African Bird Migration Theory (Steere, Fish and Phillips- all Yale's lying, bumbling fools)

[Mort Zuckerman]

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Subject: African Bird Migration Theory...Re: [SpinLyme] Seabirds
spread Lyme disease

Date: Sep 2, 2008 9:53 AM

Stand by.
This document was given in full text to James Phillips in the
fall of 2000, when I also gave him the complete, full text
hard copy of my first website:
http://www.geocities.com/kmdickson0308/lyme-dilemma.html

The report is from the MMWR.
They try to assert that Lyme flew over here on hurricanes
from Africa.

Be-wite back.
http://64.233.169.104/search?q=cache:AXsHgKXVOu8J:www.cdc.gov/ncidod/eid/vol6no4/rappole.htm+perspectives+Israeli+strain+identical+West+Nile&hl=en&ct=clnk&cd=2&gl=us

"The lineage associated with the New York outbreak has been identified
as virtually
identical with an Israeli strain of the virus by phylogenetic analysis
of E-glycoprotein
nucleic acid sequence data (13)."


Cute.

"The New York City strain of the virus was nearly identical to that
found in
the Middle East, which is different from the West African strain (13).
Despite these
considerations, normal migration remains a distinct possibility as the
mode of entry
for the disease."


Do you believe these "storms" theories, or is it likelier that ISRAELI
West Nile was an accidental release, since both AFRICAN diseases
emerged
within 10 miles of Plum Island and messing with AFRICAN vector borne
diseases
happens to be what they *DO* there on Plum Island?

http://www.actionlyme.org/BIOWEAPONEERS_CORIXA_YALE_TLRS.htm
Yale's Durland Fish:
http://www.ncbi.nlm.nih.gov/sites/entrez?Db=pubmed&Cmd=DetailsSearch&Term=9499019[uid

: J Virol. 1998 Mar;72(3):1711-24.Click here to read Click here to
read Links
African swine fever virus infection in the argasid host,
Ornithodoros porcinus
porcinus.
Kleiboeker SB, Burrage TG, Scoles GA, Fish D, Rock DL.

Plum Island Animal Disease Center, Agricultural Research Service,
U.S. Department
of Agriculture, Greenport, New York 11944, USA.

The pathogenesis of African swine fever virus (ASFV) infection in
Ornithodoros
porcinus porcinus was examined in nymphal ticks infected with the ASFV
isolate Chiredzi/83/1.
At times postinfection (p.i.) ranging from 6 h to 290 days, ticks or
dissected tick
tissues were titrated for virus and examined ultrastructurally for
evidence of virus
replication. The ASFV infection rate in ticks was 100% in these
experiments, and
virus infection was not associated with a significant increase in tick
mortality.
Initial ASFV replication occurred in phagocytic digestive cells of the
midgut epithelium.
Subsequent infection and replication of ASFV in undifferentiated
midgut cells was
observed at 15 days p.i. Generalization of virus infection from midgut
to other
tick tissues required 2 to 3 weeks and most likely involved virus
movement across
the basal lamina of the midgut into the hemocoel. Secondary sites of
virus replication
included hemocytes (type I and II), connective tissue, coxal gland,
salivary gland,
and reproductive tissue. Virus replication was not observed in the
nervous tissue
of the synganglion, Malpighian tubules, and muscle. Persistent
infection, characterized
by active virus replication, was observed for all involved tick
tissues. After 91
days p.i., viral titers in salivary gland and reproductive tissue were
consistently
the highest detected. Successful tick-to-pig transmission of ASFV at
48 days p.i.
correlated with high viral titers in salivary and coxal gland tissue
and their secretions.
A similar pattern of virus infection and persistence in O. porcinus
porcinus was
observed for three additional ASFV tick isolates in their associated
ticks.

=======


It might have even been this *particular* slob, Durland Fish,
who was responsible for West Nile accidental release.

I mean, Steere's lab workers inhaled borrelia, right?
http://www.actionlyme.org/CRYMEDISEASE_CHP3_B.htm

How could they be lying about Lyme and also protect their lab workers
by telling them the truth about these pathogens?

Lyme is aerosolizable, because the cyst or spheroplast form is
an environmental deprivation survival form:
http://www.actionlyme.org/BOGUS_RUSSIAN_NYMC_ARTICLES.htm


There is no better proof of this than Allen Steere's lab workers.
Are we expected to believe 4/9 of his lab workings unwittingly
stabbed themselves with a syringe full of spirochetes?
http://www.ncbi.nlm.nih.gov/sites/entrez?Db=pubmed&Cmd=DetailsSearch&Term=1883122[uid]

The T-cell proliferative assay in the diagnosis of Lyme disease.
Dressler F, Yoshinari NH, Steere AC.

