Re: Glycated substances & Diabetes?
Andrew B. Chung, MD/PhD
LORD GOD Almighty is the Source of all knowledge and wisdom.
May GOD continue to keep your heart beating, dear neighbor whom I love
unconditionally
Prayerfully in Christ's amazing love,
Andrew <><
--
Andrew B. Chung
Cardiologist, Atlanta, Georgia, USA
http://HeartMDPhD.com/HolySpirit
As for knowing who are the very elect, these you will know by the
unconditional love they have for everyone including their enemies
(Matthew 5:44-45, 1 Corinthians 13:3, James 2:14-17).
http://HeartMDPhD.com/Love
coon...@amestwp.com wrote:
> In response to a question about cooking methods to minimize glycation of
> foods:
>
> "Wiser to simply eat less down to the right amount and lose all the
> visceral adipose tissue (VAT) which is the proximate cause of metabolic
> syndrome (MetS), which is the precursor of type-2 diabetes:"
>
> Incorrect by being incomplete. Irrelevant by not addressing the
> question. Not useful by failing to provide applicable information
> beyond private agenda driven opinion.
>
> "Reducing intake has been shown to double the lifespan of laboratory
> animals."
>
> Again incorrect by being misinformed and not complete, it pays to keep
> up with the literature on such matters and forgo non-relevant agendas
> which distort the science involved.
Noway2
Andrew B. Chung, MD/PhD wrote:
> Your lack of wisdom does not surprise me.
>
documented.
> LORD GOD Almighty is the Source of all knowledge and wisdom.
>
Now I guess we all know where you got yours. Or didn't.
> May GOD continue to keep your heart beating, dear neighbor whom I love
> unconditionally
>
The only thing you love is the site of your own drivol.
> Prayerfully in Christ's amazing love,
>
Why don't you go back to blowing bubbles in your soup like you used to
do before discovering Usenet.
> Andrew <><
> --
> Andrew B. Chung
> Cardiologist, Atlanta, Georgia, USA
> http://HeartMDPhD.com/HolySpirit
>
Is that what you are calling your self this time around?
> As for knowing who are the very elect, these you will know by the
> unconditional love they have for everyone including their enemies
> (Matthew 5:44-45, 1 Corinthians 13:3, James 2:14-17).
> http://HeartMDPhD.com/Love
>
If you really loved everyone you would go away
Andrew B. Chung, MD/PhD
> Andrew, in the Holy Spirit, boldly wrote:
> > Bill wrote:
> > > coon...@amestwp.com wrote:
> > > > "We can dig more into microwave cooking for this purpose. As such, Can
> > > > we avoid glycation of foods by using microwave?"
> > > >
> > > > Any method can minimize this. It is mostly a product of cooking at high
> > > > temperatures such as with frying and baking for long times. Steaming,
> > > > boiling, stewing, microwaving, etc. will produce little. The best guide
> > > > is the color of or darkness of finished food. These glycation products
> > > > produce the browning effect from long fried and baked foods so any
> > > > method, including frying and baking, which produce little or none of the
> > > > browning is much safer.
> > >
> > > Boiled ham and cabbage Umm. Thanks for the info Coons makes sense.
> > >
> > >
> > > I thought the interesting point was we can pre--cook and then get our
> > > brown turkey / etc and reduce glycation.
> > >
> > > Fire good Umm Fire bad ...............
> > >
> > > Etc might be carrots??
> > >
> > > Bill who says if it is too difficult I'll change.
> >
> > Wiser to simply eat less down to the right amount and lose all the
> > visceral adipose tissue (VAT) which is the proximate cause of metabolic
> > syndrome (MetS), which is the precursor of type-2 diabetes:
> >
>
> Yes, but we can discuiss and try understanding various possibilities.
>
> Can fruits and milk/diary intake cause production of glycated
> substances endogenously?
Not in optimal amounts in folks without significant visceral adipose
tissue (VAT):
http://groups.google.com/group/sci.med.cardiology/msg/352546a7bf4a81ee?
> > Reducing intake has been shown to double the lifespan of laboratory
> > animals.
