What do you do to lower your glucose level.

[Henry Wannabe]

I am wondering... dangerous huh?

What do you think is the best exercize for lowering your glucose level? Do you have a system? For example: Each mile walked will lower my glucose level 20 points. Or, something similar.

Tanks...
are dangerous.

Hank

[outsider]

I used a treadmill till a cardiologist told me he didn't think it
a good idea to work through angina. One of my problems is that when
bg is high (and that point has been getting lower of late) I easily
get angina. When things were at their best (within the past decade)
the treadmill was my favorite torture of choice for driving down bg.

Today, alas, it is injecting insulin. I am trying to get back to
that better state, by losing some weight. Best guess is that once
I lose significant weight (I consider myself 50 pounds overweight)
everything should improve significantly.

[gord...@swbell.net]

Not an answer but another question that, hopefully, others can shed
some light on.

How does the enzyme amylase fit into this set of problems? Is it
prudent to consider taking some of those non-prescription medications
that are supposed to provide a boost of this enzyme.

I understand that as one ages their body produces less and less
amylase until finally, if there are any other contributing factors,
Type 2 diabetes may begin to be a problem. Will taking amylase head
one's Type 2 diabetes off and let them return to a diet of more
ordinary foods instead of scrimping on the carb intake? Gordon

[Cindy Wells]

On 11/11/2012 4:19 PM, gord...@swbell.net wrote:

> How does the enzyme amylase fit into this set of problems? Is it
> prudent to consider taking some of those non-prescription medications
> that are supposed to provide a boost of this enzyme.
>

Where did you come up with this idea?

> I understand that as one ages their body produces less and less
> amylase until finally, if there are any other contributing factors,
> Type 2 diabetes may begin to be a problem. Will taking amylase head
> one's Type 2 diabetes off and let them return to a diet of more
> ordinary foods instead of scrimping on the carb intake? Gordon
>

Amylase is in saliva and in the pancreatic enzymes. It breaks down
carbohydrates into their component sugars. (Did you ever do the "chew a
cracker" experiment with Benedict's solution in a science class?).
That's why here most of us find reducing carbohydrates is needed for
control, not just the "sugars in the diet". (The traditional myth of
type 2 diet plans is reduce sugars.) If you take amylase as a pill, you
might slight increase in the amino acids in your diet. If you took it as
a liquid with any carbohydrates, it would probably break down a bit more
of the carbs.

If amylase production were decreasing, the carbs we eat wouldn't have as
much of an effect. Less of the carbohydrates would be fully absorbed. (I
believe that there are limits to what carb breakdown happens in the rest
of the digestive track.)

There are some who get an increase in amylase and that can indicate a
serious problem. (IE: Pancreas inflammation, intestinal obstruction). If
a person had a high amylase level and then it dropped, it can indicate
cell death of the amylase production cells in the pancreas. (Rather like
the damage to the islets of Langerhans in type 1 diabetes.)
>


Cindy Wells

[Julie Bove]

"Henry Wannabe" <nineb...@aol.com> wrote in message
news:70fe3093-6638-4ef4...@googlegroups.com...

Exercise raises my BG. Insulin lowers it.

[Pro-Humanist FREELOVER]

"Henry Wannabe" <nineb...@aol.com> wrote ...

>I am wondering... dangerous huh?

Standard modalities utilized in the past:

o For the first 48 years after being diag-
nosed with Insulinitis (old name: type 1
diabetes) in March of 1961 (at the age
of 5), injected NPH insulin along with
Regular insulin (about 20 NPH, 10 Regu-
lar in the morning before breakfast, and
about 10 NPH and 10 Regular prior to
the dinner meal); late in that process,
due to becoming hypoglycemic unaware
and enduring repeated severe low glu-
cose events, I stopped giving the evening
shot.

o Recently (over 3 years ago), I was switched
to an intense insulin regimen which turns
out (on most days) to have a long-acting
Levemir injection of 16-20 units prior to
breakfast, and a long-acting Levemir injec-
tion of 6-8 units prior to bedtime, plus a
quick short-acting injection of Novolog
(amounts vary based on time of day and
expected Carb consumption and expected
exercise activity) prior to breakfast, lunch,
dinner, and (if needed) at bedtime. Occa-
sionally, a glucose test at the 2 hour mark
necessitates a booster Novolog injection to
handle a glucose level higher than expected/
desired.

