Mass Effect 1 - 2 - 3 Extras Only Update
Giorgina Calvello
In other words, if only the game's biggest fans would care about this, then BioWare is effectively punishing them by charging more for content only they would care about. One can quickly begin to see how fans might feel screwed over. Especially the game's "fanatics."
Site not working? Let me know @annakie. Thanks!Announcements Bioware --> Mass Effect Legendary Edition --> Save --> ME1" folder. Please email me your Char##.pcsav files at me2s...@gmail.com with a description of your Shepard (Gender, History, Level, Romance, Virmire Survivor, Councillor, Wrex Alive, Paragon/Renegade, if you did most quests, anything else important) or tweet me @annakie if you can help. Thanks!**5/14/2021 LEGENDARY RELEASE DAY***
Happy MELE release day everyone! I'm sure you're wondering how Mass Effect Saves work with MELE, and, well, there's good news and bad news.First, the bad news, and it's pretty bad for now. Original ME1 (OME1) save files used a format called .MassEffectSave, and there were also profile files called .MassEffectProfile.OME2 and OME3 used a format called .pcsav.When you imported a character from OME1 into OME2, something behind the scenes did the work to look for and convert those .MassEffectSave files to read it and start ME2 as .pcsav files.Well, MELE1 just... uses .pcsav files. I've done a little testing and as far as I can tell, MELE2 won't recognize those .MassEffectSave files to import to MELE2. I also tried just changing the filenames to see if that would work by some miracle, but no, it does not.The good news is that... from the limited amount of testing done so far, ANY OME2 save file should import just fine to MELE3, so masseffect2saves.com is still 100% useful. Also your old OME2 and OME3 files should open and work just fine in MELE2 and MELE3, respectively. I've tested this some and everything feels good, my Shepard just looked a little weird.However, unfortunately, that means the entirety of the masseffectsaves.com ME1 library doesn't work for importing to MELE2 right now.There is sort of a work around which, in theory, should work. If you import a OME1 file into OME2, then after waking up on the Cerberus base, exit the game, open that new .pcsav file in Gibbed's, and flag it as a completed save, then save it as a new save... that save should, in theory, open in MELE2 if you start it as a NG+ import. I don't know if that works yet, I'll have to test.Or, just grab a OME2 save file from masseffect2saves.com and start that as a NG+ in MELE2.**3/31/2020** Small Downtime!
Hey everyone, my hosting provider upgraded/moved some stuff around on the backend of the site, and we had to change DNS servers, so you may have experienced some short downtime while the DNS updated. If you're reading this, it should be all over now.It's also come to my attention that Shepard submitting is broken. We're not sure why. It's being looked at, may continue to be broken for a while. I'll post again when it's not! Thanks to ThinkingNessaja and TheLone_SheWolf for letting me know!
Data sources: Medline, Embase and CINAHL databases were searched and the following outcomes analyzed: associated central nervous system (CNS) and extra-CNS anomalies detected at the scan, chromosomal anomalies, additional CNS anomalies detected only at prenatal MRI, additional CNS anomalies detected only after birth, cyst progression in utero, neurological outcome and need for surgery. Two authors reviewed all abstracts independently. Results were reported as proportions, and between-study heterogeneity was explored using the I statistic; fixed or random effect models were used accordingly.
Results: Ten studies involving 47 fetuses were included in the meta-analysis. Arachnoid cysts (n = 24) had associated CNS anomalies and extra-CNS in 73% (95% CI 56-88) and 14% (95% CI 4-29), respectively. The most common associated anomalies were ventriculomegaly and callosal abnormalities. Chromosomal abnormalities were present in 6% (95% CI 0-30), but fetuses with isolated cysts were always euploid (0/7; 95% CI 0-29). Fetal MRI and postnatal examination identified 5 additional cases (21%, 95% CI 1-57). Cavum veli interpositi (CVI) cysts had associated CNS and extra-CNS anomalies in 31% (95% CI 13-52) and 6% (95% CI 0-29), respectively. No chromosomal or callosal anomalies were found in these cases. In isolated CVI cysts, no cases of associated anomalies were detected postnatally. Intrauterine regression occurred in 23% of CVI cysts and in none of the arachnoid cysts. In children with arachnoid cyst, the occurrence of hydrocephaly and mass effect on the adjacent structures were observed in 23.9% (95% CI 8.3-4.4) and 26.8% (95% CI 4.0-60.1), respectively. None of the cases included had abnormal motor outcome or intelligence. The rate of surgery was 34.7% (95% CI 16.0-56.4). None of the children with a prenatal diagnosis of isolated CVI cyst experienced any of the adverse outcomes explored in this review.
