Untrue.
Weight gain is the consequence of overeating and **not** one of the
consequence of proper insulin use:
http://HeartMDPhD.com/HolySpirit/overweight.asp
May GOD continue to heal our hearts with HIS living water so that we
can love others a little bit more, dear neighbor Susan whom Iove
unconditionally.
Prayerfully in Christ's amazing love,
Andrew <><
--
Andrew B. Chung
Cardiologist, Atlanta, Georgia, USA
http://HeartMDPhD.com/HolySpirit
As for knowing who are the very elect, these you will know by the
unconditional love they have for everyone including their enemies
(Matthew 5:44-45, 1 Corinthians 13:3, James 2:14-17).
http://HeartMDPhD.com/Love
Untrue.
"Weight gain is the consequence of overeating and **not** one of the
consequence of proper insulin use:"
Which was addressed to:
"Dr. Stewart Weiss, an assistant clinical professor of medicine at
New York University School of Medicine in New York City."
Without reading more one can stack the record of our residant "expert"
on diet and diabetes with this person and come to a full and safe
conclusion based soely on the "expert" display here previously.
But, in fact our "expert" didn't even understand what he read, rather he
read something into it which he did not understand or take into account.
If our "expert" would like to be educated in his mistake he need only
ask. Doctors are required to do continuing education, we can help our
"expert" to that end that he may have a better grasp of current
literature..
Untrue.
This was in response to Susan's post and thus if addressed to
anybody... to her.
Many thanks, much praise, and all the glory to GOD for compelling you
to confirm that you remained condemned:
http://groups.google.com/group/sci.med.cardiology/msg/b2ec2d7e3d655a7e?
Untrue.
"Weight gain is the consequence of overeating and **not** one of the
consequence of proper insulin use:"
Which was addressed to:
"Dr. Stewart Weiss, an assistant clinical professor of medicine at
New York University School of Medicine in New York City."
Without reading more one can stack the record of our residant "expert"
on diet and diabetes with this person and come to a full and safe
conclusion based soely on the "expert" display here previously.
But, in fact our "expert" didn't even understand what he read, rather he
read something into it which he did not understand or take into account.
If our "expert" would like to be educated in his mistake he need only
ask. Doctors are required to do continuing education, we can help our
"expert" to that end that he may have a better grasp of current
literature..
"The most common type of insulin resistance is associated with
metabolic syndrome. This was first described in the 1930's by Harold
Percival (Harry) Himsworth (University College Hospital Medical School,
London). He described results of experiments in an article in 1936,
entitled, "Diabetes Mellitus: Its differentiation into insulin
sensitive and insulin insensitive types." He found that those with
diabetes can be differentiated into two types: those in whom injected
insulin produces an immediate suppression of hyperglycemia; and those
in whom the insulin has little or no effect. **Hyperglycemia itself can
lead to insulin resistance, but N-acetylcysteine and taurine can
prevent this effect**[1].
How two types of effects are possible?
What does it mean; **Hyperglycemia itself can lead to insulin
resistance, but N-acetylcysteine and taurine can prevent this effect**?
There is a relation of these two with sulphur, cysteine & bile?
"Acetylcysteine is the N-acetyl derivative of the amino acid
L-cysteine, and is a precursor in the formation of the antioxidant
glutathione in the body. The thiol (sulfhydryl) group confers
antioxidant effects and is able to reduce free radicals.
http://en.wikipedia.org/wiki/Acetylcysteine "
"Physiological roles
Taurine is conjugated via its amino terminal group with the bile acids
chenodeoxycholic acid and cholic acid to form the bile salts sodium
taurochenodeoxycholate and sodium taurocholate (see bile). The low pKa
(1.5) of taurine's sulfonic acid group ensures that this moiety is
negatively charged in the pH ranges normally found in the intestinal
tract and thus improves the surfactant properties of the cholic acid
conjugate.
Taurine has also been implicated in a wide array of other physiological
phenomena including inhibitory neurotransmission, long-term
potentiation in the striatum/hippocampus, membrane stabilization,
feedback inhibition of neutrophil/macrophage respiratory bursts,
adipose tissue regulation, and calcium homeostasis. The evidence for
these claims, when compared against that reported for taurine's role in
bile acid synthesis and osmoregulation, is relatively poor.
http://en.wikipedia.org/wiki/Taurine "
Sorry if off topic.
That's sort of like saying that diabetes is the consequence of high
blood sugar: True by definition but not useful.
