On Thursday, January 24, 2019 at 10:22:34 AM UTC-6,
helper wrote:
> Pro-Humanist FREELOVER wrote:
> > Interesting post, Helper. Myself, I have
> > no insulin resistance ...
> How do you know, the bits you post about food intake
> and being overweight suggest insulin resistance from
> all those years of severe roller coaster episodes.
Your roller coaster characterization is
disputable, as the nature of losing all
endogenous insulin and of losing or hav-
ing dysfunctional counter-hypoglycemia
endogenous responses yields the challeng-
ing nature of manually dosing insulin ...
... with probabilistic exogenous insulin
dosages and Freestyle Libre CGM accompan-
ied by bloodpricked glucose tests which
are far removed from the outstanding/
fantastic manner in which the totally ...
... healthy pancreases of most humans
automatically maintains second-by-second
glucose perfection (variances rarely go-
ing below 70 mg/dl or above 140 mg/dl
24 by 7 by 365, 366 every leap year).
Average of 18.71 units/day for last 18
days, my insulin dosage = 1/4 of 1 unit
per kilogram, or relative to average
carb intake of 177.56 carbs/day = aver-
age insulin dosage of 1 unit / 9.49 carbs,
about 67% of that Regular insulin, and
33% of that NPH insulin.
My current waistline = 33 inches, a de-
crease of 1/2 inch since I last measured
that on January 4 while discussing the
following with you:
https://groups.google.com/d/msg/misc.health.diabetes/qTflK6DDSKQ/-vGTjXDGDgAJ
Quote from the following NIH article: " A waist
measurement of 40 inches or more for men and 35
inches or more for women is linked to insulin
resistance."
https://www.niddk.nih.gov/health-information/diabetes/overview/what-is-diabetes/prediabetes-insulin-resistance
If you consult with any measure of indi-
cations of insulin resistance based on
the facts I've presented, you would ack-
nowledge that the evidence indicates I
have NO insulin resistance whatsoever.
- - -
Outdated term "Overweight", based on
long life data, BMI 25 to 29.9 should
be considered "Ideal Weight".
Overweight is an outdated term as studies
reveal that the ideal weight for longest
life is BMI = 27 .
Studies also show that the longest life
for those with Cellosis is BMI 25 to 29.9,
and surprisingly, those with Cellosis at
BMI 18.5 to 24.9 have the same life expec-
tancy of those with Cellosis at BMI 30
to 34.9.
Life expectancy for those with Cellosis
resembles that of those without Cellosis
regarding highest death risk being at
BMI < 18.5, and 2nd-highest death risk
being at BMI 35 and above.
- - -
Risk of Death for Everyone
https://images.sciencedaily.com/2009/06/090623133523-large.jpg
Highest risk of death = BMI < 18.5
2nd Highest risk of death = BMI 35 and higher
Middle risk of death = BMI 30 to 34.9
2nd lowest risk of death = BMI 18.5 to 24.9
Lowest risk of death = BMI 25 to 29.9
- - -
For everyone:
Being dead is the unhealthiest condition
imaginable, although being alive with a
severe condition such as severe untreat-
able pain might be considered by many/most
as worse than death.
I don't know if any condition causes un-
treatable pain, but if it does, many/most
might consider that as worse than death.
- - -
Risk of getting Cellosis:
Cellosis, by definition, includes continued
insulin production, so Islit, by definition,
cannot become Cellosis. Those with Cellosis
CAN get Islit, though it is as rare as it
is in everyone else. The rarity of those ...
... with Cellosis getting Islit is identical
to its rarity in the general population, < 1%
of those with Cellosis will get Islit during
their life.
