Can a lemon stop a cold sore?
Giorgio Vlad
that the main difference between the genital and oral herpes triggers,
can be the food that makes the acidity/alcalinity in our mouth/lips areas,
a proper environment for the HV.
A simple lemon can be a very alcalinic protector, then?
(like soaking half a lemon on your lips)
*(Did anyone say 'Potato Chips'?!) ;O) *
Perl Molson
http://www.4quantumhealing.com/acidalk.html
http://www.digitalnaturopath.com/data/Lysine_Arginine_Food_List.html
Next its some Tim's old post, related:
Erm if the pH is high the is low acidity of more likely high alkalinity...
pH (stands for potency/power of H+) is basically measuring free hydrogen
ions.....its a log scale (so open ended), with 7 being neutral at 22
degrees (6.8 at 37 degrees)...as you increase the number you go alkaline,
and as you decrease it its increasingly acidic.
The stomach is somewhere round about pH 1 - thats basically hydrochloric
acid, and moderately concentrated....and its really really heavily
protected for good reason (can we say peptic ulcers)...that would digest
virus...other important pH's blood pH is around 7.4, if the mouth goes
below 5.5 thats when saliva stops protecting. (I pHed a can of cola once
and it came out at 3 by the way,.....)
They really dont do too well in acid conditions..if I grow a stock and the
medium starts to get acidic the stock concentration Id get back drops a
whole load. There was a group who specifically mutated a virus strain to
grow in high acid conditions to look at things like the mucosal genital
surfaces and so on as the acidity is part of the protection (obviouly not
100%).
I guess it depends on where you want to be on the scale when you are
talking too much acid...inside cells and organs the pH is regulated and
controlled for various reasons (lysozymes have a different pH to
endoplamic reticulum in the cell, salivary glands and stomach glands
produce different things) so when talking about it being out of place or
too acid you really need to be putting it in perspective....the stomach
cavity being at pH3 is far too alkaline, the mouth being at 5 is far to
acid but one is far less acid than the other.
A tissue being out of pH balance for some reason (either way) is going to
stress it and cause problems but lining it to an absolute pH number will
be I think problematic. Stressed tissues on the other hand are likely to
be sending all sorts of signals ripe for reactivation - that is after all
the basic idea behind herpes gladiatorum....roughly abraded tissue at the
surface triggers viral reactivation in the deeper ganglia....you'll have
sorts of chemical floating around there, histamines, nitrous oxide,
oxidative damage, fibrin formation all the repair and damage signals. What
that does to the pH I dont know.
And we roll back to a host of general trigger systems, and what can
reactivate and how it does it....and thats in the area of 'not fully
described' and 'the person who works this out properly is staring at a
Nobel'...ie some things are known but the whole isnt.
Best ideas I can come up with on this one.
Tim
When playing rugby, its not the winning that counts, but the taking apart
ICQ: 5178568
Perl Molson
New Treatment Options are Available for the Prevention of Cold-Sores Without
the Use of Prescription Drugs.
An Independent Study Demonstrates an Effective New Model for the Prevention
of HSV-1 Infections.
Individuals suffering from chronic infections of HSV-1 are invited to
participate in the first phase of research to study the effectiveness of a
new model for the prevention of HSV-1 infections without the use of
prescription drugs in the general populace. Individuals wishing to
participate should respond including their name, age, city, state, and
address or telephone number along with information concerning the severity
and duration of their symptoms.
----------------------------------------------------------------------------
----
Herpes Simplex Type 1; Suppression of Symptoms Through Restriction of
Dietary Nitrites and Nitrates.
INTRODUCTION:
1 in 5 or 50 million people in this country suffer from cold-sores. 50 to
75% of adults and children have herpes simplex virus, type 1 (HSV-1). By age
50, 75-90% of American adults have HSV-I or HSV-II antibodies in their
blood. About half of those affected are never troubled again. The other half
have recurrent outbreaks. Treatment includes local anesthetics, antibiotics
for secondary infections, and ointments to soften the crusts of the
blisters.
Elements which may trigger an onset of symptoms include: lowered physical
resistance from illness, fever, trauma, emotional stress, fatigue and
overexposure to the sun. Outbreaks are frequently accompanied by fatigue,
aches, and lack of energy as a result of the immune system's response to the
infection. Patients who are subject to incessant over-lapping, never quite
healing herpes virus infections at the site of the initial outbreak may
harbor a particularly strong strain of the virus -- or have an unusually
weak resistance -- or a combination of both. In chronic infections, somehow
the herpes viruses outnumber or resist the immune system and remain active
with new lesions breaking out as old ones heal.