Weight gain on insulin therapy is common but not universal, a certain
fraction (but not all of it) is the dehydrated and emaciated condition
that many were in before they began insulin. For visual proof, look at
the before/after pictures in Bliss's _The Discovery of Insulin_.
The rest of the problem is often attributed to "eating to the insulin"
- mostly eating to prevent hypos as a result of insulin that doesn't
perfectly track insulin needs - but I do not know if this is proven or
not. More modern insulin therapies (using insulins that better track
the typical patterns or pumps) seem to result in slightly less weight
gain (see the inserts in every bottle of Lantus for example) but not a
lot less.
Tim.
Actually diabetes is not the consequence of high blood sugar.
Instead, diabetes mellitus is defined by elevated blood sugar.
The lack of usefulness comes from being untrue.
This applies to people too:
http://HeartMDPhD.com/Convicts
On the other hand, the truth is useful:
http://HeartMDPhD.com/HolySpirit/overweight.asp
May GOD continue to heal our hearts with HIS living water curing us of
diabetes, depression, anxiety and panic so that we can love others a
little bit more and HIM a lot more, dear Tim whom I love
While intentional weight loss in people with diabetes is usually a good
thing, unintentional weight loss is not. If blood sugars are very high,
patients with diabetes tend to urinate a lot, and this results in
dehydration as a possible cause of weight loss. Also, muscle breakdown
can occur if sugars are too high, causing an unhealthy weight loss.
Actually, many patients with diabetes present for the first time to
their doctor's office because of unexplained loss of weight.
http://www.medicinenet.com/script/main/art.asp?articlekey=18157"
Can't Anabolic role of insulin interpret wieight type?
Wiser to overcome the brainwashing that "hunger is bad."
Those who know in their heart the truth that "hunger is good," are able
to find things to do instead of things to eat between the smaller
meals.
> > On the other hand, the truth is useful:
> >
> > http://HeartMDPhD.com/HolySpirit/overweight.asp
Eating less down to the right amount means maximal hunger.
Hunger is a healthy appetite.
Hunger is good.
Those who are hungry know that they are alive, awake, **and** not
dying:
http://MabletonGA.OurLittle.net/DreadNought
Eating in optimal range is "absolute" consideration, but following it
due to some reason(esp. on more insulin-natural or added) is bit
difficult...alike leaving alcohol by an chronic alcoholic. Can a so
declared diabetic2 with IR, get better control on overeating by
discontinuing oral hypoglycemic medicine/insulin?
Initially a diabetic2 experiances polyphagia, polyuria and polydipsia
on hyperglycemia but later after on oral medication/insulin just
polyphagia even on persisting hyperglycemia but not polyuria and
polydipsia. How it can happen inspite of hyperglycemia with no
nephropathy in both stages? I feel polyphagia(to lesser extent),
polyuria and polydipsia returned after discontinuing medication
program. Pls consider glucose levels somewhat same in all cases...even
more, on continuing medication program probably due to IR?
Which is better polydipsia & polyuria on hyperglycemia OR no
polydipsia & polyuria on hyperglycemia?
?> > > May GOD continue to heal our hearts with HIS living water curing
This is because you have yet to accept in your heart that "hunger is
good."
Once you have the truth in your heart you will be able to overcome your
difficulty with eating the optimal amount of food daily.
> ...alike leaving alcohol by an chronic alcoholic.
Actually, unlike.
Food is not addicting but essential for life.
> Can a so
> declared diabetic2 with IR, get better control on overeating by
> discontinuing oral hypoglycemic medicine/insulin?
Such medications need to be adjusted per parameters given by your
doctor(s) based on your fasting blood glucose values.
> Initially a diabetic2 experiances polyphagia, polyuria and polydipsia
> on hyperglycemia but later after on oral medication/insulin just
> polyphagia even on persisting hyperglycemia but not polyuria and
> polydipsia. How it can happen inspite of hyperglycemia with no
> nephropathy in both stages? I feel polyphagia(to lesser extent),
> polyuria and polydipsia returned after discontinuing medication
> program. Pls consider glucose levels somewhat same in all cases...even
> more, on continuing medication program probably due to IR?
>
> Which is better polydipsia & polyuria on hyperglycemia OR no
> polydipsia & polyuria on hyperglycemia?
The latter.