Unfortunately, with all insulin injection regimens,
severe low glucose risks are present, day and
night, and from time to time, a severe low glu-
cose event transpires (it has been estimated that
up to 11 percent of those with Insulinitis will die
due to a severe low glucose event, though most
estimates are lower than that, and often, when
someone with Insulinitis dies, that actual nature
of their death is either unreported / unknown or
suspected but unverified).

> What do you think is the best exercize for
> lowering your glucose level? Do you have
> a system? For example: Each mile walked
> will lower my glucose level 20 points. Or,
> something similar.

Exercise, when an expectation of an unusual
amount of exercise increase or decrease arises,
I modify the Levemir and Novolog dosages to
accomodate that increase or decrease.

As always, important to recognize that in an
individual with Insulinitis, insulin-giving is a
probabilistic guessing enterprise, with the re-
sults probably meeting expectations, but at
times, due to metabolic unpredictability, the
results are lower (sometimes, dangerously
lower) or higher than expected/desired, and
it also must be mentioned that unless exact
carbohydrate intake is known (which is much
easier to accomplish with carb information re-
quired on labels, but in the past, was naught
but a guessing game, and even in the present,
there are times when food/drink ingestion is
a guessing game due to nutrition information
being unavailable, typical of ingestion at social
events), and when unknown, must be guessed
at.

Also, unlike the perfect every second of every
day and night glucose-sensing insulin-responding
glucagon-responding modality of those without
a glucose anomaly, the insulin guessing of those
with Insulinitis is an intense 24 hour 7 day a week
365 days per year (366 ever leap year) enterprise,
with no time off, ever, a constant unrelenting night-
mare (but, of course, efforts are underway to reduce
this burden, with dramatic advances in treatment
being researched, and efforts to cure this condition
being researched, and hopefully, some day or night,
sooner rather than later, a cure will become widely
and affordably available, and the quality of life for
those with Insulinitis will return to that grand state
of metabolic perfection we all enjoyed prior to the
unwelcome arrival of Insulinitis).

- - -
Pro-Humanist FREELOVER
C.ure I.nsulinitis A.ssociation
http://prohuman.net/cureinsulinitisassociation.htm
- - -

> Tanks...
> are dangerous.
>
> Hank

["pill popper"]

He was asking about exercise to lower glucose. Your response was
irrelevant
to his situation.

[Jim Dumas]

On Sun, 11 Nov 2012 12:08:02 -0800, Henry Wannabe wrote:

> What do you think is the best exercize for lowering your glucose level?

Hi Henry,

Thought I'd chime in. When I was first Dx'd, I used exercise to control
BG without any meds. I had a cross-country ski machine in the basement
and I would use it for 30 minutes in the morning before going to work,
and then 30 minutes in the evening before dinner. I had a small TV in
the basement to distract me. This worked very well for two years in the
"honeymoon phase" of T1. (I also jogged in the summer/good weather.)

This no-meds honeymoon phase approximates the T2 physiology where the
liver dumps glucose into the bloodstream under low stress levels.
Exercise stimulates at least 4 stress hormones: adrenaline, nor-
adrenaline, cortisol and glucagon. So the trick to using exercise to
lower BG is not to stimulate these stress hormones in the workout.

As an example, if I sprint in a workout, my BG rises from these stress
hormones. So the ideal workout to lower BG is very calm, "long, slow
distance." Today, if my BG is 200-300 mg/dl, I'll dose a very small
amount of insulin (1-2U Humalog) and use this "long, slow distance"
method to keep stress hormones low. BG will drop if I do not sprint.

So the trick is stay calm, and use long, slow workouts.
--
Jim
Still kick'n! Low tech T1 4/86, no complications. T.75? 7/2011
Email mangled: change SeeSig2Fix to mindspring for utopia. (Where?)

["pill popper"]

That is a question best answered by your meter. Eat a meal and then at
about 1 hour do a set exercise for a set time and measure before and after.

I have a set of exercises using heavy hand weights. 10 minutes can chop 20
points off a peak.