I am wondering how the last sentence can be made sense of. First of all, what would it even mean to push on the two-particle system (as opposed to pushing on each particle)? I can make sense of the "acceleration of the two-particle system" by taking the center of mass as the representative point of the two-particle system. However, we can't push on the center of mass since we can only push on the particles.
In classical mechanics, even if the force acted directly on single particle only, that force would "feel it has to accelerate the other particle as well". More correctly, the effect of the external force on the particle it acts on (its acceleration) is smaller when the other particle is attached or keeps close to the first particle. The acceleration is as if the external force acted on a hypothetical particle with greater mass, equal to sum of masses of the particles. In EM theory, this rule - the force effect being as if there was a particle with mass that is sum of masses of the particles - is no longer necessarily true.
This "acceleration electric field" may act on other nearby charged particles. In inductors, it is responsible for the induced electric field and thus induced EMF pointing against the acceleration, thus adding more inertia to mobile charge carriers forming the current, and thus greater effective mass. This is because all the accelerating charges have the same charge sign, so the induced field is proportional to acceleration, but points in the opposite direction.
If there is just a pair of positive and negative charge, the same kind of acceleration field is present due to both particles, and acts on the other particle in direction of its acceleration, thus decreasing effective mass of both particles. This is the simplest system manifesting "electromagnetic mass effect", an increase or decrease of effective mass due to EM interaction in the system.
A classical analogy may be useful. Consider two masses $m_1$ and $m_2$ connected by a spring with mass $m_s$. Apply a force to $m_1$. Initially, only $m_1$ accelerates, as it takes time to accelerate the spring. As $m_1$ accelerates, its motion excites a wave on the spring. You may call the wave speed $c$. The force launching the wave reacts back on $m_1$, reducing its acceleration. Part of the spring accelerates while part remains in its original state of motion until the wave reaches $m_2$. Then, $m_2$ begins to accelerate.
Local tumor spread (2)
Another important consideration is the effect on the surrounding structures.
Primary brain tumors are derived from brain cells and often have less mass effect for their size than you would expect, due to their infiltrative growth.
This is not the case with metastases and extra-axial tumors like meningiomas or schwannomas, which have more mass effect due to their expansive growth.
On the left is an image of a diffusely infiltrating intra-axial tumor occupying most of the right hemisphere with only a minimal mass effect.
This is typical for the infiltrative growth seen in primary brain tumors.
There is no enhancement so this would probably be a low-grade astrocytoma.
On the left are images of a 52-year-old female who, over the period of one year, complained of headache and neck pain.
There is a recent onset of tonic-clonic seizures.
The CT shows a mass with calcifications, which extends all the way to the cortex.
Although this is a large tumor there is only limited mass effect on surrounding structures, which indicates that this is an infiltrating tumor.
The most likely diagnosis is oligodendroglioma.
The differential diagnosis includes a malignant astrocytoma or a glioblastoma.
On the left is an image of an intra-axial tumor in an adult.
It is centered in the temporal lobe and involves the cortex.
Although there is massive infiltrative growth involving a large part of the right cerebral hemisphere, there is only minimal mass effect.
There is no enhancement.
These features are typical for a low-grade astrocytoma.
Patchy enhancement (2)
On the left are images of a tumor located in the right hemisphere.
Although is a large tumor, the mass-effect is limited.
This indicates that there is marked infiltrative growth, a characteristic typical for gliomas.
Notice the heterogeneity on both T2WI and FLAIR.
There is patchy enhancement.
All these findings are typical for a GBM.
Virtually no other tumor behaves in this way.
Ok so I completed mass effect 1 did every sidequest everything but I only got to level 27 or 28 on the new leveling curve or lvl 55 of the original curve. Which means I think is start at lvl 4 with some money and such in mass effect 2. I'd need to do another complete 100% playthrough making all the same choices to max out my Shep in m.e 1 to bring over to start with a lvl 5 in m.e 2 and more money and such
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