http://www.neuroeconomics.nyu.edu/papers/Bickel1.pdf
One neurobehavioral process evident in a variety of disorders
and suboptimal behaviors is a high level of delay discounting
(1). Delay discounting refers to the decrease in
value of a reward as a function of the delay to its receipt (2). An
individual’s rate of discounting can be measured by assessing
preferences
between a sooner, smaller reward or a later, larger one.
Higher rates of discounting are associated with drug dependence
(2), problem gambling (3), obesity (4,5), and human immunodeficiency
virus risk behaviors (6,7). Given that delay discounting underlies
a wide variety of disadvantageous behaviors, itmayfunction
as a transdisease process (8).
The correlation between rates of delay discounting and suboptimal
behaviors has led, in part, to efforts to gain a better understanding
of discounting in terms of its relationship to other decision-
making and neurocognitive processes. Consideration and
valuation of the future has been shown to overlap with processes
and brain regions associated with memory or valuation of the past
(9,10). For example, discounting of past and future rewards have
been found to be qualitatively and quantitatively comparable (11–
13) by conforming to the same signature hyperbolic function and
magnitude effect, and bilateral damage to the hippocampus impairs
the ability to remember the past and to imagine future personal
experiences (14). More directly, recent demonstrations indicate
significant correlations between measures of working memory
and delay discounting (15,16). Thus, if consideration of and valuation
of the past and future are linked (17,18), then it might be
possible to decrease an individual’s discounting of future events by
increasing his or her ability to remember past events.
In this study, we employed neurocognitive rehabilitation approaches
that are proven to be effective with individuals with
schizophrenia (19,20) to examine the effects of working memory
training on measures of delay discounting, working memory, and
related assessments in individuals in treatment for psychomotor
stimulants (e.g., cocaine, methamphetamine). Frequent or heavy
users of stimulants have been shown to exhibit neurocognitive
deficits, including deficiencies in working memory (21,22) and high
rates of delay discounting (23,24).
In this study, participants receiving treatment for their stimulant
use received either experimental or control memory training.
Experimental
(Active) Training consisted of working memory tasks
with monetary reinforcement for performance. Control Training
consisted of presenting the same working memory tasks and cueing
the correct response. Reinforcement for each participant in the
control group was yoked to performance of a participant in the
active group; yoking ensured that the amount of reinforcement
obtained by each experimental group participant during each session
is also obtained by each participant in the control group. Preand
posttraining assessments on a variety of decision-making and
cognitive functions quantified the effects of training and determined
whether the effects were selective or more general.
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There was no before and after IQ taken for the active and control
groups and dual n back was not explicitly used in the study. However,
the research does suggest that generic working memory training
improves a person's will to overcome instant gratification urges and
improves executive functioning.