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Subject: Oncovirus Nobel Winner Neglects Myco-Type VBDs and Other
Fungal-Viral Assaults
Date: Sep 14, 2011 7:01 AM
Oncovirus Nobel Winner Neglects Myco-Type VBDs and Other Fungal-Viral
Assaults
Below.
Nice article, and which supports the Harold
Varmus criticism of Yale'd criminal shenanigans
with the diagnostic standard for Lyme - falsified
in order to falsely qualify LYMErix - in the NYTimes:
http://www.actionlyme.org
But we're neglecting to mention the
fungal causes of cancer thru the
activation of latent Epstein Barr
(mentioned below), here.
And this HPV expert forgets to mention that
while Papilloma virus causes cancer, there
is a much higher rate of infertility
due to fungal infections such as chlamydia
and plain old mycoplasma.
That is, the silent STDs. The ones promoted
via the false sense of security re the sexual
promiscuity prescribed by psychiatry as a
cure for all diseases (especially vector
borne diseases), the HPV vaccine affords.
Allow me to elaborate:
[CHLAMYDIA and INFERTILITY]
http://www.ncbi.nlm.nih.gov/pubmed?term=%28%22chlamydia%22[MeSH%20Terms]%20OR%20%22chlamydia%22[All%20Fields]%29%20AND%20%28%22infertility%22[MeSH%20Terms]%20OR%20%22infertility%22[All%20Fields]%29&cmd=DetailsSearch
And as regards mycoplasma and infertility,
well, that was the specialty of the murdered
Iraq Plum Island scientist:
http://www.actionlyme.org/PIIB.htm
OUTLINE:
I. Mycoplasma and Vector-Pathogen Competence Studies performed on Plum
Island
Selling such fungi and other biological and chemical weapons to Saddam
Hussein
http://www.actionlyme.org/MIDDLETOON.pdf
and Robin Cook);
Dead Iraqi scientist who worked with mycoplasma and allegedly on Plum
Island (run over while changing a flat, much like Don Wiley)
http://www.ncbi.nlm.nih.gov/sites/entrez?Db=pubmed&Cmd=DetailsSearch&Term=Al-Aubaidi+jm[Author
Mycoplasma cause infertility and reduced milk production in livestock
Mycoplasma-Membrane Associated Lipoproteins inhibit the auto-kill
kinases resulting in the activation of latent Epstein-Barr:
http://www.ncbi.nlm.nih.gov/sites/entrez?Db=pubmed&Cmd=DetailsSearch&Term=16889623[uid]
Mycoplasma and Leukemias
http://www.ncbi.nlm.nih.gov/pubmed/14276278
Mycoplasma and other tumors:
http://www.pubmedcentral.nih.gov/articlerender.fcgi?tool=pubmed&pubmedid=14329455
Isolation of mycoplasma from Leukemic bone marrow:
http://www.ncbi.nlm.nih.gov/pubmed/14287426
"Pleuropneumonia-like organisms associated with neoplastic disease":
http://www.ncbi.nlm.nih.gov/pubmed/14243415
Mycoplasma are molds that cause plant rot
Stinking Smut and Iraq
http://www.antiwar.com/prather/?articleid=10989
And so on...
The whole thing is silly. Not one
scientist standout in the entire
USA and especially not in the entire
NIH.
How hard is it to tabulate the scientifically
valid biomarkers? Has anyone ever heard of...
wait for it..
a computer database?
KMDickson
http://www.actionlyme.org
==========================
http://www.familypracticenews.com/news/more-top-news/single-view/oncovirus-expert-cattle-may-transmit-human-cancers/5c864766dc.html
Current estimates hold that 21% of all human cancers are linked to
infections. But some experts believe this figure is too low and is
headed substantially higher.
Fueled by the success of vaccines for the hepatitis B virus and high-
risk human papillomaviruses, which offer the first-ever means of
preventing specific widespread cancers by vaccination, investigators
are hunting for additional infectious causes of common malignancies.
