No paradox here: saturated fatty acids protect the liver.

[mont...@lycos.com]

It's been noted (as insiders already know):

"The protective effect of dietary saturated fatty acids against the
development of alcoholic liver disease has long been known..."

Source: Hepatology. 2005 Sep;42(3):568-77.

Role of adiponectin in the protective action of dietary saturated fat
against alcoholic fatty liver in mice.

You M, Considine RV, Leone TC, Kelly DP, Crabb DW.

Such studies as the following are common:

Dietary saturated fat reduces alcoholic hepatotoxicity in rats by
altering fatty acid metabolism and membrane composition.

Ronis MJ, Korourian S, Zipperman M, Hakkak R, Badger TM.

Arkansas Children's Nutrition Center, Department of
Pharmacology/Toxicology, University of Arkansas for Medical Sciences,
Little Rock, AR 72205, USA. Ronis...@uams.edu

Rats fed a saturated fat diet are protected from experimentally
induced alcoholic liver disease, but the molecular mechanisms
underlying this phenomenon remain in dispute. We fed male
Sprague-Dawley rats intragastrically by total enteral nutrition using
diets with or without ethanol. In 1 control and 1 ethanol group, the
dietary fat was corn oil at a level of 45% of total energy. In other
groups, saturated fat [18:82 ratio of beef tallow:medium-chain
triglyceride (MCT) oil] was substituted for corn oil at levels of 10,
20, and 30% of total energy, while keeping the total energy from fat at
45%. After 70 d, liver pathology, serum alanine aminotransferase (ALT),
biochemical markers of oxidative stress, liver fatty acid composition,
cytochrome P450 2E1 (CYP2E1) expression and activity and cytochrome
P450 4A (CYP4A) expression were assessed. In rats fed the corn oil plus
ethanol diet, hepatotoxicity was accompanied by oxidative stress. As
dietary saturated fat content increased, all measures of hepatic
pathology and oxidative stress were progressively reduced, including
steatosis (P < 0.05). Thus, saturated fat protected rats from alcoholic
liver disease in a dose-responsive fashion. Changes in dietary fat
composition did not alter ethanol metabolism or CYP2E1 induction, but
hepatic CYP4A levels increased markedly in rats fed the saturated fat
diet. Dietary saturated fat also decreased liver triglyceride, PUFA,
and total FFA concentrations (P < 0.05). Increases in dietary saturated
fat increased liver membrane resistance to oxidative stress. In
addition, reduced alcoholic steatosis was associated with reduced fatty
acid synthesis in combination with increased CYP4A-catalyzed fatty acid
oxidation and effects on lipid export. These findings may be important
in the nutritional management and treatment of alcoholic liver disease.

In contrast, however:

Free Radic Biol Med. 2002 Jan 1;32(1):38-45.

Lipid peroxidation contributes to immune reactions associated with
alcoholic liver disease.

Mottaran E, Stewart SF, Rolla R, Vay D, Cipriani V, Moretti M, Vidali
M, Sartori M, Rigamonti C, Day CP, Albano E.

Increasing evidence indicates the involvement of immune reactions in
the pathogenesis of alcoholic liver disease. We have investigated
whether ethanol-induced oxidative stress might contribute to immune
response in alcoholics. Antibodies against human serum albumin modified
by reaction with malondialdehyde (MDA), 4-hydroxynonenal (HNE),
2-hexenal, acrolein, methylglyoxal, and oxidized arachidonic and
linoleic acids were measured by ELISA in 78 patients with alcoholic
cirrhosis and/or hepatitis, 50 patients with nonalcoholic cirrhosis, 23
heavy drinkers with fatty liver, and 80 controls. Titers of
IgG-recognizing epitopes derived from MDA, HNE, and oxidized fatty
acids were significantly higher in alcoholic as compared to
nonalcoholic cirrhotics or healthy controls. No differences were
instead observed in the titers of IgG-recognizing acrolein-,
2-hexenal-, and methylglyoxal-modified albumin. Alcoholics showing high
IgG titers to one adduct tended to have high titers to all the others.
However, competition experiments showed that the antigens recognized
were structurally unrelated. Anti-MDA and anti-HNE antibodies were
significantly higher in cirrhotics with more severe disease as well as
in heavy drinkers with cirrhosis or extensive fibrosis than in those
with fatty liver only. We conclude that antigens derived from lipid
peroxidation contribute to the development of immune responses
associated with alcoholic liver disease.

Understand the scientfic reality of health and "disease" at:

http://groups.msn.com/TheScientificDebateForum-/

No need to be confused by all the conflicting "studies" the mainstream
media likes to tout (most people don't realize that for every study the
media emphasizes there are hundreds if not thousands of others on the
same subject that have never been mentioned by them).

