[RML]: Durland Fish's "bogus articles" is the reason for his stalking and hysteria
Mort Zuckerman
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Subject: [RML]: Durland Fish's "bogus articles" is the reason for his
stalking and hysteria
Date: Feb 27, 2010 7:41 AM
Rocky Mountain Bioweapons Lab says
OspA vaccination does the opposite
of what Yale and SmithKline claimed
(they claimed OspA vaccination of humans
disinfected ticks; ie. Yale wanted to
turn humans into walking canisters of
tick disinfectant).
http://www.actionlyme.org/TICK_BITE_CONSPIRACY.htm
This must be the reason Durland Fish
started his own journal. They wanted
to have a platform to continue their
LIES about OspA in order to avoid
prosecution for scientific fraud
and racketeering.
KMDickson
http://www.actionlyme
===============================
http://www.ncbi.nlm.nih.gov/pubmed?term=18779341[uid]&cmd=DetailsSearch&log$=details
Infect Immun. 2008 Nov;76(11):5228-37. Epub 2008 Sep 8.
Outer surface protein A protects Lyme disease spirochetes from
acquired host immunity in the tick vector.
Battisti JM, Bono JL, Rosa PA, Schrumpf ME, Schwan TG, Policastro PF.
Rocky Mountain Laboratories, NIAID, NIH, 903 S. 4th Street, Hamilton,
MT 59840, USA.
The Lyme disease spirochete Borrelia burgdorferi alters the expression
of outer surface protein (osp) genes as the bacterium cycles between
ticks and mammals. OspA is produced as borreliae enter the tick vector
and remains a major surface antigen during midgut colonization. To
elucidate the role of OspA in the vector, we created an insertional
deletion of ospA in strain B31-A3. The ospA mutant infects mice when
it is injected intradermally and is acquired by larval ticks fed on
these mice, where it persists through the molt to the nymph stage.
Bacterial survival rates in artificially infected tick larvae fed on
naïve mice were compared with those in the vector fed on immune mice.
The ospA mutant proliferates in larvae if it is exposed to blood from
naïve mice, but it declines in density after larval feeding if the
blood is from immune mice. When uninfected larvae are fed on B-cell-
deficient mice infected with the ospA mutant, larvae show borrelial
densities and persistence that are significantly greater than those
fed on infected, immunocompetent mice. We conclude that OspA serves a
critical antibody-shielding role during vector blood meal uptake from
immune hosts and is not required for persistence in the tick vector.
PMID: 18779341 [PubMed - indexed for MEDLINE]
PMCID: PMC2573341
Publication Types, MeSH Terms, Substances
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