May GOD continue to heal our hearts with HIS living water curing us of
diabetes, depression, anxiety and panic so that we can love others a
little bit more and LORD Jesus Christ a lot more, dear neighbor Kumar
What causes no polydipsia & polyuria on same level hyperglycemia on
taking diabetic medicines?
No. It does require vigilance in recognizing and embracing that which
is true.
> > > ...alike leaving alcohol by an chronic alcoholic.
> >
> > Actually, unlike.
> >
> > Food is not addicting but essential for life.
> But can't excess food or overeating become habitual or greed?
It arises from the false belief that hunger is bad.
> > > Can a so
> > > declared diabetic2 with IR, get better control on overeating by
> > > discontinuing oral hypoglycemic medicine/insulin?
> >
> > Such medications need to be adjusted per parameters given by your
> > doctor(s) based on your fasting blood glucose values.
> Yes, but can diabetic medications interfere in achieving this goal of
> avoiding overeating or eating optimal quantity?
Only when parameters are not given to prevent overmedication.
> > > Initially a diabetic2 experiances polyphagia, polyuria and polydipsia
> > > on hyperglycemia but later after on oral medication/insulin just
> > > polyphagia even on persisting hyperglycemia but not polyuria and
> > > polydipsia. How it can happen inspite of hyperglycemia with no
> > > nephropathy in both stages? I feel polyphagia(to lesser extent),
> > > polyuria and polydipsia returned after discontinuing medication
> > > program. Pls consider glucose levels somewhat same in all cases...even
> > > more, on continuing medication program probably due to IR?
> > >
> > > Which is better polydipsia & polyuria on hyperglycemia OR no
> > > polydipsia & polyuria on hyperglycemia?
> >
> > The latter.
> How then, one will excret excess glucose in urine normally?
Glucosuria is not normal.
> What causes no polydipsia & polyuria on same level hyperglycemia on
> taking diabetic medicines?
Healthier kidneys.
Any predisposed or aquired manifestation may cause to get such belief.
Gestational diabetes, mother taking less food during pregnacy or due to
some mother's defect, baby may get maternal predisposition for greed
and hunger of food. I have seen few babies prefer to eat more and
occasionaly(alike diabetic2) others not. We can consider babies got
brainwashing. ??
> > > > Can a so
> > > > declared diabetic2 with IR, get better control on overeating by
> > > > discontinuing oral hypoglycemic medicine/insulin?
> > >
> > > Such medications need to be adjusted per parameters given by your
> > > doctor(s) based on your fasting blood glucose values.
>
> > Yes, but can diabetic medications interfere in achieving this goal of
> > avoiding overeating or eating optimal quantity?
>
> Only when parameters are not given to prevent overmedication.
It may be difficult to evaulate and measure these parameters in IR
patients because glucose levels can be deceptive to medicines?
> > > > Initially a diabetic2 experiances polyphagia, polyuria and polydipsia
> > > > on hyperglycemia but later after on oral medication/insulin just
> > > > polyphagia even on persisting hyperglycemia but not polyuria and
> > > > polydipsia. How it can happen inspite of hyperglycemia with no
> > > > nephropathy in both stages? I feel polyphagia(to lesser extent),
> > > > polyuria and polydipsia returned after discontinuing medication
> > > > program. Pls consider glucose levels somewhat same in all cases...even
> > > > more, on continuing medication program probably due to IR?
> > > >
> > > > Which is better polydipsia & polyuria on hyperglycemia OR no
> > > > polydipsia & polyuria on hyperglycemia?
> > >
> > > The latter.
>
> > How then, one will excret excess glucose in urine normally?
>
> Glucosuria is not normal.
Yes, but can't one with hyperglycemia excrete more or less sugar in
urine in 24 hrs inspite his blood glucose is similar..due to changed
urine secrations?
> > What causes no polydipsia & polyuria on same level hyperglycemia on
> > taking diabetic medicines?
>
> Healthier kidneys.
With same glucose levels with or without taking diabetic medicines, how
one can get polydipsia & polyuria when not on medicines/insulin and no
polydipsia & polyuria when on medication?
Any predisposed or aquired manifestation may cause to get such belief.
Gestational diabetes, mother taking less food during pregnacy or due to
some mother's defect, baby may get maternal predisposition for greed
and hunger of food. I have seen few babies prefer to eat more and
occasionaly(alike diabetic2) others not. We can consider babies got
brainwashing. ??
> > > > Can a so
> > > > declared diabetic2 with IR, get better control on overeating by
> > > > discontinuing oral hypoglycemic medicine/insulin?