Viruses may infect cells without instructing them to produce more viruses.
But once an invaded cell has incorporated the virus's genetic information,
it manufactures a new set of mutated genes every time it divides. Such
infected cells may suddenly begin replicating the virus. One cell, taken
over by a viral invader can produce 20,000 to 50,000 new herpes virus
particles every day causing the cell to lyse, that is, its exterior membrane
deteriorates. New-formed viral particles spill out when the cell ruptures
and then the cell dies. Viral particles affix to healthy cells in the nearby
cellular environment, and the cycle repeats itself. Viral activity results
in ruptured cells. The area of infection becomes swollen and inflamed.
Blisters form filled with lymph fluids, dead and dying cells, virus
particles, and white blood-cells. It may take one to four days for the
phagocytes, macrophages, T-cells, B-cells, plasma cells, antibodies and
interferon of the immune system to turn the tide and get viral activity back
under control. The symptoms, extent and duration of lesions thus vary widely
from person to person.
During the outbreak, some herpes virus particles find their way into a nerve
cell leading from the skin's surface and somehow gain entry to the nerve
axons to migrate along neural pathways. During latency, HSV-I virions reside
in the trigeminal ganglia; clusters of neural roots located in the upper
cheek near each temple, and remain insulated from the body's immune
defenses. Within the nerve cells, viruses don't replicate. It is suggested
that the virions may somehow travel back along the neuron to cause new
outbreaks in response to general immune suppression.
Acyclovir speeds healing of sores in HSV-II infections, reducing the
severity of initial and recurrent outbreaks. Acyclovir was at one time
prescribed to treat HSV-I and has since been shown to be ineffective.
Valtrex has since been shown to exhibit partial effectiveness in inhibiting
viral repication.
Dr. Linus Pauling related that his wife, Alva Helen Pauling, reported that
large concentrations of oral and topical ascorbic acid powder exhibited
partial effectiveness in treating cold-sores by dramatically spreading the
healing time and reducing the severity and duration of recurrent outbreaks,
often arresting the process within a few hours. Others have reported similar
results.
Other topical and oral medications which are reported to exhibit partial
effectiveness in reducing the severity of symptoms and the frequency of
outbreaks currently undergo clinical trials.
CASE STUDY:
A dietary correlation of nitrites and nitrates to HSV-I expression was
observed in one subject experiencing epidermal hypersensitivity due to
treatment with accutane. The severity of outbreaks, length of healing time
and rapidity of onset of prodromes after consumption were found to be
proportional to the quantity ingested, with lesions appearing within 24 to
72 hours of the ingestion of affective dosages. Total restriction of dietary
nitrites and nitrates resulted in a gradual dissipation of the number and
severity of prodromes and the recurrence of outbreaks over a three to four
month period, with remission occurring in the fourth month.
The history of the subject is consistent with herpes simplex type 1
expression, with characteristic localized lesions recurring after the
appearance of the prodromes, and gradually healing after the abatement of
the accompanying inflammatory immune responses within a one to two week
period. Symptoms were active in the subject for a period of 10 years, with
characteristic lesions appearing monthly and often weekly, and with the
symptoms and effects of the prodromes often persisting between episodes.
L-lysine which is reputed by to have partial effectiveness for some in
relieving the symptoms of oral herpes was ingested at the beginning of one
outbreak and did not appear to exhibit any effectiveness, although the
effects of long-term usage had not been determined prior to the remission.
Prior to the remission outbreaks had occurred regularly in response to a
number of generally recognized environmental stimuli which tend to trigger
infections including excessive sunlight, heat, cold, dryness, fatigue,
illness and mechanical abrasion. Behavioral modifications had been made to
avoid stimulus characteristically related to the condition, and while this
did not prevent outbreaks from occurring, experience demonstrates that this
may help to prevent more severe outbreaks from occurring. It was determined
that the outbreaks, occurring regularly in response to environmental
stimuli, were the result of a pre-disposing factor of sensitivity, and it is
widely regarded that these stimuli play the role of exacerbatory cofactors.
This underlying sensitivity appeared to indicate that ongoing low-level
viral replication was initiating cellular eruptions resulting in an
accompanying localized inflammatory immune response, and it was realized
that owing to the severity of this condition at a given time, depended the
degree of response to those exacerbating stimuli.