[Mr. Natural-Health]

Will the spammer known as Monty 'the flatuence Kook' kindly stop
spamming smn?

I have absolutely no risk of developing alcoholic liver disease.

Just thought that you might want to know.

[mont...@lycos.com]

You are just too funny, nature boy. A month or so ago you were
claiming that because I didn't have a web site that I was not as
credible as you are, which is hilarious on two counts; one, because
anyone who has ever posted on a newsgroup has more credibility than
you, and two, because when I put together a web site so that I don't
have to tell people to go searching through my old posts, you call it
spam, which relates to someone trying to sell a service or product. I
have neither to sell, and am providing an alternative interpetation of
the scientfic evidence. There would be no reason for anyone to read a
newsgroup such as this if they were just going to listen to everything
their doctors told them to do without question, but you don't have the
ability or the temperment to understand something as simple as that
point. I just hope others find your rantings, free of citations from
the appropriate literature and usually incoherent, to be as humorous as
I do. Forn me, and I suspect for almost everyone else who reads these
posts, you are the "village idiot" of sci.med.nutrition

[Mr. Natural-Health]

mont...@lycos.com wrote:

> You are just too funny, nature boy. A month or so ago you were
> claiming that because I didn't have a web site that I was not as
> credible as you are

And, you still do NOT. A group is NOT a web site. It is just another
form of a newsgroup.

Nor, is a Blog a web site for that matter.

Of course, all of this information is above your intellectual capacity.

> because when I put together a web site so that I don't
> have to tell people to go searching through my old posts, you call it
> spam, which relates to someone trying to sell a service or product.

Sorry, but you are the village idiot, here. You do NOT even know the
definition of spam after being on a ng for over 3 years.

You have my condolences.

And, STOP trying to sell your little group of Flatulence Freaks on this
ng ... Spammer!

Who says so? I do. And, I do not mind saying so. :)

[mont...@lycos.com]

"What is spam?

Spam is flooding the Internet with many copies of the same message, in
an attempt to force the message on people who would not otherwise
choose to receive it. Most spam is commercial advertising, often for
dubious products, get-rich-quick schemes, or quasi-legal services. Spam
costs the sender very little to send -- most of the costs are paid for
by the recipient or the carriers rather than by the sender.

There are two main types of spam, and they have different effects on
Internet users. Cancellable Usenet spam is a single message sent to 20
or more Usenet newsgroups. (Through long experience, Usenet users have
found that any message posted to so many newsgroups is often not
relevant to most or all of them.) Usenet spam is aimed at "lurkers",
people who read newsgroups but rarely or never post and give their
address away. Usenet spam robs users of the utility of the newsgroups
by overwhelming them with a barrage of advertising or other irrelevant
posts. Furthermore, Usenet spam subverts the ability of system
administrators and owners to manage the topics they accept on their
systems.

Email spam targets individual users with direct mail messages. Email
spam lists are often created by scanning Usenet postings, stealing
Internet mailing lists, or searching the Web for addresses. Email spams
typically cost users money out-of-pocket to receive. Many people -
anyone with measured phone service - read or receive their mail while
the meter is running, so to speak. Spam costs them additional money. On
top of that, it costs money for ISPs and online services to transmit
spam, and these costs are transmitted directly to subscribers.

One particularly nasty variant of email spam is sending spam to mailing
lists (public or private email discussion forums.) Because many mailing
lists limit activity to their subscribers, spammers will use automated
tools to subscribe to as many mailing lists as possible, so that they
can grab the lists of addresses, or use the mailing list as a direct
target for their attacks."

Source: http://spam.abuse.net/overview/whatisspam.shtml

[Mr. Natural-Health]

It is now Monty and his postings to promote his group, such as this
thread.
http://groups.google.com/group/sci.med.nutrition/msg/95bd8e62060d1f10?hl=en&

[msamso...@yahoo.ca]

mont...@lycos.com wrote:
. I just hope others find your rantings, free of citations from
> the appropriate literature and usually incoherent, to be as humorous as
> I do.

Yep.

Forn me, and I suspect for almost everyone else who reads these
> posts, you are the "village idiot" of sci.med.nutrition

Indeed.

I read the nature boy's website and it's a complete joke. He obviously
has to resort to immature name calling since his intellect comes
nowhere near yours.

Ignore him for he is a troll. Don't waste your time Monty. I may not
completely agree with your diet, but know you are alot smarter than the
nature boy.

[Mr. Natural-Health]

Monty, Jr.,

Let me introduce you to your leader, the King of Flatulence: Montygram.

You two deserve each other.

Now, kindly go to Monty's group and stop pestering me you, Kook!