Subject: [SpinLyme] USDOJ's Phantom Limb DSM Disorder
Date: Nov 16, 2009 7:58 PM
Way, way back a hunderd yearn ago
in 1988, there was 2 kindza Lyme,
as schoen hearin nis repord:
http://www.ncbi.nlm.nih.gov/pubmed?term=3190104[uid]&cmd=DetailsSearch&log$=details
Now watches keerfully:
"Ann N Y Acad Sci. 1988;539:56-64.
Borrelia burgdorferi in the nervous system: the new "great imitator".
Pachner AR.
Department of Neurology, Georgetown University School of Medicine,
Washington, D.C. 20007.
There are many obvious similarities between Lyme disease and syphilis.
The major ones are their spirochetal etiology, the ability of the
spirochetes to stay alive in human tissue for years, occurrence of
clinical manifestations in stages, early disease in the skin and later
disease in the brain, and susceptibility to antibiotic treatment.
Thus, one can assume that many of the same lessons learned from the
centuries of experience with syphilis will apply to Lyme disease. One
of these lessons that should be constantly borne in mind is that
spirochetal disease of the brain can mimic many other neurological
diseases. Thus, the "effective clinician" must take special care to
consider Lyme disease primarily because of the excellent response to
antibiotics early in its course in relationship to some of the
diseases it mimics. Lyme meningitis, occurring in the "second stage"
of the disease, usually is fairly easily recognized because it occurs
in the summer or early fall, often is associated with ECM or a recent
history of it, and has a characteristic clinical picture of
lymphocytic meningoradiculoneuritis. Many patients with Lyme
meningitis or ECM have very mild symptoms, and it is likely that a
large percentage of patients go undiagnosed and untreated. The
frequency of progression of these patients to third-stage disease is
unknown but may be quite high. This can be inferred from a similar
situation in the other major late manifestation of Lyme disease: Lyme
arthritis. A large number of patients present with joint involvement
as their only manifestation of Lyme disease. Similarly, patients may
present with symptoms of third-stage Lyme disease affecting the CNS,
but they may not be recognized because of the lack of earlier stages
usually associated with the disease. *** Thus, serology has become a
very important tool for identifying patients exposed to B.
burgdorferi. At the present time, serologic tests are the key to
diagnosis of Lyme disease in its later stages, since, as in
neurosyphilis, cultures and tests for antigen have not proven useful.
Lyme arthritis and acrodermatitis atrophicans (ACA) both are
associated with quite high antibody titers to the organism, while the
test is understandably unreliable for identification of patients with
ECM. Antibody titers in Lyme meningoradiculoneuritis are generally
positive but often are not as high as those in ACA or arthritis.***
http://www.actionlyme.org/USDOJ_COMPLAINT_RICO.htm
The antibody response in serum in CNS Lyme disease seems to be related
to the presence of other manifestations; patients who have had both
arthritis and CNS disease have quite high titers, while those with
only CNS disease sometimes do not."
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http://www.actionlyme.org/MKLEPMER.htm
http://www.journals.uchicago.edu/doi/pdf/10.1086/432733?cookieSet=1
"There appear to be at least 2 distinct syndromes in patients with
persistent symptoms after antibiotic treatment. One syndrome has
localized symptoms that are similar to pretreatment symptoms.
Patients with this syndrome often have recurrent episodes of
arthritis/synovitis. Results of synovial fluid cultures and polymerase
chain reaction (PCR) for B. burgdorferi are negative [2].
***Patients generally feel well aside from their arthritis
symptoms."***
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"'Lyme Disease' is just the knee; 'Lyme Borreliosis' is everything
else"- Yale and IDSociety.org:
http://www.actionlyme.org/MUNCHAUSENS.htm
There ^^ it is in black and white.
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Most of my visitors are from
university domains in Europe,
Japan, and South America because
nobody in America knows how to
think. And the lawyers in the USDOJ,
in particular, don't have any balls
despite their cerebropenile inversion.
Their mental disorder is kinda like
phantom limb. They don't have any
maleness, so they obsess over it.
And Anthony Fauci clearly has
Asperger's since that's the mental
disorder where all you do is talk
and talk, but your Right Brain
is shut down:
http://kidshealth.org/teen/diseases_conditions/learning/learning_disabilities.html
"People with nonverbal learning disabilities may have difficulty
processing what they see. They may have trouble making sense of visual
details like numbers on a blackboard. Someone with a nonverbal
learning disability may confuse the plus sign with the sign for
division, for example. Some abstract concepts like fractions may be
difficult to master for people with nonverbal learning disabilities."
I don't mind Aspergery people like
Fauci having jobs, just not as the
head of the NIAID because that's a
scientific leadership position.
It's too dangerous.
Kathleen M. Dickson
http://www.actionlyme.org
http://www.relapsingfever.org
"[Real] scientists are *fiercely* independent. That's the good
news."-- NIH's Top Fool, Anthony Fauci
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