Tufts University School of Medicine, New England Medical Center,
Boston, Massachusetts.

OBJECTIVE: To determine the sensitivity and specificity of the T-cell
proliferative
assay as a diagnostic test in Lyme disease. DESIGN: Cross-sectional
study of patients
with Lyme arthritis or chronic neuroborreliosis who had a history of
erythema migrans,
positive antibody responses to Borrelia burgdorferi by enzyme-linked
immunosorbent
assay (ELISA), or both; patients with other diseases; and healthy
subjects. SETTING:
Diagnostic Lyme disease clinic in a university hospital. PATIENTS:
Forty-two of
the 67 patients with active Lyme arthritis or chronic neuroborreliosis
who were
seen during the study period; 16 patients with inactive late Lyme
disease; 77 patients
with other rheumatologic or neurologic diseases; 9 workers from the
Borrelia laboratory;
and 9 healthy subjects. MEASUREMENTS AND MAIN RESULTS: Nineteen of 42
patients with
Lyme arthritis or chronic neuroborreliosis and 4 of 77 patients with
other diseases
had positive T-cell proliferative responses to B. burgdorferi
antigens. The sensitivity
of the proliferative assay was 45% (95% Cl, 30% to 60%) and the
specificity was
95% (95% Cl, 87% to 99%). Twelve of 27 patients with active Lyme
arthritis, 7 of
15 patients with chronic neuroborreliosis, 4 of 16 patients with
inactive Lyme disease,
*** 4 of 9 healthy Borrelia laboratory workers,*** and 0 of 9 healthy
subjects had
positive responses. Three of five patients with Lyme disease who had
negative or
indeterminant antibody responses by ELISA had positive T-cell
proliferative responses.
CONCLUSION: The T-cell proliferative assay may be a helpful diagnostic
test in the
small subset of patients with late Lyme disease who have negative or
indeterminant
antibody responses by ELISA.

Kathleen

-----Original Message-----
>From: Miguel <miki...@pacifier.com>
>Sent: Sep 2, 2008 12:24 AM
>To: Spin...@yahoogroups.com
>Subject: [SpinLyme] Seabirds spread Lyme disease
>
> Studies from D. Duneau and co-researchers yield new data on borrelia.
> Biotech Week. August 27, 2008 p149.
>
>
>
>
> Full Text: COPYRIGHT 2008 NewsRX
>
>
>
>
>
> According to recent research published in the journal Infection Genetics
and Evolution, "A potential role of seabirds in spreading Lyme disease
(LB)
spirochetes over large spatial scales was suggested more than 10 years
ago when
Borrelia garinii was observed in marine birds of both hemispheres.
Since then, there
have been few studies examining the diversity of Borrelia spp.
circulating in seabirds,
or the potential interaction between terrestrial and marine disease
cycles."
>
> "To explore these aspects, we tested 402 Ixodes uriae ticks collected
from five colonial seabird species by amplification of the flaB gene.
Both the average
prevalence (26.0% +/- 3.9) and diversity of LB spirochetes was high.
Phylogenetic
analyses grouped marine isolates in two main clades: one associated
with B. garinii
and another with B. lusitaniae, a genospecies typically associated
with lizards.
One sequence also clustered most closely with B. burgdorferi! sensu
stricto. Prevalence
in ticks varied both among seabird species within colonies and among
colonies. However,
there was no clear association between different Borrelia isolates and
a given seabird
host species. Our findings indicate that LB spirochetes circulating in
the marine
system are more diverse than previously described and support the
hypothesis that
seabirds may be an important component in the global epidemiology and
evolution
of Lyme disease," wrote D. Duneau and colleagues (see also Borrelia).
>
> The researchers concluded: "Future work should help determine the
extent to which isolates are shared between marine and terrestrial
systems."
>
> Duneau and colleagues published their study in Infection Genetics and
Evolution (Prevalence and diversity of Lyme borreliosis bacteria in
marine birds.
Infection Genetics and Evolution, 2008;8(3):352-359).
>
> For additional information, contact D. Duneau, IRD, CNRS IRD, UMR 2724,
911 Avenue Agropolis, BP 64501, F-34394 Montpellier, France.
>
> The publisher's contact information for the journal Infection Genetics
and Evolution is: Elsevier Science BV, PO Box 211, 1000 AE Amsterdam,
Netherlands.
>
>
>
>[Non-text portions of this message have been removed]
>
>
>------------------------------------