The Queen Says not to believe any of the dot guvs on "Somatoform Diseases"
Mort Zuckerman
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Subject: The Queen Says not to believe any of the dot guvs on
"Somatoform Diseases"
Date: Mar 2, 2010 9:57 AM
http://www.actionlyme.org/index.htm
The Queen of America says all those diseases which are said to be
"psychiatric," such as Fibromyalgia (a trap diagnosis- don't fall for
http://www.actionlyme.org/081030.htm
it), Chronic Fatigue Syndrome, Gulf War Illness
http://www.actionlyme.org/ROCKET_SCIENCE.htm
, Chronic Lyme (better described as LYMErix-AIDS to call attention to
the fact that Yale staff abused Lyme and LYMErix victims and lied to
the FDA about their unreadable Western Blots, instead of investigating
why LYMErix failed),
http://www.actionlyme.org/DICKSON_FDA_SUBMISSION_FULL.htm
http://www.actionlyme.org/Pam3Cys_Version15.htm
the immune damage from hypervaccination of children, and even many
autoimmune diseases are not caused by the primary infection, such as
is the case with Diabetes Type I, but are caused by the secondary
opportunistics. The reason for this USA.gov abuse is to hide the
known mechanisms of Stealth Disablers.
http://www.actionlyme.org/JohnDunn_Brookhaven.htm
This is the reason the SUNY-SB Medical Library threw out all their
older medical journals. They did not want scientists looking back for
evidence, for example, that all borrelioses are permanent brain
infections.
http://www.actionlyme.org/RICOCHRON.htm
http://www.actionlyme.org/BRAIN_PERMANENT.htm
Across-the-board, "American" "scientists" have obscured the facts:
1) "These results extended previous studies with ceftriaxone,
indicating that antibiotic treatment is unable to clear persisting
spirochetes, which remain viable and infectious, but are nondividing
or slowly dividing."
http://www.ncbi.nlm.nih.gov/pubmed?term=19995919[uid]&cmd=DetailsSearch&log$=details
2) "Maternal infection with Trypanosoma cruzi and congenital Chagas
disease induce a trend to a type 1 polarization of infant immune
responses to vaccines."
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2796860/?tool=pubmed
Sort by Pub Date:
http://www.ncbi.nlm.nih.gov/sites/entrez?db=pubmed&cmd=link&linkname=pubmed_pubmed&uid=19396036&ordinalpos=3
3) "Screening for B- and T-cell defects in Egyptian infants and
children with suspected primary immunodeficiency."
http://www.ncbi.nlm.nih.gov/pubmed?term=19396036[uid]&cmd=DetailsSearch&log$=details
4) "Measles virus (MV) causes transient but profound immunosuppression
resulting in increased susceptibility to secondary bacterial and viral
infections. Due to the development of these opportunistic infections,
measles remains the leading vaccine-preventable cause of child death
worldwide. Different immune abnormalities have been associated with
measles, including disappearance of delayed-type hypersensitivity
reactions, impaired lymphocyte and antigen-presenting cell functions,
down-regulation of pro-inflammatory interleukin 12 production and
altered interferon alpha/beta signalling pathways. Several MV proteins
have been suggested to hinder immune functions: hemagglutinin, fusion
protein, nucleoprotein and the non-structural V and C proteins. This
review will focus on the novel functions attributed to MV proteins in
the immunosuppression associated with measles. Here, we highlight new
advances in the field, emphasising the interaction between MV proteins
and their cellular targets, in particular the cell membrane receptors,
CD46, CD150, TLR2 and FcgammaRII in the induction of immunological
abnormalities associated with measles."
http://www3.interscience.wiley.com/journal/112125331/abstract?CRETRY=1&SRETRY=0
5) "Since the possibility of interruption of latent EBV infection has
been suggested by the induction of the lytic virus cycle with chemical
substances, other viruses, and by immunosuppression, we hypothesized
that the same effect might happen in B. burgdorferi sensu lato
infection as happens in Lyme disease patients with positive serology
for both agents. We have observed EBV replication in lymphoblastoid
cells after superinfection with B. garinii and B. afzelii strains
after 1 and 4 h of their interaction. We found that viral and
borrelial antigens persisted in the lymphoblasts for 3 and 4 days.
