It was also more than once in my mens magazine. I'm pretty sure it was in
Pubmed. There was nothing about either Zinc or Carnosine.
J Infect Dis. 2012 Apr 15;205(8):1195-202. Epub 2012 Mar 13.
Association between gastric Helicobacter pylori colonization and glycated
hemoglobin levels.
Abstract
BACKGROUND: Few studies have evaluated the potential influence of
Helicobacter pylori on biomarkers for diabetes. METHODS: We conducted
cross-sectional analyses using data from 7417 participants in the National
Health and Nutrition Examination Survey (NHANES) III (aged ≥18 years) and
6072 participants in NHANES 1999-2000 (aged ≥3 years) to assess the
association between H. pylori and levels of glycosylated hemoglobin (HbA1c).
RESULTS: There was no association between H. pylori and history of
self-reported diabetes. Helicobacter pylori seropositivity, especially H.
pylori cagA positivity, was positively associated (P < .01, NHANES III; P =
.02, NHANES 1999-2000) with HbA1c levels after excluding individuals with
history of diabetes and controlling for potential confounders. There was
also a synergistic interaction between H. pylori and higher body mass index
(BMI), such that increased levels of HbA1c associated with having both H.
pylori and higher BMI were greater than the sum of their individual effects
(P for interaction < .01). This interaction was observed consistently in
both NHANES III and NHANES 1999-2000 and for H. pylori cagA positivity in
NHANES III. CONCLUSIONS: The findings indicate a role of H. pylori in
impaired glucose tolerance in adults that may be potentiated by higher BMI
level.
PMID: 22427676
J Indian Med Assoc. 2010 Mar;108(3):140-3.
Role of Helicobacter pylori in causation of diabetic gastropathies and
non-gastrointestinal complications in type 2 diabetes.
A cross-sectional case-control study was conducted in 80 diabetic patients,
to evaluate the incidence of gastropathy by endoscopy in type 2 diabetes
mellitus. An association between Helicobacter pylori infection and
non-gastrointestinal complication of diabetes mellitus was also looked into.
Gastric biopsies were subjected to rapid urease test for demonstration of
Helicobacter pylori. The fasting blood glucose levels among Helicobacter
pylori positive diabetes were 175 +/- 36.5 mg %, and in Helicobacter pylori
negative diabetics were 138 +/- 39.4 mg %. The prevalence of endoscopically
detectable gastro-intestinal complications were higher in Helicobacter
pylori infected diabetics (odd's ratio 4:2; p < 0.05). The total prevalence
of Helicobacter pylori positive in diabetics by rapid urease test was
statistically significant (p < 0.05). Coronary heart disease was more
prevalent in diabetics with Helicobacter pylori infection than those without
Helicobacter pylori (57%). The prevalence of H. pylori positivity in other
complications such as peripheral vascular diseases, cerebrovascular diseases
was not significant. The association between nephropathy, retinopathy and
neuropathy with Helicobacter pylori, was also observed and the strong
association was seen in diabetic retinopathy (p < 0.001), diabetic
neuropathy (p < 0.01) and nephropathy (p < 0.001).
PMID: 21043350