Harris Web Portal

[Amelie Robertos]

Belowyou will find answers to commonly asked questions concerning the patient portal. For additional questions or technical issues with this portal during normal business hours, please contact our office.

The Patient Portal is an online service that provides patients secure access to their health information. Various features may be available on the portal at your practice's discretion, including the ability to send messages to your health care providers, schedule appointments, and pay bills online.

Any active patient may be eligible to register for and use the Patient Portal. If you are authorized, a family access account can be created that will allow you to access selected family members' health information.

All communications between you and your provider's office are carried over a secure, encrypted connection. This secure connection utilizes industry standard Secure Socket Layer (SSL) encryption to ensure secure data transmission as well as server-side digital certificate authentication. To prohibit unauthorized access, all medical information is stored behind our firewall in our electronic medical record system.

You should always make sure that the email address on file for your account is accurate, as notifications from the portal are sent to the email address on file. Make sure to sign out of your account each time you are finished using the portal.

Click the Sign Out link at the top right of the screen. Alternatively, if your keyboard remains idle for 10 minutes or more, you will receive a pop-up window asking if you are still actively using the portal. If you do not click the OK button, you will be signed out automatically. Any information you have typed and not saved or sent will be lost.

It is at your practice's discretion to allow online appointment scheduling. If your practice does not allow online appointment scheduling, you can request an appointment by sending a message to your provider. To do so:

Your provider's office will make every effort to respond to your messages within a timely manner. Please do not expect a response on weekends or holidays. If you need to speak with the office sooner, please call the office directly. Urgent matters should not be dealt with via the Patient Portal.

Adobe Acrobat is required to view and print statements and forms on the Patient Portal. When you click the Billing tab, you will see a note indicating this requirement, along with a link to download this program for free.

It is at your provider's discretion to make test results available. Your provider must authorize the release of your test results in order for them to post to your Patient Portal account. Only test results which are considered appropriate for release will be accessible through the Patient Portal.

We use cookies to make our site work, including measuring elements related to the site's performance as intended. By using our site you agree that information about you and your visit may be collected and/or sent to third parties for these purposes. We do not sell or share your information, nor do we use analytics, advertisement, or other non-necessary cookies. For more information about our online privacy practices, please visit our online Privacy Policy.

The Patient Portal, offered by Harris Regional Hospital, is a convenient, secure health management tool you can use anywhere. It provides access to important information about you and your visit(s) to our hospital.

Harris Regional Hospital has partnered with Syntellis to provide this service. Online healthcare information is available to patients 18 years and older and who have been a patient at the hospital on or after January 1, 2014.

All Discharged Patients who supplied a valid email address will receive an electronic invitation from the hospital and a link to register online for Syntellis shortly after discharge.

If you have any difficulty registering or using the service, please contact Syntellis Customer Support at 1-800-851-5043 or by e-mail at patientpor...@syntellis.com.

The patient portal, offered by Harris Regional Hospital, is a convenient, secure health management tool you can use anywhere. It provides access to important information about you and your visit(s) to our hospital. Please note: If you add any information to your personal health record, we cannot access those changes. All questions regarding test results should be directed to the patients primary care or ordering physician.

Yes, secure-socket layer encryption technology is utilized. When will health information/results be available on the Patient Portal? All information will be available within 36 hours of discharge. In most cases, information is available immediately following discharge.

In addition to revolutionising our understanding of the mechanisms that control the key endocrine systems and peptide synthesis, release and action, the neuroendocrine revolution led to major developments in the clinical diagnosis and treatment of conditions such as infertility, central precocious puberty (CPP), acromegaly, dwarfism, Cushing's syndrome, neuroendocrine and hormone dependent cancers, hypertension and other cardiovascular disorders and metabolic syndrome (e.g. Fink 1976, Fink et al. 2012, Fliers et al. 2014).

