I agree with Ian that if you expect a background check it makes sense to send the information proactively, explaining you used the Major GPA and wanted for full transparency show them also the total GPA.
Are they doing a background check? If so, I recommend you take a pro-active approach. They will almost certainly check your GPA. Flag it as you having put a preminilary calculation prior to being informed of the true GPA.
The thing is that the transcript will likely be asked. So my advice is to be transparent with them, because lying would not get you anywhere. And considering the fact that you are worried so much, this could help.
Hi Gaurav, thanks for your answer! To clarify, are you saying I should point out the discrepancy if they request my transcript? I'm planning on explaining this honest mistake whether I take initiative or if they come to me about it. Thanks again!
You should be fine and wont be penalised for this. Provide the transcripts and if they ask during the checks just explain like you have done in the question you posted. Apologise for the error. No need to do anything proactively at this stage.
I hope you have learnt a lesson. In future avoid any such irregularities. Your integrity, reputation and personal brand will play a BIG part in your success in Consulting. Hold yourself to the highest standards please. Its easy to get carried away and stretch the facts here and there but if you get caught out, the damage can be severe.
Perhaps an unpopular opinion, but I have never heard of quoted GPAs for purposes of employment selection being some subset of a cumulative GPA. I have seen and participated in many cases of CV reviews of undergrads at 2 firms, and while this was several years ago, I never came across this distinction once. So, on the slim chance it is noted, it would almost certainly come off as a deception.
That said, the chance that your stated GPA will be compared to your documented GPA does not appear to be great. I have less visibility into the background check process, but from my limited experiences when internal HR teams were conducting their document checks (distinct from an external background check), they weren't checking for comparisons; rather, they were checking that minimum hurdles had been scaled. For example, if the role called for undergrads from Princeton University with GPAs of 3.25 of better, graduated in the last 12 months, et cetera, et cetera; then if you reported 3.77 (but actually had 3.44), reported graduation in May (but really graduated in June), and so on, you would have scaled the minimum hurdles and despite the discrepancies, would be fine.
I'm torn on what to advise you to do and the wide dispersion in the previous comments (not something that occurs frequently on this forum which should tell you something) should underline how murky this is:
+ On the on hand, keeping silent feels best if you believe this is merely a credentials check and not a fact-finding mission. Was a minimum GPA specified when you applied? Is it above your cumulative GPA?
+ On the other hand, though, if this is an application check (where information you have submitted, say, on an online form) where checked, any recruiter would find the sizeable discrepancy rather difficult to explain. Yes, in such a case, proactive disclosure (as someone - or a few people, actually - have mentioned), but you would really need to have an airtight explanation (I'm personally not swayed by the one you claim) AND a credible composure to make it work.
Perhaps you need to find out what checks are being expected and proceed from there. Once can only hope that the required GPA is below your final cumulative because if it is not, it could cause rather sticky situations in the future.
I am a California native through and through. I am also a first generation Mexican- American queer Latina and first generation college and medicine graduate. Professionally I am interested in creating a safe space that embraces the diversity of our healthcare workers and the communities we serve. I am working to create a community for LGTBQIA+ identifying GME staff through events, curriculum, and mentoring. If you are also interested in this, please let me know! We are looking to build our community further and would love to have you involved. This year I will be applying into gastroenterology. Outside of the hospital I love to go on nature adventures with my dog, binge watch shows/movies, read queer romance novels, explore new speakeasies, and find new hobbies to enjoy!
I am a Southern California native, grew up in Monterey Park, and stuck around Los Angeles for undergrad at UCLA (still a huge Bruins fan!). Thereafter I worked for a few years in LA as a medical scribe, started dating one of my best friends in high school (now wife!!), and checked off some bucket list items (saw the Northern lights in Alaska, cross-country road tripped, ran a marathon, coached a high school basketball team). I stayed in LA for medical school, but at the rival institution (USC). In medical school, I realized that I loved caring for both adults and children, so I applied to Med-Peds! I did an away elective as a 4th year med student here at UCSD and quickly realized that this would be the perfect place to learn and grow as a resident physician! In terms of career aspirations, I plan on going into adult GI and have an interest in Inflammatory Bowel Disease. In my free time, I love to travel, find epic sights/views, catching as many sunsets as I can here in SD, spending quality time with my now family of 3, playing golf, and trying all the amazing food in San Diego.
