Michael Raegan is Ronald Reagan's adopted son, not his biological son. On
the other hand Ron Reagan Jr., Raegan's biological son, is in his early 40's
and has a full untouched head of hair like his father. Reagan is a freak ,
he has an unreceeded teen age hairline at 90. That's the hairline that a
pseudohermaphrodite or a castrate would have at that age.
Alex
"Alex1250" <Alex...@hotmail.com> wrote in message
news:rMbg6.204540$JT5.7...@news20.bellglobal.com...
Too bad he can't remember what hair is.
--
nw3
Sent via Deja.com
http://www.deja.com/
> Michael Raegan is Ronald Reagan's adopted son, not his biological
> son. On the other hand Ron Reagan Jr., Raegan's biological son, is in
> his early 40's and has a full untouched head of hair like his father.
> Reagan is a freak , he has an unreceeded teen age hairline at 90.
> That's the hairline that a pseudohermaphrodite or a castrate would
> have at that age.
>
> Alex
I'd be curious to see what kind of body hair he has... amount-wise, and
how sexual of a guy he is libido-wise... just for information sake.
Kevin
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Steven...@webtv.net wrote in message <12916-3A...@storefull-104.iap.bryant.webtv.net>...The bald son is adopted, he has another son that has a full head of
hair. I believe the son with hair is a step son or may also be adopted.Please explain to me how a step-son is a Jr.. Ronald Prescott Raegan Jr. is Ronald Raegan's biological son with Nancy. Patti Davis is his full sister. If your going to say that somebody is wrong make sure you have the facts straight first.Alex
<< I'd be curious to see what kind of body hair he has... >>
Rumor has it that Nancy has always plucked out his body hairs with tweezers
and applied saw palmetto solution. So for national security, possibly the
FBI has a gag order preventing Ernie from naming him and any others as being
successful with his method. <g>
too bad he can't remember...
>I'll take a bald head any day over the disease that he has. My
>grandmother and my aunt both died from alzheimer's disease. <gulp>
Interestingly enough, the same mechanisms likely operate in Altzheimers as
operate in pattern hair loss. And the same drugs, such as PBN, may be
effective. E.g.:
: Ann N Y Acad Sci 2000;899:222-37
"Nitrone inhibition of age-associated oxidative damage."
Floyd RA, Hensley K
Free Radical Biology and Aging Research Program, Oklahoma Medical Research
Foundation, Oklahoma City, USA. Robert...@omrf.ouhsc.edu
The mechanistic basis of the neuroprotective activity of the nitrone-based free
radical trap PBN (alpha-phenyl-N-tert-butyl nitrone) has been investigated
extensively. Key observations exclude its simple mass action spin trapping of
free radicals activity as the key mechanism of action. These include: A) the
fact that it protects in experimental stroke even if administered several hours
after the event and B) the fact that its chronic low-level administration to old
experimental animals reverses their age-enhanced susceptibility to stroke even
several days after the last dosage. PBN was found to inhibit gene induction in
several models including stroke and an LPS-mediated septic shock model. Stoke
causes inducible nitric oxide synthase (iNOS) to be expressed. High levels of
nitric oxide and peroxynitrite (formed from nitric oxide), produced by iNOS, is
particularly neurotoxic. PBN inhibits iNOS induction. Therefore, it seems that
prevention of the formation of neurotoxic products is a rational mechanism of
action of PBN in the stroke model. There is strong rationale to consider that
there is an enhanced propensity for a "smoldering" neuro-inflammatory state in
the old brain. Reversal of this state by PBN may explain its action in
preventing age-enhanced stroke susceptibility in old experimental animals.
Significant new findings underscore the importance of neuro-inflammatory
processes in neuronal death or dysfunction in Alzheimer's disease.
Neuro-inflammatory processes implicate enhanced signal transduction processes.
Strong evidence for this is the enhanced p38 kinase activation in neurons near
plaques and tangles of the Alzheimer's brain in contrast to normal aged-matched
control brain which did not show p38 activation. In rat primary astrocytes p38
activation by the pro-inflammatory cytokine IL-1 beta, as well as by H2O2, was
significantly suppressed by PBN. Mechanistically it was shown that PBN
suppresses the amount of reactive oxygen species (ROS) produced in mitochondrial
respiration. Much evidence indicates that ROS are signaling molecules and that
they also are involved to maintaining brain phosphatases in an inactive state.
We argue that finding a specific high affinity site mechanism for the
neuroprotective action of PBN is unlikely based on the complexity of the system
reflecting ROS generation and signal transduction processes that have apparently
evolved to maintain adaptive responses. The promising pharmacological activity
of molecules like PBN is not diminished by this however, for only excessive
amounts of ROS is considered detrimental. The action of PBN in suppressing
signal transduction processes, most likely by suppressing ROS production in
mitochondrial respiration, effectively controls excessive oxidative damage and
prevents induction of genes that form neurotoxic products.
So why isn't he losing his hair?
>In article
><4F687876C3AF4A31.D4FF65C9...@lp.airnews.net>,
> ppro...@proctorgamble.com (Peter H. Proctor) wrote:
>> In article <qu848t4qv08jvgo3g...@4ax.com> Herve'
>Joncour <u84...@yahoo.com> writes:
>> >From: Herve' Joncour <u84...@yahoo.com>
>> >Subject: Re: Ronald Reagan's Hair AT 90!
>> >Date: Thu, 08 Feb 2001 04:48:47 GMT
>>
>> >I'll take a bald head any day over the disease that he has. My
>> >grandmother and my aunt both died from alzheimer's disease. <gulp>
>>
>> Interestingly enough, the same mechanisms likely operate in
>Altzheimers as operate in pattern hair loss.
>
>So why isn't he losing his hair?
The _mechanisms_ are similar on the molecular level, the disease
is not, being limited to brain. However, systemic amyloidosis
(Altzheimers is sort of a brain version of this) IS associated with
alopecia. Interestingly, this is reported to respond to a hydroxyl
radical scavenger, DMSO.
Peter H Proctor, PhD, MD
http://www.drproctor.com
Jpn J Med 1987 Aug;26(3):393-5
"Amyloidosis presented with whitening and loss of hair which improved
after dimethylsulfoxide (DMSO) treatment."
Hsieh SD, Yamamoto R, Saito K, Iwamoto Y, Kuzuya T, Ohba S, Kobori S,
Saito K
A 67-year-old male patient presented with rapid progression of
whitening and loss of hair in past 2 months was consulted due to the
suspicion of hypothyroidism. He had been told to have cardiomegaly for
3 years. Thyroid function was within normal limit. Prostate biopsy was
performed because of prostatic hypertrophy and mild elevation of serum
acid phosphatase. Amyloid accumulation was observed in the biopsy
specimen. Subsequent skin biopsies revealed the same result. The scalp
hair and beard grew and turned to black gradually several months after
dimethyl sulfoxide (DMSO) treatment. These findings suggest that some
of the manifestation of amyloidosis may respond to DMSO treatment.
>The _mechanisms_ are similar on the molecular level, the disease
>is not, being limited to brain. However, systemic amyloidosis
>(Altzheimers is sort of a brain version of this) IS associated with
>alopecia. Interestingly, this is reported to respond to a hydroxyl
>radical scavenger, DMSO.
Dr. P, do you have any idea how the DMSO was used?
Bryan
He is not President Reagans biological son.In any case,baldness can skip
generations.If you ever see pics of Pres.Reagan without his shirt,he has no
body hair or 5 o'clock shadow.Ernie
Or someone who removes his body/facial hair from the folicles.I expect to
have that hair when I am 70.I will keep you posted.Ernie