SUNY
Tulane
CDC
The Mayo Clinic
All reported "POSITIVE FOR BORRELIA" either by staining or by DNA
methods.
And then the New York Medical Board tortured Ken what else is new.
Kaiser supplies the New York Medical Board's "experts" from New
York Medical College. It's sorta like the George Bush, Karl Rove, US
Attorneygate,
Alberto Gonzales and John Yoo scenario. Stack the deck, then commit
the crime.
==========================================================================
14) Again, in a repeat of Robert Schoen's scare-mongering of the
already
mortally scared of having nothing to talk about at cocktail parties,
East Lyme,
Connecticut's Vijay Sikand said at the 1998 FDA LYMErix Vaccine
Meeting
"...the specter of asymptomatic infection is something that
troubles
me a great deal and troubles a great number of my colleagues who need
to treat Lyme
disease. The obvious analogy with syphilis infection with Treponema
pallidum is
there to consider. It is well known that Borrelia burgdorferi indeed
after asymptomatic
infection can lurk or secrete itself in certain areas of the body,
perhaps the central
nervous system or perhaps the joint spaces, only to reappear months or
maybe years
later in the form of late stages of illness which are harder to
diagnosis and treat."
---Vijay Sikand
"OOoohh!
"Scare me!
"Do that again!!!"
<squeal of delight>
["Harder to diagnose and treat?" I thought Lyme was "easily
diagnosed and cured?" Sikand sounds remarkably like myself when I say
"TROJAN
HORSE." Or maybe it's the other way around.]
==========================================================================
15) The Liegner Autopsies Found to be culture and DNA positive
for B. burgdorferi
after extensive courses of treatments:
by Mayo Clinic, SUNY-SB, CDC, Fort Collins
==========================================================================
16) The combined National Institutes say: --
http://intramural.nimh.nih.gov/inip/call4proposals.htm
"8. Infectious diseases of the CNS mediated through immune
mechanisms,
including acute and chronic Lyme disease and neuroAIDS;"
Heavens! Who are we to argue with all of the combined National
Institutes?
==========================================================================
17) Early Brain Invasion by Lyme crook, Jorge Benach.
Benach later sent a letter to the editor of the New York Times
stating that
Allen Steere was right, that we Lyme victims are delusional, and that
"Allen
Steere has the science on his side," when you can clearly see that we
have
Jorge Benach's science as well as Allen Steere's on our side.
==========================================================================
18) Early Brain Invasion by Ray Dattwyler-
Dattwyler is now the author of the new IDSA "guidelines," where
Lyme
is a non-disease.
But this is what as told to the FDA in 1994.
Hence, Lyme is not a knee disease, and the Klempner report is
bogus, so there
is no data to support the new IDSA guidelines on Lyme as an imaginary,
self-limited
disease that is cured by the placebo effect of antibiotics as Mark
Klempner asserted.
This has other indictment value as regards Steere and what he did
in Europe,
since clearly we want to diagnose the earliest case of Lyme to
possibly prevent
brain invasion. (I know. Steven Malawista and the Yale Psychiatry
Department would
not agree with me.)
==========================================================================
19) Pachner_Brains_1990 Antigenic variation in the brain.
This means you can't use the Dressler-Steere antibody method to
determine
late Lyme in the brain and neither can you say what Mark Klempner now
says. This
is just one more well-established report on Lyme as a brain disease by
a well-acknowledged
authority. This report adds nothing as regards permanence of the
infection, since
it is not a treatment study, but it's simply the pursuit of the
obvious to anyone
who cares about reality.
==========================================================================
*20) IDSA REVIEWS 1989 IDSA reviews demonstrate that the REAL
expert, Russell
Johnson claims that some spirochete cultures could take months to re-
grow intact
spirochetes from the spheroplast form (serum-starvation or survival in
harsh conditions
form). This negates the Klempner report.
"Treatment fails in more than half the cases"- Dattwyler, Luft,
Sigal
and Steere.
RUSSELL JOHNSON, CULTURING "spheroplasts could take months to
revert to
intact spirochetes in BSK media"
NEW "GREAT IMITATOR," PACHNER (We have enough data to work this
up now. We had it over a decade ago. The creeps kept the lid on the
truth and
now American scientists are known as "stupid and incomprehensible" in
Europe. The CT Attorney General had to sue the bastards, they refused
to turn over
their own self-incriminating data to him, and the settlement looks
very much like
they want to avoid criminal charges. They threw a fit when we laughed
at their
response to the settlement with Richard Blumenthal. We laughed some
more. Now
they say nothing, except for Edward McSweegan who has become more
virulent against
myself, in particular, which is in itself a clue as to what he doesn't
want
the world to see. We say "Grow the Eff up and accept responsibility
for what
you have done, since there's no better example of a coward than he who
blames
the victim and the world is obviously watching...")
DURAY, CLINICAL PATHOLOGICAL CORRELATES
ABNORMALITIES OF THE NERVOUS SYSTEM, HALPERIN
DATTWYLER and LUFT, IMMUNOLOGICAL ASPECTS
TMT of SYPHILIS, CEFTRIAXONE RECOMMENDED, ENDPOINT UNKNOWN
"Recent evaluations
of ceftriaxone for early syphilis therapy are promising; however, the
optimal dose
and duration of therapy are unknown."
CDC's GEORGE SCHMID on LYME and SYPHILIS' SIMILARITIES
Thirty days of IV ceftriaxone was in the first place, an arbitrary
decision
by the crooks, themselves, and that was because they thought it was a
brain disease,
and the same for syphilis.
Then, again, came Mark Klempner.
==========================================================================
21) John Dunn at Brookhaven et al, say that Lyme is "the perfect
stealth
pathogen" that can "mistaken for MS, Lupus, can cause excruciating
headaches."
Oh.
It's always nice to have the Department of Defense and the
Department of
Energy on your side :)))
==========================================================================
*22) Alan Barbour on what happens if you wait long than 7 days to
treat Lyme
very aggressively
Remember, now, there is the issue with mice not being the best
human brain example
[fill in the blank self-ass-biting comment]:
http://www.ncbi.nlm.nih.gov/ 8913478
Antimicrob Agents Chemother. 1996 Nov;40(11):2632-6
In vivo activities of ceftriaxone and vancomycin against Borrelia
spp. in the
mouse brain and other sites.
Kazragis RJ, Dever LL, Jorgensen JH, Barbour AG.
Department of Medicine (Infectious Diseases), University of Texas
Health Science
Center at San Antonio 78284, USA.
Borrelia burgdorferi, the agent of Lyme disease, and B. turicatae,
a neurotropic
agent of relapsing fever, are susceptible to vancomycin in vitro, with
an MIC of
0.5 microgram/ml. To determine the activity of vancomycin in vivo,
particularly
in the brain, we infected adult immunocompetent BALB/c and
immunodeficient CB-17
scid mice with B. burgdorferi or B. turicatae. The mice were then
treated with vancomycin,
ceftriaxone as a positive control, or normal saline as a negative
control. The effectiveness
of treatment was assessed by cultures of blood and brain and other
tissues. Ceftriaxone
at a dose of 25 mg/kg of body weight administered every 12 h for 7 to
10 days eliminated
cultivable B. burgdorferi or B. turicatae from all BALB/c or scid mice
in the study.
Vancomycin at 30 mg/kg administered every 12 h was effective in
eliminating infection
from immunodeficient mice if treatment was started within 3 days of
the onset of
infection. If treatment with vancomycin was delayed for 7 days or
more, vancomycin
failed to eradicate infection with B. burgdorferi or B. turicatae from
immunodeficient
mice. The failure of vancomycin in eradicating established infections
in immunodeficient
mice was associated with the persistence of viable spirochetes in the
brain during
antibiotic treatment. PMID: 8913478 [PubMed - indexed for MEDLINE]
See the Chronology of the Lyme crimes for the older references
Identify the disease early and treat it aggressively, but always
remember the
Trojan Horse, so avoid psychiatry, because they, in their hysteria and
hissy-fitting
response to this website and the proofs that Lyme is a permanent brain
infection,
dared to say, "There will be no more spirochete-like discoveries."
