The Chemical Mechanisms leading to EMF Sensitivityis?

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news....@googlemail.com

ungelesen,
02.02.2010, 02:50:1402.02.10
an omeg...@googlegroups.com
Here we are... nitric oxide (NO)... in between other biochimical
mechanisms...

Well, I already sent to our Community compiles I've done some times ago.

Wireless communications are practical, useful and facilitate life, right?

Some very specific electric, magnetic and electromagnetic waves (EMF
to be short) can cure some very specific ailments, right?

Sure, a lot of things are bad for our health. But when the castle
burns, we don't care about the stable, right?

But the point is that humans and wildlife were not built, nor evolved
to support all that mass of mixed spread EMF by wireless technologies
we are obliged to sustain without any place to hide.

For example, the total radiation (terrestrial and cosmic) in the 900
MHz band were evaluated to:
0.000'005 uW/m2.
It must be compared with the 4.500.000 uW/m2 authorized by authorities
for the human pollution.
If you divide the second number by the first, I hope your screen is
large enough to shows the result...

Back to NO...
In the 3 compiles attached (2 old and an update), you'll find
interesting studies about EMF and NO or relative studies.

For example:

Ann N Y Acad Sci. 2001 Mar;933:323-9.
Elevated nitric oxide/peroxynitrite mechanism for the common etiology of
multiple chemical sensitivity, chronic fatigue syndrome, and post
traumatic stress disorder.

Pall ML, Satterlee JD.


Otolaryngol Head Neck Surg. 2005 May;132(5):713-6.

Nitric oxide level in the nasal and sinus mucosa after exposure to
electromagnetic field.

Yariktas M, Doner F, Ozguner F, Gokalp O, Dogru H, Delibas N.


Oh my divine, oh my divine, oh my diiiivine melatonin...

But wait, in that one, conclusions say:
These results strongly suggest that ELF-MF attenuates antioxidative
actions of melatonin on cellular level.

Look:
Biol Trace Elem Res. 2004 Winter;102(1-3):227-43.
Influence of extremely-low-frequency magnetic field on antioxidative
melatonin properties in AT478 murine squamous cell carcinoma culture.
Zwirska-Korczala K, Adamczyk-Sowa M, Polaniak R, Sowa P, Birkner E,
Drzazga Z, Brzozowski

T, Konturek SJ.


I wish you a good lecture. Don't we know enough to say STOP to EMF?
Are there not enough studies showing so many biological effects after
EMF exposition and enough studies telling us the direct or indirect
interaction with all the diseases development growing on this world on
exponential manner ?

Just allow me a question for my own use:
Which one of you don't own a mobile phone, nor a DECT, nor WiFi, nor
any kind of wireless technology ? I will keep it in my secret records !
You have the right to remain silent. Anything you say...


Philippe Hug
Chairman of ARA
http://www.alerte.ch

PS: I have thousands of other studies I never compiled. Too much work
for a single man...

NO subject is not close. Other studies are available, like that one:

One of the barriers to our understanding of the mechanisms involved in
fibromyalgia (FM) is the lack of any animal models of FM. So whereas
proposed animal models for chronic fatigue syndrome (CFS), multiple
chemical sensitivity (MCS) and posttraumatic stress disorder (PTSD)
are available which suggest a role for excessive nitric oxide in each
of these conditions, there is no similar animal model to study for FM.

See:
http://www.prohealth.com/fibromyalgia/library/showarticle.cfm?id=4350&t=CFIDS_FM

Yes, EMF trigger NO that trigger EHS, CFS, MCS, PTSD, COPD, Alzheimer,
and more and more !

Mayday, mayday, mayday...

1. 0 Compile Oxydative stress 1.2.10.pdf
2. 0 Compile Oxydative stress 26.2.7.pdf
3. 00 NO LISTE des �tudes au 15.1.7.pdf

Gary, George, all

This has always been an interesting strand to me, because (and in danger
of sounding like a one-string-violin - I'm not!) there is a common
thread in nitric oxide (NO) imbalance via synthase disruption, that
still intrigues. It connects and explains the similarities in MCS, EHS,
CFS etc. and also connects with mitochondrial disease, eg:

http://www.nature.com/ncb/journal/v6/n11/full/ncb1188.html

It's not that MCS triggers EHS, EHS triggers CFS, etc., but that the
commonalities mean different triggers can compound for the same results.
Set a fire off from an electrical fault,in one room, a gas leak in
another, a cigarette in a third, and the house will burn in the same
way. No good just blaming the smoker.

I see too little about the role of NO, apart from a few references to
RNS, but the amount of research evidence of it being central, and being
triggered also by EM fields, is substantial.