When doing moderate to heavy exercise muscel cells don't need insulin to
take glucose into themselves.

["pill popper"]

"Exercise raises my BG. Insulin lowers it."

Are you type 2?

[gord...@swbell.net]

On Sun, 11 Nov 2012 21:46:17 -0600, Cindy Wells
<lcwel...@invalid.invalid> wrote:

>On 11/11/2012 4:19 PM, gord...@swbell.net wrote:
>
>> How does the enzyme amylase fit into this set of problems? Is it
>> prudent to consider taking some of those non-prescription medications
>> that are supposed to provide a boost of this enzyme.
>>
>Where did you come up with this idea?
>
>> I understand that as one ages their body produces less and less
>> amylase until finally, if there are any other contributing factors,
>> Type 2 diabetes may begin to be a problem. Will taking amylase head
>> one's Type 2 diabetes off and let them return to a diet of more
>> ordinary foods instead of scrimping on the carb intake? Gordon
>>
>
>Amylase is in saliva and in the pancreatic enzymes. It breaks down
>carbohydrates into their component sugars. (Did you ever do the "chew a
>cracker" experiment with Benedict's solution in a science class?).
>That's why here most of us find reducing carbohydrates is needed for
>control, not just the "sugars in the diet". (The traditional myth of
>type 2 diet plans is reduce sugars.) If you take amylase as a pill, you
>might slight increase in the amino acids in your diet. If you took it as
>a liquid with any carbohydrates, it would probably break down a bit more
>of the carbs.
>

Thanks, Cindy. This is pretty much in agreement with what I've been
reading on this subject. I still wonder what would be the result of
having a chroncally low level of stomach acidity. Would this cause the
amylace action to continue while the food is in the stomach, with the
end result being a high BG spike?

Amylase is one of the primary starch digesting enzymes, and it is
secreted in the saliva as well as the pancreas. The salivary amylace
starts digesting the starches in the food as it is being chewed. But,
this salivary amylace digestion process is slowed down or halted in a
normal stomach because of the stomach's acid level. The starch
digestion process has to wait until the food moves on into the
duodenum and on into the small intestine.

If my stomach acid level is very low, as my doctors have repeatedly
told me, the amylace starch digestion process may go on at a high
rate, even while the food is still in my stomach, causing a very rapid
transition from the ingested starches to the fully digested
glucose...and a high BG spike. Am I on track here, or is there
something I've overlooked? Gordon

[Cindy Wells]

On 11/12/2012 11:31 AM, gord...@swbell.net wrote:

> Thanks, Cindy. This is pretty much in agreement with what I've been
> reading on this subject. I still wonder what would be the result of
> having a chroncally low level of stomach acidity. Would this cause the
> amylace action to continue while the food is in the stomach, with the
> end result being a high BG spike?
>

How low is that acid level? Normal stomach conditions include .1M HCl
(pH ~ 2). The body produces buffering agents that keep the pH fairly
constant but it will vary depending on your recent meal. (It's not
unusual for the pH to vary from 1 to 5 depending on the meal but those
are usually temporary swings.)

The proteases in the stomach break down proteins fairly quickly in an
acid environment (pH<7). Part of the reaction releases H+ (or H3O+);
thus decreasing the pH. That will break down any other enzymes as well.
So unless your stomach pH is routinely above 6; I doubt the amylase
lasts very long after reaching the stomach.


The low-acid level is more likely to give you problems with mineral
absorption than with the amylase working longer. (Most of the minerals
we eat are in the form of salts; in the stomach, the salts dissociate
and the free ions are absorbed in the intestines.)

Cindy Wells

[outsider]

But not irrelevant to others participating in this group.

[gord...@swbell.net]

Cindy, I don't remember what my doctors found regarding my stomach
acid levels. They just told me that I had a low stomach acid level and
that was all I remember having been said about it. At that time I was
working in a clinic with a group of doctors who were researching some
medications that were intended for use as stomach acid control, if
they could be approved for such. They asked me to volunteer and in the
initial process they tested my stomach acid many times but always
reported that it was too low to give any meaningful test results for
the medications they were evaluating.