To which we reply, "We think it's a free-for-all, now, and anyone
can
make up any diseases they want, as long as you psychiatrists claim to
have the magical
ability to diagnose us with anything without the use of any
scientifically valid
tests."
I say, "RETROCHONDRIA!!" in your general direction.
I say, "The AMA has FLACCITUDE!"
"Your ideas are Steereborn!!"
We say, "Meet me at the Yale Center for the Study of Erections,
and we
can have a discussion about all your uncomplicated variables ."
==========================================================================
23) An independent study on spirochetes in the brain from
dentists and they
say:
Molecular and immunological evidence of oral Treponema in the
human brain
and their association with Alzheimer's disease.
http://www.ncbi.nlm.nih.gov...11929559 medline link to verify
Riviere GR, Riviere KH, Smith KS.
Department of Pediatric Dentistry, School of Dentistry, Oregon
Health and
Sciences University, Portland, OR 97201-3097, USA.
The purpose of this investigation was to use molecular and
immunological
techniques to determine whether oral Treponema infected the human
brain. Pieces
of frontal lobe cortex from 34 subjects were analyzed with species-
specific PCR
and monoclonal antibodies. PCR detected Treponema in 14/16 Alzheimer's
disease
(AD) and 4/18 non-AD donors (P < 0.001), and AD specimens had more
Treponema
species than controls (P < 0.001). PCR also detected Treponema in
trigeminal
ganglia from three AD and two control donors. Cortex from 15/16 AD
subjects and
6/18 controls contained Treponema pectinovorum and/or Treponema
socranskii species-specific
antigens (P < 0.01). T. pectinovorum and/or T. socranskii antigens
were also
found in trigeminal ganglia and pons from four embalmed cadavers, and
2/4 cadavers
also had Treponema in the hippocampus. These findings suggest that
oral Treponema
may infect the brain via branches of the trigeminal nerve.
PMID: 11929559 [PubMed - indexed for MEDLINE]
"PCR detected Treponema in 14/16 Alzheimer's disease (AD) and
4/18
non-AD donors."
"Cortex from 15/16 AD subjects and 6/18 controls contained
Treponema pectinovorum
and/or Treponema socranskii species-specific antigens."
We wondered why this information never makes breaking headline
news as regards
new developments in Alzheimer's but then we remember the American
Psychiatric
Association has become like the Pope in his treatment of Galileo.
Such is heresy
against the dogma that "no more spirochete-like discoveries will be
made."
Despite the history and syphilis and the insane asylums and
Ehrlich's Compound
606, the arsenic bullet and so on and so forth...
==========================
24) Mousehausen's is now official CDC admits ceftriaxone fails to
eradicate
all spirochetes.
http://aac.asm.org/cgi/content/abstract/AAC.01050-07v1
Persistence of Borrelia burgdorferi Following Antibiotic
Treatment in Mice
Emir Hodzic, Sunlian Feng, Kevin Holden, Kimberly J. Freet,
and Stephen
W. Barthold*
Center for Comparative Medicine, Schools of Medicine and
Veterinary Medicine,
University of California at Davis, One Shields Avenue, Davis, CA 95616
The effectiveness of antibiotic treatment was examined in a
mouse model
of Lyme borreliosis. Mice were treated with ceftriaxone or saline for
one month,
commencing during the early (3 weeks) or chronic (4 months) stages of
infection
with Borrelia burgdorferi. Tissues from mice were tested for infection
by culture,
polymerase chain reaction (PCR), xenodiagnosis, and transplantation of
allografts
at 1 and 3 months after completion of treatment. In addition, tissues
were examined
for spirochetes by immunohistochemistry. In contrast to saline-treated
mice, mice
treated with antibiotic were consistently culture-negative, but
tissues from some
of the mice remained PCR-positive, and spirochetes could be visualized
in collagen-rich
tissues. Furthermore, when some of the antibiotic treated mice were
fed upon by
Ixodes scapularis ticks (xenodiagnosis), spirochetes were acquired by
the ticks,
based upon PCR, and ticks from those cohorts transmitted spirochetes
to naïve SCID
mice, which became PCR-positive, but culture-negative. Results
indicated that following
antibiotic treatment, mice remained infected with non-dividing but
infectious spirochetes,
particularly when antibiotic treatment was commenced during the
chronic stage of
infection.
Ya gonna argue with the CDC, now?
spirochetes were acquired by the ticks
Culture negative means nothing, as we know. It could take
months in vitro.
They should have completed the Mouse Infectivity Test.
25)
http://www.ncbi.nlm.nih.gov/pubmed/17045505?ordinalpos=11&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_RVDocSum
-
CDC on intracellular nerve and brain borrelia spirochetes
We all wonder how the hell the CDC can talk about this phenomenon
and not come
right and say "Lyme is a relapsing fever organism and is incurable due
to their
intracellularity and the formation of dormant cyst or spheroplast
forms, which are
also inhalable," - since this is all either their own data, or the
NIH's
data, or the US Army's data.
================================
26) Willy Burgdorfer discussing the cyst or the spheroplast
form. Combine
that with the reality that CDC officers discuss intracellular
spirochetes (Mark
Klempner and the one above). While Willy Burgdorfer is discussing
European articles,
however - disclaimed as invalid by the CDC except when Allen Steere
does it (Chapter
3) - recall that Willy Burgdorfer was himself recruited by the NIH
Rocky Mountains
Bioweapons Lab (which had a moat dug around it hmmm strangely like a
mini island,
like a mini Plum Island) from Switzerland, and the NIH also recruited
the German
scientist Roland Martin to head up the NINDS-Multiple Sclerosis group.
So, everyone can discount CDC's discount of foreign research since
obviously
no matter what they say, it's a lie.
==================================================
As we Lyme warriors cannot engage any earthly assistance;
As the entire US medical community chooses not to assist sick and
abused people
and put a stop to this;
As there is not one MD or group in the entire USA who will take
these criminals
to court;
As there is not one lawyer or Department of Justice official who
will do the
job they were hired to do and protect us from corporate crime;
As there are no men left among us:
http://www.ourladyswarriors.org/prayer/michael.htm
Saint Michael the Archangel,
defend us in battle.
Be our protection against the wickedness and snares of the devil.
May God rebuke him, we humbly pray;
and do Thou, O Prince of the Heavenly Host -
by the Divine Power of God -
cast into hell, satan and all the evil spirits,
who roam throughout the world seeking the ruin of souls.
Amen.
kshe...@calea.org, fit...@gmail.com, patrick.f...@usdoj.gov,
model...@sbcglobal.net, jdr...@nejm.org, let...@courant.com,
Jgerb...@cdc.gov, len...@courant.com, michae...@po.state.ct.us,
con...@po.state.ct.us, executiv...@nytimes.com, managing-
edi...@nytimes.com, news...@nytimes.com, the-...@nytimes.com,
biz...@nytimes.com, for...@nytimes.com, me...@nytimes.com,
nati...@nytimes.com, dv...@cdc.gov, brigidc...@optonline.net,
tr...@hotmail.com, ubi...@courant.com, ma...@concentric.net,
camp...@courant.com, jhorn...@fff.org, thomas...@usdoj.gov,
thoma...@po.state.ct.us, kur...@washpost.com,
georg...@washpost.com, hor...@courant.com,
commissi...@po.state.ct.us, cohen...@aol.com,
FalN...@aol.com, brans...@comcast.net, vts...@comcast.net,
o...@po.state.ct.us, da...@davila-dilzer.com,
scott....@po.state.ct.us, govern...@po.state.ct.us,
attorney...@po.state.ct.us, randall...@usdoj.gov
Cc: fra...@ucia.gov, dr-ahma...@president.ir,
eugener...@washpost.com, hor...@courant.com,
bmi...@newstimes.com, tr...@hotmail.com, rast...@aol.com,
billc...@gmail.com, thomas...@usdoj.gov, amcg...@rms-law.com,
rjmu...@aol.com, paulcrai...@yahoo.com,
sidney_b...@yahoo.com, criminal...@usdoj.gov,
karla.d...@usdoj.gov, christophe...@usdoj.gov
Subject: Wormser sounds very bioweaponeery
Date: Jul 5, 2008 11:22 AM
I never really looked too closely at the IDSA 2006 regurgitation of
their 2000 batch
of excrement, having known what went on in 2000, and knowing all along
they were
doing a 180 on their own previously published data.