A correlation of HSV-1 expression to the ingestion of sodium nitrite was
observed and a determination was made to restrict this compound from dietary
consumption. This resulted in a way of controlling the degree of the
predisposition to respond to environmental stimuli by reducing the degree of
sensitivity indicating active infection, and this indicates a possible means
of controlling the level of viral replication. This model proposes a basis
for explaining why relative degrees of active infection may predispose
individuals differently to such traumatizations as may tend to result in the
production of cold-sores.
Since these correlations and dietary modifications were made, the subject
has found that the pre-disposing sensitivity to all of the stimuli which
would initiate outbreaks has disappeared, and few precautions need now be
taken. The subject is asymptomatic and routinely experiences exposure to UV
rays, the elements, fatigue, and topical irritations including mechanical
abrasion without consequence, whereas all of these had previously produced
nearly immediate inflamatory reactions while symptomatic.
Episodes have occurred on an average of once or twice a year since dietary
modification, and always within a 24 to 72 hour period of either accidental
or deliberate ingestion of an appreciable quantity of either sodium nitrite
or sodium nitrite and nitrate, during which time the prodromes and
accompanying sensitivity to stimuli were observed, with lesions occurring in
the expected time-frame. Reactions were found to be typically more severe
on an empty stomach, and proportional to the quantity ingested. On several
occasions, a large quantity of sodium nitrite was ingested specifically to
determine the effect of various single dosages, with the expected outcomes.
Despite general restriction of dietary nitrites and nitrates, discretionary
consumption of very small infrequent single doses of sodium nitrite,
particularly when not taken on an empty stomach have not always produced
side-effects, while large single doses have done so invariably. This is all
the more compelling in that on three of these occasions, the determination
that sodium nitrite had been ingested was ascertained only after the onset
of symptoms. Nevertheless, the lesions from such episodes healed on the
average of twice as quickly and were of a much less severe character, while
the underlying inflammatory response would dissipate within twenty-four
hours allowing the lesions to heal, and did not persist between episodes.
The subject continues to employ moisturizers and sunscreens and remains
normally asymptomatic and lesion-free after 8 years.
DISCUSSION:
In this subject study, it had been determined that of the compounds of
interest as study candidates, being common to most of the food products
implicated as being responsible for initiating reactivity, including: sodium
nitrite, sodium nitrate, sodium phosphate and sodium erythorbate, sodium
nitrite and sodium nitrate were prevelent; but only sodium nitrite was
common to all reactive agents. Nevertheless the relative reactivity of the
subject indicated a readier response to bacon for example than to foods such
as ham, hot-dogs and pepperoni, which, though in much larger quantities, had
also demonstrated reactivity. This effect has been tentatively attributed
to the concentrating effects resulting from the reduction of mass that
occurs during the cooking process.
Food manufacturers use nitrites and nitrates as inhibitors of botulism and
aspergillis flavus mold which produces aflatoxin organisms in bacon and
other meats, and is a carcinogenic toxin. Nevertheless, preliminary
indications seem to indicate a possible association between sodium nitrate
and/or nitrite and a number of conditions suggestive of immunological
compromise including the relationship esteemed to exist between hot-dog
consumption and certain leukemias for example. Nitrates should be avoided
without vitamin C supplementation. Vitamin C can block formation of
carcinogenic nitrosamines from nitrites and nitrates in foods and cigarette
smoke and amines such as nicotine and other amines found in foods. Nitrates
are found to be a leading factor in cancer of the colon. Nitrites and
Nitrates may accumulate in the body's fatty tissues and remain present for
months after ingestion. Only certain viral proteins can attach themselves to
certain receptors. Herpes virus proteins can attach more readily to cells in
the mucous membranes than tougher cells of the epidermal layers. Herpes
simplex type 1 lesions are thus expressed in the lips' fatty tissues.
CONCLUSION:
It is concluded that cumulative and incidental concentrations of sodium
nitrite and/or nitrate present systemically and in affected tissues may
somehow interfere in a significant way with the body's local immune response
to herpes simplex type 1 virus in infected individuals, encouraging
replication. Long-term total dietary restriction of nitrites and nitrates as
well as supplementation with vitamin C may significantly improve the body's
immune response to herpes simplex type 1 virus, and may thus provide a
significant prophylactic aid in the inhibition of viral replication for the
prevention of recurrent infections. Further research and testing may
therefore be warranted as a possible source of new insight into herpes
virology as well as for deriving a prospectively effective approach to
patient care as an alternative to the limited selection of treatment options
available over the counter to reduce pain and inflamation as analgesics as
well as by prescription to reduce the severity and frequency of outbreaks as
antiviral therapies.