The history of neuroendocrinology has been the subject of several reviews (Harris 1955, Guillemin 1967, 1978, 2011, Harris 1972, Fink 1976, 1986, 1988, 2012, Charlton 2008). Friedgood (1936) and Hinsey (1937) were arguably the first to postulate formally that the anterior pituitary gland was controlled by substances liberated into the hypophysial portal vessels from nerve terminals in the median eminence. However, this hypothesis was not accepted until work from Harris' laboratory in Cambridge established that: i) the direction of blood flow in the portal vessels of living mammals was from the hypothalamus to the pituitary (Green & Harris 1949); ii) after severance of the pituitary stalk, the function of the anterior pituitary gland could be correlated with the degree of its revascularisation by the hypophysial portal vessels (Harris 1950); and iii) the morphological and functional integrity of pituitary grafts were maintained (or developed in the case of tissues from immature donor animals) when these grafts were vascularised by the hypophysial portal but not the systemic circulation (Harris & Jacobsohn 1952). The results of the last study, which constituted the most important biological evidence for the neurohumoral hypothesis, were soon confirmed by the equally elegant pituitary grafting experiments of Nikitovitch-Winer & Everett (1958, 1959).

Schally, with Murray Saffran at McGill, and Guillemin in Houston, started their isolation studies on corticotrophin-releasing factor (CRF). But two decades passed before the indomitable Wylie Vale at the Salk obtained the amino acid residue sequences of CRF-41 and the related urocortins (Vale et al. 1981, Bale & Vale 2004). Vale's success in sequencing CRF was a special landmark in neuroendocrine history, and it was perhaps brought into sharper focus by his untimely and tragic death on January 3, 2012 (Fink 1981, Bale & Chen 2012).

In addition to controlling anterior pituitary hormone release, hypothalamic neurohormones are also essential for pituitary hormone synthesis. This has been obvious since the original graft experiments of Harris & Jacobsohn (1952), and it was reinforced, for example, by the hypogonadal (hpg) mouse, which has an autosomal recessive mutation in the GnRH gene that results in an isolated, massive deficiency in LH and FSH (Cattanach et al. 1977).

Harris' first papers were on the induction of pseudopregnancy in the rat and ovulation in the rabbit (Harris 1936, 1937), and this major interest in reproductive physiology continued throughout his life. Thus, after adducing physiological evidence for the neurohumoral hypothesis of anterior pituitary control, Harris' personal research interests focused largely on the neural control of gonadotrophin secretion. And it is on this topic that the remainder of my review focuses, because reproductive neuroendocrinology encompassed my graduate research under Harris' supervision as well my subsequent research at Monash University Australia and then at Oxford University. Furthermore, it has heuristic value for other neuroendocrine and neurotransmitter mechanisms and ultimately led to research on the steroid hormone control of central neurotransmission, which has relevance for our understanding of mental disorders (Fink 1995a, Fink & Sumner 1996, Fink et al. 1996, 1999, Sumner et al. 2007).

In his Oxford laboratory, Harris promoted research on the isolation and characterisation of LH-releasing factor, now GnRH, and the existence of GnRH-activity in hypophysial portal vessel blood. The latter was investigated in blood collected from the cut pituitary stalk of the urethane-anaesthetised rat using the transpharyngeal approach (Worthington 1966, Fink 1967, Fink et al. 1967). Our findings showed significant GnRH activity in unextracted and acid-ethanol extracts of portal blood from intact and hypophysectomised (to exclude LH contamination) rats, as assessed by three different bioassays, including the induction of ovulation in the rabbit (Fink & Harris 1970).

Our bioassay findings (see the previous section) were confirmed by specific GnRH RIA, which showed that the concentration of GnRH in rat hypophysial portal was significantly greater than that in systemic jugular venous blood (Fink & Jamieson 1976). Using HPLC and two specific anti-GnRH sera, we found that a single immunoreactive peak was present in rat hypophysial portal blood and in hypothalamic extracts from rats and normal mice, and it corresponded in retention time to synthetic GnRH (Sheward et al. 1985). No GnRH immunoreactivity was detected in hypothalamic extracts from the hpg mouse (Sheward et al. 1985).

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