However, before too many days go by (ActionLyme activity is pretty hot
of its own,
via referral), all Lyme victims and their worthy attendants should
keep a copy of
this (the webpage I link below) to their harddrives, and better yet,
also email
it to your foreign terrorist friends in other nations (I'm a
"terrorist,"
according to Yale and the State of Corrupticut for having exposed
their crimes-
that being the object of the Bushie/Mossad illegal wiretapping).
The best outcome is Russia infiltrates Canadian and Mexican business,
such that
when NAFTA happens, they can take American Fraud Corporations (who are
at present
being protected by the USDOJ KarlRovesque US Attorneygate Soap Opera
Digest US Attorneys
and the distracted FBI porn-watchers) for all they've got.
Kathleen M. Dickson
http://www.actionlyme.org/BRAIN_PERMANENT.htm
CHAPTER 7, Lyme Crooks Report Treatment Failure about 20 times
(This chapter is still working, however the reference below
are obviously
available to the interested- 080705, 10:00 AM EST KMDickson)
This is data (20+ reports of persistence past treatment and in
the brain,
by the crooks, themselves), the Lyme crooks (Yale/NYMC/ALDF/IDSA/
Kaiser cabal) refused
to turn over to the Connecticut Attorney General for a year and a half
before settling
out of court.
SAY THE IDSA GUIDELINES (GARY WORMSER, et al, or the Original
ALDF.com "entrepreneurial
trio" gang of:
"The entrepreneurial trio are Durland Fish, Ph.D.,
former director
of the College's Lyme Disease Center and now a research scientist at
Yale; Gary
P. Wormser, M.D., still professor of medicine and pharmacology and
chief of the
Division of Infectious Diseases at the College; and John J. Connolly,
Ed.D., former
College president and current chairman of the board of the American
Lyme Disease
Foundation, Inc., which had its genesis on the Valhalla campus in
1990."
http://www.journals.uchicago.edu/doi/full/10.1086/508667 ◄ "The
Guidelines"
"The notion that symptomatic, chronic B. burgdorferi infection
can
exist despite recommended
treatment courses of antibiotics (tables 2 and 3) in the
absence of objective
clinical signs of
disease, is highly implausible as evidenced by (1) the lack of
antibiotic
resistance in this genus
[39, 40, 310], (2) the lack of correlation of persistent
symptoms with laboratory
evidence of
inflammation or with the eventual development of objective
physical signs
[223, 257, 288, 289],
and (3) the lack of precedent for such a phenomenon in other
spirochetal
infections [315-317].
[Lyme is a "STEALTH DISABLER" and as shown in the Yale
Congenital
Lyme Autopsy report, there was "NO INFLAMMATION." SEE THE BIOMARKERS
and the MARK KLEMPNER CHAPTER; NOT CAPTURING THEIR OWN IDENTIFIED
BIOMARKERS of
disease and instead, deploying invalid mumbo-jumbos is a research
fraud crime.]
Additional compelling evidence against the hypothesis that
persistent symptoms
are the result of
persistent infection is the fact that the concentrations of
antibodies against
B. burgdorferi in
many of these patients diminish to undetectable levels [257,
286, 288, 318].
The panel is unaware
of any chronic infection in which antibody titers diminish
despite persistence
of the causative
organism. [VERY DAMNING statement. This phenomenon is related
to the "stealth
disabler" mechanisms of fungal or mycoplasmal or mycobacterial
antigens- no
antibodies are made against the infection due to TLR2 tolerization and
the downregulation
of HLA or antigen-presenting molecules Wormser is aware of the
immune suppression
aspects of OspA vaccination, since he reported about it:
Gary Wormser reporting the blunting of the immune
response in
vaccinated animals:
http://www.ncbi.nlm.nih.gov/pubmed/10865170?ordinalpos=5&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_RVDocSum
"OspA interferes with the response of lymphocytes
to proliferative
stimuli including a blocking of cell cycle phase progression."
'In syphilis, patients who are regarded as having treatment
failure
typically have persistent or rising titers of antibodies [319]. [THE
IMMUNE SUPPRESSING
PlumIsland PAM3CYS LIPOPEPTIDE OSPA IS NOT FOUND IN TREPONEMA
PALLIDUM]
"Finally, Lyme disease lacks characteristics of other
infections that
justify longer treatment courses, such as infections in
immunodeficient hosts, [YOU
ARE REALLY GONNA BE SHOCKED when you see the Plum Island Mycoplasmal-
immune suppressing
data, the activation of latent viruses data (auto-kill turned off) the
HIV Pam3Cys
immune depression data, and the Fungal Vaccines data] infections in
which a pathogen
is inhibited but not killed by antimicrobial therapy or in which
available antimicrobials
are minimally active in vitro [JOHNSON CULTURING FROM SPHEROPLAST
COULD TAKE MONTHS],
infections caused by an intracellular pathogen [KLEMPNER- "cef
fails"],
infections involving a biofilm, infections on a heart valve, or
infections involving
a clinical site in which there is ischemia, a foreign body, a
sequestrum***, or
frank pus [170]. The “cystic” forms of B. burgdorferi that have been
seen under
certain growth conditions in vitro have not been shown to have any
clinical significance
[320]. [INTRACELLULAR CYSTS are the KEY TO LATENCY, well-known
historically, and
this is an application of the NEGATIVE DATA RULE, which is another
research fraud
crime indictment point; THE MOUSE INFECTIVITY TEST HAS NOT BEEN
PERFORMED ON TREATED
MICE OR MONKEYS]
CDC and NIH Rocky Mountain antibody-gold sphere method
as applied
to borreliosis:
The Method Developed:
http://www.pubmedcentral.nih.gov/picrender.fcgi?artid=261539&blobtype=pdf
*** Applied to Borreliosis by NIH Rocky Mountain Lab:
http://www.pubmedcentral.nih.gov/picrender.fcgi?artid=269963&blobtype=pdf
The Dorward/Garon Gold Test Apparently detects plenty
of shed antigen.
And it looks pretty sequestery to me, not to mention we could never
determine sequestery
sites in a human unless we had a lot of brains to biopsy, although the
dentists
did a pretty good job detecting oral treponemes sequestering in the
brains of Alzheimer's
patients- See Item 23, below.
1975 at an international spirochetal diseases conference
for which Willy
Burgdorfer was present; "The ability of the borrelia, especially tick-
borne
strains to persist in the brain and in the eye after treatment with
arsenic or with
penicillin or even after apparent cure is well known (1). The
persistence of treponemes
after treatment of syphilis is a major area which currently requires
additional
study (3,5,10,11)." -Jay Sanford, US Military Hospital, Bethesda,
MD
As you can see, Gary Wormser, ad IDSA et al, are trying to
deny everything
that Lyme actually is. Lyme is stealth (no antibodies late in the
disease), latent,
intracellular, is resistant to antibiotics (goes into a self-
protective spheroplast
form upon exposure to antibiotics which is not an "end stage" as the
crooks claim), it goes into a "dormant" or serum-starved or
spheroplast
mode (known to the crooks themselves as a "Trojan Horse"), Lyme
belongs
to the class of diseases where there is typically no inflammation. It
does not
belong to NIAID or Anthony Fauci's division, but the parasitic
diseases division
- but it's not going to belong to either the CDC or the NIH at all
after not
too long, because when they're done being investigated we will find
that that
NIH and the CDC are the same as the NEJM "peer-reviewers" - not a one
of them isn't interest conflicted. In fact, we will find out what is
the exact
relationship between the CDC and Kaiser or else the NAFTA-eers of
Canada and Mexico
will discover it and prosecute it.
At this time I recommend that Russia and China infiltrate
Canadian and
Mexican business structures such that when the NAFTA NationRape of the
North American
Continent takes place, Russia can return the favor for the Israeli
Oligarchic NationRape
Escapade under Yeltsin.
