CDC says the pandemic swine flu is the Don Wiley H9N5 (Danish scientists say "BS" to American "medicine.")

[Mort Zuckerman]

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Subject: CDC says the pandemic swine flu is the Don Wiley H9N5 (Danish
scientist say "BS" to American "medicine.")

Date: Jan 17, 2012 12:07 PM

ARTICLE BELOW
==================================


If I am not mistaken, I read this
to be the transformation to H9N5:
Virology. 2012 Jan 5;422(1):105-13. Epub 2011 Nov 5.
In vitro evolution of H5N1 avian influenza virus toward human-type
receptor specificity.

Chen LM, Blixt O, Stevens J, Lipatov AS, Davis CT, Collins BE, Cox NJ,
Paulson JC, Donis RO.
Source
Influenza Division, ***Centers for Disease Control and Prevention,***
1600 Clifton Road, Atlanta, GA 30333, United States.

Abstract
***"Acquisition of α2-6 sialoside receptor specificity by α2-3
specific highly-pathogenic avian influenza viruses (H5N1) is thought
to be a prerequisite for efficient transmission in humans.*** By in
vitro selection for binding α2-6 sialosides, we identified four
variant viruses with amino acid substitutions in the hemagglutinin
(S227N, D187G, E190G, and Q196R) that revealed modestly increased α2-6
and minimally decreased α2-3 binding by glycan array analysis.
However, a mutant virus combining Q196R with mutations from previous
pandemic viruses (Q226L and G228S) revealed predominantly α2-6
binding. ***Unlike the wild type H5N1, this mutant virus was
transmitted by direct contact in the ferret model although not by
airborne respiratory droplets.*** However, a reassortant virus with
the mutant hemagglutinin, a human N2 neuraminidase and internal genes
from an H5N1 virus was partially transmitted via respiratory droplets.
The complex changes required for airborne transmissibility in ferrets
suggest that extensive evolution is needed for H5N1 transmissibility
in humans.
http://www.ncbi.nlm.nih.gov/pubmed?term=22056389


VS the Murdered Don Wiley's Observation:


"α2,6-Linked sialosides bind in a cis conformation, exposing the
glycosidic oxygen to solution and nonpolar atoms of the receptor to
Leu-226, a human-specific residue. ...

"Evidently, the “closed” geometry of the avian H5 HA, which prefers
α2,3 linkages, results from the Gln-226/Gly-228 pair. This geometry
appears optimal for positioning Gln-226 to hydrogen-bond to the α2,3
trans motif composed of the 4-OH of Gal-2 and the glycosidic oxygen
(Fig. ​(Fig.22c). ***The human H3 HA with the Leu-226/Ser-228 pair is
at the opposite extreme, more “open” at both 228 and 226, which may be
optimal for Leu-226 to make nonpolar contacts to α2,6 cis linkages.***
Swine H9 HA (Leu-226/Gly-228) and the L226Q variant of human H3 HA
(Gln-226/Ser-228) appear to be intermediate, with partial avian and
partial human character and the nonstandard Leu/Gly and Gln/Ser pairs
(Fig. ​(Fig.22f)."

http://www.ncbi.nlm.nih.gov/pmc/articles/PMC58807/?tool=pubmed

And the graphic I included in the
Oct 4, 2003 datapackage to Blumenthal
and the USDOJ:
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC58807/figure/F1/

========

This information was given to the US Attorney
with whom I filed the RICO complaint about
Yale's falsifying the testing for Lyme in
order to falsify LYMErix' outcome:
http://www.actionlyme.org

It was a data set for the US Attorney's office
marked "Oct 4, 2003," a copy of which was stolen off
of Senator Blumenthal's desk by the criminally
insane AAG Jessica Gauvin and you can see her list
it here:
http://www.actionlyme.org/BLUMENTHALS_MAIL_STOLEN_BY_JESSICA_GAUVIN.htm

Confirming to the casual observer that
CT AAG Jessica Gauvin is insane.


I was merely trying to splain to da 'tornies
how schientificals like biomarkers is done
- or was done by the same gang who says Lyme
is the result of not-enough-sex:
http://www.actionlyme.org/BIOMARKERS2.htm

I also show it in the movie, here (from 2006):
http://www.youtube.com/watch?v=6Pc8odvc9Z0


Once again, here is the CDC apparently revealing
what they don't want revealed:

"By in vitro selection for binding α2-6 sialosides, we identified four
variant viruses with amino acid substitutions in the hemagglutinin
(S227N, D187G, E190G, and Q196R) that revealed modestly increased α2-6
and minimally decreased α2-3 binding by glycan array analysis.
However, a mutant virus combining Q196R with mutations from previous
pandemic viruses (Q226L and G228S) revealed predominantly α2-6
binding. ***Unlike the wild type H5N1, this mutant virus was
transmitted by direct contact in the ferret model although not by
airborne respiratory droplets.***"


"Unlike the wild type H5N1, this mutant virus was transmitted by
direct contact in the ferret model although not by airborne
respiratory droplets."

In other words, take a bird H5N1 and
infect ferrets... and keep infecting
ferrets until the virus makes the species
jump. Then you have a strain of virus
that adopts to the human pandemic formation.

Now, all of this we knew to be what they
do on Plum Island thanks to Durland Fish:
http://www.actiolyme.org/PIIB.htm

I could be wrong but to the casual observer
of the binding sites, Don Wiley explained
what would be the pandemic conformation in
2001. Or some months before he was murdered
in the same way the Iraqi scientist working
on Plum Island was bumped off:
http://www.actionlyme.org/PIIB.htm

"Changing a flat tire."


KMDickson
http://www.actionlyme.org
===================
http://www.presstv.ir/detail/221444.html

A researcher inspecting samples in a lab. (file photo)The Dutch
scientists who created a mutated strain of bird flu have criticized
the US government's decision to censor their ground breaking
research.


Ron Fouchier and Ab Osterhaus of Erasmus Medical Centre in Rotterdam,
Netherlands, created a form of the H5N1 bird flu strain that can pass
easily between mammals.

The breakthrough, announced by the duo last fall, can be helpful in
the early-warning surveillance of new strains of flu virus, as well as
in creating new vaccines and anti-viral drugs.

The US National Science Advisory Board for Biosecurity (NSABB),
however, asked the world's leading scientific journals, Science and
Nature, to withhold publication of key details of the methodology and
results of the scientists' experiment, citing fears that the virus
could fall into the hands of terrorists wanting to use it as a bio-
weapon of mass destruction.

NSABB was formed in the aftermath of the 2001 anthrax attacks in the
US and is tasked with monitoring the scientific community for
bioterrorist threats.

The university-based scientists agreed to omit from their reports
certain details on how to construct the new strain but said the US
should not have a global veto on sensitive science.

“We … question whether it is appropriate to have one country dominate
a discussion that has an impact on scientists and public-health
officials worldwide," Fouchier and Osterhaus wrote in the journal
Nature.

"It is not clear whether an international discussion would lead to
different recommendations ... We don't know the worldwide opinion
until a group of experts from all parts of the globe is formed. An
issue this big should not be decided by one country, but all of us,"
they said.

The work was paid for by the US National Institutes of Health as part
of a large portfolio of research aimed at "pandemic preparedness.”

H5N1virus was first spotted in 1996 at a goose farm in Guangdong
province in China, and was reported the next year in Hong Kong, along
with the first human cases. Reports of infected birds then came in
from Korea, Thailand, Vietnam, Japan and Indonesia by 2004, Turkey and
Iraq by 2006, then Egypt.

About 600 have reportedly caught the virus worldwide, nearly 336 of
whom died as the result.

MN/MA