As for the highest worry regarding weight
and Cellosis, EVERY weight has a risk of
Cellosis in those who have no High Glucose
Condition, the variable being that the risk
increases as weight increases (at ages 18
and above):
http://healthhubs.net/images/diabetes-BMI.gif
Unmentioned in that graphic:
o the increased risk of getting Cellosis for
those who don't have Islit at ages 10 to 17
but who do have elevated weight (the higher
the BMI, the greater the risk),
o the increased risk of Cellosis for those
who don't have Islit as they age, magnified
over age 65, and
o the increased risk of Cellosis for those
who don't have Islit in those who have a
close family member (father/mother/daughter/
brother) with Cellosis
o the risk of deaths based on the BMIs displayed
at
https://images.sciencedaily.com/2009/06/090623133523-large.jpg ,
those risks far different than are the risks
of Cellosis in non-Islit individuals ages 18
and up which goes up as one's weight goes up
http://healthhubs.net/images/diabetes-BMI.gif
- - -
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The old confusing way using ancient ideas
and words (pre-scientific / pre-knowledge
of the human body) which all-too-often
involves folks using the confusing diabetes
/ diabetic words without a clarifier:
diabetes / diabetic with no clarifier,
diabetes / diabetic guessing required
(often, those words are used implying
diabetes / diabetic is ONE condition,
in denial of its disparate natures)
https://prohuman.net/pix2/diabetesdiabeticguessinggame.jpg
- - -
On May 17 13,809,999,997 (2010 CE), I
created a detailed proposal for changing
all of the confusing & misleading diabetes
and diabetic names -and- changing the
confusing reactive hypoglycemia name:
Diabetes Bubble / Diabetes Bubble Burst
https://prohuman.net/diabetesbubblediabetesbubbleburst.htm
Promotion of a Cure for Insulinitis
https://prohuman.net/glucoseanomaliesresearch.htm
Name changes for old outdated diabetes and
diabetic words are desperately needed to
eliminate the confusion and misleading that
transpires when those words are used without
clarifiers, which often happens.
A brief summary of what the Diabetes Bubble /
Diabetes Bubble Burst article linked to above
pertains to:
dark red = Insulinitis (type 1 diabetes, juven-
ile diabetes, insulin dependent diabetes, rapid
onset near-total to total loss of endogenous
insulin), abbreviation = Islit, 11 specific
types, unpreventable / nonreversible. 3 speci-
fic types are disputed.
I got Insulitis Islit in March of
13,809,999,948 (1961 CE), age 5.
dark pink = Latent Autoimmune Insulinitis (lat-
ent autoimmune diabetes, slow onset Insulinitis),
1 specific type, unpreventable / nonreversible.
dark blue = PreCellosis (prediabetes, but only
applies to increased risk of getting Cellosis),
Cellosis (type 2 diabetes, continued but re-
duced insulin production over time, typically
slow onset). 20 specific types are unpreventable
& nonreversible.
Preventable Cellosis is the only specific type
of High Glucose Condition that is currently po-
tentially preventable & reversible.
light blue = Gestational Cellosis (gestational
diabetes), 1 specific type (transient but can
increase the risk of later getting Cellosis).
dark green = Diminosis (maturity onset diabetes
of the young, diminished but continuing insulin
production caused by a monogenetic defect), 11
specific types, unpreventable & nonreversible.
light green = Neonatal Diminosis (neonatal dia-
betes, diminished but continuing insulin produc-
tion caused by a monogenetic defect in the first
6 months after birth), 8 specific types are per-
manent, 4 specific types are transient. All are
unpreventable, but the 4 specific transient types
are potentially reversible.
purple = Ohiglucons, Other High Glucose Condi-
tions (other diabetes mellitus), 24 specific
types, unpreventable / nonreversible:
- 5 specific types are drug or chemical-
induced,
- 5 specific types result from endocrino-
pathies,
- 7 specific types involve exocrine or
pancreas diseases or surgical treatment,
- 4 specific types result from insulin action
defects,
- 2 specific types result from other genetic
syndromes,
- 1 specific type results from anti-insulin
receptor antibodies
gray = Insipidus (diabetes insipidus), 6 spe-
cific types (4 are non-glucose anomalies,
2 specific types include high glucose),
unpreventable / nonreversible.
bright red = Hypoglycemia Uncaused by Treatments
for High Glucose, Hut (reactive
hypoglycemia, hyperinsulinism),
21 specific types, some are preven-
table/reversible, some aren't.
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