More of these clear rebuttals below and in the
RICOCHRON.htm page.
Remember, the IDSA keywords: "INFLAMMATION" and "OBJECTIVE
SIGNS" (meaning they are still referring to "Lyme Disease" as the
Dearborn definition of "autoimmune bad knee" as seen in the RICO
graphics),
because that later comes in in Congenital Lyme ("lack of
inflammation")
and the Biomarkers chapter as regards the description of the immune
suppression
outcomes of Lyme, which is the key to all of their crimes, especially
as regards
this fungal vaccine, LYMErix or ImmuLyme, or rOspA and the activation
of latent
viruses of all kinds.
The crooks' own treatment failure or persistence articles
are below.
The IDSA statement about the cysts or the spheroplasts is
yet another
criminal matter, since they all referenced Dave Nelson's "reversion
to
intact spirochete form from spheroplast within one minute of the
addition of rabbit
blood" report, Russell Johnson said it could take months in culture
for the
cyst form to regenerate in media, and the Mouse Infectivity Test has
never been
tried as a treatment efficacy trial, ever, to my knowledge in lab
animals. They
cannot make the claim that the cyst form hasn't any clinical
significance, since
here we have again the Crooks' Negative Data Rule.
At the present time, there are a whole slew of Russian
scientists now
studying these intracellular serum-starvation ("Stringent Response")
forms
at New York Medical College.
http://iai.asm.org/cgi/reprint/70/6/3061
The difficulty in testing for congenital syphilis- lest we
forget, syphilis
testing was always terrible, despite Wormser's attempt to re-write
history
http://www.pubmedcentral.nih.gov/picrender.fcgi?artid=262629&blobtype=pdf
It's pretty hard to find a cohort nowadays of non-HIV
infected syphilis
patients.
COMPREHENSIVE ARTICLE ON SYPHILIS:
http://www.pubmedcentral.nih.gov/articlerender.fcgi?tool=pubmed&pubmedid=16418521
25 PERSISTENCE ARTICLES (brain, intracellular, spheroplast) by the
CROOKS and/or
THEIR ASSOCIATES
BSK is not the only media these spirochetes grow in. The gold
standard
of persistence is still the Mouse Infectivity Test (MIT), although it
should probably
be the Hamster or some other rodent Infectivity Test, which is the
transfer of SPINAL
FLUID (not synovial fluid, since who cares about knees) from one
infected rodent
to an uninfected one and allow the infection to disseminate elsewhere,
and look
for whole spirochetes by staining, etc or re-ear puncture transfer,
etc., to determine
treatments' efficacies..
CDC's Alan Barbour published in 1986 that rodent brains was
the actual
former storage media for spirochetes.
Yes.
http://mmbr.asm.org/cgi/reprint/50/4/381?view=long&pmid=3540570
"Borrelia spirochetes so reliably infected rodent brains that
we used
to use rodent brains as the main storage media."
For a clarification, hamster/guinea pig/rat/whatever brains
are better than
mouse brains, which is why the crooks use only mouse brains for their
treatment
studies, since they don't want the truth about Lyme revealed,
apparently, ever:
http://www.pubmedcentral.nih.gov/picrender.fcgi?artid=441260&blobtype=pdf
"Borrelia do not survive long in mouse brains."
1944, relapsing fever during WWII, Lancet, Sept 30, 1944, page
436
NEUROLOGICAL COMPLICATIONS OF relapsing fever
Ronald Bodley Scott, DM, OXFD, FRCP
LIEUT.-COLONEL RAMC; OFFICER I/C A MEDICAL DIVISION
THE campaigns in the Lybyan Desert yielded cases of
relapsing fever
of a type not previously known in Egypt. They were due to a infection
with Treponema
recurrentis, whose immunological characteristics have not yet been
determined, transmitted
by the bite of an unnamed argasid tick of the genus ornithodoros,
bearing a close
resemblence to O. erraticus, the vector of the Tunisand strain of
treponema (Adler,
1942). This tick probably inhabited the burrows of desert rodents and
infection
was commonly acquired in caves, slit trenches and tombs.
Characteristic of this fever was the high proportion of
cases in which
the central nervous system was invaded. Most descriptions of the
disease include
the comment that nervous system sequelae occur and that they are more
common in
the tick-botne than in the louse-borne fever; but theirt frequency and
variety are
not generally recognized. This paper is concerned with a small series
of cases
seen in 1941 and 1942; its observations consequently apply to the
disease of the
north-west Africa coast. It is likely, however, that this reservation
is not absolute"
the characteristics of Tr. recurrentus are so labile that strains from
the same
locality, and even from different relapses in the same case, may show
immunological
variations (Ashbel 1943). This the clinician is probably justified in
discounting
the importance of differences of strain and in regarding the tick-
borne relapsing
fever as an entity, however heretical the immunologist may consider
this view.
NEUROTROPIC CHARACTER OF Tr. recurrentis
In the animal the neurotropism of this treponema is well-
established.
Ashbel (1943), investigating 17 strains of Tr. persica, found that
organisms could
be isolated from the brains of guinea pigs 117-398 days after apparent
recovery
from the infection. In some cases this cerebral invasion proved fatal
and post-mortem
examination showed small perivascular hemorrhages and infiltrations
with lymphocytes,
monocytes and macrophages. The neurotropism of various strains has
been shown to
be equally great in other animals (Heronimus 1928).
The predilection of the treponema for nervous tissue in
the animals
raises the question of whether it is similarly neurotropic in man.
Data are not plentiful; but as long ago as 1874 Ponfick
reported petechial
hemorrhages [LIEGNER] in the brains of cases dying in the Berlin
epidemic. Belezky
and Umanskaja (1930) have recorded the findings in 8 fatal cases:
clinical observations
are scanty in their paper but only one had symptoms of disease of the
nervous system.
In all instances microscopy showed a patchy perivascular infiltration
of the pia
with monocytes, lymphocytes and plasma cells, and in places the
cerebral vessels
were encircled by a similar cellular halo. In 3 cases treponemata
were found in
the brain substance, diffusely distributed and in no constant relation
to vessels.
More recently, Ungar has described the case of a woman
dying in the
puerperium with relapsing fever and cerebral symptoms. The post-
mortem findings
included a cholesteatoma of the lateral recess edema of the cisternal
pia-arachnoid
and hemorrhages in the caudal part of the pons and the floor of the
4th ventricle.
Sections showed the Virchow-Robin spaces distended with erythrocytes,
lymphocytes
and monocytes; treponemata were recovered from the cerebrospinal fluid
and from
the tumor.
Evidence of a less convincing nature is provided by
Spielmeyer and Jahnel
(1926) from examination of the brains of paretics in whom therapeutics
infection
with Tr. duttoni had proved lethal. These authors describe a
perivascular infiltration
which differed from that seen in general paresis by virtue of the
scanty plasma
cells and the numerous macrophages.
These observations show that Tr. recurrentis has
neurotropic characters
in man as well as in the animal, and they furnish a morbid anatomical
foundation
for the varied neurological guises the disease may assume...
Treatment.-Details of treatment have been omitted from the
case reports;
but in each instance an organic arsenical was given. There was no
evidence that
these druigs reduced the number of relapses or the ability to invasion
of the nervous
system. Adler, Theodor and Schieber (1936) found that untreated cases
had fewer
relapses than those that received intravenous arsphenamine. Cooper
(1942), while
admitting that the dosage was insufficient, noted a relapse in 41 of
57 cases after
arsenic had been gievn; in 17 Tr. recurrentis was again present in
blood films.
Arsphenamine often appears to cut short a bout of fever and it is
perhaps possible
that more energetic treatment was required. More recently
tryparsamide and bismuith
have been found effective (McAlpine 1943).
Prognosis.- It is not easy to assess prognosis in a
disease of long
duration under the conditions necessarily operative in military
practice; so many
patients are transferred to other hospitals before full recovery.