My thinking on the amylase action process that I mentioned is that
amylase is a fast acting enzyme that really gets busy breaking the
complex carbs down into simple carbs and then into glucose. This
process starts in the mouth as saliva is mixed with the food, but, in
people with normal stomach acid levels, the amylase prcess may be
severely slowed down while the food is in the stomach and the stomach
acid curtails the amylase process.

In persons such as myself, a low stomach acid level may permit the
amylase process to go ahead at its rapid rate all the way through the
stomach. If this happens, it could result in a significant BG spike
while the food is still in the stomach. I usually see a BG spike that
peaks around 75 minuters after I begin a meal. I would think that most
of the food I've just eaten would still be in my stomach at this time.

People with a normal stomach acid level might not get this rapid
movement of glucose into their blood while the food is still in the
stomach, and later on as the food moves into the small intestins the
process may not be nearly as robust, resulting in a normal, slow
movement of glucose into the blood.

Am I anywhere near right in this school of thought?

Gordon

[terryc]

On 13/11/12 11:01, gord...@swbell.net wrote:

> In persons such as myself, a low stomach acid level may permit the
> amylase process to go ahead at its rapid rate all the way through the
> stomach. If this happens, it could result in a significant BG spike
> while the food is still in the stomach. I usually see a BG spike that
> peaks around 75 minuters after I begin a meal. I would think that most
> of the food I've just eaten would still be in my stomach at this time.
>
> People with a normal stomach acid level might not get this rapid
> movement of glucose into their blood while the food is still in the
> stomach, and later on as the food moves into the small intestins the
> process may not be nearly as robust, resulting in a normal, slow
> movement of glucose into the blood.
>
> Am I anywhere near right in this school of thought?

Timewise you might be right. I often ceck out the time response graphs
of the various insulins avaialble. the mixed stuff here is timed to peak
at two hours which is when my quack says that the first effect of "food"
will be seen in the blood stream.

[Cindy Wells]

On 11/12/2012 6:01 PM, gord...@swbell.net wrote:

> In persons such as myself, a low stomach acid level may permit the
> amylase process to go ahead at its rapid rate all the way through the
> stomach. If this happens, it could result in a significant BG spike
> while the food is still in the stomach. I usually see a BG spike that
> peaks around 75 minuters after I begin a meal. I would think that most
> of the food I've just eaten would still be in my stomach at this time.
>
>

I don't know enough about the amylase breakdown but I worked on a diet
menu that avoids spikes in the first place. Based on my meter readings,
simple carbohydrates that didn't need amylase to breakdown and crackers
spiked at nearly the same time for me. However, I didn't do many tests
without proteins and fats in the meal so I may not have enough data.

Cindy Wells

[RandyF]

Here's two recent papers on the issue.

Moderate walking after meals is best for immediate lowering of glucose
levels.
Exercise on an empty stomach has other benefits, but if after-meal bg
levels are your concern - moderate exercise shortly after after is the
way to go.

Randy

//*************
1.
The Effect of Walking on Postprandial Glycemic Excursion in Patients
With Type 1 Diabetes and Healthy People.
Manohar C, Levine JA, Nandy DK, Saad A, Man CD, McCrady-Spitzer SK,
Basu R, Cobelli C, Carter RE, Basu A, Kudva YC.

Source
Center for Clinical Investigation, Case Medical School, Case Western
Reserve University, Cleveland, Ohio.

Abstract
OBJECTIVEPhysical activity (PA), even at low intensity, promotes
health and improves hyperglycemia. However, the effect of low-
intensity PA captured with accelerometery on glucose variability in
healthy individuals and type 1diabetes has not been examined.
Quantifying the effects of PA on glycemic variability would improve
artificial endocrine pancreas (AEP) algorithms.RESEARCH DESIGN AND

METHODSWe studied 12 healthy control subjects (five males, 37.7 ± 13.7
years of age) and 12 patients with type 1 diabetes (five males, 37.4 ±
14.2 years of age) for 88 h. Participants performed PA approximating a
threefold increase over their basal metabolic rate. PA was captured
using a PA-monitoring system, and interstitial fluid glucose
concentrations were captured with continuous glucose monitors. In
random order, one meal per day was followed by inactivity, and the
other meals were followed by walking. Glucose and PA data for a total
of 216 meals were analyzed from 30 min prior to meal ingestion to 270
min postmeal.

