More lies on Congenital Lyme by Dr. Donohue- quoting spin

[Mort Zuckerman]

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Subject: More lies on Congenital Lyme by Dr. Donohue- quoting spin

Date: Oct 16, 2008 6:55 AM

http://www.southcoasttoday.com/apps/pbcs.dll/article?AID=/20081016/LIFE/810160305

Congenital Lyme by Yale, Steere, NIH's and the US Army's Paul Duray
(worked with Steven Hatfill)
http://www.actionlyme.org/Duray.htm
and Willy Burgdorfer:
http://www.actionlyme.org/MOMS_CAN_GIVE_LYME_TO_BABIES.htm
[I have to use baby-talk, since I am trying to educate commoners
and serfs (to use my Rockefeller cousins' terminology) so that's
why such a long string for a URL.]


Here, Bergstrom is CDC officer Alan Barbour's business partner:
http://www.ncbi.nlm.nih.gov/pubmed/17054065?ordinalpos=1&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_DefaultReportPanel.Pubmed_RVDocSum

: J Infect Dis. 2006 Nov 15;194(10):1367-74. Epub 2006 Oct 3.Click
here to read
Links
Complications of pregnancy and transplacental transmission of
relapsing-fever
borreliosis.
Larsson C, Andersson M, Guo BP, Nordstrand A, Hagerstrand I,
Carlsson S, Bergstrom
S.

Department of Molecular Biology, Umea University, Umea, Sweden.

Relapsing-fever borreliosis caused by Borrelia duttonii is a
common cause of
complications of pregnancy, miscarriage, and neonatal death in sub-
Saharan Africa.
We established a murine model of gestational relapsing fever infection
for the study
of the pathological development of these complications. We demonstrate
that B. duttonii
infection during pregnancy results in intrauterine growth retardation,
as well as
placental damage and inflammation, impaired fetal circulation, and
decreased maternal
hemoglobin levels. We show that spirochetes frequently cross the
maternal-fetal
barrier, resulting in congenital infection. Furthermore, we compared
the severity
of infection in pregnant and nonpregnant mice and show that pregnancy
has a protective
effect. This model closely parallels the consequences of human
gestational infection,
and our results provide insight into the mechanisms behind the
complications of
pregnancy that have been reported in human relapsing-fever infection.

=================

See the 563 related articles on congenital relapsing fever:

http://www.ncbi.nlm.nih.gov/sites/entrez?Db=pubmed&DbFrom=pubmed&Cmd=Link&LinkName=pubmed_pubmed&LinkReadableName=Related%20Articles&IdsFromResult=17054065&ordinalpos=1&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_DiscoveryPanel.Pubmed_Discovery_RA&log$=relatedarticles&logdbfrom=pubmed


I note that it is only LYME relapsing fever that is a non-disease
for which we all should be vaccinated every year, but that no
other relapsing fever has a "vaccine" because vaccination is
impossible for an infection whose name implies antigenic
variation.

Antigenic Variation by CDC officer Alan Barbour:

http://www.ncbi.nlm.nih.gov/pubmed/10998374?ordinalpos=9&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_DefaultReportPanel.Pubmed_RVDocSum
: Emerg Infect Dis. 2000 Sep-Oct;6(5):449-57.Links

Comment in:
Emerg Infect Dis. 2001 May-Jun;7(3):486.

Antigenic variation in vector-borne pathogens.
Barbour AG, Restrepo BI.

University of California Irvine, Irvine, California 92697-4025,
USA. abar...@uci.edu

Several pathogens of humans and domestic animals depend on
hematophagous arthropods
to transmit them from one vertebrate reservoir host to another and
maintain them
in an environment. These pathogens use antigenic variation to prolong
their circulation
in the blood and thus increase the likelihood of transmission. By
convergent evolution,
bacterial and protozoal vector-borne pathogens have acquired similar
genetic mechanisms
for successful antigenic variation. Borrelia spp. and Anaplasma
marginale (among
bacteria) and African trypanosomes, Plasmodium falciparum, and Babesia
bovis (among
parasites) are examples of pathogens using these mechanisms. Antigenic
variation
poses a challenge in the development of vaccines against vector-borne
pathogens.

---------

For the same reason, the Dearborn diagnostic criteria is bullshit
in late Lyme borreliosis according to Alan Barbour himself:
http://www.actionlyme.org/BARBOUR_MUTANTS_1992.htm
who states in that article that even OspA undergoes antigenic
variation (meaning he would know his patented ImmuLyme OspA vaccine
was a hoax) and therefore the only antibody we can use is Bb Specific
41 or flagellin- a test developed by Yale's Erol Fikrig in
1991.


Perhaps Pam Weintraub can re-write all of this because it
is all so illogical and confusing that no one can follow
the argument, logic, or even more illogically, look at the
proofs for themselves !


I would like everyone to note that not one other single
person ever complained directly to the USDOJ about this
FRAUD, except myself, yet everyone else involved intends
to make money off of this disease- ONLY:
http://www.actionlyme.org/USDOJ_COMPLAINT_RICO.htm

And yet they're *all* my critics.


THAT'S America.

This is a nation full of *nothing* other greedy cowards.


Kathleen M. Dickson
http://www.actionlyme.org
http://www.relapsingfever.org