None of this
series died; indeed it is rarre for tick-borne relapsing fever to be
fatal. The
majority appeared to recover completely but case 9 illustrates the
frequency of
repeated relapses and the chronic ill heatlh which may result from
this disorder.
http://www.actionlyme.org/1944,%20Relapsing%20Fever%20During%20WWII.htm
So, it is a little hard to believe that we don't have the same
disease
they had during World War II and so on and so forth and arsenic as a
treatment for
spirochetes since the time of Paul Ehrlich and so on and that all of a
sudden spirochetes
behave better now and will only attack knees for a month and then roll
over and
die. Were we Lyme victims not really sick, we would have no reason to
search for
the evidence that we knew would exist to prove we are. It's that
simple.
We absolutely knew the data would exist that proves we are as
sick as we
say we are. Think about it. We absolutely knew we could prove these
bastards at
Yale were lying,
Get the whole picture in your mind:
Borreliae are permanent, intracellular brain and nerve
infections
which go dormant in a spheroplast form, and these facts are all
acknowledged and/or
researched and reported, and/or described, and/or referenced by the
Lyme criminals
themselves.
The shed Osps suppress the immune response due to
tolerization,
and..
...the shed Osps suppress the immune system by
anchoring the auto-kill
kinases, or, the immune suppression caused by these Osps results in
the activation
of latent viruses of all kinds.
Lyme Borreliae are usually delivered by a microscopic
tick (US Army:
"ticks could be used to spread diseases"), resulting in chronic,
ongoing
neurologic damage,
But the testing for Lyme only allows for a person to
be diagnosed
with the KNEE-Disease or LYMErix Disease, or the hypersensitivity
reaction to OspA,
which was Steere's new, Dearborn Definition...
Lyme Borreliosis is a triple stealth disabler:
1) The worst cases are not detectable
(deliberately)
2) It's permanent and incurable, so in currently
healthy
people it can be like a Trojan Horse, emerging later as any of the
Great Imitator's
outcomes (ALS, Lupus, MS, etc)
3) Is delivered by microscopic, stealth ticks
Worse yet, the Lyme criminals literally stalk,
wiretap, harass,
order the false arrests of, sic the CPS on, report falsely to medical
boards, and
otherwise attack Lyme victims and their treaters, constantly
persecuting them, especially
the smarter and louder ones and especially as regards congenital Lyme.
as if the
medical community can't figure out that the same thing happens in
congenital
syphilis.
Well, what am I saying? The "medical community"
can't figure anything out.
All of this would have been believable as either a
natural development
"evolutionary bottleneck" or an accidental release from Plum Island,
but
for the harassment.
Why the harassment? Why is the CDC allowing for
this harassment?
Is it because they're protecting their own criminal CDC officers, Alan
Steere
and Allen Barbour, and their own patents with SmithKline in Europe, or
is this bioweapons
information that they don't want to get out?
Or is it because all these secret outcomes have
some meaning
as regards all childhood vaccines damage?
Or is it because the childhood vaccines damage is
some sort
of a deliberate campaign to get rid of the genetically weak, since it
has been tried
before by this same clique of British and American Bankers?
Since the CDC is the CIA of bioweapons and the CIA
works for
the Bigs (Rockefeller), and the Mossad works for the British Bigs
(Rothschild),
it does not matter, since we the people are not the Bigs. We don't
have any
aristocrats in this country, and even if we did, we don't see or know
any intelligent
ones, so they can't claim brains as the essence of their superiority.
==================================================================
*1)
In 1975 there was apparently an international conference on
spirochetal diseases
and a Dr. Jay Sanford of the Uniformed Services Military Hospital in
Bethesda, Maryland
reported:
"The ability of the borrelia, especially tick-borne strains to
persist
in the brain and in the eye after treatment with arsenic or with
penicillin or even
after apparent cure is well known (1). The persistence of treponemes
after treatment
of syphilis is a major area which currently requires additional study
(3,5,10,11)."
-Jay Sanford, US Military Hospital, Bethesda, MD
This textbook was edited by Russell Johnson who was an advisor to
the criminal
gang of the ALDF.com
Russell Johnson later published for the Infectious Diseases
Society of America
that: "Although spirochetes can often be detected in culture media
after 3
weeks of culture, some isolates may not be visible for several
months."
IDSA says we need to culture these suckers for several months.
Did Mark Klempner
do that in his bogus long term treatment "study?"
NO.
=================================================================
2)
http://www.pubmedcentral.nih.gov/picrender.fcgi?artid=373079&blobtype=pdf
By CDC Officer Alan Barbour, in 1986, The Biology of Borrelia Species:
"The propensity for borrelia to go to the brain of infected
mammals suggests
that the relationship between these spirochetes and neural tissues is
not trivial.
Further study of this attraction and the interaction that follows may
reveal the
basis for the significant nerve and brain involvement in Lyme
borreliosis"--
This is self-explanatory. We have previously seen that rodent
brains so reliably
were a home for spirochetes that they were actually the culture
media. We also
acknowledge that ceftriaxone for brain diseases was a treatment IDSA
themselves
used and offered and still use, so Lyme must not be a self-limiting
knee disease
as Allen Steere and Yale's Steven Malawista claim.
And here we have in the same CDC officer's 1986 report that there
are, gasp,
"gemma:"
[You want to GO HERE for other older references published
before the
Great Whore of Medicine, Allen Steere emerged onto the scene]
=================================================================
3) Russell Johnson's patent for the very first Lyme vaccine
"The chronic forms of the disease such as arthritis (joint
involvement),
acrodermatitis chronica atrophicans (skin involvement), and Bannwart's
syndrome
(neurological involvement) may last for months to years and are
associated with
the persistence of the spirochete. A case of maternal-fetal
transmission of B. burgdorferi
resulting in neonatal death has been reported. Domestic animals such
as the dog
also develop arthritis and lameness to this tick-borne infection. For
every symptomatic
infection, there is at least one asymptomatic infection. Lyme disease
is presently
the most commonly reported tick-borne disease in the United States."
--
The patent also says:
"The infection may be treated at any time with antibiotics
such as
penicillin, erythromycin, tetracycline, and ceftriaxone. Once
infection has occurred,
however, the drugs may not purge the host of the spirochete but may
only act to
control the chronic forms of the disease. Complications such as
arthritis and fatigue
may continue for several years after diagnosis and treatment."
================================================================
4) At the time of the writing of this book, CDC officer Alan
Barbour has on
his website, the following statement:
"We are taking a multi-discipline approach, including methods
of genetics,
cell biology, and immunology, to study in depth two spirochetal
diseases: Lyme disease
and relapsing fever. These tick-borne infections are notable for
multiphasic antigenic
variation through DNA recombinations in the case of relapsing fever,
the occurrence
of chronic arthritis in the case of Lyme disease, and invasion of and
persistence
in the brain in the case of both diseases. ---Alan Barbour
============================================================
*5) In 1994 Alan Steere and other published a report entitled:
http://www.annals.org/cgi/content/full/121/8/560
The Long-Term Clinical Outcomes of Lyme Disease: A Population-
based Retrospective
Cohort Study
right arrow Nancy A. Shadick; Charlotte B. Phillips; Eric L.
Logigian; Allen
C. Steere; Richard F. Kaplan; Victor P. Berardi; Paul H. Duray; Martin
G. Larson;
Elizabeth A. Wright; Katherine S. Ginsburg*; Jeffrey N. Katz; and
Matthew H. Liang
15 October 1994 | Volume 121 Issue 8 | Pages 560-567
In that report:
"Patient 12 had had high fever, meningeal symptoms, and
subsequent
arthritis in 1982. She was noted to have a positive serologic test
result for Lyme
disease 4 years later and was treated with 2 weeks of parenteral
penicillin. She
later developed a progressive speech disorder, bradykinesia, and
abnormal ocular
motor function. Magnetic resonance imaging of the brain showed
scattered white matter
lesions in the hemispheres and pons, and she was diagnosed with
supranuclear palsy.
Lumbar puncture showed no selective concentration of antibody in the
spinal fluid.
Nevertheless, she was re-treated with 2 weeks of parenteral
ceftriaxone in 1989
that had no effect on her neurologic symptoms. During the time of
observation, this
patient died. At autopsy, lymphoid mononuclear cells were observed
surrounding the
intracerebral vessels in one section. Using Dieterle silver stain, a
spirochete
was present in the cortex and another was exterior to a leptomeningeal
vessel."