----------------------------------------------------------------------------
----
A Note From the Author --
It is my hope that through further research and by encouraging research by
others, that substantial relief may be obtained by those suffering from
chronic HSV-I infections without the expense and side-effects of ongoing
drug and herbal therapies all of which seem at present to exhibit limited
effectiveness.
The following test has been proposed: In subjects with any active HSV-I
infection, all active inflamation should be allowed to subside. Subjects are
then directed to consume 16 ounces (1 lb.) of bacon on an empty stomach,
preferably in a divided dose. Any reaction is expected to occur within a 24
to 72 hour period as indicated. If this occurs, it is then adviseable that
all dietary consumption of sodium nitrites and nitrates be eliminated in
order for this treatment to be effective. The condition should subside and
a remission of symptoms is expected to occur within a few months after
dietary modifications have been implimented. In the subject study 16 ounces
of bacon had been sufficient to demonstrate specific reactivity but it
should be borne in mind that the subject had been experiencing epidermal
hypersensitivity and reactivity due to treatment with accutane. Circulating
systemic levels of the implicated compounds while possibly contributing to
the overall predisposition of a subject to develop eruptions could also, due
to an attending tolerence, contribute to an elevation of the threshold
level determining quantities necessary for initiating specific reactivity,
the reactivity threshold determining affective dosages for individual
subjects having not yet been determined.
I am particularly interested in learning of those who while asymptomatic
have been able to induce the indicated response from the implicated
compounds in the proscribed period, as well as from those who have found
substantial long-term relief as a result of sustained total dietary
restriction, and more particularly those with chronic recurrent infections.
Anyone wishing to report their results from the proposed test or who would
like to participate in the study group are invited to respond. Only
individuals with active chronic HSV-1 infections exhibiting reactivity to
the test are considered as fitting those criteria suitable of candidates
required for participation in long-term study. Study participants will be
provided with information they may require concerning sources of sodium
nitrite and nitrate along with other dietary recommendations, and will
receive individual follow-up and consultation to evaluate their candidacy
for participation as well as the effectiveness of treatment.
beat...@email.com (Giorgio Vlad) wrote in message news:<813d1c43.03031...@posting.google.com>...
Perl Molson
From: nwa (no...@again.com)
Subject: ?
View: Complete Thread (15 articles)
Original Format
Newsgroups: alt.support.herpes
Date: 2000/01/09
"Thus, a herpes simplex virus once present in a cold sore, although
healed and leaving no evidence of lip pathology, could ignite later by
simple exposure to ultraviolet light. How many mothers are endangering
the lives of their children by sunbathing, laboring under the belief
that they are improving their health". Roizman [9] believes that all
children are infected by age 5, but that only 1 percent experience
true clinical illness. For many years investigators thought that each
recurrence of fever blisters represented a new infection. Evidence is
accumulating that shows the herpes simplex virus is harbored in
dormant form until a physiological or emotional event provokes the
virus to produce the typical herpetic lesion. *In one case with five
repeats of herpes virus erupting at yearly intervals and at the same
site, 7- 10 gm ascorbic acid by mouth, daily, was found to eliminate
this pathology.*
Effecting a cure when a virus is the offending agent, and many times
bringing about this change in the short space of 24 hours, is a
rewarding moment in medicine. Vitamin C treatment must be intensive to
be successful. Use veins when practical, otherwise give vitamin C
intramuscularly. Never give less than 350 mg/kg body weight. This must
be repeated every hour for 6 to 12 times, depending upon clinical
improvement, then every two to four hours until the patient has
recovered. Ice cubes held to the gluteal muscle before and after
injection will reduce or eliminate pain and induration. When treatment
continues for several days, the child can be placed on an ice cap
between injections. When employing vitamin C intravenously, it is best
to use sodium ascorbate and the solution free of all additives except
sodium bisulfite. The dose of vitamin C using a syringe should range
between 350 mg and 400 mg/kg body weight. In older patients or when
very high doses are required the vitamin can be added to 5 percent
dextrose in water, in saline solution or in Ringer's solution. The
concentration should be approximately 1 gm to 18 cc fluid. Bottle
injections will need 1 gm calcium gluconate one to two times each day
to replace calcium ions removed by the high intravenous schedule. One
quart of milk daily will suffice when using the vitamin
intramuscularly. In place of milk one can substitute calcium gluconate
tablets. Supplemental vitamin C is always given by mouth. As a guide
in determining the amount and frequency of injections we recommend our
Silver Nitrate-Urine test [10]. This is done by placing ten drops of 5
percent silver nitrate in a Wasserman tube and adding ten drops urine.
