Lie Alone Blanco White _VERIFIED_ Download
Lauretta Jaffray
White Ibises gather in groups in shallow wetlands and estuaries in the southeastern United States. At each step, their bright red legs move through the water and their curved red bill probes the muddy surface below. As adults, these striking wading birds are all white save for their black wingtips, but watch out for young birds that are brown above and white below. White Ibises nest in colonies in trees and shrubs along the water's edge, changing locations nearly every year.
A visit to a coastal wetland in the Southeast any time of the year will likely be dotted with large white and dark wading birds. White Ibises stand out in the crowd with their reddish pink legs and bills. White Ibises tend to move around in large groups depending on water depth, so look for shallow wetlands or flooded fields to find foraging birds. Unlike larger herons who stalk their prey, White Ibises spend more time walking through wetlands. In some areas they now forage in urban parks and lawns, so don't be surprised if you find one outside of a wetland, especially in southern Florida.
Deana, I gasped when I scrolled down to the picture! It is absolutely beautiful! :o A work of art really and I love your floral version. I didn't realise that there was a white version of gazpacho too :)
HOLY COW....wait.....this has got to be one of the most creative recipes I have seen all summer yet. Spain is the land of my father. The RED STUFF is a staple here but AJO BLANCO????? Why did I never hear of this?
And then, the lavender sautée almonds? I need to go over to Penzey's Spices this weekend in St. Paul and get me some culinary lavender my dear...those little nuggets alone would be a wonder in MY SALAD or alone for guests! The photo of the soup....how do you do it all? You are a poet of food. That's all there is to it!!!
Thank you again my friend, for taking the time to come visit me. I wish you a WONDERFUL and most delicious FOURTH OF JULY!! Anita
Oh dear, I am going to be thinking about those almonds alone all day!! Anita
Such interesting flavours. I have never seen a white gazpacho. Hopefully those plants coming in my garden are borage. I love those flowers. Lavender almonds...mmmm. I must try that. So many interesting flavour combinations.
Wonderful read on the history of gazpacho! I guess it would make sense given the available ingredients of the time. I have never tried a white gazpacho, but this soup is just heavenly...and gorgeous! Wow!
What a feast for the eyes and taste buds, Deana! I so enjoyed ready about the history of white gazpacho and the recipe is going into my recipe file!
I hate to admit Borage is an herb I've never tasted.
Better late than never, right? I agree with Lazaro this is hands down the best soup of the round up. Can you believe I have never had ajo blanco?? It actually looks more appealing to me than tomato based gazpacho.
Some Senator(s) are alleged to be conflicted on the basis that actual inventors are among the ranks of their citizenry, and calling for their execution would be, somehow, wrong. I hope good sense overwhelms that particular Senator and they, alone if necessary, put a stop to this mindlessness.
I would suggest, in the absolute and complete alternative to the present proposals to eliminate the patent system, this: before a patent suit can be brought, the PTO works with a patent owner, like the ITC, to certify an infringement. Cost: under $ 5000.00. Once that is certified, a District Court complaint can be filed with notice to State AGs about who and how many accused exist within their state, the process is then automatically stayed until validity is likewise certified, also by the PTO, on the basis of the accused, alone or in combination, paying for and challenging validity. Once validity is determined up and through CAFC appeal, the court proceeds to enjoin and/or mete out damages. All of this occurs within 2 years. Inasmuch as the PTO is the gate keeper for infringement and validity, the claim interpretation does not change. The District Court does what they do best, determine damages, not claim scope and meaning. The CAFC runs it all, subject to occasional interference (guidance) from the S.Ct. Everyone is a winner. Both sides have predictable timelines, costs and outcomes. It is not, as it is now, rigged so that the victory goes to the well capitalized, and merit is of little or no import, and the patent is an afterthought.
Under Florida's death penalty scheme, a convicted defendant cannot qualify for the death sentence unless one or more statutory aggravators are found to exist in addition to *14 the conviction for first-degree murder. See Elam v. State, 636 So. 2d 1312 (Fla.1994). Hence, a defendant convicted of intentional premeditated murder will not be eligible for the death penalty unless some additional aggravating circumstance, in addition to the premeditated murder, is found to exist. On the other hand, a person found guilty of felony murder automatically becomes eligible for a death sentence. By reason of the felony murder aggravator, the underlying felony is used not only as a legal substitute for premeditation to support a first-degree murder conviction; but is also used as a statutory aggravator to immediately make the defendant eligible for the death sentence. In other words, Florida's felony murder aggravator permits a defendant convicted of felony murder to be sentenced to death by virtue of his conviction for felony murder alone.