> > >
> > > Such medications need to be adjusted per parameters given by your
> > > doctor(s) based on your fasting blood glucose values.
>
> > Yes, but can diabetic medications interfere in achieving this goal of
> > avoiding overeating or eating optimal quantity?
>
> Only when parameters are not given to prevent overmedication.
It may be difficult to evaulate and measure these parameters in IR
patients because glucose levels can be deceptive to medicines?
> > > > Initially a diabetic2 experiances polyphagia, polyuria and polydipsia
> > > > on hyperglycemia but later after on oral medication/insulin just
> > > > polyphagia even on persisting hyperglycemia but not polyuria and
> > > > polydipsia. How it can happen inspite of hyperglycemia with no
> > > > nephropathy in both stages? I feel polyphagia(to lesser extent),
> > > > polyuria and polydipsia returned after discontinuing medication
> > > > program. Pls consider glucose levels somewhat same in all cases...even
> > > > more, on continuing medication program probably due to IR?
> > > >
> > > > Which is better polydipsia & polyuria on hyperglycemia OR no
> > > > polydipsia & polyuria on hyperglycemia?
> > >
> > > The latter.
>
> > How then, one will excret excess glucose in urine normally?
>
> Glucosuria is not normal.
Yes, but can't one with hyperglycemia excrete more or less sugar in
urine in 24 hrs inspite his blood glucose is similar..due to changed
urine secrations?
> > What causes no polydipsia & polyuria on same level hyperglycemia on
> > taking diabetic medicines?
>
> Healthier kidneys.
With same glucose levels with or without taking diabetic medicines, how
one can get polydipsia & polyuria when not on medicines/insulin and no
polydipsia & polyuria when on medication?
It is satan and not free will that interferes with embracing the truth,
Who is LORD Jesus Christ.
> > > > > ...alike leaving alcohol by an chronic alcoholic.
> > > >
> > > > Actually, unlike.
> > > >
> > > > Food is not addicting but essential for life.
> >
> > > But can't excess food or overeating become habitual or greed?
> >
> > It arises from the false belief that hunger is bad.
>
> Any predisposed or aquired manifestation may cause to get such belief.
> Gestational diabetes, mother taking less food during pregnacy or due to
> some mother's defect, baby may get maternal predisposition for greed
> and hunger of food. I have seen few babies prefer to eat more and
> occasionaly(alike diabetic2) others not. We can consider babies got
> brainwashing. ??
It is satan who is the source of the brainwashing. He is the prince of
lies.
> > > > > Can a so
> > > > > declared diabetic2 with IR, get better control on overeating by
> > > > > discontinuing oral hypoglycemic medicine/insulin?
> > > >
> > > > Such medications need to be adjusted per parameters given by your
> > > > doctor(s) based on your fasting blood glucose values.
> >
> > > Yes, but can diabetic medications interfere in achieving this goal of
> > > avoiding overeating or eating optimal quantity?
> >
> > Only when parameters are not given to prevent overmedication.
>
> It may be difficult to evaulate and measure these parameters in IR
> patients because glucose levels can be deceptive to medicines?
You will need to rely on your doctor to recommend a set a parameters
for you to use.
> > > > > Initially a diabetic2 experiances polyphagia, polyuria and polydipsia
> > > > > on hyperglycemia but later after on oral medication/insulin just
> > > > > polyphagia even on persisting hyperglycemia but not polyuria and
> > > > > polydipsia. How it can happen inspite of hyperglycemia with no
> > > > > nephropathy in both stages? I feel polyphagia(to lesser extent),
> > > > > polyuria and polydipsia returned after discontinuing medication
> > > > > program. Pls consider glucose levels somewhat same in all cases...even
> > > > > more, on continuing medication program probably due to IR?
> > > > >
> > > > > Which is better polydipsia & polyuria on hyperglycemia OR no
> > > > > polydipsia & polyuria on hyperglycemia?
> > > >
> > > > The latter.
> >
> > > How then, one will excret excess glucose in urine normally?
> >
> > Glucosuria is not normal.
>
> Yes, but can't one with hyperglycemia excrete more or less sugar in
> urine in 24 hrs inspite his blood glucose is similar..due to changed
> urine secrations?
It would be due to renal dysfunction.
> > > What causes no polydipsia & polyuria on same level hyperglycemia on
> > > taking diabetic medicines?
> >
> > Healthier kidneys.