The search is focused on viruses, since they are already implicated in
64% of the known infection-related cancer burden, with bacteria and
parasites accounting for the remainder.
Dr. Harald zur Hausen
One of the world’s most celebrated oncovirus hunters is Dr. Harald zur
Hausen, winner of the 2008 Nobel Prize in Medicine for his work on
human papillomaviruses as the major cause of cervical cancer. He sees
a number of other malignancies as being prime candidates for potential
linkage to infections, including childhood lymphoblastic leukemias,
basal cell carcinomas, Epstein-Barr virus–negative Hodgkin’s
lymphomas, colorectal and breast cancers, and lung cancers in
nonsmokers.
The TTV (torque teno virus) family shows particular promise in this
regard. The TTVs, first described by Japanese investigators in 1997
(Rev. Med. Virol. 2007;17:45-57), are an extremely heterogeneous
family of single-strand DNA viruses. There are well over 100
genotypes. The torque teno viruses are widespread in all human
populations. They have been found in umbilical cord blood and are
vertically transmitted from mother to child, even prenatally. They
frequently rearrange their genomes; and transmissibility and
replication have been demonstrated to occur even for small portions of
the TTV genome, according to Dr. zur Hausen, professor emeritus at the
German Cancer Research Center, Heidelberg.
The TTVs are known to replicate in lymphatic and bone marrow cells –
the precursor cells of leukemia. In every cell line of acute
lymphoblastic leukemia and Epstein-Barr virus–negative Hodgkin’s
lymphoma analyzed by Dr. zur Hausen and his coworkers, they have found
the same highly conserved region of TTV DNA.
Of note, TTV load is known to be reduced by interferon. This finding
is consistent with the epidemiologic observation that infants who
experience multiple upper respiratory infections and other infections
during their first year of life seem to be protected against childhood
leukemias. The hypothesis is that bursts of increased interferon
production in response to multiple infections prevents levels of an
interferon-sensitive putative leukemogenic agent – perhaps a TTV –
from reaching critical mass.
Dr. zur Hausen is particularly attracted to the possibility that
cattle may play a role in some human cancers. He hypothesizes that
some as-yet unidentified bovine virus, which is nononcogenic in its
normal host, can become carcinogenic when transmitted to humans.
Multiple lines of circumstantial evidence support this notion. For
example, basal cell carcinomas are known to have a predisposition to
arise in smallpox vaccination scars. Smallpox vaccines were prepared
by inoculating vaccinia virus into the skin of calves and then
harvesting the crusted skin, which could in theory contain
contaminating bovine viruses.
Also, colorectal cancer, and to a lesser extent breast cancer and lung
cancer in nonsmokers, have repeatedly been associated with beef
consumption in epidemiologic studies. Countries with high consumption
of goat or pork have relatively low rates of these malignancies.
The observed link between a red meat–rich diet and increased rates of
colorectal and other cancers is often attributed to the formation of
aromatic hydrocarbons and other known carcinogens during cooking or
meat curing. But Dr. zur Hausen believes this interpretation might be
inadequate. He noted that grilled, fried, and smoked poultry contain
similar concentrations of these carcinogens, yet heavy consumption of
poultry hasn’t been associated with an increased cancer risk.
It is of interest that the temperature achieved in the center of a
piece of beef cooked rare is only 40 -50° C, yet torque teno viruses,
papillomaviruses, and polyomaviruses are able to survive in a protein
environment at temperatures of 80° C for 30 minutes or more.
"These are just suggestions. They do not prove anything, of course.
But, for now, we can speculate that beef consumption plays a
significant role in the development of colorectal cancer," Dr. zur
Hausen said.
If even a small portion of these speculations turn out to be true, the
implications for cancer prevention could be huge. It is estimated that
if both the hepatitis B vaccine and the HPV vaccine were to be applied
globally, the overall cancer burden in women could be reduced by
12%-14% and in men by 4%-5%, he noted.
He presented his theory at the World Congress of Dermatology in Seoul,
South Korea.
Dr. zur Hausen declared having no financial conflicts.
KMDickson