Andy Davidson

Gary Kohls wrote: HELLO ALL: THANKS FOR THE INFORMATION ABOUT
MITOCHONDRIAL MEMBRANE DYSFUNCTION AND THE RELATIVELY GOOD NEWS ABOUT
THE UNIVERSAL TREATMENT (WITH OMEGA 3 FATTY ACIDS AND PLENTY OF
ANTIOXIDANTS, I PRESUME) THAT MUST BE DIRECTED AT THE POISONED BILIPID
CELL WALL OF THE DAMAGED MITOCHONDRIA (AND ALSO THE NEURONS, IN THE CASE
OF THE CENTRAL NERVOUS SYSTEM AND, FOR THAT MATTER, EVERY CELL OF THE
BODY). TWO YEARS AGO I ATTENDED A SEMINAR IN DULUTH, MN THAT WAS
ABOUT SO-CALLED \"CONGENITAL\" MITOCHONDRIAL SYNDROMES. IN THE AUDIENCE
WAS A RESEARCHER WHOSE AREA OF EXPERTISE WAS MITOCHONDRIAL DISEASES. AT
THE Q&A HE MENTIONED THAT A RESEARCH COLLEAGUE OF HIS HAD IDENTIFIED
72 COMMONLY PRESCIBED DRUGS THAT WERE MITOCHONDRIAL TOXINS. THE
EXPLOSIVE DATA HAD NOT BEEN PUBLISHED YET (I WONDER WHY?). THE ONLY TWO
MITOCHONDRIAL-POISONING DRUGS OF THE 72 THAT HE MENTIONED WERE PROZAC
(DISTA) AND LIPITOR (PFIZER). I TALKED TO HIM AT THE BREAK AND HE GAVE
ME HIS EMAIL ADDRESS WHEN I ASKED IF I COULD COMMUNICATE WITH HIM
FURTHER ABOUT THIS EXTREMELY IMPORTANT ISSUE (WHICH COULD BRING DOWN
BIGPHARMA IF IT EVER CAME OUT). I HAVE EMAILED HIM ON SEVERAL OCCASIONS
SINCE THEN AND HE HAS NEVER RESPONDED. I LATER FOUND OUT THAT HE HAD
RESEARCH GRANTS FROM PFIZER, HENCE THE SILENCE. I HAVE TRIED TO TRACK
DOWN THE RESEARCH ON THIS TOPIC AND HAVE ASKED OTHERS WHO WOULD BE
INTERESTED IN THIS TOPIC TO DO THE SAME AND NONE OF US HAVE FOUND
ANYTHING ONLINE ABOUT THIS TOPIC. I AM THEREFORE ASKING THOSE ON THIS
ELIST FOR HELP IN THIS EFFORT. THANKS FOR ANY INFORMATION YOU MIGHT BE
ABLE TO FIND. GARY G. KOHLS, MD. MY MAILING ADDRESS IS 1306 E 8TH ST,
DULUTH, MN 55805. PHONE # IS 218-728-9756. PS: THIS IS INFORMATION THAT
COULD BE LETHAL TO THE RESEARCHER, PUBLISHER OR PROPAGATOR OF IT, SO BE
CAREFUL OUT THERE. GARY G. KOHLS, MD ----- ORIGINAL MESSAGE -----


FROM: G. L. Carlo TO: xxx

SENT: Sunday, January 31, 2010 8:03 AM SUBJECT: Re: Chemical Mechanisms
leading to EMF Sens...Dallas 6-3 thru

6-6-2010 As we continue to learn more and more based on causal studies
(published and unpublished), efficacy experience with various
intervention protocols, clinical observations, and feedback from those
both adversely effected and helped, the following emerge as fundamental
to the mechanism based on our work:

* A common mechanistic thread: cellular level oxidative stress reactions
that cause cell and mitochondrial membrane permeability changes. These
changes are measurable in the laboratory and can be both adverse and
adaptive. Symptoms occur when the adverse impact becomes greater than
the adaptive capacity.

* Virtually all of the resultant biological effect cascades --
intracellular (e.g. DNA repair), extracellular (e.g. heat shock
proteins) and intercellular (e.g. communication) -- that lead to
symptoms derive initially from ROS and RNS impacts

* Our data show that there are many pathways to cell membrane-level
oxidative stress response -- physical, chemical, biological. These
include environmental chemicals, internal biochemical imbalances caused
by anxiety and life-stresses, EMR, viral and bacterial infections, etc.
However, once the resultant cascades ensue, it appears that the
pathophysiological condition is the same. This is good news on the
treatment side.