A color pattern will develop showing white, beige, smoke gray or one
that looks like fine grain charcoal. Charcoal is the color needed and
the test is performed at least every four hours. The test itself is
read in one minute. These large doses of ascorbic acid will also bring
all body tissue back to saturation which means that the white blood
cells will now be capable of destroying other pathogens that might be
clouding the picture. Unless the white blood cells are saturated with
ascorbic acid, they are like soldiers without bullets. Research on
this is now under way at the Bowman Gray School of Medicine by McCall
and Cooper [11]. White cells ingest bacteria and in the process
produce hydrogen peroxide. Hydrogen peroxide will combine with
ascorbic acid to produce a substance which is lethal to bacteria. I
have seen diphtheria, hemolytic streptococcus and staphylococcus
infections clear with within hours following injections of ascorbic
acid in a dose range of from 500 mg to 700 mg/kg body weight given
intravenously and run in through a 20G needle as fast as the patients
cardiovascular system will allow.
The killing power of ascorbic acid is not limited to just herpes
simplex and the adenovirus. When proper amounts are used it will
destroy all virus organisms. We found measles to be a medical
curiosity. Specifically we observe that vitamin C prophylactically, by
mouth, was not protective unless 1 gram was given every two hours
around the clock. One gram every four hours would modify the attack.
One gram given every four hours intramuscularly was also protective.
With our own children we kept the measle syndrome going off and on for
30 days by giving 1gm every two hours for two days, then off for two
days. The disease was then stopped by continuing 1 gm every two hours,
by mouth, for four days. By 1950 we learned that we could kill the
measles virus in 24 hours by giving intramuscular injections in a dose
range of 350 mg/kg body weight every 2 hours. We also found that we
could dry up chicken pox in the same time, but more dramatic results
were obtained by giving 400 mg/kg body weight intravenously. Two to
three injections in 24 hours were all that was required. We published
these results in 1951 [16]. Recently, we cured a man weighing 85 kg in
four days taking 30 gm each day by mouth. In conclusion, the killing
power of ascorbic acid on virus bodies has been demonstrated by me in
hundreds of cases, many of which were treated in our hospital with
nothing but vitamin C. We have published some 28 papers on this
matter.
In certain individuals some virus conditions have a slower response.
Herpes zoster and mumps belong to this group. We found that in these
conditions equally rapid destruction of the virus could be effected
through the use of adenosine-5-monophosphate. Adenosine was given
according to age and weight, 25 mg in children and 50-100 mg
intramuscularly in adults. This was given every 12 hours along with
ascorbic acid. Adenosine will sometimes precipitate a mild reaction in
that the patient will feel a fullness in his or her head with varying
degrees of nausea. Inhalation of aromatic spirits of ammonia will
quickly relieve and, if used before injection, will prevent this
condition. Their response, when adenosine was administered, led us
theorize that when a cell has been invaded by a foreign substance,
like virus nucleic acid, enzymatic action fostered by ascorbic acid
contributes to the breakdown of virus nucleic acid to adenosine
dearminase which converts adenosine to inosine. Some individuals
cannot manufacture sufficient adenosine to cope with this phase of
purine metabolism under certain stress conditions associated with
virus pathology. The net result from this chemical action is to
catabolize purines rendering them unavailable for making additional
virus nucleic acid. Ascorbic acid is further unique in that it
possesses the capability of entering all cells. After entering a virus
infected cell, ascorbic acid proceeds to take up the protein coats
being manufactured by the virus nucleic acid, thus preventing the
assembly of new virus units. These newly made macromolecules within
the host cell soon create a situation where the tensile strength of
the cell membrane is exceeded with resulting rupture and cell death.
Ascorbic acid, when given in the massive amounts that accomplish full
tissue saturation, will also enter those cells harboring the so-called
dormant virus. Where the vitamin C removes the protective protein coat
of the virus the macromolecule formed will act in the capacity of a
repressor factor inhibiting further activity of the virus nucleic acid
which is then destroyed by additional vitamin C. We offer as proof of
this the instance of a patient having herpetic lesions for five years
and being cured with continuous high daily intake of ascorbic acid. In
acute virus infection, associated with a virusemia, ascorbic acid
given intravenously will remove the protein protective coat from the
virus body, leaving the denuded virus unit vulnerable to the
leukocytes for destruction.
http://www.vitamincfoundation.org/mega_1_1.html#FISHES
at http://www.orthomed.com , the website of medical doctor Robert F.