May GOD continue to heal your heart by curing your diabetes, dear
neighbor Kumar whom I love unconditionally.
Prayerfully in Christ's amazing love,
Andrew <><
--
Andrew B. Chung
Cardiologist, Atlanta, Georgia, USA
http://HeartMDPhD.com/HolySpirit
As for knowing who are the very elect, these you will know by the
Andrew B. Chung, MD/PhD
May GOD continue to keep your heart beating, dear neighbor whom I love
unconditionally.
Prayerfully in Christ's amazing love,
Andrew <><
--
Andrew B. Chung
Cardiologist, Atlanta, Georgia, USA
http://HeartMDPhD.com/HolySpirit
As for knowing who are the very elect, these you will know by the
unconditional love they have for everyone including their enemies
(Matthew 5:44-45, 1 Corinthians 13:3, James 2:14-17).
http://HeartMDPhD.com/Love
coon...@amestwp.com wrote:
> "> Can fruits and milk/diary intake cause production of glycated
> > substances endogenously?
>
> Not in optimal amounts in folks without significant visceral adipose
> tissue (VAT):"
>
> Simply untrue, displays continuing failure to keep up with the
> literature, a willful obsession to mislead to achieve an agenda driven
> non-scientific goal, and more positive proof the source can be safely
> ignored.
Andrew B. Chung, MD/PhD
coon...@amestwp.com wrote:
> Can fruits and milk/diary intake cause production of glycated
> ubstances endogenously?
>
> in optimal amounts in folks without significant visceral adipose
> sue (VAT):"
>
> ply untrue, displays continuing failure to keep up with the
> erature, a willful obsession to mislead to achieve an agenda driven
> -scientific goal, and more positive proof the source can be safely
> ored.
>
> ditto
>
> ditto
T
lo...@thetruth.com says...
Sneaky little bastard, you changed domains again.
Plonk!
kumar
Andrew B. Chung, MD/PhD wrote:
Whether overeating causes increased nutrients availability to tissues
resulting more/early growth and replications, thus decreasing the
lifespan?
Can more or early replications of body cells effect aging?
Andrew B. Chung, MD/PhD
> Seems like some of you are "seeing ghosts." It is the lipid
> peroxidation that is causing the glycation problems (in vivo). Here is
> yet another study that makes this point:
>
> Diabetes, Obesity and Metabolism
> Volume 7 Page 448 - July 2005
> doi:10.1111/j.1463-1326.2004.00387.x
> Volume 7 Issue 4
>
> Short Report
> High-fat diet enhances visceral advanced glycation end products,
> nuclear O-Glc-Nac modification, p38 mitogen-activated protein kinase
> activation and apoptosis
> S.-Y. Li1, Y. Liu1, V. K. Sigmon1, A. McCort1 and J. Ren1*
>
> High-fat diet intake often leads to obesity, insulin resistance and
> hypertension, which present a common and detrimental health problem.
> However, precise mechanism underlying tissue damage due to high-fat
> diet-induced obesity has not been carefully elucidated. The present
> study was designed to examine the effect of high-fat diet intake on
> visceral advanced glycation end products (AGEs) formation, nuclear
> O-Glc-NAc modification and apoptosis in heart, liver and kidney. Adult
> male Sprague-Dawley weight-matched rats were fed for 12 weeks with a
> high-fat diet (45% kcal from fat) or an isocaloric low-fat diet (10%
> kcal from fat). High-fat diet feeding significantly elevated body
> weight. Blood pressure and heart rate were comparable between the two
> rat groups. Competitive enzyme-linked immunosorbent assay showed
> significantly elevated serum AGE levels, visceral AGE formation,
> caspase-3 activation and cytoplasmic DNA fragmentation in heart and
> liver but not kidney samples of high-fat diet fed rats compared with
> those from low-fat diet fed group. Western blot analysis further
> revealed that high-fat diet feeding induced overt nuclear O-Glc-NAc
> modification and p38 mitogen-activated protein kinase activation in
> heart and liver although not in kidney samples of the high-fat diet-fed
> rats. Collectively, our results indicated that high-fat diet intake is
> associated with obesity accompanied by elevated serum and visceral
> AGEs, visceral post-translational nuclear O-Glc-NAcylated modification
> and apoptosis, which may contribute to high-fat diet-induced tissue
> damage.