Common sense alone tells us the scheme described above is patently inconsistent with the United States Supreme Court's strict requirements in Zant v. Stephens for a rational and narrowing scheme for selecting those who will be subject to the death penalty. Recently, the U.S. Supreme Court reiterated the requisite narrowing function that must be served by proper aggravating factors, explaining again that where a weighing state, like Florida, uses aggravating factors to determine who is eligible for the death penalty, "it cannot use factors which as a practical matter fail to guide the sentencer's discretion." Stringer v. Black, 503 U.S. 222, 235, 112 S. Ct. 1130, 1139, 117 L. Ed. 2d 367 (1992). By making a defendant convicted of felony murder automatically eligible for the death penalty based upon the same felony that was used to establish the defendant's conviction for murder, this scheme simply does not narrow the class of convicted persons who become eligible for the death penalty. Rather, if anything, it clearly enlarges the eligible class in an irrational way.
Incubation of chondrocytes with rhIL-1β (1 ng/ml) resulted in the production of high concentrations of NO, as compared with untreated controls (Fig. 1,A). The production of NO above basal levels was not observed during the first 4 h of rhIL-1β exposure, but increased within 12 h, and continued to rise in a time-dependent manner during the ensuing 96 h. Coapplication of CTS and rhIL-1β to chondrocytes consistently and significantly (p < 0.05) suppressed IL-1β-induced NO production at all time points tested (Fig. 1,A). The culture supernatants of chondrocytes subjected to CTS in the absence of rhIL-1β and unstressed controls exhibited NO levels similar to that of medium alone (
To determine whether the suppression of IL-1β-dependent iNOS mRNA by CTS was due to the inhibition of iNOS mRNA expression, chondrocytes were subjected to CTS regimen for 4 h. The assessment of iNOS mRNA abundance by RT-PCR revealed that CTS alone did not induce expression of iNOS mRNA, whereas rhIL-1β treatment induced significant expression of iNOS mRNA within 4 h (Fig. 2,B). The relative expression of iNOS mRNA in chondrocytes subjected to costimulation with rhIL-1β and CTS was markedly suppressed, as compared with chondrocytes treated with rhIL-1β alone (Fig. 2,B). Parallel experiments demonstrated that inhibition of rhIL-1β-dependent iNOS mRNA expression by CTS during the first 4 h was followed 4 h later by suppression of iNOS synthesis (Fig. 2,C). The sustained suppression of iNOS synthesis by CTS was also confirmed by the observation that CTS inhibited 66% of the total IL-1β-induced NO accumulation in the culture supernatants 24 h later (Fig. 2 D).
The exact extent of IL-1β-induced iNOS mRNA suppression by CTS was then determined by quantitative competitive PCR. As expected, control untreated cells and cells treated with stress alone did not exhibit the presence of iNOS mRNA. Whereas, mRNA from chondrocytes treated with IL-1β alone and treated with IL-1β and CTS exhibited equimolar gene products in competitive PCR at 0.62 and 0.156 attomoles of MIMIC DNA, respectively (Fig. 2,E). Deduced from this data, cells treated with IL-1β alone, and cells treated with IL-1β and CTS, exhibited 112.7 and 28.3 molecules of iNOS mRNA per μg of total RNA, respectively. Thus, CTS suppressed IL-1β-induced iNOS mRNA expression by 75%. In these experiments, total accumulation of NO in the culture supernatants was reduced by 66% in cells exposed to CTS and IL-1β, as compared with cells exposed to IL-1β alone (Fig. 2 D).
To determine whether the reduction in IL-1β-dependent iNOS mRNA expression by CTS was paralleled by a net decrease in the synthesis of iNOS, chondrocytes were subjected to CTS regimen for 8 h in the presence of IL-1β. Thereafter, the cells were harvested, and total iNOS synthesis in cells was assessed by Western blot analysis. As expected, untreated control cells and cells subjected to stress alone did not exhibit iNOS synthesis (Fig. 2,C). However, chondrocytes exposed to IL-1β exhibited significantly increased iNOS synthesis, which was suppressed in cells costimulated with IL-1β and CTS. In parallel confirmatory experiments, cells subjected to costimulation with IL-1β, and CTS exhibited a significant reduction in NO production as compared with cells exposed to IL-1β alone (Fig. 2 D).
Immunochemical analysis of chondrocytes exhibiting suppression of rhIL-1β-dependent iNOS synthesis by CTS. The iNOS synthesis in chondrocytes was compared in cells subjected to medium alone (A), CTS alone (B), rhIL-1β (C) (1 ng/ml), or costimulation with CTS and IL-1β (D) for 24 h. Histomorphometric analysis of cells showed absence of iNOS in A and B. IL-1β-treated cells (C) show presence of iNOS by intense peroxidase staining in 35 4% of chondrocytes, while cells subjected to simultaneous IL-1β and CTS (D) showed a 62 6% lower staining intensity for iNOS, as compared with iNOS-positive cells in C.
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