Cathcart ; http://www.vitamincfoundation.org ; and
http://www.cforyourself.com
Over 85 articles (and hundreds of scientific references) on
nutritional
therapeutics posted at http://www.doctoryourself.com ; still more at
http://www.rath.nl/sitemap/usa/main.htm http://www.orthomed.com
http://www.orthomed.org http://pcrm.org http://www.pnc.com.au/~cafmr/
beat...@email.com (Giorgio Vlad) wrote in message news:<813d1c43.03031...@posting.google.com>...
Perl Molson
Vitamin C has been an essential ingredient to life since before
evolution made its split into the animal and plant kingdoms. It is
utilized throughout the body for so many things that it is probably
not an overstatement to say that there are virtually no functions that
it is not involved in. To quote Dr. Emanuel Cheraskin, Dr. Ringsdorf
and Dr. Sisley from THE VITAMIN C CONNECTION:
"There are more than ten thousand published scientific papers that
make it quite clear that there is not one body process (such as what
goes on inside cells or tissues) and not one disease or syndrome (from
the common cold to leprosy) that is not influenced -- directly or
indirectly -- by vitamin C."
By inference, tissues that maintain a higher concentration of vitamin
C would be most susceptible to chronic problems as a result of chronic
deficiency. From Vitamin C in Health and Disease, "Plasma ascorbic
acid concentration of a healthy person is 8-14 mg/L, while adrenal
glands, pituitary, thymus, corpus luteum, and retina have
concentrations more than 100 times higher. The brain, spleen, lung,
testicle, lymph glands, liver, thyroid, small intestinal mucosa,
leukocytes, pancreas, kidney, and salivary glands have concentrations
10-50 times that of plasma. The skeletal, smooth and cardiac muscle,
and erythrocytes have concentrations about 10 times that of plasma."
Vitamin C supplementation would probably have a very positive affect
on these organs particularly.
Among the most important areas where C plays a crucial role are
building and maintaining our tissues and fortifying our immune
systems.
Antioxidant
We here a lot these days about the antioxidant vitamins, C, E and A.
Oxygen is a highly reactive element. We see the result of oxidation
all around us. Rust, brittle rubber, food spoilage, these are all the
result of oxidation. Now oxidation is not always bad. The iron in
your blood's hemoglobin oxidizes or "rusts" in order to carry oxygen
to all the cells of the body. But much oxidation is damaging,
accelerating aging and contributing to tissue and organ damage.
Oxidation is also a contributor to heart disease (LDL oxidation has
been linked to atherosclerosis) and cancer. As research continues,
the more free-radical damage appears to contribute to chronic
conditions and the more we realize that antioxidant nutrition
supplementation is essential.
Balz Frei, in his paper Vitamin C as an Antiatherogen: Mechanisms of
Action published in Vitamin C in Health and Disease states
"Vitamin C (ascorbic acid) has been shown to be the most effective
water-soluble antioxidant in human plasma."
"...that ascorbate is useful as part of the treatment of almost all
diseases. This almost universal benefit is because massive doses of
ascorbate neutralize massive amounts of free radicals and free
radicals mediate all inflammations. Most acute infectious diseases
can be cured if the free radicals are eliminated."
- Dr. Robert Cathcart
Homeodynamics
Homeodynamics is a fairly recent term that updates "homeostasis",
defined in the Bantam Medical Dictionary as:
"the physiological process by which the internal systems of the body
(e.g. blood pressure, body temperature, acid-base balance) are
maintained at equilibrium, despite variations in the external
conditions."
Homeodynamics attempts to add emphasis to the dynamic, ever-adjusting
nature of these processes.
When our body's are exposed to stress (stress means any influence
which disturbs our calm balance, therefore all the physical and
emotional pressures that we are exposed to make demands on our
systems) our stores of Vitamin C are depleted, mostly by our adrenal
gland (it takes a molecule of vitamin C to produce a molecule of
adrenaline). It is an old wives tale that getting soaked in the rain
causes us to catch cold. But, like so many of these, there is a basis
of truth. Being wet and cold is stressful. It puts an additional
strain on us to keep our body temperature up. This reduces our
"resistance". Since vitamin C is vital to both our immune system and
our natural balance, the day after coming in from a cold rain, we find
ourselves with a scratchy throat and its downhill from there.