>
> Now, if they were more knowledgable they would do another experiment,
> this time using fresh coconut oil, and giving the animals a "high fat"
> diet. What they would find is that this diet is not a problem at all,
> but because they are assuming that "saturated fat is bad," they
> apparently don't even consider this possibility, and this is why much
> "science" today is unscientific. You must control for all possibly
> relevant factors in order to follow the scientific method.
The p38 MAP kinase activation would be mediated by the inflammatory
cytokines from the visceral adipose tissue (VAT). Also a high fat (low
carb) diet would also lead to the hyperketonemia which is associated
with accelerated lipid peroxidation.
May GOD continue to heal our hearts with HIS living water, dear
neighbor Monty whomI love unconditionally.
Kumar
Andrew B. Chung, MD/PhD wrote:
systemic inflammations?
Kumar
palpitated:
> Spindly woman of loose morals with horrid frothing gash and ruined
> gagas goes hungry for junior needle for torturing stuff-my-bunny fun
> times. Mail me at <lordsh...@rediffmail.com>
Andrew B. Chung, MD/PhD
Disseminated tuberculosis.
Kumar
> > > > > Reducing intake has been shown to double the lifespan of laboratory
> > > > > animals.
> >
> > Other than VAT & possibily glycated substances, what other can cause
> > systemic inflammations?
>
> Disseminated tuberculosis.
Can glycation also occur in extravascular comparments? If yes, which
conditions are related to it?
The Lord of Fair Punishments
"Andrew B. Chung, MD/PhD" <lo...@thetruth.com> wrote in message
news:1163051577.2...@k70g2000cwa.googlegroups.com...
I wish your answers to non-medical questions would have been so straight
forward.
Andrew B. Chung, MD/PhD
Disseminated malignancies.
> Can glycation also occur in extravascular comparments?
Yes.
> If yes, which
> conditions are related to it?
Hyperglycemia.
Kumar
> > Can glycation also occur in extravascular comparments?
>
> Yes.
>
> > If yes, which
> > conditions are related to it?
>
> Hyperglycemia.
Hyperglycemia is not glycation but it is a reason to glycation? How
extravascular glycations can effect us?
Andrew B. Chung, MD/PhD
> Some other?
Gum disease.
> > > Can glycation also occur in extravascular comparments?
> >
> > Yes.
> >
> > > If yes, which
> > > conditions are related to it?
> >
> > Hyperglycemia.
>
> Hyperglycemia is not glycation but it is a reason to glycation? How
> extravascular glycations can effect us?
As per earlier discussions.
Kumar
local inflammation or a cause to it?
> > > > Can glycation also occur in extravascular comparments?
> > >
> > > Yes.
> > >
> > > > If yes, which
> > > > conditions are related to it?
> > >
> > > Hyperglycemia.
> >
> > Hyperglycemia is not glycation but it is a reason to glycation? How
> > extravascular glycations can effect us?
>
> As per earlier discussions.
tissues or excessive fat store in VAT--greter visceral adiposity),
glycated substances and absorbed fats which may encourage inflammatory
responses( as per other topics) as causes to systemic inflammatory
responses in diabetics?
Btw, whether sugar, carbs, triglycirides(glyceral part)(other are Mg
and NaCl) attract and absorb water in intestines relieving from
constipation and resulting into clear motions and increase motility?
Andrew B. Chung, MD/PhD
> Which gum disease?
Gingivitis.
> Btw, wheher gum disease is a result of systemic or
> local inflammation or a cause to it?
Goes both ways.
> > > > > Can glycation also occur in extravascular comparments?
> > > >
> > > > Yes.
> > > >
> > > > > If yes, which
> > > > > conditions are related to it?
> > > >
> > > > Hyperglycemia.
> > >
> > > Hyperglycemia is not glycation but it is a reason to glycation? How
> > > extravascular glycations can effect us?
> >
> > As per earlier discussions.