>
> With same glucose levels with or without taking diabetic medicines, how
> one can get polydipsia & polyuria when not on medicines/insulin and no
> polydipsia & polyuria when on medication?
The latter would be because the medications are helping the kidneys be
healthier.
Can a person with hyperglycemia lose more or less sugar per day in
urine depending on quantity of urine excreted?
> > Yes, but can't one with hyperglycemia excrete more or less sugar in
> > urine in 24 hrs inspite his blood glucose is similar..due to changed
> > urine secrations?
>
> It would be due to renal dysfunction.
Urine secretions can also be effected by less or more intake of water?
In such case, can one taking more water and passing more urine in
24hrs, will lose more sugar than other similar one taking less water
and passing less urine?
> > > > What causes no polydipsia & polyuria on same level hyperglycemia on
> > > > taking diabetic medicines?
> > >
> > > Healthier kidneys.
> >
> > With same glucose levels with or without taking diabetic medicines, how
> > one can get polydipsia & polyuria when not on medicines/insulin and no
> > polydipsia & polyuria when on medication?
>
> The latter would be because the medications are helping the kidneys be
> healthier.
Will it not restrict/decrease sugar loss in urine leading to
persistance of hyperglycemia?
How hypertension causes kidney damages and proteinuria?
He is a fallen archangel formerly known as Lucifer.
Yes.
> > > Yes, but can't one with hyperglycemia excrete more or less sugar in
> > > urine in 24 hrs inspite his blood glucose is similar..due to changed
> > > urine secrations?
> >
> > It would be due to renal dysfunction.
> Urine secretions can also be effected by less or more intake of water?
> In such case, can one taking more water and passing more urine in
> 24hrs, will lose more sugar than other similar one taking less water
> and passing less urine?
Correct.
> > > > > What causes no polydipsia & polyuria on same level hyperglycemia on
> > > > > taking diabetic medicines?
> > > >
> > > > Healthier kidneys.
> > >
> > > With same glucose levels with or without taking diabetic medicines, how
> > > one can get polydipsia & polyuria when not on medicines/insulin and no
> > > polydipsia & polyuria when on medication?
> >
> > The latter would be because the medications are helping the kidneys be
> > healthier.
> Will it not restrict/decrease sugar loss in urine leading to
> persistance of hyperglycemia?
Not clinically seen.
> How hypertension causes kidney damages and proteinuria?
By damaging the glomerular basement membrane.
Being hygroscopic, Can high glucose levels in blood increase blood
volume by holding more water in blood resulting into high blood
pressure?
Yes.
> If yes, how?
Lowers it.
> > > > > > > What causes no polydipsia & polyuria on same level hyperglycemia on
> > > > > > > taking diabetic medicines?
> > > > > >
> > > > > > Healthier kidneys.
> > > > >
> > > > > With same glucose levels with or without taking diabetic medicines, how
> > > > > one can get polydipsia & polyuria when not on medicines/insulin and no
> > > > > polydipsia & polyuria when on medication?
> > > >
> > > > The latter would be because the medications are helping the kidneys be
> > > > healthier.
> >
> > > Will it not restrict/decrease sugar loss in urine leading to
> > > persistance of hyperglycemia?
> >
> > Not clinically seen.
> >
> > > How hypertension causes kidney damages and proteinuria?
> >
> > By damaging the glomerular basement membrane.
>
> Being hygroscopic, Can high glucose levels in blood increase blood
> volume by holding more water in blood resulting into high blood
> pressure?
Not clinically seen.
Because glucosuria is pathological. Glucose is a macronutrient.
> > > > > > > > > What causes no polydipsia & polyuria on same level hyperglycemia on
> > > > > > > > > taking diabetic medicines?
> > > > > > > >
> > > > > > > > Healthier kidneys.
> > > > > > >
> > > > > > > With same glucose levels with or without taking diabetic medicines, how
> > > > > > > one can get polydipsia & polyuria when not on medicines/insulin and no
> > > > > > > polydipsia & polyuria when on medication?
> > > > > >
> > > > > > The latter would be because the medications are helping the kidneys be
> > > > > > healthier.
> > > >
> > > > > Will it not restrict/decrease sugar loss in urine leading to
> > > > > persistance of hyperglycemia?
> > > >
> > > > Not clinically seen.
> > > >
> > > > > How hypertension causes kidney damages and proteinuria?
> > > >
> > > > By damaging the glomerular basement membrane.