One of Steere's multiply-treated patients died anyway with
spirochetes in
her brain.
How amazing that that suddenly doesn't happen any more, despite no
new breakthroughs
in antibiotic treatments or their delivery.
The Primer's Shell Game is where these Lyme crooks use the
wrong DNA
(or RNA) primers (they use the variable Osp primers instead of the non-
variable
chromosomally encoded) to test Lyme victims. These same crooks use
the correct
DNA primers when they want to find Lyme or borrelia in ticks (to
patent). It's
a shell game. They all know that all that they can use for treatment
outcomes is
non-variable DNA or RNA.
Mark Klempner never reported his primers in his Chronic Lyme
"study,"
and he would not tell me, either, in an email correspondence I had
with him. Therefore,
the Lyme crooks have never validly reported on the presence of
borrelia in humans,
with the exception of Gary Wormser's study of EMs in which he found
2/9 patients
who had EM, who had been treated with antibiotics still had
spirochetes in their
tissues. This data can be found on the HOW RICO WILL BE CHARGED PAGE
===============================================================
*6, 7) In 1994 and in 1996, Steere and Nocton published a reports
of treatment
failure and brain invasion using RNA and DNA methods and they found
treatment failed
in a third of the patients with their OspA primer sets. In the first
report, in
1994, published in the New England Journal of Medicine, that they used
4 primer
sets. Three were for the OspA gene (which we know undergoes antigenic
variation
and therefore these are the wrong probes). The fourth set was for 16S
RNA intragenic
spacer (remember now, for Borrelia burgdorferi, ignoring all the other
possibilities).
These were rather valuable reports, overall, since they prove that
Allen Steere
and the ALDF cabal is saying something much different today about the
what the disease
actually is (brain infection) and about treatment outcomes.
http://content.nejm.org/cgi/content/full/330/4/229
Detection of Borrelia burgdorferi DNA by polymerase chain reaction
in cerebrospinal
fluid in Lyme neuroborreliosis.
"of 73 patients with Lyme arthritis who were untreated or
treated with
short courses of oral antibiotics before testing, 70 (96 percent) had
positive PCR
results. In contrast, of 19 patients who received either parenteral
antibiotics
or long courses of oral antibiotics, only 7 (37 percent) had positive
test results
after treatment (P<0.001). In the 29 patients for whom serial samples
were available,
all pretreatment samples were positive."
Self-explanatory.
About a third of the patients still had spirochetes in their knees
after antibiotic
treatment.
- - - -
The following is not a treatment outcomes assessment, but merely
demonstrates
that Allen Steere acknowledges the brain disease called "chronic
neuroborreliosis"
and that he previously had proven with the synovial fluid DNA post-
treatment analysis,
that treatment failed in a third of the cases. As I do not have the
full text of
this report, and the probes were not reported, I can't comment on
whether or
not they were the correct ones. As the correct ones by this time had
been used
by Gary Wormser and Robert Schoen when assessing tick bite treatment
outcomes and
whether or not the spirochete missing the OspA-B plasmid belonged to
this new New
England deer tick relapsing fever group, respectively, we can guess
that Allen Steere
maybe knew which primer probes to use: 16S and 23S RNA, but better
from more from
the genera:
http://www.ncbi.nlm.nih.gov/pubmed/8769624
Detection of Borrelia burgdorferi DNA by polymerase chain
reaction in cerebrospinal
fluid in Lyme neuroborreliosis.
: J Infect Dis. 1996 Sep;174(3):623-7.
Nocton JJ, Bloom BJ, Rutledge BJ, Persing DH, Logigian EL, Schmid
CH, Steere
AC.
Division of Rheumatology/Immunology, New England Medical Center,
Boston, Massachusetts02111,
USA.
A polymerase chain reaction (PCR) assay that detects Borrelia
burgdorferi DNA
in cerebrospinal fluid (CSF) was evaluated as a diagnostic test for
acute or chronic
Lyme neuroborreliosis. In one laboratory, 102 samples were tested
blindly, and 40
samples were retested in a second laboratory. In the first laboratory,
B. burgdorferi
DNA was detected in CSF samples in 6 (38%) of 16 patients with acute
neuroborreliosis,
11 (25%) of 44 with chronic neuroborreliosis, and none of 42 samples
from patients
with other illnesses. There was a significant correlation between PCR
results and
the duration of previous intravenous antibiotic therapy. The overall
frequency of
positive results was similar in the second laboratory, but concordance
between the
laboratories and among primer-probe sets was limited because many
samples were positive
with only one primer-probe set. Thus, PCR testing can sometimes detect
B. burgdorferi
DNA in CSF in patients with acute or chronic neuroborreliosis, but
with current
methods, the sensitivity of the test is limited.
PMID: 8769624 [PubMed - indexed for MEDLINE]
Perhaps the best way to prove antibiotic efficacy is with monkeys
treated with
ceftriaxone and then subject to the Mouse (monkey) Infectivity Test.
=======================================================================
*8, 9) As reported in the previous chapter, and in the
introduction of this
one, Mark Klempner says the IV drug ceftriaxone, which is used for
meningitis (and
not knee-only diseases), does not kill all the spirochetes (click here
for full
text journal article)
"Fibroblasts protected B. burgdorferi for at least 14 days of
exposure
to ceftriaxone. Mouse keratinocytes, HEp-2 cells, and Vero cells but
not Caco-2
cells showed the same protective effect. Thus, several eukaryotic cell
types provide
the Lyme disease spirochete with a protective environment contributing
to its long-term
survival." HERE on Medline (same report as above)
Mark Klempner here describes a special nerve and brain
degrading enzyme,
Matrix-metalloproteinase 130 that he found in chronic Lyme patients.
Be sure to
read why he performed this study.
Reports 8 and 9 here are the Klempner reports. There are more
that 20 studies
either by the crooks or their associates which proves treatment fails,
or rather,
that IDSA is reporting falsely that "there is no evidence of
persisting infection
after antibiotic treatment." This is their own evidence.
=======================================================================
10) In 1999, Mark Klempner reported with Denise Huber at Tufts,
"Autoimmunity;
Is is Me or Thee?"
http://www.nature.com/nm/journal/v5/n12/abs/nm1299_1346.html;jsessionid=47122A0C58C911613E8BDE31ED7C9609
Klempner reported that OspA or LYMErix could cause brain
disease as if that
needs an explainer.
"T cells that react to OspA, OspC and p 22 epitopes also
recognize
myelin basic protein, SST-R1m and IL-1R, respectively, possibly
leading to encephalopathy
and radiculopathy..."
We're not too impressed with the T cell data to support that
hypothesis,
but we are impressed with Mark Klempner's non-reporting the possible
association
between OspA vaccination and brain damage, since in 1999, LYMErix had
come onto
the market. We're also impressed with the fact that in 2003 Mark
Klempner said
there was no such thing as cognitive impairment associated with Lyme
infection.
We claim that myelin has something to do with brains and nerves, and
that the spinal
fluid was from whence Mark discovered the very special MMP-130.
NINDS' Roland Martin did not prove T cell autoimmunity, and so he
went home
to Germany. Martin failed to prove that the Multiple Sclerosis
version of "Lyme
Disease" is caused by T cell autoimmunity, just as Allen Steere failed
to find
T cell autoimmunity was the cause of the knee kind of Lyme. If Lyme-
Knee and Lyme-Brain
are not really T cell autoimmunity diseases, then what are they ya
think?
Read more about that on this page: 080515.htm
See Mark Klempner's nice flow chart (more detailed version,
HERE):
==========================================================================
*11) Yale Pathology and the Congenital Brain Infection of Newborn
Resulting
in Death "The death of the newborn was probably due to respiratory
failure
as a consequence of perinatal brain damage."
The child and mother were seronegative, they were treated, and
there was remarkably
"no inflammation." Yale's Eugene Shapiro claimed in PBS' Life
on Earth Series that the only way you can have a disease is if you
have inflammation.
The baby died of congenital Lyme brain damage, anyway. This is
not the only
such report, and they report on several babies who died from
congenital Lyme.