Thus, our data do not suggest that one type of sensitivity leads to
another. Certainly there are additive and synergistic effects when
multiple stressors are involved. However, it appears that there are
diverse pathways to the pathophysiologic effect, but once that effect
occurs, the disease conditions are essentially similar. That is one
reason why, at least in our experience, intervention protocols that work
for EHS also work for MCS, PTSD etc.. One key appears to be eliminating
the myriad sources of oxidative stress while the interventions are being
tried..... the proverbial "stop the bleeding" first.... G

Dr. George L. Carlo
The Science and Public Policy Institute The Institute for Healthful
Adaptation Washington, D.C.
866-620-4459


-----Original Message-----
From: Don Sent: Sun, Jan 31, 2010 4:54 am Subject:
Re: Chemical Mechanisms leading to EMF Sens...Dallas 6-3 thru 6-6-2010

An excellent book to read is Gunni Nordstrom's "The Invisible Disease:
The dangers of Environmental Illness caused by Electromagnetic Fields
and Chemical Emissions". People working in electrical environments are
also usually posed to a wide range of chemical emissions that out-gas
from the equipment. The Swedish Union of Clerical and Technical
Employees in Industry did a lot of excellent research, and funding
researchers into these issues up to the late 1990's. What apparently was
the case with the EHS problem in Sweden was exposure to chemicals may
have been a factor in sensitizing some workers to EMF as well. In other
words, in some cases where chemical exposure existed (such as working
with computers) the resultant chemical sensitivity may have led to
EHS.MCS can be quite unrelated to EMF exposure. Of course the increasing
EMF exposure worldwide with microwave technologies is enough to make
many people EHS without any additional co-factors.The conference looks
like an excellent event, with some good presenters. Too bad its not in
Australia!

Don Maisch

On 31/01/2010, at 8:12 PM, a...@alerte.ch wrote:
excerpt
The title: The Chemical Mechanisms leading to EMF Sensitivityis
confusing for me. I would prefer: EMF leading to Biochemical mechanisms,
because all humans are EMF sensitive, and MCS has it's origin on EMF.

Philippe Hug


excerpt

Subject: Chemical Mechanisms leading to EMF Sens...
Dallas 6-3 thru6-6-2010 - Anmual Symposium Environmental Health and
Disease

List of faculty is "very encouraging!!!" Prof. Donald Hillman, Prof.
Andrew Marino, Dr. William Rea, Prof. Cyril Smith (UK) and
more.......Would love to attend and do know I have a lot of VIP facts to
share;however, simply can not afford even travel and hotel let alone
registration feethat I have not yet determined. Would have to bring Bud
along, whichwould be o.k. except that would also require another
registration fee. I do see such a gathering of EMF/EMR-savvy persons as
offering hope insofaras "informing public," but, as always, "media
silence" is a formidable obstacle! We know the release of the EMF RAPID
Interagency Committee Report to Congress should be followed with "public
service announcements" by EPA, NIEHS/NIH, H&H. If immune deficiency and
Leukemia links are notsufficient to put "Precautionary Principle" in
place, I guess I do not knowwhat to say. Problem, as we already know, is
that government is tooconcerned already and that is why we must force
release of EMF RAPID Interagency Committee Report! I usually write and
think "one day at a time!" But, as we know, rates of cancer and other
health problems are increasing at such a rapid pace, we,as society, can
not be complacent on any level particularly re close, chronic, prolonged
electric field exposures that will obviously be major concerns at this
Symposium in Dallas.Take care - JoanneJoanne C. MuellerGuinea Pigs R
Us731 - 123rd Avenue N.W.Minneapolis, Minnesota 55448-2127 USAPhone:
763-755-6114E (1-28-10)Website: http://www.guineapigsrus.org/)ARE YOU
AND YOUR CHILDREN GUINEA PIGS?
http://www.guineapigsrus.org/letters.html)See also:
http://www.radiationrescue.org/)
Joanne C. Mueller FACEBOOK public profile page:
http://www.facebook.com/people/Joanne-C-Mueller/1454397859)In a message
dated 1/29/2010 12:21:31 P.M. Central Standard Time,evans...@att.net
writes:"P" (name & email address removed) and Joanne,I hope you can go:
http://www.aehf.com/symposium_2010/2010Symposium.pdf)</excerpt></excerpt>

0 Compile Oxydative stress 1.2.10.pdf
0 Compile Oxydative stress 26.2.7.pdf
00 NO LISTE des études au 15.1.7.pdf

omega_news

ungelesen,
04.02.2010, 01:55:0304.02.10
an World-News
Re: Anybody care to respond to this question about prescription drugs
being mitochondrial toxins?

Thanks for this information.

Unfortunately, I haven't made any investigation in that matter.

But about microwaves, you can see the effects on mitochondrial DNA
here:
http://www.ncbi.nlm.nih.gov/pubmed/19879861
http://www.ncbi.nlm.nih.gov/pubmed/15130438

This appear to be part of the aging process, see:
http://www.ncbi.nlm.nih.gov/pubmed/11499335

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