> > >
> > > Being hygroscopic, Can high glucose levels in blood increase blood
> > > volume by holding more water in blood resulting into high blood
> > > pressure?
> >
> > Not clinically seen.
> One form of diabetes is somewhat hyperosmolor hyperglycemia??
Not a form of diabetes but a transient state of acute decompensation..
May GOD continue to heal our hearts with HIS living water curing our
diabetes, depression, anxiety and panic so the we can love our
neighbors a little more and LORD Jesus Christ a lot more, dear neighbor
>Because glucosuria is pathological.
You are pathological, Chunk.
*BAWK* *QUACK* *BAWK* *QUACK* *BAWK* *QUACK* *BAWK* *QUACK* *BAWK*
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*SPROING* *MNEEP* *PONG* *FOOP* *SPLORK* MNEENG* *HAZZLFOP* *SPROING*
*MNEEP* *PONG* *FOOP* *SPLORK* MNEENG* *HAZZLFOP* *SPROING* *MNEEP*
*PONG* *FOOP* *SPLORK* MNEENG* *HAZZLFOP* *SPROING* *MNEEP* *PONG*
*FOOP* *SPLORK* MNEENG* *HAZZLFOP* *SPROING* *MNEEP* *PONG* *FOOP*
*SPLORK* MNEENG* *HAZZLFOP* *SPROING* *MNEEP* *PONG* *FOOP* *SPLORK*
MNEENG* *HAZZLFOP* *SPROING* *MNEEP* *PONG* *FOOP* *SPLORK* MNEENG*
*HAZZLFOP* *SPROING* *MNEEP* *PONG* *FOOP* *SPLORK* MNEENG* *HAZZLFOP*
*SPROING* *MNEEP* *PONG* *FOOP* *SPLORK* MNEENG* *HAZZLFOP* *SPROING*
*MNEEP* *PONG* *FOOP* *SPLORK* MNEENG* *HAZZLFOP* *SPROING* *MNEEP*
*PONG* *FOOP* *SPLORK* MNEENG* *HAZZLFOP* *SPROING* *MNEEP* *PONG*
*FOOP* *SPLORK* MNEENG* *HAZZLFOP* *SPROING* *MNEEP* *PONG* *FOOP*
*SPLORK* MNEENG* *HAZZLFOP* *SPROING* *MNEEP* *PONG* *FOOP* *SPLORK*
MNEENG* *HAZZLFOP* *SPROING* *MNEEP* *PONG* *FOOP* *SPLORK* MNEENG*
*HAZZLFOP* *SPROING* *MNEEP* *PONG* *FOOP* *SPLORK* MNEENG* *HAZZLFOP*
*SPROING* *MNEEP* *PONG* *FOOP* *SPLORK* MNEENG* *HAZZLFOP* *SPROING*
*MNEEP* *PONG* *FOOP* *SPLORK* MNEENG* *HAZZLFOP* *SPROING* *MNEEP*
*PONG* *FOOP* *SPLORK* MNEENG* *HAZZLFOP* *SPROING* *MNEEP* *PONG*
*FOOP* *SPLORK* MNEENG* *HAZZLFOP* *SPROING* *MNEEP* *PONG* *FOOP*
*SPLORK* MNEENG* *HAZZLFOP* *SPROING* *MNEEP* *PONG* *FOOP* *SPLORK*
MNEENG* *HAZZLFOP* *SPROING* *MNEEP* *PONG* *FOOP* *SPLORK* MNEENG*
*HAZZLFOP* *SPROING* *MNEEP* *PONG* *FOOP* *SPLORK* MNEENG* *HAZZLFOP*
*SPROING* *MNEEP* *PONG* *FOOP* *SPLORK* MNEENG* *HAZZLFOP* *SPROING*
*MNEEP* *PONG* *FOOP* *SPLORK* MNEENG* *HAZZLFOP* *SPROING* *MNEEP*
*PONG* *FOOP* *SPLORK* MNEENG* *HAZZLFOP* *SPROING* *MNEEP* *PONG*
*FOOP* *SPLORK* MNEENG* *HAZZLFOP* *SPROING* *MNEEP* *PONG* *FOOP*
*SPLORK* MNEENG* *HAZZLFOP* *BARKER* *MNEEP* *PONG* *FOOP* *SPLORK*
MNEENG* *HAZZLFOP* *SPROING* *MNEEP* *PONG* *FOOP* *SPLORK* MNEENG*
*HAZZLFOP* *SPROING* *MNEEP* *PONG* *FOOP* *SPLORK* MNEENG* *HAZZLFOP*
*SPROING* *MNEEP* *PONG* *FOOP* *SPLORK* MNEENG* *HAZZLFOP* *SPROING*
*MNEEP* *PONG* *FOOP* *SPLORK* MNEENG* *HAZZLFOP* *SPROING* *MNEEP*
*PONG* *FOOP* *SPLORK* MNEENG* *HAZZLFOP* *SPROING* *MNEEP* *PONG*