RESULTSIn healthy subjects, the incremental glucose area under the
curve was 4.5 mmol/L/270 min for meals followed by walking, whereas it
was 9.6 mmol/L/270 min (P = 0.022) for meals followed by inactivity.
The corresponding glucose excursions for those with type 1 diabetes
were 7.5 mmol/L/270 min and 18.4 mmol/L/270 min, respectively (P <
0.001).CONCLUSIONSWalking significantly impacts postprandial glucose
excursions in healthy populations and in those with type 1 diabetes.
AEP algorithms incorporating PA may enhance tight glycemic control end
points.

//***********************
2.
Effects of post-absorptive and postprandial exercise on
glucoregulation in metabolic syndrome.
Derave W, Mertens A, Muls E, Pardaens K, Hespel P.

Source
Research Center for Exercise and Health, Faculty of Kinesiology and
Rehabilitation Sciences, K.U. Leuven, Tervuursevest 101, B-3001
Leuven, Belgium.
Abstract

OBJECTIVE:
The aim of this study was to investigate the effects of an acute
exercise bout in the morning in the post-absorptive or postprandial
state on the glycemic and insulinemic response to three standardized
meals throughout the day. It is hypothesized that post-absorptive
exercise enhances fat oxidation rate during exercise and thereafter
attenuates the glucose and insulin response to subsequent meals.

RESEARCH METHODS AND PROCEDURES:
Seven sedentary males with metabolic syndrome (age, 45 +/- 11 years;
BMI, 34 +/- 3 kg/m2) were studied in a crossover design comparing
three conditions: no exercise, postprandial and post-absorptive
exercise (at approximately 60% of the individual VO2max for 45
minutes). Substrate use was evaluated by indirect calorimetry during
exercise. Venous blood samples were taken at regular (30- to 60-
minute) intervals throughout the day, and glucose, insulin, and
triglyceride concentrations were determined.

RESULTS:
During exercise, a higher fat oxidation rate was observed in the post-
absorptive than the postprandial state. The glycemic response to a
standardized high-carbohydrate breakfast was lower when exercising
after breakfast than when exercising before breakfast. There was no
effect of either exercise mode on glucose and insulin response to
lunch and supper.

DISCUSSION:
Post-absorptive exercise has the advantage of promoting fat use,
whereas postprandial exercise can attenuate the glycemic response to
breakfast. Neither exercise mode acutely induces improved
glucoregulation later during the day. The impact of meal timing on the
effects of regular exercise training on glycemic control in this
population remains to be studied.

PMID: 17372321 [PubMed - indexed for MEDLINE] Free full text

[W. Baker]

terryc <newsnine...@woa.com.au> wrote:

what? 2 hours is when most of the high bg should be over adn the bg is
returning toward normal, definitely not the first effect! Take some fast
acting carb like orange juice adn test 15-30 mins later and see what
happens to the bg. that is the standart time for seeign if a hypo is is
beginning to pass and the bg is risingas a result of eating a quick carb
like OJ.

Wendy

[Pro-Humanist FREELOVER]

>"pill popper" wrote:

>> He was asking about exercise to lower glucose. Your response was irrelevant
>> to his situation.

"outsider" responded:

> But not irrelevant to others participating in this group.

- - -

Coincidentally, on the very day of this post,
I went to work and neglected to take my
insulin pen which has Novolog with me. I
glucose tested at my first break, and was
at 202, unusual in that I'm usually battling
a level in the 40s or 50s at that time of day.

Normally, I'd give a booster shot of 3 units
of Novolog to get the glucose down, but
since I didn't have my insulin pen, I em-
barked on trying to increase my exercise
level.

Thankfully, by lifting heavy bags and doing
other tasks, my glucose dropped to 126 prior
to lunch (an hour an a half later), and I went
home for lunch. Glucose was within normal
ranges until at bedtime, it was once again
above 200, to which I responded with a
higher than normal Novolog and a normal
Levemir bedtime dosage.

