The "Lyme-is-only-a-bad-knee" Autopsy Keepers
kathleen
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Subject: [SpinLyme] The "Lyme-is-only-a-bad-knee" Autopsy Keepers
Date: Aug 25, 2007 12:24 AM
Here is Gary Wormser's report where he says the CDC's IgG method to
diagnose
Lyme misses 85% of the cases (detects 9/59), and he says in this
report, that he
is specifically assessing the CDC's (Steere's phoney method) IgG
method:
http://www.pubmedcentral.nih.gov/picrender.fcgi?artid=266355&blobtype=pdf
Here is Imugen, part of the racketeering entity, reporting to the CDC
at the 1994
Dearborn conference, that the CDC's current IgG method to diagnose
Lyme misses
86% of the cases (only detects 14% of the known cases):
http://www.actionlyme.org/FDA_JAN_2001%20027.jpg
Dontchya think that's pretty funny that IDSA says you can only be
treated for
Lyme if you test positive when they all clearly know only 15% of all
people will
ever test positive, condemning the other 85% (who are sick and try to
get some kind
of a diagnosis and therefore TREATMENT for you know, "end-of-sickness"
purposes) to permanent disability?
(I know its silly of me to suggest that "medical treatment is meant to
end
misery" since I live in Corrupticut, Insurance Capitol of Earth and
home of
the psychopathic Bushies.)
Yale's DEAD CONGENITALLY INFECTED BABY autopsy report:
http://www.actionlyme.org/Congenital_Brain_Infection_of_Newborn_Resulting_in_Death.htm
"The death of the newborn was probably due to respiratory failure as a
consequence
of perinatal brain damage." "Mother and infant were 'seronegative'
and there was remarkably no inflammation."
Here is the Lyme infected monkeys autopsy report:
http://www.geocities.com/kmdickson0308/lyme-dilemma.html
http://www.geocities.com/kmdickson0308/3-17.txt
"Nerve changes. A detailed survey of the central nervous system
lesions was
carried out, which included 50 sampling sites. The lesions observed
are listed in
Table 4. Sensory ganglia of the dorsal root and trigeminal ganglia of
animals J
831 and K 216 had individual neurons that immunostained positive with
anti-Bb 7.5kD
lipoprotein mAb (fig 16). These neurons were swollen and were
undergoing chromatolysis.
The dorsal root ganglia of the thoracic and cervical regions were
especially affected.
Nerve sheath fibrosis within the spinal cord was limited to the
thoracic segment
in animal J 831. Positive staining with anti-Bb mAb, accompanied by
vacuolization
of peripheral nerves, was observed in three of four animals. This
change occurred
as a radiculoneuropathy of the thoracic segment and peripheral nerves
(Fig 17).
Animal L 131 had five peripheral nerves affected. When the same nerves
were stained
for fat, focal vacuolization was observed (Figs 18 and 19). Focal
demyelination
of the cervical cord was limited to J 831. Lymphocyte infiltration of
the affected
nerves was mild in extent and confined to perivascular spaces. In
animals L 131
and K 383, Bb could be demonstrated by immunostaining (Fig 20).
"The pathogenesis of Lyme disease neuropathies is poorly
understood...Sensory
ganglia involvement has previously been described in human borreliosis
(27-28).
In our study, many of the ganglia positive for Bb by immunostaining
were undergoing
necrosis. This staining was seen in two out of five animals and was
not accompanied
by a cellular infiltrate.
" Nerve tissues with perivascular lymphocyte infiltrate were present
in three
out of five animals. This was the only lesions where extracellular Bb
could be demonstrated.
Peripheral cutaneous nerves were prominantly affected in the early
phase of borreliosis
of rhesus macaques(20, 28-29). Changes of the nervous system include
the full range
of changes observed in neuroborreliosis, which suggests a variety of
disease mechanisms,
including sensitization or mimicry as suggested by the vacuolization
of peripheral
nerves and cytokine-mediated destruction of nerves as a result of the
infiltrating
lymphocytes..."
But "Lyme is only a bad knee" says all of Yale, all of New York
Medical
College, all of Baxter, all of SmithKline, all the insurance
companies, all of the
CDC, Durland Fish and IDSA and everyone who agrees with IDSA,
including the 9000
Neurologists who signed onto Lou Gehrig's John Halperin:
http://www.actionlyme.org/ALS_&_Lyme_47%25.htm
(Lou Gehrig was a well-known hypochondriac, and in fact, he really
wanted to be
on Broadway because he was a drama queen like all the rest of the
Yuppie Lymies.
This was yet another shameful chapter in the history of baseball,
exposed and debunked
by the famous researchers at Yale.)
Allen Steere CDC "OcCiFer" says: http://www.annals.org/cgi/content/full/121/8/560
"Patient 12 had had high fever, meningeal symptoms, and subsequent
arthritis
in 1982. She was noted to have a positive serologic test result for
Lyme disease
4 years later and was treated with 2 weeks of parenteral penicillin.
