No Cure Latex Allergy

ironjustice

unread,

Jul 24, 2009, 7:52:16 PM7/24/09

to

http://www.mechdir.com/press/catalog/1079/index.html

"18 percent of health care workers have some level of latex allergy"

---------------
Excess dietary iron is the root cause for increase in childhood Autism
and allergies
Medical Hypotheses, Volume 61, Issue 2, Pages 220-222
Medical Hypotheses
Abstract
Autism is a profoundly and poorly understood developmental disorder
that impairs a person’s social and communication abilities.
I propose a hypothesis that the excessive dietary iron consumed by
today’s infants is the root cause of increased cases of Autism,
allergies and other childhood diseases. Iron is a powerful immune
system modulator.
Excess iron causes hyperactive immune system.
This hyperactive immune system attacks undigested food peptides.
The chemicals released during these intense allergic reactions can
damage surrounding tissue. Neurodegeneration is caused by combination
of, oxidative stress induced by free iron radicals and intense immune
reactions.
Iron chelators have shown beneficial results in Autism and allergies.

doi:10.1016/S0306-9877(03)00126-9

-------------------------------

"Loading cells with Fe-D increased their response to latex"

Years ago a guy wondered if his iron overload had anything to do with
his spina .. bifida.

"Up to 73% of children and adolescents with Spina Bifida are
sensitive
to latex"

Iron uptake and release by macrophages is sensitive to propranolol.
Mol Cell Biochem. 2006 Aug;288(1-2):213-7. Epub 2006 May 23.
Komarov AM, Hall JM, Chmielinska JJ, Weglicki WB.
Division of Experimental Medicine, Departments of Biochemistry &
Molecular Biology, The George Washington University Medical Center,
2300 Eye St., NW, Ross Hall, Rm. 441A, Washington, DC 20037,
USA. phy...@gwumc.edu

In this study we have tested the effects of d-propranolol (D-Pro) on
the iron uptake, iron release and oxidative response of iron-loaded
cells
in a cellular model of iron-overload using isolated rat peritoneal
macrophages incubated with iron-dextran (Fe-D).
Pretreatment of macrophages with D-Pro (5-200 microM) prior to Fe-D
exposure decreased the cellular iron content and partially prevented
iron release from latex-activated macrophages.
Release of reactive oxygen species from activated cells was detected
by dichlorodihydrofluorescein (DCDHF, 5 microM) oxidation.
We found that loading cells with Fe-D increased their response to
latex, which was prevented by the lysosomotropic antioxidant agent D-
Pro
(10 microM).

PMID: 16718379

http://www.spinabifidaassociation.org/site/c.liKWL7PLLrF/b.2664425/ap...

Who loves ya.
Tom

Jesus Was A Vegetarian!
http://tinyurl.com/2r2nkh

Man Is A Herbivore!
http://tinyurl.com/a3cc3

DEAD PEOPLE WALKING
http://tinyurl.com/zk9fk