This morning, awoke with a glucose level
of 20 (it was 134 an hour after giving my
Novolog at bedtime, and I ate a diet choc-
alate - 8 carbs, hoping that would counter
the 'too low' risk without risking the glucose
going 'too high', but apparently, that was
not enough), and I don't plan on doing much
in the way of exercise today (an 'off' day),
so we'll see how things work out on this, my
(well, it's been since March of 1961 that I've
been forced into doing this to stay alive, so)
18,866th (or thereabouts) day and night of
dealing with this damnable disease.

[Julie Bove]

<pill popper> wrote in message
news:50a11f56$0$12888$1c46...@news.club.cc.cmu.edu...

>
>
> He was asking about exercise to lower glucose. Your response was
> irrelevant
> to his situation

Of course it was!

[Julie Bove]

<pill popper> wrote in message

news:50a124ed$0$12888$1c46...@news.club.cc.cmu.edu...

>
> "Exercise raises my BG. Insulin lowers it."
>
> Are you type 2?

Yes.

[terryc]

On 14/11/12 01:30, W. Baker wrote:

> what? 2 hours is when most of the high bg should be over adn the bg is
> returning toward normal, definitely not the first effect! Take some fast
> acting carb like orange juice adn test 15-30 mins later and see what
> happens to the bg. that is the standart time for seeign if a hypo is is
> beginning to pass and the bg is risingas a result of eating a quick carb
> like OJ.

Please revise food knowledge, specifically sugars, starches, and
carbohydrates. Each has differing effects and response times.

[Pro-Humanist FREELOVER]

> <pill popper> wrote ...

>> He was asking about exercise to lower glucose.

Actually, the title states "What do you do to lower
your glucose level", so pill popper was incorrect
in his reference to only a part of Henry's post
which pertained to a question about exercise,
ignoring the title which opened the appropriate
response to include *all* and *any* methods
used to lower glucose levels, the most import-
ant, of course, for those with Insulinitis (old
name: type 1 diabetes) being insulin injections
or insulin pumped in for those who wear an
insulin pump.

[terryc]

On 14/11/12 02:42, Susan wrote:
> x-no-archive: yes

>
> On 11/13/2012 10:38 AM, terryc wrote:
>
>> Please revise food knowledge, specifically sugars, starches, and
>> carbohydrates. Each has differing effects and response times.
>>
>>
>>
>

> Sugars, starches, vegetables are all carbohydrates,

Blink, not when I went to school. Shrug.
and the fourth is cellulose,

Hint, form and availability.

[Henry Wannabe]

I'm sorry to all for the confusion. I'll try to be more specific next time.

Hank

[terryc]

On 14/11/12 03:24, Susan wrote:
> x-no-archive: yes
>

> On 11/13/2012 11:06 AM, terryc wrote:
>
>> Blink, not when I went to school. Shrug.
>> and the fourth is cellulose,
>>
>> Hint, form and availability.
>

> Hint: they're all carbohyrates and each person responds to them
> differently.
>
> Complexity makes no difference in glucose response to foods.

Think about it.

["pill popper"]

> He was asking about exercise to lower glucose. Your response was
> irrelevant

> to his situation.

"
Actually, the title states "What do you do to lower
your glucose level", so pill popper was incorrect
in his reference to only a part of Henry's post
which pertained to a question about exercise,"

He is type 2. Your then typical narcissistic wanderings into type 1
experiences was irrelevant. Most of it did not pertain to excersize and
that only about your problems with the roller coaster effect which don't
apply to him.

[Pro-Humanist FREELOVER]

<pill popper> wrote ...

> "Actually, the title states "What do you do to lower
> your glucose level", so pill popper was incorrect
> in his reference to only a part of Henry's post
> which pertained to a question about exercise,"
>

> He is type 2. [...]

Well, as you well know, 25% or so of those
with Cellosis (old name: type 2 diabetes)
include insulin injections in their treatment
in America (unknown, the percentages in
areas outside of America), and insofar as
Insulinitis (old name: type 1 diabetes) is of
interest to those who have Cellosis, I suspect
the post was of value even to those who have
Cellosis, as well as those who have other glu-
cose anomalies, especially Insulinitis, and to
everyone interested in the disparate nature
of the various treatments to deal with the
(in many critical areas, not all) disparate
glucose anomalies.