I grew up in Marin County (just north of San Francisco, across the Golden Gate Bridge) and went to UC Berkeley for undergrad. I went to medical school at USC in Los Angeles and continued the journey south to San Diego for residency. I plan to pursue a career in Cardiology, ideally focused in advanced heart failure and cardiac transplantation. Outside of the hospital, I love to practice yoga, go on hikes, spend time at the beach, and hang out with my incredible friends.
I grew up in Aurora, IL and moved to Chicago, IL for undergrad and medical school where I had an amazing time. I moved to San Diego, CA with my now husband and we have made this our new home with new friends and exciting experiences. I started out interested in Infectious disease and Endocrinology and I'm finishing residency hoping to go into Pulm Crit! For fun I like to run along the beach, paddleboard, roller skate, yoga, binge watch TV, and find new bars/breweries with my husband.
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Using online surveys, we collected data regarding COVID-19-related loss of smell or taste from 69,841 individuals. We performed a multi-ancestry genome-wide association study and identified a genome-wide significant locus in the vicinity of the UGT2A1 and UGT2A2 genes. Both genes are expressed in the olfactory epithelium and play a role in metabolizing odorants. These findings provide a genetic link to the biological mechanisms underlying COVID-19-related loss of smell or taste.
Loss of sense of smell (anosmia) or taste (ageusia) are distinctive symptoms of COVID-19 and are among the earliest and most often reported indicators of the acute phase of SARS-CoV-2 infection. It is notable from other viral symptoms in its sudden onset and the absence of mucosal blockage1. While a large fraction of COVID-19 patients report loss of smell or taste, the underlying mechanism is unclear2. In this study, we conducted a genome-wide association study (GWAS) of COVID-19-related loss of smell or taste, having collected self-reported data from over 1 million 23andMe research participants as described previously3. By asking study participants to report the symptoms they encountered during their COVID-19 experience, we identified SARS-CoV-2 test-positive individuals who reported a loss of smell or taste and contrasted them with test-positive individuals who did not report a loss of smell or taste.
While mechanistic explanations have been proposed7 for COVID-19-related loss of smell, experimental studies suggest that loss of smell is related to damage to the cilia and olfactory epithelium but not infection of the olfactory neurons. For example, in an experiment where hamsters were nasally infected with SARS-CoV-2, the olfactory epithelium and cilia became very damaged, which can completely inhibit the ability to smell, but no infection was observed in the olfactory neurons8. Recent evidence suggests that SARS-CoV-2 enters and accumulates in olfactory support cells, specifically, sustentacular cells, which unlike olfactory neurons abundantly express the viral entry proteins angiotensin-converting enzyme 2 (ACE2) and transmembrane protease serine 2 (TMPRSS2; refs. 9,10). These support cells are metabolically and functionally associated with olfactory neurons and with odorant signal transduction (processing odorants by endocytosing the odorant-binding protein complex, detoxifying, maintaining the cilia of mature olfactory receptor neurons and maintaining epithelial integrity). It has been proposed that olfactory sensation is impaired when these essential functions are disrupted, causing ciliary impairment7. How UGT2A1 and UGT2A2 are involved in this process is unclear but given their localization and essential function in metabolizing and detoxifying such compounds, these genes may play a role in the physiology of infected cells and the resulting functional impairment that contributes to loss of ability to smell. Notably, the variant identified in this study also appears to be associated with general ability to smell, which may suggest that those with heightened smell or taste sensitivity may be more prone to notice a loss of these senses resulting from a SARS-CoV-2 infection.
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