MORE HERE
==========================================================================
12) When trying to strike fear into the hearts of the New England
Munchausers,
hypochondriacs, trophy-wives who co-suffer Viagra-deficit syndrome,
Fibrofemzalgiacs,
"what I call Lyme Paranoia, " antibiomaniacs, members of the "Lyme
internet cult," and other delusional newsgroup posters, Yale's Robert
Schoen
said about the LYMErix vaccine:
http://www.annals.org/cgi/reprint/132/8/661.pdf
In late-stage disease [a disease now called Munchausen's by proxy,
etc],
the central nervous system [which we know to be a complicating
variable and should
be thrown out] may be involved. A new diagnostic test [not ever to be
used, since
it is a scientifically valid marker of real illness and we Lyme crooks
never go
there, preferring the services of the professional, perpetual pee-pee
whacking Voo-Dooists
of medicine, psychiatry] measuring glial fibrillary acidic protein in
the cerebrospinal
fluid may prove to be a useful tool in measuring such involvement.
"Oh," we say.
"My, my. A new disease for us bored housewives to wear. GFAp-
itis. We
can talk about it at cocktail parties..."
==========================================================================
*13) Having had the nearly dead dumped on him repeatedly, the very
famous and
brilliant (truly) Kenneth B. Liegner of Armonk, New York, and having
had the experience
and training to go with the bodies, decided to send samples of
multiply-treated
human bodies to the crooks, to have THEM determine whether or not Lyme
infection
persists past treatment and VIOLA!
CDC participates in (Liegner) autopsy and identifies persisting
DNA in samples
of patients treated many times for Lyme, which therefore proves
antibiotic treatment
does not completely eradicate the Lyme infection.
SUNY
Tulane
CDC
The Mayo Clinic
All reported "POSITIVE FOR BORRELIA" either by staining or by DNA
methods.
And then the New York Medical Board tortured Ken what else is new.
Kaiser supplies the New York Medical Board's "experts" from New
York Medical College. It's sorta like the George Bush, Karl Rove, US
Attorneygate,
Alberto Gonzales and John Yoo scenario. Stack the deck, then commit
the crime.
==========================================================================
14) Again, in a repeat of Robert Schoen's scare-mongering of the
already
mortally scared of having nothing to talk about at cocktail parties,
East Lyme,
Connecticut's Vijay Sikand said at the 1998 FDA LYMErix Vaccine
Meeting
"...the specter of asymptomatic infection is something that
troubles
me a great deal and troubles a great number of my colleagues who need
to treat Lyme
disease. The obvious analogy with syphilis infection with Treponema
pallidum is
there to consider. It is well known that Borrelia burgdorferi indeed
after asymptomatic
infection can lurk or secrete itself in certain areas of the body,
perhaps the central
nervous system or perhaps the joint spaces, only to reappear months or
maybe years
later in the form of late stages of illness which are harder to
diagnosis and treat."
---Vijay Sikand
"OOoohh!
"Scare me!
"Do that again!!!"
<squeal of delight>
["Harder to diagnose and treat?" I thought Lyme was "easily
diagnosed and cured?" Sikand sounds remarkably like myself when I say
"TROJAN
HORSE." Or maybe it's the other way around.]
==========================================================================
15) The Liegner Autopsies Found to be culture and DNA positive
for B. burgdorferi
after extensive courses of treatments:
by Mayo Clinic, SUNY-SB, CDC, Fort Collins
==========================================================================
16) The combined National Institutes say: --
http://intramural.nimh.nih.gov/inip/call4proposals.htm
"8. Infectious diseases of the CNS mediated through immune
mechanisms,
including acute and chronic Lyme disease and neuroAIDS;"
Heavens! Who are we to argue with all of the combined National
Institutes?
==========================================================================
17) Early Brain Invasion by Lyme crook, Jorge Benach.
Benach later sent a letter to the editor of the New York Times
stating that
Allen Steere was right, that we Lyme victims are delusional, and that
"Allen
Steere has the science on his side," when you can clearly see that we
have
Jorge Benach's science as well as Allen Steere's on our side.
==========================================================================
18) Early Brain Invasion by Ray Dattwyler-
Dattwyler is now the author of the new IDSA "guidelines," where
Lyme
is a non-disease.
But this is what as told to the FDA in 1994.
Hence, Lyme is not a knee disease, and the Klempner report is
bogus, so there
is no data to support the new IDSA guidelines on Lyme as an imaginary,
self-limited
disease that is cured by the placebo effect of antibiotics as Mark
Klempner asserted.
This has other indictment value as regards Steere and what he did
in Europe,
since clearly we want to diagnose the earliest case of Lyme to
possibly prevent
brain invasion. (I know. Steven Malawista and the Yale Psychiatry
Department would
not agree with me.)
==========================================================================
19) Pachner_Brains_1990 Antigenic variation in the brain.
This means you can't use the Dressler-Steere antibody method to
determine
late Lyme in the brain and neither can you say what Mark Klempner now
says. This
is just one more well-established report on Lyme as a brain disease by
a well-acknowledged
authority. This report adds nothing as regards permanence of the
infection, since
it is not a treatment study, but it's simply the pursuit of the
obvious to anyone
who cares about reality.
==========================================================================
*20) IDSA REVIEWS 1989 IDSA reviews demonstrate that the REAL
expert, Russell
Johnson claims that some spirochete cultures could take months to re-
grow intact
spirochetes from the spheroplast form (serum-starvation or survival in
harsh conditions
form). This negates the Klempner report.
"Treatment fails in more than half the cases"- Dattwyler, Luft,
Sigal
and Steere.
RUSSELL JOHNSON, CULTURING "spheroplasts could take months to
revert to
intact spirochetes in BSK media"
NEW "GREAT IMITATOR," PACHNER (We have enough data to work this
up now. We had it over a decade ago. The creeps kept the lid on the
truth and
now American scientists are known as "stupid and incomprehensible" in
Europe. The CT Attorney General had to sue the bastards, they refused
to turn over
their own self-incriminating data to him, and the settlement looks
very much like
they want to avoid criminal charges. They threw a fit when we laughed
at their
response to the settlement with Richard Blumenthal. We laughed some
more. Now
they say nothing, except for Edward McSweegan who has become more
virulent against
myself, in particular, which is in itself a clue as to what he doesn't
want
the world to see. We say "Grow the Eff up and accept responsibility
for what
you have done, since there's no better example of a coward than he who
blames
the victim and the world is obviously watching...")
DURAY, CLINICAL PATHOLOGICAL CORRELATES
ABNORMALITIES OF THE NERVOUS SYSTEM, HALPERIN
DATTWYLER and LUFT, IMMUNOLOGICAL ASPECTS
TMT of SYPHILIS, CEFTRIAXONE RECOMMENDED, ENDPOINT UNKNOWN
"Recent evaluations
of ceftriaxone for early syphilis therapy are promising; however, the
optimal dose
and duration of therapy are unknown."
CDC's GEORGE SCHMID on LYME and SYPHILIS' SIMILARITIES
Thirty days of IV ceftriaxone was in the first place, an arbitrary
decision
by the crooks, themselves, and that was because they thought it was a
brain disease,
and the same for syphilis.
Then, again, came Mark Klempner.
==========================================================================
21) John Dunn at Brookhaven et al, say that Lyme is "the perfect
stealth
pathogen" that can "mistaken for MS, Lupus, can cause excruciating
headaches."
Oh.
It's always nice to have the Department of Defense and the
Department of
Energy on your side :)))
==========================================================================
*22) Alan Barbour on what happens if you wait long than 7 days to
treat Lyme
very aggressively
Remember, now, there is the issue with mice not being the best
human brain example
[fill in the blank self-ass-biting comment]:
http://www.ncbi.nlm.nih.gov/ 8913478
Antimicrob Agents Chemother. 1996 Nov;40(11):2632-6
In vivo activities of ceftriaxone and vancomycin against Borrelia
spp. in the
mouse brain and other sites.
Kazragis RJ, Dever LL, Jorgensen JH, Barbour AG.
Department of Medicine (Infectious Diseases), University of Texas
Health Science
Center at San Antonio 78284, USA.