["pill [popper"]

> "Actually, the title states "What do you do to lower
> your glucose level", so pill popper was incorrect
> in his reference to only a part of Henry's post
> which pertained to a question about exercise,"
>
> He is type 2. [...]

"Well, as you well know, 25% or so of those with Cellosis (old name: type 2
diabetes) include insulin injections in their treatment"

He has posted he uses metformin.


And the narcissistic self indulgence went on and on with a bit about your
roller coaster caused exercise responses thrown in as almost an
afterthought.

And in this post you continued, snipped.

It ain't about you.

[Pro-Humanist FREELOVER]

<pill [popper> wrote ...

>> "Actually, the title states "What do you do to lower
>> your glucose level", so pill popper was incorrect
>> in his reference to only a part of Henry's post
>> which pertained to a question about exercise,"
>>
>> He is type 2. [...]

> "Well, as you well know, 25% or so of those with
> Cellosis (old name: type 2 diabetes) include insulin
> injections in their treatment"
>
> He has posted he uses metformin.
>

> [...]

Both Henry's Cellosis -and- his metformin
usage were absent from his initial post,
and the response was, as previously noted,
pertinent to the title.

I suspect your stance revolves around
your dislike of using new names for the
various glucose anomalies, and not on
the details of using insulin to deal with
lowering glucose levels, said details of
interest to anyone who currently uses
insulin, and that applies to everyone who
has Insulinitis, as well as to the already
mentioned individuals who don't have
Insulinitis.

["pill popper"]

And the narcissistic self indulgence continues apace.

[Pro-Humanist FREELOVER]

- - -

So, thus far, in this thread, variations in
dealing with glucose have been discussed,
with issues pertaining to type of glucose
condition, whether or not insulin injections
or an insulin pump are used (required for
Insulinitis, old name: type 1 diabetes, some
form of exogenous insulin administration to
deal with the total or near-total loss of endo-
genous insulin production, but only used by
a minority of those with Cellosis, old name:
type 2 diabetes, as the insulin production
of those with Cellosis continues and is oft-
times adequate enough to not require sup-
plementation with insulin injections, initially,
and it's unclear the degree to which insulin
injections become a part of the treatment
profile of some or most with Cellosis as time
passes).

Also discussed by some, impacts of ingestion
of food and drink, and impacts of exercise.
As for posting about the impacts of other
medications, or the impacts of stress, or the
impacts of other medical conditions, or the
impacts of drugs (legal or illegal) or alcohol,
all of which can impact glucose levels in vary-
ing degrees, those topics given less attention
relative to their large potential import regard-
ing glucose levels (noting, of course, the anec-
dotal reports of impacts often vary from person
to person, and of course, impacts of everything
are individually variable, with metabolic conse-
quences both unique to each individual and
condition and unique to the happenstance of
metabolic variability which transpires in an oft-
times difficult to predict manner within each
individual, especially in those whose insulin
production is totally or near-totally lost, i.e.,
in those who have Insulinitis).

["pill popper"]

Thus far we see continued narcissistic indulgence far afield from exercise
and a recitation of opinions lame and long ago ignored.

[Pro-Humanist FREELOVER]

- - -

What does pill popper do to lower his
glucose level? Well, since pill popper
rarely (if ever) discusses what High
Glucose Condition he has, or how he
deals with it (though he promotes met-
formin, for everyone with any High
Glucose Condition), it's mysterious,
although for some reason, he seems
fascinated in this thread with Henry's
question about exercise.

Since pill popper acts as if his way of
dealing with his unknown High Glucose
Condition yields a propensity to assert
that all with any High Glucose Condition
can (with enough effort and willpower
and the 'right' approach) have 'control',
something that people with Insulinitis,
old name: type 1 diabetes, every last
one of us, lost, totally, when we came
down with Insulinitis, our pancreases,
you see, having totally or near-totally
lost their ability to produce insulin, oft-
times at a very young age (myself, diag-
nosed at the age of 5), and along with
that, having dysfunctional responses to
low glucose that goes hand-in-hand with
having Insulinitis.

