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Usenet Abuse: Someone at IP address 141.234.16.129 is impersonating me and posting nonsense

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Radium

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Sep 5, 2007, 6:32:32 PM9/5/07
to
common causes of pseudocoma."

#randsent

"It is important to remember that none of the historical data
absolutely include or exclude the possibility of pseudocoma. However,
there are some clinical findings suggestive of psychogenic origin,
such as conditions precipitated by stress. Pseudocoma usually begins
or persists when an observer is present. Patients with pseudocoma
slump to the floor and protect themselves from hitting their heads and
other body parts."

#randsent

"During examination, patients with pseudocoma usually make
semipurposeful avoiding movements. They have normal pupils, corneal
reflexes and plantar reflexes. They may keep their eyes firmly shut
and resist the opening of the eye by examiners. Because eyelid tone
cannot be changed at will, in patients with true coma passive eyelid
opening is easy and is followed by slow eyelid closure. Blinking also
increases in feigned coma, but decreases in true coma. Passive eye
opening in a sleeping or an actually comatose person results in
mydriasis if the pupillary reflex mechanisms are intact. Conversely,
opening the eyes of a person who is awake produces miosis. The eyes
roll up when the lids are raised, known as Bell's phenomenon as
mentioned before, in patients with psychogenic pseudocoma, while the
eyes remain in the neutral position in patients with real coma. Roving
eye movements cannot be imitated and their presence indicates true
coma. In contrast, voluntary saccadic eye movements seen in feigned
coma are usually faster and briskly with a well-defined endpoint.
Pseudocoma patients may respond with purposeful movement to painful
stimulation and avoid unpleasant stimuli such as a nasal tickle. The
presence of nystagmus during cold caloric testing suggests that coma
is either feigned or hysterical, because nystagmus requires an intact
cerebral cortex and


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