Morphological and functional transformation of both agents facilitate
their transfer to daughter cells. Association with lymphoblasts and
internalization of B. garinii by tube phagocytosis increased
replication of viruses more successfully than B. afzelii and chemical
inductors. Demonstration of such findings must be interpreted
cautiously, but may prove a mixed borrelial and viral cause of severe
neurological disease."
http://www.ncbi.nlm.nih.gov/pubmed?term=12630667[uid]&cmd=DetailsSearch&log$=details
6) "These revised recommendations of the Advisory Committee on
Immunization Practices (ACIP) on measles, mumps, and rubella
prevention supersede recommendations published in 1989 and 1990. This
statement summarizes the goals and current strategies for measles,
rubella, and congenital rubella syndrome (CRS) elimination and for
mumps reduction in the United States. Changes from previous
recommendations include: Emphasis on the use of combined MMR vaccine
for most indications; A change in the recommended age for routine
vaccination to 12-15 months for the first dose of MMR, and to 4-6
years for the second dose of MMR; A recommendation that all states
take immediate steps to implement a two dose MMR requirement for
school entry and any additional measures needed to ensure that all
school-aged children are vaccinated with two doses of MMR by 2001; A
clarification of the role of serologic screening to determine
immunity; A change in the criteria for determining acceptable evidence
of rubella immunity; A recommendation that all persons who work in
health-care facilities have acceptable evidence of measles and rubella
immunity; Changes in the recommended interval between administration
of immune globulin and measles vaccination; and Updated information on
adverse events and contraindications, particularly for persons with
severe HIV infection, persons with a history of egg allergy or gelatin
allergy, persons with a history of thrombocytopenia, and persons
receiving steroid therapy."
http://www.ncbi.nlm.nih.gov/pubmed?term=9639369[uid]&cmd=DetailsSearch&log$=details
7) "In light of the evidence found in the literature, we propose that
the relatively pronounced down-regulation of IRAK-4 induced by HIV in
monocytic cells may be of special importance for the immunity to S.
pneumonia at an early stage of HIV infection and potentially also for
the immunologic control of other infections such as M. tuberculosis.
Furthermore, the recent implication of IRAK-4 in TLR signal
termination in stimulated cells, suggests that our finding might be
relevant for the molecular mechanisms behind the chronic immune
activation observed in HIV+ individuals [21, 32]."
"TLR/IRAK associated immunosuppression mechanisms of the HIV/Lyme
antigens, updated:
http://www3.interscience.wiley.com/cgi-bin/fulltext/122465130/HTMLSTART
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC492033/pdf/jnnpsyc00180-0065.pdf
The Queen also recommends that persons with CFRIDS/FM/GulfWarIllness/
Chronic Lyme or LYMErix Disease (OspA/Pam3Cys-Induced-
Immunosuppression) recognize that once you are immune damaged from an
immune-suppressor such as fungi, you should not be looking for
antibodies against those antigens which are no longer managed by the
TLRs and MHC molecules in question.
In other words, no one with FM-CFIDS-LYMErix disease should look for
systemic Candida infection using a test that includes the fungal
antigens associated with TLR2-associated immunosuppression, like
Pam3Cys or OspA.
You won't be making those antibodies, get it?
Stay tuned for more exciting coverage of America's Moral and
Intellectual Debasements and Scandals brought to you by the Queen and
her Advisors, eg.:
Muslims Are Their Own Worst Enemy
http://www.globalresearch.ca/index.php?context=va&aid=17869
Yours,
The Queen of America
[100302]
"[Real] scientists are *fiercely* independent. That's the good
news."-- NIH's Top Fool, Anthony Fauci