What do we with Insulinitis rely on to
lower our glucose levels, in 100% of
the cases? Insulin guessing, either in-
jected or pumped in, with other meta-
bolic activity interacting with those
insulin guesses, and unpredictable
variability in metabolic activity making
those guesses yield naught but a pro-
babilistic outcome, sometimes too
high, sometimes too low, sometimes
so low that our lives are placed at risk.

Lowering glucose levels?

Always, 100% of the time, with every
insulin injection or insulin pumping deci-
sion, with every food/drink intake deci-
sion, with every last bit of exercise (the
more, the riskier), with every other con-
dition / disease that we with Insulinitis
have, with every unknown or guessed
at impact on metabolic activity drug /
medication we take, with every increase
in stress, with every unknown and un-
knowable metabolic activity that trans-
pires in our bodies, the degree of severe
low glucose risk increases as the degree
of lowering glucose levels increases.

["pill popper"]

What is the benefit for a type 1 or any diabetic to exercise?

Aside from the obvious health benefits all share, it is one way to level
off glucose peaks. To do so helps with the roller coaster effect from
which some type 1's suffer because of large carb intake being chased by
large insulin intake. For others it does the same post meals generally.

During moderate to high levels of physical activity muscle cells don't
require insulin to uptake glucose from the blood. It is this effect which
knocks off the peaks post meals and during liver glucose releases that has
benefit for a diabetic.

One's meter will show how much this effect happens with which level of
physical activity and to know when peaks happen. This can be done in place
of using more insulin and promoting the roller coaster effect. Because it
lowers glucose taking into account
of how much exercise with what glucose level before and after should be
considered.

This effect works for all types of diabetes.

[Charly Coughran]

"pill popper" wrote in
news:50a3ed28$0$12891$1c46...@news.club.cc.cmu.edu:

>
> During moderate to high levels of physical activity muscle
> cells don't require insulin to uptake glucose from the
> blood. It is this effect which knocks off the peaks post
> meals and during liver glucose releases that has benefit
> for a diabetic.

Be a little careful here. While it is true that moderate levels
of exercise open non-insulin mediated pathways for glucose entry
into skeletal and muscle cells which result in an effective
reduction of insulin resistance, it is not the case that these
pathways account for all the glucose transfer into cells during
exercise. Insulin still accounts for a major proportion of the
glucose transfer.

--
______________________
ccou...@DELETE-TO-RESPOND-UCSD.EDU

["pill popper"]

> During moderate to high levels of physical activity muscle
> cells don't require insulin to uptake glucose from the
> blood. It is this effect which knocks off the peaks post
> meals and during liver glucose releases that has benefit
> for a diabetic.

"Be a little careful here. While it is true that moderate levels of
exercise open non-insulin mediated pathways for glucose entry into skeletal
and muscle cells which result in an effective reduction of insulin
resistance, it is not the case that these pathways account for all the
glucose transfer into cells during exercise. Insulin still accounts for a
major proportion of the glucose transfer."

Yes, but enough to have a significant impact on glucose levels. A simple
before and after meter read will show this easily, it will knock the peak
off a post meal glucose level.

This happens regardless of insulin resistance, even in non-diabetic people.

It is not intended to be the entire treatment approach but one added to
balancing the carb intake with available insulin.

[Charly Coughran]

"pill popper" wrote in
news:50a509be$0$12894$1c46...@news.club.cc.cmu.edu:

You are absolutely correct and I suspected that you understood
the nuance. However, your original statement says that cells
don't require insulin to uptake glucose during exercise. That
is a sufficient oversimplfication to make the statement
incorrect. Thus my comment to be a little careful, although I
probably should have said, "be a little careful with your
wording", rather than assuming that constraint as implied.

Answering questions about diabetes without oversimplifing to
misleading or making the answers unintelligable as by talking at
the level of the GLUT4 mediated pathway vs. the insulin mediated
pathway is hard. It takes significant effort to produce clear,
understandable, and useful answer to any question about
diabetes. If you had replaced the single paragraph I quoted
with the three in your response to me, you would have produced
an answer which would have stood out as a prime example of how
questions should be answered in this forum.


--
______________________
ccou...@DELETE-TO-RESPOND-UCSD.EDU