>
> Can we combine VAT(please clear, is it VAT--excessive visceral adipose
> tissues or excessive fat store in VAT--greter visceral adiposity),
> glycated substances and absorbed fats which may encourage inflammatory
> responses( as per other topics) as causes to systemic inflammatory
> responses in diabetics?
It is the inflammatory cytokines produced by the visceral adipose
tissue (VAT) that promote vascular inflammation thereby causing
increased insulin resistance.
> Btw, whether sugar, carbs, triglycirides(glyceral part)(other are Mg
> and NaCl) attract and absorb water in intestines relieving from
> constipation and resulting into clear motions and increase motility?
Not clinically seen.
Kumar
Andrew B. Chung, MD/PhD wrote:
> Kumar wrote:
>
> > Which gum disease?
>
> Gingivitis.
Pyorria?
Which gum disease is more common in diabetics?
> > Btw, wheher gum disease is a result of systemic or
> > local inflammation or a cause to it?
>
> Goes both ways.
>
> > > > > > Can glycation also occur in extravascular comparments?
> > > > >
> > > > > Yes.
> > > > >
> > > > > > If yes, which
> > > > > > conditions are related to it?
> > > > >
> > > > > Hyperglycemia.
> > > >
> > > > Hyperglycemia is not glycation but it is a reason to glycation? How
> > > > extravascular glycations can effect us?
> > >
> > > As per earlier discussions.
I think those were for intravascular glycations?
> > Can we combine VAT(please clear, is it VAT--excessive visceral adipose
> > tissues or excessive fat store in VAT--greter visceral adiposity),
> > glycated substances and absorbed fats which may encourage inflammatory
> > responses( as per other topics) as causes to systemic inflammatory
> > responses in diabetics?
>
> It is the inflammatory cytokines produced by the visceral adipose
> tissue (VAT) that promote vascular inflammation thereby causing
> increased insulin resistance.
Can't above other conditions/factors add to such inflamation?
> > Btw, whether sugar, carbs, triglycirides(glyceral part)(other are Mg
> > and NaCl) attract and absorb water in intestines relieving from
> > constipation and resulting into clear motions and increase motility?
>
> Not clinically seen.
They are hygroscopic..somewhat may act as MOM acts?
Whether bile is hygroscopic? Can bile's abnormal secretion(less) in
intestine make a person constipated, lazy esp. in morning, sleeping
more, waking late etc.? Two aspects may be related, abnormal bile
comong from liver and abnormal secretion from gall bladder(can't say
about abnormal re-absorption)?
Andrew B. Chung, MD/PhD
> Andrew B. Chung, MD/PhD wrote:
> > Kumar wrote:
> >snip> > > Gum disease.
> >
> > > Which gum disease?
> >
> > Gingivitis.
>
> Pyorria?
>
> Which gum disease is more common in diabetics?
The following link should help you:
http://www.nlm.nih.gov/medlineplus/gumdisease.html
> > > Btw, wheher gum disease is a result of systemic or
> > > local inflammation or a cause to it?
> >
> > Goes both ways.
> >
> > > > > > > Can glycation also occur in extravascular comparments?
> > > > > >
> > > > > > Yes.
> > > > > >
> > > > > > > If yes, which
> > > > > > > conditions are related to it?
> > > > > >
> > > > > > Hyperglycemia.
> > > > >
> > > > > Hyperglycemia is not glycation but it is a reason to glycation? How
> > > > > extravascular glycations can effect us?
> > > >
> > > > As per earlier discussions.
> I think those were for intravascular glycations?
No.
> > > Can we combine VAT(please clear, is it VAT--excessive visceral adipose
> > > tissues or excessive fat store in VAT--greter visceral adiposity),
> > > glycated substances and absorbed fats which may encourage inflammatory
> > > responses( as per other topics) as causes to systemic inflammatory
> > > responses in diabetics?
> >
> > It is the inflammatory cytokines produced by the visceral adipose
> > tissue (VAT) that promote vascular inflammation thereby causing
> > increased insulin resistance.
> Can't above other conditions/factors add to such inflamation?
It starts with the VAT.
> > > Btw, whether sugar, carbs, triglycirides(glyceral part)(other are Mg
> > > and NaCl) attract and absorb water in intestines relieving from
> > > constipation and resulting into clear motions and increase motility?
