News on the MS-Version of Lyme / LYMErix (Pam3Cys) or the Activation of Latent EBV
Mort Zuckerman
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Subject: News on the MS-Version of Lyme / LYMErix (Pam3Cys) or the
Activation of Latent EBV
Date: Jun 12, 2010 5:13 AM
Treat with MS-Lyme with MS drugs
http://www.ncbi.nlm.nih.gov/pubmed/20530322
AND antibiotics (and perhaps antivirals)
like Roland Martin (NINDS) did.
Don't forget, there are no "Guidelines,"
not when these Yale guideliners gave
people Pam3Cys - the EBV-inducer - and
called that a "vaccine." They also 1) *lied*
to the FDA about their Pam3Cys outcomes,
2) re the testing (Dearborn), and 3) re
Mark Klempner's self-alleged "study."
Now the whole world - the AMA, the FDA,
the WHO, the EU, and Big Pharma - has *trashed*
this Lyme cryme gang, the Colorado-CDC Lyme
crooks have been *defunded,* too (as have these
Lyme crooks, who were also sued 3 times) and
Anthony Fauci had to admit he was a dang foo
re the HIV-LYMErix-Tb Pam3Cys TLR2-agonist
non-vaccines.
Use the diagnostic codes for MS and Relapsing
http://www.relapsingfever.org
Fever, and use band 41 in the absence of Syphilis
for diagnostic codes. These will imply long
term, as-needed treatment.
Kathleen M. Dickson
http://www.actionlyme.org
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News on the MS-Version of Lyme being caused by LYMErix (Pam3Cys) or
the Activation of Latent EBV:
Epstein-Barr Virus Latent Infection and BAFF Expression in B Cells in
the Multiple Sclerosis Brain: Implications for Viral Persistence and
Intrathecal B-Cell Activation.
http://www.ncbi.nlm.nih.gov/pubmed/20535037
"A cardinal feature of multiple sclerosis (MS) is the persistent
intrathecal synthesis of antibodies. Our previous finding that a large
fraction of B cells infiltrating the MS brain are infected with
Epstein-Barr virus (EBV) raises the possibility that this virus,
because of itsability to establish a latent infection in B cells and
interfere with their differentiation, contributes to B-cell
dysregulation in MS. The aim of this study was to gain further insight
into EBV latency programs and their relationship to B-cell activation
in the MS brain. Immunohistochemical analysis of postmortem MS brain
samples harboring large EBV deposits revealed that most B cells in
white matter lesions, meninges, and ectopic B-cell follicles are CD27
antigen-experienced cells and coexpress latent membrane protein 1and
latent membrane protein 2A, 2 EBV-encoded proteins that provide
survival and maturation signals to B cells. By combininglaser-capture
microdissection with preamplification reverse transcription-polymerase
chain reaction techniques, EBV latency transcripts (latent membrane
protein 2A, EBV nuclear antigen 1) were detected in all MS brain
samples analyzed. We also found that B cell-activating factor of the
tumor necrosis factor family is expressed in EBV-infected B cells in
acute MS lesions and ectopic B-cell follicles. These findings support
a role for EBV infection in B-cell activation in the MS brain and
suggest that B cell-activating factor of the tumor necrosis factor
family produced by EBV-infected B cells may contribute to this process
resulting in viral persistence and, possibly, disruption of B-cell
tolerance."
"[Real] scientists are *fiercely* independent. That's the good
news."-- NIH's Top Fool, Anthony Fauci