> On Sep 6, 9:54 pm, bake
...@yahoo.com wrote:
> > People have been attributing every other mental illness to head
> > injury.http://en.wikipedia.org/wiki/Kleptomania
> > has a citation:http://www.ncbi.nlm.nih.gov/sites/entrez?cmd=Retrieve&db=pubmed&dopt=...
> > There is a research report:
> > Kleptomania After Head Trauma: Two Case Reports and Combination
> > Treatment Strategies
> > By Aizer, Anat MD; Lowengrub, Katherine MD; Dannon, Pinhas N MD>From The Rehovot Community Mental Health & Rehabilitation Center, Tel
> > Aviv University, Rehovot Israel.
> > and here is the actual article:http://ljfind.com/post/57031263/
> > There are 2 case reports. One is an army major and the other is a
> > Cyprus resident. Lot of things can happen after a head injury while
> > the brain tries to recover and goes into an overactive state and
> > remain in that state for months. Were those people had any co-morbid
> > condition in addition to stealing such as hyperactivity etc? Also,
> > were they on medication and what type?
> > I don't see any co-relation between kleptomania and head injury. There
> > is a connection to psychopathy and head injury. However putting the
> > Kleptomaniacs also in the psycopathic category may be considered
> > misdiagnosis.
> > '`'`'`'`'`'`'`'`'`'`'`'`'`''`'`'`'`'`'`'`'`'`'`'`'`'`'`'`'`'`'`'`'`'`'`'`
> > sci.psychology.research is a moderated newsgroup.
> > Before submitting an article, please read the guidelines which are posted
> > here bimonthly or the charter on the web athttp://psychcentral.com/spr/
> > Submissions are acknowledged automatically.
> A neural basis for collecting behaviour in humans
> http://brain.oxfordjournals.org/cgi/content/full/128/1/201
> Steven W. Anderson, Hanna Damasio and Antonio R. Damasio
> Collecting behaviour is commonplace in the normal population, but
> there has been little investigation of its neural basis in humans. The
> observation that collecting behaviour can assume pathological
> proportions in patients with certain patterns of brain damage led us
> to hypothesize that dysfunction in a system encompassing mesial
> prefrontal cortices accounts for abnormal collecting and may guide
> normal collecting. We tested the hypothesis in 86 subjects with focal
> lesions of the telencephalon, by relating the neuroanatomical
> placement of the lesions to the presence of repetitive and
> indiscriminate acquisition behaviour and impaired discard behaviour.
> The subjects had no history of psychiatric disease or abnormal
> collecting behaviour prior to lesion onset. Lesions were analysed with
> high-resolution three-dimensional MRI. Collecting behaviour was
> evaluated with a standardized questionnaire completed by a close
> relative of each subject. Thirteen subjects exhibited abnormal
> collecting, characterized by massive and disruptive accumulation of
> useless objects. In all cases, the abnormality of collecting behaviour
> was severe and persisted despite attempted interventions and obvious
> negative consequences. There were no differences between pathological
> collectors and non-collectors on tests of executive functions or
> anterograde memory. All subjects with pathological collecting
> behaviour had damage to the mesial frontal region (including the right
> polar sector and the anterior cingulate), but there was no damage to
> most of the subcortical structures that, in species such as rodents,
> are known to drive the acquisition and retention of objects. The
> evidence suggests that damage to the mesial frontal region disrupts a
> mechanism which normally modulates subcortically driven
> predispositions to acquire and collect, and adjusts these
> predispositions to environmental context.
> '`'`'`'`'`'`'`'`'`'`'`'`'`''`'`'`'`'`'`'`'`'`'`'`'`'`'`'`'`'`'`'`'`'`'`'`
> sci.psychology.research is a moderated newsgroup.
> Before submitting an article, please read the guidelines which are posted
> here bimonthly or the charter on the web athttp://psychcentral.com/spr/
> Submissions are acknowledged automatically.- Hide quoted text -
> - Show quoted text -
area of the brain.
On Table 2, I don't see any data that supports that hypothesis. Then
metabolism in the brain but there is no data to support it. Where is