> >
> > Not clinically seen.
>
> They are hygroscopic..somewhat may act as MOM acts?
Still not clinically seen.
> Whether bile is hygroscopic? Can bile's abnormal secretion(less) in
> intestine make a person constipated, lazy esp. in morning, sleeping
> more, waking late etc.? Two aspects may be related, abnormal bile
> comong from liver and abnormal secretion from gall bladder(can't say
> about abnormal re-absorption)?
Clinically not seen.
Kumar
Andrew B. Chung, MD/PhD wrote:
> Kumar wrote:
> > Andrew B. Chung, MD/PhD wrote:
> > > Kumar wrote:
> > >snip> > > Gum disease.
> > >
> > > > Which gum disease?
> > >
> > > Gingivitis.
> >
> > Pyorria?
> >
> > Which gum disease is more common in diabetics?
>
> The following link should help you:
>
> http://www.nlm.nih.gov/medlineplus/gumdisease.html
Thanks.
> > > > Btw, wheher gum disease is a result of systemic or
> > > > local inflammation or a cause to it?
> > >
> > > Goes both ways.
Can gum diseases in diabetic be due to cell's swelling interfering in
blood flow and supply resulting into necrosis?
> > > > > > > > Can glycation also occur in extravascular comparments?
> > > > > > >
> > > > > > > Yes.
> > > > > > >
> > > > > > > > If yes, which
> > > > > > > > conditions are related to it?
> > > > > > >
> > > > > > > Hyperglycemia.
> > > > > >
> > > > > > Hyperglycemia is not glycation but it is a reason to glycation? How
> > > > > > extravascular glycations can effect us?
> > > > >
> > > > > As per earlier discussions.
>
> > I think those were for intravascular glycations?
>
> No.
Soory but if you can tell me again?
> > > > Can we combine VAT(please clear, is it VAT--excessive visceral adipose
> > > > tissues or excessive fat store in VAT--greter visceral adiposity),
> > > > glycated substances and absorbed fats which may encourage inflammatory
> > > > responses( as per other topics) as causes to systemic inflammatory
> > > > responses in diabetics?
> > >
> > > It is the inflammatory cytokines produced by the visceral adipose
> > > tissue (VAT) that promote vascular inflammation thereby causing
> > > increased insulin resistance.
>
> > Can't above other conditions/factors add to such inflamation?
>
> It starts with the VAT.
Whether VAT as you mention, is increase of visceral adipose tissues or
greater adiposity/fat stores in visceral adipose tissues?
> > > > Btw, whether sugar, carbs, triglycirides(glyceral part)(other are Mg
> > > > and NaCl) attract and absorb water in intestines relieving from
> > > > constipation and resulting into clear motions and increase motility?
> > >
> > > Not clinically seen.
> >
> > They are hygroscopic..somewhat may act as MOM acts?
>
> Still not clinically seen.
Whether soluble or not solubles fibers are also hygroscopic or releases
some chemicals in intestines?
> > Whether bile is hygroscopic? Can bile's abnormal secretion(less) in
> > intestine make a person constipated, lazy esp. in morning, sleeping
> > more, waking late etc.? Two aspects may be related, abnormal bile
> > comong from liver and abnormal secretion from gall bladder(can't say
> > about abnormal re-absorption)?
>
> Clinically not seen.
Can abnormal or obstructed flow of bile from liver to intestine make a
person lazy, more sleepy, waking late etc.?
I feel abnormalities in bile flow are related to many conditions
couldn't yet evalued?
Which out of bile and gastric acid ir related to either unclear or
clear motions? Billary diahrria is thought?
Andrew B. Chung, MD/PhD
> Andrew B. Chung, MD/PhD wrote:
> > Kumar wrote:
> > > Andrew B. Chung, MD/PhD wrote:
> > > > Kumar wrote:
> > > >snip> > > Gum disease.
> > > >
> > > > > Which gum disease?
> > > >
> > > > Gingivitis.
> > >
> > > Pyorria?
> > >
> > > Which gum disease is more common in diabetics?