She later developed
a progressive speech disorder, bradykinesia, and abnormal ocular motor
function.
Magnetic resonance imaging of the brain showed scattered white matter
lesions in
the hemispheres and pons, and she was diagnosed with supranuclear
palsy. Lumbar
puncture showed no selective concentration of antibody in the spinal
fluid. Nevertheless,
she was re-treated with 2 weeks of parenteral ceftriaxone in 1989 that
had no effect
on her neurologic symptoms. During the time of observation, this
patient died. At
autopsy, lymphoid mononuclear cells were observed surrounding the
intracerebral
vessels in one section. Using Dieterle silver stain, a spirochete was
present in
the cortex and another was exterior to a leptomeningeal vessel."
Because the cortex is in your knee and leptomeninges means the left
knee.
(One of Steere's multiply-treated patients died anyway with
spirochetes in her
brain.)
http://www.actionlyme.org/Biology%20of%20Borrelia%20Species,%201986,%20Barbour.htm
"The propensity for borrelia to go to the brain of infected mammals
suggests
that the relationship between these spirochetes and neural tissues is
not trivial.
Further study of this attraction and the interaction that follows may
reveal the
basis for the significant nerve and brain involvement in Lyme
borreliosis"-
Alan Barbour (CDC Occifer and Wiobeapons Level IV Lab Maganer.)
http://www.ucihs.uci.edu/microbio/index.html?top.html&menu.html&facultyResearch/faculty/barbour.html
"We are taking a multi-discipline approach, including methods of
genetics,
cell biology, and immunology, to study in depth two spirochetal
diseases: Lyme disease
and relapsing fever. These tick-borne infections are notable for
multiphasic antigenic
variation through DNA recombinations in the case of relapsing fever,
the occurrence
of chronic arthritis in the case of Lyme disease, and invasion of and
persistence
in the brain in the case of both diseases." Alan Barbour's sebwite.
CDC Level IV Wiobeapons Occifer Mark Klempner explains the Placebo
Effect:
"Fibroblasts protected B. burgdorferi for at least 14 days of exposure
to ceftriaxone.
Mouse keratinocytes, HEp-2 cells, and Vero cells but not Caco-2 cells
showed the
same protective effect. Thus, several eukaryotic cell types provide
the Lyme disease
spirochete with a protective environment contributing to its long-term
survival."
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=pubmed&cmd=Retrieve&dopt=AbstractPlus&list_uids=1634816&query_hl=29&itool=pubmed_docsum
He meant to say "contributing to the spirochetes' long-term
hystrionics
and playing not-dead."
Vijay Sikand discussing 911-stunt-like matters collectively seen in
persons who
are a "Lyme internet cult," even before there was an internet, and
also
observed in neurosyphilis patients, even before there telephones:
http://www.fda.gov/ohrms/dockets/ac/98/transcpt/3422t1.rtf
"...the specter of asymptomatic infection is something that troubles
me a great
deal and troubles a great number of my colleagues who need to treat
Lyme disease.
The obvious analogy with syphilis infection with Treponema pallidum is
there to
consider. It is well known that Borrelia burgdorferi indeed after
asymptomatic
infection can lurk or secrete itself in certain areas of the body,
perhaps the central
nervous system or perhaps the joint spaces, only to reappear months or
maybe years
later in the form of late stages of illness which are harder to
diagnosis and treat."
The combined National Institutes say: http://intramural.nimh.nih.gov/inip/call4proposals.htm
"8. Infectious diseases of the CNS mediated through immune mechanisms,
including
acute and chronic Lyme disease and neuroAIDS;"
But thanks to Yale, everyone knows HIV is a somatoform illness caused
by a lack
of sex and that Viagra is the cure for HIV. In fact, the high cost of
Viagra is
the nature of the controversy over AIDS.
http://www.actionlyme.org/EARLY_CNS_INVASION.htm
Early Brain Invasion by Jorge Benach. Benach later sent a letter to
the editor
of the New York Times stating that Allen Steere was right, that we
Lyme victims
are delusional, and that "Allen Steere has the science on his side,"
when
you can clearly see that we have Jorge Benach's science as well as
Allen Steere's
on our side.
http://www.actionlyme.org/Dattwyler_Luft_Bb_DNA_in_CSF.htm
Early Brain Invasion by Ray Dattwyler- author of the IDSA "guidelines"
as told to the FDA in 1994.
http://www.actionlyme.org/PACHNER_BRAINS_1990_2.htm
Pachner_Brains_1990; Antigenic variation in the brain. This means you
can't
use the Dressler-Steere antibody method to determine late Lyme in the
brain.
http://www.actionlyme.org/JohnDunn_Brookhaven.htm
John Dunn at Brookhaven says that It's "the perfect stealth pathogen"
that can mistaken for MS, Lupus, can cause excruciating headaches.
Because everyone knows, the brain is either the knee or the penis.
Brought to you by the Yale University School of Uncomplicated
Variables.
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