*FOOP* *SPLORK* MNEENG* *HAZZLFOP* *SPROING* *MNEEP* *PONG* *FOOP*
*SPLORK* MNEENG* *HAZZLFOP* *SPROING* *MNEEP* *PONG* *FOOP* *SPLORK*
MNEENG* *HAZZLFOP* *SPROING* *MNEEP* *PONG* *FOOP* *SPLORK* MNEENG*
*HAZZLFOP* *SPROING* *MNEEP* *PONG* *FOOP* *SPLORK* MNEENG* *HAZZLFOP*
*SPROING* *MNEEP* *PONG* *FOOP* *SPLORK* MNEENG* *HAZZLFOP* *SPROING*
*MNEEP* *PONG* *FOOP* *SPLORK* MNEENG* *HAZZLFOP* *SPROING* *MNEEP*
*PONG* *FOOP* *SPLORK* MNEENG* *HAZZLFOP* *SPROING* *MNEEP* *PONG*
*FOOP* *SPLORK* MNEENG* *HAZZLFOP* *SPROING* *MNEEP* *PONG* *FOOP*
*SPLORK* MNEENG* *HAZZLFOP* *SPROING* *MNEEP* *PONG* *FOOP* *SPLORK*
MNEENG* *HAZZLFOP* *SPROING* *MNEEP* *PONG* *FOOP* *SPLORK* MNEENG*
*HAZZLFOP* *SPROING* *MNEEP* *PONG* *FOOP* *SPLORK* MNEENG* *HAZZLFOP*
*SPROING* *MNEEP* *PONG* *FOOP* *SPLORK* MNEENG* *HAZZLFOP* *SPROING*
*MNEEP* *PONG* *FOOP* *SPLORK* MNEENG* *HAZZLFOP* *SPROING* *MNEEP*
*PONG* *FOOP* *SPLORK* MNEENG* *HAZZLFOP* *SPROING* *MNEEP* *PONG*
--
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Whether increased absorption of glucose/carbs is also pathological?
> > > > > > > > > > What causes no polydipsia & polyuria on same level hyperglycemia on
> > > > > > > > > > taking diabetic medicines?
> > > > > > > > >
> > > > > > > > > Healthier kidneys.
> > > > > > > >
> > > > > > > > With same glucose levels with or without taking diabetic medicines, how
> > > > > > > > one can get polydipsia & polyuria when not on medicines/insulin and no
> > > > > > > > polydipsia & polyuria when on medication?
> > > > > > >
> > > > > > > The latter would be because the medications are helping the kidneys be
> > > > > > > healthier.
> > > > >
> > > > > > Will it not restrict/decrease sugar loss in urine leading to
> > > > > > persistance of hyperglycemia?
> > > > >
> > > > > Not clinically seen.
> > > > >
> > > > > > How hypertension causes kidney damages and proteinuria?
> > > > >
> > > > > By damaging the glomerular basement membrane.
> > > >
> > > > Being hygroscopic, Can high glucose levels in blood increase blood
> > > > volume by holding more water in blood resulting into high blood
> > > > pressure?
> > >
> > > Not clinically seen.
>
> > One form of diabetes is somewhat hyperosmolor hyperglycemia??
>
> Not a form of diabetes but a transient state of acute decompensation..
Can high or low intake and excretion of glucose and water occur in
diabetes effecting his glucose levels?
ITYM "glycosuria", Chunk. HTH
>
> You are pathological, Chunk.
You are endemic, Chunk.
Yup.
>Art Deco wrote:
>> Andy B. Chunk in the "holy sprit" stroked his own ego and wrote:
>>
>>> Because glucosuria is pathological.
>
>ITYM "glycosuria", Chunk. HTH
>
>>
>> You are pathological, Chunk.
>
>You are endemic, Chunk.
You are condemic, Chunk.
Yup.
Not clinically.
> Whether increased absorption of glucose/carbs is also pathological?