> >
> > The following link should help you:
> >
> > http://www.nlm.nih.gov/medlineplus/gumdisease.html
>
> Thanks.
All thanks and praises belong to GOD, Whom I love with all my heart,
soul, mind, and strength:
http://HeartMDPhD.com/HolySpirit/fear.asp
Fear GOD and dread nothing that is of this world:
http://MabletonGA.OurLittle.net/DreadNought
> > > > > Btw, wheher gum disease is a result of systemic or
> > > > > local inflammation or a cause to it?
> > > >
> > > > Goes both ways.
> Can gum diseases in diabetic be due to cell's swelling interfering in
> blood flow and supply resulting into necrosis?
In the type 2 diabetic, it is due in part to the inflammatory cytokines
from viseral adipose tissue (VAT).
> > > > > > > > > Can glycation also occur in extravascular comparments?
> > > > > > > >
> > > > > > > > Yes.
> > > > > > > >
> > > > > > > > > If yes, which
> > > > > > > > > conditions are related to it?
> > > > > > > >
> > > > > > > > Hyperglycemia.
> > > > > > >
> > > > > > > Hyperglycemia is not glycation but it is a reason to glycation? How
> > > > > > > extravascular glycations can effect us?
> > > > > >
> > > > > > As per earlier discussions.
> >
> > > I think those were for intravascular glycations?
> >
> > No.
> Soory but if you can tell me again?
The glycosylations are happening everywhere and not just
intravascularly.
> > > > > Can we combine VAT(please clear, is it VAT--excessive visceral adipose
> > > > > tissues or excessive fat store in VAT--greter visceral adiposity),
> > > > > glycated substances and absorbed fats which may encourage inflammatory
> > > > > responses( as per other topics) as causes to systemic inflammatory
> > > > > responses in diabetics?
> > > >
> > > > It is the inflammatory cytokines produced by the visceral adipose
> > > > tissue (VAT) that promote vascular inflammation thereby causing
> > > > increased insulin resistance.
> >
> > > Can't above other conditions/factors add to such inflamation?
> >
> > It starts with the VAT.
>
> Whether VAT as you mention, is increase of visceral adipose tissues or
> greater adiposity/fat stores in visceral adipose tissues?
VAT is simply visceral adipose tissue which can be quantified by
measuring waist circumference.
> > > > > Btw, whether sugar, carbs, triglycirides(glyceral part)(other are Mg
> > > > > and NaCl) attract and absorb water in intestines relieving from
> > > > > constipation and resulting into clear motions and increase motility?
> > > >
> > > > Not clinically seen.
> > >
> > > They are hygroscopic..somewhat may act as MOM acts?
> >
> > Still not clinically seen.
>
> Whether soluble or not solubles fibers are also hygroscopic or releases
> some chemicals in intestines?
Soluble fiber in the diet comes with its own water so that it is not
hygroscopic.
> > > Whether bile is hygroscopic? Can bile's abnormal secretion(less) in
> > > intestine make a person constipated, lazy esp. in morning, sleeping
> > > more, waking late etc.? Two aspects may be related, abnormal bile
> > > comong from liver and abnormal secretion from gall bladder(can't say
> > > about abnormal re-absorption)?
> >
> > Clinically not seen.
> Can abnormal or obstructed flow of bile from liver to intestine make a
> person lazy, more sleepy, waking late etc.?
Not clinically seen.
> I feel abnormalities in bile flow are related to many conditions
> couldn't yet evalued?
You would be wise to inform your doctor(s) about your symptoms rather
than attributing them to bile flow.
> Which out of bile and gastric acid ir related to either unclear or
> clear motions? Billary diahrria is thought?
No clinical association.
Kumar
Andrew B. Chung, MD/PhD wrote:
> Kumar wrote:
interfering in
> > blood flow and supply resulting into necrosis?
>
> In the type 2 diabetic, it is due in part to the inflammatory cytokines
> from viseral adipose tissue (VAT).
> > > > > > > > > > Can glycation also occur in extravascular comparments?
> > > > > > > > >
> > > > > > > > > Yes.