InSCIght magazine has an article of interest, Fortifying Against
Stress that reinforces this topic.
Any trauma we are exposed to utilizes vitamin C. Best to have as much
as possible all the time.
Collagen
Symptoms of scurvy include bleeding gums, easy bruising and a tendency
toward bone fractures. All these symptoms are a result of the
requirement for Vitamin C in the development of the ground substance
between our cells. This ground substance, primarily collagen, is the
cement that gives our tissues form and substance (see description at
left). Collagens are principal components of tendons, ligaments,
skin, bone, teeth, cartilage, heart valves, intervertebral discs,
cornea, eye lens, in addition to the ground substance between cells.
Some collagen forms in the absence of ascorbic acid, but the fibers
are abnormal, resulting in skin lesions and blood vessel fragility,
characteristics of scurvy.
Any tissue-related malady will have some basis in Vitamin C. There is
a long continuum between scurvy and optimum tissue integrity. As most
all of us suffer from sub-clinical scurvy, how is this evident from a
tissue-integrity standpoint? Let's look at the conditions that are
tissue related. Gum problems are an obvious first choice since they
relate directly to scurvy, but there are many others.
A Japanese study concluded that most disc herniations are the result
of Vitamin C deficiency. This makes sense. The discs in our spinal
column are like donuts, with a tough, gristle-like exterior and a soft
interior to provide cushioning. Lack of proper amounts of Vitamin C
will produce a disc with compromised integrity. The tough exterior
won't be so tough. Over time and much wear and tear, this compromised
exterior will wear down and a pinhole will result. Moving just the
right way (or should I say wrong way) will push some of the soft
interior material out this pinhole. That is a disc herniation. If
this squished-out material touches a nerve in your spinal column, it
causes pain and usually a lot of it. I had this myself almost seven
years ago and let me tell you the pain kept me from sleeping for three
days! Adequate Vitamin C will toughen up the outside portion of the
disc and a herniation is much less likely. By the way, laser surgery
is now quite common to remove this balloon of squished-out disc
material. I did not have surgery and I can't tell that I ever had
the herniation. I attribute that, at least in part, to the better
shape my discs must be in due to my increased C intake over the past
six years.
Tissues under stress will suffer the most from vitamin C deficiency.
Please refer to the cancer and heart disease sections. Both of these
chronic conditions have a strong tissue-integrity component.
Similar to the example above concerning disc integrity, our blood
vessels are quite susceptible to lack of Vitamin C. My father died of
a ruptured aorta. This is all too common. I firmly believe that that
would not have happened if he had not been suffering from sub-clinical
scurvy.
Immune System
Vitamin C is a requirement for the proper functioning of our immune
systems. It is involved in white blood cell production, T-cells and
macrophages. Without Vitamin C in adequate quantities, our own body's
best defense against disease is left without ammunition. This has a
distinct bearing on how much Vitamin C to take (please see How Much to
Take.) A sickness such as a cold or virus is analogous to a brush
fire. Destruction in its path, just as an infection wrecks havoc to
our own internal ecology. If the sickness is the fire, then our
immune systems are the fire department and Vitamin C is the water. If
I may take this analogy a little further, the fire department may use
chemical fire retardents, which are loosely analogous to medicine.
Now we would all agree that these chemicals are sometimes warranted,
but surely we also know that they will have some consequences to the
environment or ecology. This is similar to nutrition and medicine.
Nutrition is the natural bolstering of our own systems. Medicine, on
the contrary, is foreign and needs to be used with corresponding care.
Due to the strong relationship between C and our immune systems, it is
not surprising that viral and bacterial infections can be dealt with
by our own systems when adequate C is present. Ascorbic acid is toxic
to viruses, bacteria, and many types of cultured cells, because of its
prooxidant activity. It is particularly toxic to malignant tumor
cells but much less toxic to nonmalignant normal cells, thus its
therapeutic use in cancer.
No matter what medicine or other treatment you and your doctor may
choose to treat your illness, Vitamin C should be part of the therapy.
As mentioned other places, Dr. Cathcart refers to disease by the
Vitamin C levels needed for treatment.
http://www.cforyourself.com/Overview/Primer/What_C_Does/what_c_does.html
beat...@email.com (Giorgio Vlad) wrote in message news:<813d1c43.03031...@posting.google.com>...
dedale
> The killing power of ascorbic acid
...
> Where the vitamin C removes the protective protein coat
> of the virus the macromolecule formed will act in the capacity of a
> repressor factor inhibiting further activity of the virus nucleic acid
> which is then destroyed by additional vitamin C.
