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CHE Diabetes and Obesity News
and Updates
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Reviews
This review explores the proliferator-activated receptor gamma (PPARγ) mechanism in detail, and examines the obesogenic effect of prenatal exposure to BPA during critical windows of vulnerability. Hoepner, Environ Res.
Environmental Chemicals: Human Studies
Exposure to phthalates was positively associated with type 2 diabetes, fasting glucose, and long-term blood glucose (HbA1c) levels, and the associations differed based on sex, BMI, and age. Duan et al. Sci Total Environ.
Prenatal exposure to PFBS, a replacement chemical for PFOS, was associated with increased adiposity in 5 year old girls. Chen et al. Chemosphere.
Found an association between short-chain perfluoroalkyl carboxylates (PFCA) exposure and both gestational diabetes risk and impaired glucose homeostasis in pregnant women (China). Liu et al. Environ Int.
Environmental Chemicals: Laboratory Studies
These results may have implications for the epidemiologic association between arsenic exposure and diabetes. Jochem et al. Mol Cell Biol.
DEHP induces apoptosis in beta cells in part via oxidative stress, and taurine, an amino acid, helps reverse DEHP-induced oxidative stress. Li et al. Toxicol Mech Methods.
PBDE 99 may be a potential obesogen. Wen et al. Sci Total Environ.
Air Pollution
PM2.5 exposure was associated with adverse lipid level changes (U.S.). Sci Total Environ.
To see how these studies relate to existing research, or for more on environmental chemicals and diabetes/obesity, visit www.diabetesandenvironment.org
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