Borrelia burgdorferi, the agent of Lyme disease, and B. turicatae,
a neurotropic
agent of relapsing fever, are susceptible to vancomycin in vitro, with
an MIC of
0.5 microgram/ml. To determine the activity of vancomycin in vivo,
particularly
in the brain, we infected adult immunocompetent BALB/c and
immunodeficient CB-17
scid mice with B. burgdorferi or B. turicatae. The mice were then
treated with vancomycin,
ceftriaxone as a positive control, or normal saline as a negative
control. The effectiveness
of treatment was assessed by cultures of blood and brain and other
tissues. Ceftriaxone
at a dose of 25 mg/kg of body weight administered every 12 h for 7 to
10 days eliminated
cultivable B. burgdorferi or B. turicatae from all BALB/c or scid mice
in the study.
Vancomycin at 30 mg/kg administered every 12 h was effective in
eliminating infection
from immunodeficient mice if treatment was started within 3 days of
the onset of
infection. If treatment with vancomycin was delayed for 7 days or
more, vancomycin
failed to eradicate infection with B. burgdorferi or B. turicatae from
immunodeficient
mice. The failure of vancomycin in eradicating established infections
in immunodeficient
mice was associated with the persistence of viable spirochetes in the
brain during
antibiotic treatment. PMID: 8913478 [PubMed - indexed for MEDLINE]
See the Chronology of the Lyme crimes for the older references
Identify the disease early and treat it aggressively, but always
remember the
Trojan Horse, so avoid psychiatry, because they, in their hysteria and
hissy-fitting
response to this website and the proofs that Lyme is a permanent brain
infection,
dared to say, "There will be no more spirochete-like discoveries."
To which we reply, "We think it's a free-for-all, now, and anyone
can
make up any diseases they want, as long as you psychiatrists claim to
have the magical
ability to diagnose us with anything without the use of any
scientifically valid
tests."
I say, "RETROCHONDRIA!!" in your general direction.
I say, "The AMA has FLACCITUDE!"
"Your ideas are Steereborn!!"
We say, "Meet me at the Yale Center for the Study of Erections,
and we
can have a discussion about all your uncomplicated variables ."
==========================================================================
23) An independent study on spirochetes in the brain from
dentists and they
say:
Molecular and immunological evidence of oral Treponema in the
human brain
and their association with Alzheimer's disease.
http://www.ncbi.nlm.nih.gov...11929559 medline link to verify
Riviere GR, Riviere KH, Smith KS.
Department of Pediatric Dentistry, School of Dentistry, Oregon
Health and
Sciences University, Portland, OR 97201-3097, USA.
The purpose of this investigation was to use molecular and
immunological
techniques to determine whether oral Treponema infected the human
brain. Pieces
of frontal lobe cortex from 34 subjects were analyzed with species-
specific PCR
and monoclonal antibodies. PCR detected Treponema in 14/16 Alzheimer's
disease
(AD) and 4/18 non-AD donors (P < 0.001), and AD specimens had more
Treponema
species than controls (P < 0.001). PCR also detected Treponema in
trigeminal
ganglia from three AD and two control donors. Cortex from 15/16 AD
subjects and
6/18 controls contained Treponema pectinovorum and/or Treponema
socranskii species-specific
antigens (P < 0.01). T. pectinovorum and/or T. socranskii antigens
were also
found in trigeminal ganglia and pons from four embalmed cadavers, and
2/4 cadavers
also had Treponema in the hippocampus. These findings suggest that
oral Treponema
may infect the brain via branches of the trigeminal nerve.
PMID: 11929559 [PubMed - indexed for MEDLINE]
"PCR detected Treponema in 14/16 Alzheimer's disease (AD) and
4/18
non-AD donors."
"Cortex from 15/16 AD subjects and 6/18 controls contained
Treponema pectinovorum
and/or Treponema socranskii species-specific antigens."
We wondered why this information never makes breaking headline
news as regards
new developments in Alzheimer's but then we remember the American
Psychiatric
Association has become like the Pope in his treatment of Galileo.
Such is heresy
against the dogma that "no more spirochete-like discoveries will be
made."
Despite the history and syphilis and the insane asylums and
Ehrlich's Compound
606, the arsenic bullet and so on and so forth...
==========================
24) Mousehausen's is now official CDC admits ceftriaxone fails to
eradicate
all spirochetes.
http://aac.asm.org/cgi/content/abstract/AAC.01050-07v1
Persistence of Borrelia burgdorferi Following Antibiotic
Treatment in Mice
Emir Hodzic, Sunlian Feng, Kevin Holden, Kimberly J. Freet,
and Stephen
W. Barthold*
Center for Comparative Medicine, Schools of Medicine and
Veterinary Medicine,
University of California at Davis, One Shields Avenue, Davis, CA 95616
The effectiveness of antibiotic treatment was examined in a
mouse model
of Lyme borreliosis. Mice were treated with ceftriaxone or saline for
one month,
commencing during the early (3 weeks) or chronic (4 months) stages of
infection
with Borrelia burgdorferi. Tissues from mice were tested for infection
by culture,
polymerase chain reaction (PCR), xenodiagnosis, and transplantation of
allografts
at 1 and 3 months after completion of treatment. In addition, tissues
were examined
for spirochetes by immunohistochemistry. In contrast to saline-treated
mice, mice
treated with antibiotic were consistently culture-negative, but
tissues from some
of the mice remained PCR-positive, and spirochetes could be visualized
in collagen-rich
tissues. Furthermore, when some of the antibiotic treated mice were
fed upon by
Ixodes scapularis ticks (xenodiagnosis), spirochetes were acquired by
the ticks,
based upon PCR, and ticks from those cohorts transmitted spirochetes
to naïve SCID
mice, which became PCR-positive, but culture-negative. Results
indicated that following
antibiotic treatment, mice remained infected with non-dividing but
infectious spirochetes,
particularly when antibiotic treatment was commenced during the
chronic stage of
infection.
Ya gonna argue with the CDC, now?
spirochetes were acquired by the ticks
Culture negative means nothing, as we know. It could take
months in vitro.
They should have completed the Mouse Infectivity Test.
25)
http://www.ncbi.nlm.nih.gov/pubmed/17045505?ordinalpos=11&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_RVDocSum
-
CDC on intracellular nerve and brain borrelia spirochetes
We all wonder how the hell the CDC can talk about this phenomenon
and not come
right and say "Lyme is a relapsing fever organism and is incurable due
to their
intracellularity and the formation of dormant cyst or spheroplast
forms, which are
also inhalable," - since this is all either their own data, or the
NIH's
data, or the US Army's data.
================================
26) Willy Burgdorfer discussing the cyst or the spheroplast
form. Combine
that with the reality that CDC officers discuss intracellular
spirochetes (Mark
Klempner and the one above). While Willy Burgdorfer is discussing
European articles,
however - disclaimed as invalid by the CDC except when Allen Steere
does it (Chapter
3) - recall that Willy Burgdorfer was himself recruited by the NIH
Rocky Mountains
Bioweapons Lab (which had a moat dug around it hmmm strangely like a
mini island,
like a mini Plum Island) from Switzerland, and the NIH also recruited
the German
scientist Roland Martin to head up the NINDS-Multiple Sclerosis group.
So, everyone can discount CDC's discount of foreign research since
obviously
no matter what they say, it's a lie.
==================================================
As we Lyme warriors cannot engage any earthly assistance;
As the entire US medical community chooses not to assist sick and
abused people
and put a stop to this;
As there is not one MD or group in the entire USA who will take
these criminals
to court;
As there is not one lawyer or Department of Justice official who
will do the
job they were hired to do and protect us from corporate crime;
As there are no men left among us:
http://www.ourladyswarriors.org/prayer/michael.htm
Saint Michael the Archangel,
defend us in battle.
Be our protection against the wickedness and snares of the devil.
May God rebuke him, we humbly pray;
and do Thou, O Prince of the Heavenly Host -
by the Divine Power of God -
cast into hell, satan and all the evil spirits,
who roam throughout the world seeking the ruin of souls.
Amen.
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