This is pure speculation, but how about this approach: I will
administer vit. C mixed with DMSO to the area of recurrence DAILY.
(DMSO is a substance that will carry any substance mixed with it
straight across cell boundaries.) A high vitamin dose will enter into
the nerve cell daily; the virus will never get its protein coats ready
and I will live herpes free, having reduced the infection to the
status of chicken pox--permanent latency.
Dedale
M.L.S.
>Interesting, these Vitamin C effects.
>> The killing power of ascorbic acid
>> Where the vitamin C removes the protective protein coat
>> of the virus the macromolecule formed will act in the capacity of a
>> repressor factor inhibiting further activity of the virus nucleic acid
>> which is then destroyed by additional vitamin C.
>This is pure speculation, but how about this approach: I will
>administer vit. C mixed with DMSO to the area of recurrence DAILY.
>(DMSO is a substance that will carry any substance mixed with it
>straight across cell boundaries.) A high vitamin dose will enter into
>the nerve cell daily; the virus will never get its protein coats ready
>and I will live herpes free, having reduced the infection to the
>status of chicken pox--permanent latency.
You are completely at your own risk if you rely on Molson's
recommendations and doubly so if you start throwing DMSO into the mix.
Maybe you should ask whether he'll assume liability should his
assorted hypotheses prove less substantial than the bytes used in
positing them.
On the other hand, maybe you'll become a medical legend. ;-)
Mike
dedale
Frederick Klenner, given in is paper, "Observations On the Dose and
Administration of Ascorbic Acid When Employed Beyond the Range Of A
Vitamin In Human Pathology". Molson provided the reference.
M.L.S. <mso...@newsguy.com> wrote in message
krz
administer vit. C mixed with DMSO to the area of recurrence DAILY.
(DMSO is a substance that will carry any substance mixed with it
straight across cell boundaries.) A high vitamin dose will enter into
the nerve cell daily; the virus will never get its protein coats ready
and I will live herpes free, having reduced the infection to the
status of chicken pox--permanent latency.
What proof do you have that Vitamin C will kill the herpes DNA once inside the
cell membrane?
Greg Evans
the
> cell membrane?
I'm no expert (though now that I'm HSV-positive I'm trying hard to become
one!), and I'm definitely not qualified to prove or disprove that claim.
But I do know that Vitamin C is an antioxidant, and I have heard that
antioxidants have an effect against viruses of many types.
Here's a few websites talking about this:
http://www.healthy.net/asp/templates/article.asp?PageType=article&ID=2134 (a
little more than halfway down the page)
http://www.eclecticphysician.com/supplements/vitamin_c.shtml (references at
bottom of page)
http://freespace.virgin.net/health.news/page27.html
Arlyn Grant
"krz" <krz...@aol.comkrzgirl> wrote in message
news:20030331134443...@mb-fq.aol.com...
A high vitamin dose will enter into
> the nerve cell daily; the virus will never get its protein coats ready
> and I will live herpes free, having reduced the infection to the
> status of chicken pox--permanent latency.
Hate to tell you this, but chicken pox does sometimes reactivate. It's
called Shingles.
Tim Fitzmaurice
> This is pure speculation, but how about this approach: I will
> administer vit. C mixed with DMSO to the area of recurrence DAILY.
> (DMSO is a substance that will carry any substance mixed with it
> straight across cell boundaries.)
Its a great penetrant - but its not infinite - so you are only assuming it
reaches the nerve cell would be my concern - knowing how much DMSO is a
fine art (it is used for one of the eye formulations of antiviral).
Also make sure you get the decent stuff - cheap DMSO from a hardware store
sold as a solvent can have all sort of stuff in it which it also would
carry across you bodies lipid barriers....when they talk about clean DMSO
and pharm grade this is one of those where they really really mean it.
> What proof do you have that Vitamin C will kill the herpes DNA once inside the
> cell membrane?
I can dig out various bits on ascorbate and effect on virus growth etc -
none on taking out the DNA in the host cell specifically. The closest for
anything is preventing its replicating properly and I cant even find that
for ascorbate in the searches I've done before.....
What pubmed keywords have you tried, see if we are duplicating searches...
dedale
> On 31 Mar 2003, krz wrote:
> What pubmed keywords have you tried, see if we are duplicating searches...
I haven't tried to look on pubmed. My only reference is the paper by
Dr. Klenner I read at
http://www.vitamincfoundation.org/mega_1_1.html#FRED
Dedale