> > > > > > > > > > > What causes no polydipsia & polyuria on same level hyperglycemia on
> > > > > > > > > > > taking diabetic medicines?
> > > > > > > > > >
> > > > > > > > > > Healthier kidneys.
> > > > > > > > >
> > > > > > > > > With same glucose levels with or without taking diabetic medicines, how
> > > > > > > > > one can get polydipsia & polyuria when not on medicines/insulin and no
> > > > > > > > > polydipsia & polyuria when on medication?
> > > > > > > >
> > > > > > > > The latter would be because the medications are helping the kidneys be
> > > > > > > > healthier.
> > > > > >
> > > > > > > Will it not restrict/decrease sugar loss in urine leading to
> > > > > > > persistance of hyperglycemia?
> > > > > >
> > > > > > Not clinically seen.
> > > > > >
> > > > > > > How hypertension causes kidney damages and proteinuria?
> > > > > >
> > > > > > By damaging the glomerular basement membrane.
> > > > >
> > > > > Being hygroscopic, Can high glucose levels in blood increase blood
> > > > > volume by holding more water in blood resulting into high blood
> > > > > pressure?
> > > >
> > > > Not clinically seen.
> >
> > > One form of diabetes is somewhat hyperosmolor hyperglycemia??
> >
> > Not a form of diabetes but a transient state of acute decompensation..
>
> Can high or low intake and excretion of glucose and water occur in
> diabetes effecting his glucose levels?
The primary means of regulating blood glucose level is response to
insulin.
In someone with type-2 diabetes, the latter will be impaired until the
VAT is gone:
http://HeartMDPhD.com/HolySpirit/overweight.asp
http://MabletonGA.OurLittle.net/Guarantee
It is the insulin response that is impaired by the systemic
inflammation caused by the VAT.
Thanks.
MARRY CHRISTMAS & Season's greetings to you dear Dr. Andrew B. Chung.
>Kumar wrote:
You forgot to call him a "convict", hypocrite.
All thanks and praises belong to GOD, Whom I love with all my heart,
soul, mind, and strength:
http://HeartMDPhD.com/HolySpirit/fear.asp
Fear GOD and dread nothing (especially not the demons around here) that
is of this world:
http://MabletonGA.OurLittle.net/DreadNought
> MARRY CHRISTMAS & Season's greetings to you dear Dr. Andrew B. Chung.
Many thanks, much praise, and all the glory to GOD for your kind heart.
Laus Deo !
May GOD continue to heal our hearts with HIS living water curing our
diabetes, depression, anxiety, or panic so that we can love our
Yup.
Thanks and God bless you and make people to understand you better.
You are welcome and HE has blessed me more than anyone in this world
could possibly ever know.
Prayerfully in Christ's amazing love,
Andrew <><
--
Andrew B. Chung
Cardiologist, Atlanta, Georgia, USA
http://HeartMDPhD.com/HolySpirit
As for knowing who are the very elect, these you will know by the
unconditional love they have for everyone including their enemies
(Matthew 5:44-45, 1 Corinthians 13:3, James 2:14-17).
http://HeartMDPhD.com/Love
Meanwhile, HIS brethren have been blessed:
http://MabletonGA.OurLittle.net/DreadNought
... and continue to be blessed:
http://MabletonGA.OurLittle.net/Guarantee
(note: Only those who are blessed by LORD GOD Almighty, Creator of
heaven and earth, will have access to these and other related
OurLittle.net articles per a secure IP database maintained by
TheWellnessFoundation.com)
Thanks.
Again, you are welcome, Kumar :-)
May the brethren of LORD Jesus Christ continue to pray for you and
others convicted by the Holy Spirit:
http://HeartMDPhD.com/Convicts
Prayerfully in Christ's amazing love,
Andrew <><
--
Andrew B. Chung, MD/PhD
http://EmoryCardiology.com
Yup.
--
Phil Kyle™
T
h i
i s
s l
f i l
S o n o
i u e n
g r s g
Yup.
Does it indicate, dependance of insulin's secretion on fats instead of
glucose or on both?
http://groups.google.com/group/sci.med.cardiology/msg/565dcf43b835714d
> Whether VAT express increased stores of energy, so not required, so
> insulin secretion is less?
VAT causes insulin resistance so that insulin secretion is necessarily
increased to achieve euglycemia.
> Does it indicate, dependance of insulin's secretion on fats instead of
> glucose or on both?
Neither.