> > > > > > > > >
> > > > > > > > > > If yes, which
> > > > > > > > > > conditions are related to it?
> > > > > > > > >
> > > > > > > > > Hyperglycemia.
> > > > > > > >
> > > > > > > > Hyperglycemia is not glycation but it is a reason to glycation? How
> > > > > > > > extravascular glycations can effect us?
> > > > > > >
> > > > > > > As per earlier discussions.
> > >
> > > > I think those were for intravascular glycations?
> > >
> > > No.
>
> > Soory but if you can tell me again?
>
> The glycosylations are happening everywhere and not just
> intravascularly.
In case of vasoconstriction or reduced blood flow?
> > > > > > Can we combine VAT(please clear, is it VAT--excessive visceral adipose
> > > > > > tissues or excessive fat store in VAT--greter visceral adiposity),
> > > > > > glycated substances and absorbed fats which may encourage inflammatory
> > > > > > responses( as per other topics) as causes to systemic inflammatory
> > > > > > responses in diabetics?
> > > > >
> > > > > It is the inflammatory cytokines produced by the visceral adipose
> > > > > tissue (VAT) that promote vascular inflammation thereby causing
> > > > > increased insulin resistance.
> > >
> > > > Can't above other conditions/factors add to such inflamation?
> > >
> > > It starts with the VAT.
> >
> > Whether VAT as you mention, is increase of visceral adipose tissues or
> > greater adiposity/fat stores in visceral adipose tissues?
>
> VAT is simply visceral adipose tissue which can be quantified by
> measuring waist circumference.
Can't they store more or less fats? Whether such store is in form of
triglyciride or fatty acids?
> > > > > > Btw, whether sugar, carbs, triglycirides(glyceral part)(other are Mg
> > > > > > and NaCl) attract and absorb water in intestines relieving from
> > > > > > constipation and resulting into clear motions and increase motility?
> > > > >
> > > > > Not clinically seen.
> > > >
> > > > They are hygroscopic..somewhat may act as MOM acts?
> > >
> > > Still not clinically seen.
> >
> > Whether soluble or not solubles fibers are also hygroscopic or releases
> > some chemicals in intestines?
>
> Soluble fiber in the diet comes with its own water so that it is not
> hygroscopic.
>
> > > > Whether bile is hygroscopic? Can bile's abnormal secretion(less) in
> > > > intestine make a person constipated, lazy esp. in morning, sleeping
> > > > more, waking late etc.? Two aspects may be related, abnormal bile
> > > > comong from liver and abnormal secretion from gall bladder(can't say
> > > > about abnormal re-absorption)?
> > >
> > > Clinically not seen.
>
> > Can abnormal or obstructed flow of bile from liver to intestine make a
> > person lazy, more sleepy, waking late etc.?
>
> Not clinically seen.
How bile secretion is triggered? Can there be different type or
quantity of bile secreted in different conditions? > > I feel
abnormalities in bile flow are related to many conditions
> > couldn't yet evalued?
>
> You would be wise to inform your doctor(s) about your symptoms rather
> than attributing them to bile flow.
I was just evaulating. In ancient systems bile was of great importance,
constitutional(personality type), but I think it is not given much
importance?
> > Which out of bile and gastric acid ir related to either unclear or
> > clear motions? Billary diahrria is thought?
>
> No clinical association.
Diahrria can occur when medication for resisting bile absorption is
given?
Anyway, what increased RBC Dist Width(MCV normal) indicates?
Andrew B. Chung, MD/PhD
> Andrew B. Chung, MD/PhD wrote:
> > Kumar wrote:
> snip> > Can gum diseases in diabetic be due to cell's swelling
> interfering in
> > > blood flow and supply resulting into necrosis?
> >
> > In the type 2 diabetic, it is due in part to the inflammatory cytokines
> > from viseral adipose tissue (VAT).
> How inflammatory cytokines causes gum diseases?
By promoting inflammation there.
> > > > > > > > > > > Can glycation also occur in extravascular comparments?
> > > > > > > > > >
> > > > > > > > > > Yes.
> > > > > > > > > >
> > > > > > > > > > > If yes, which
> > > > > > > > > > > conditions are related to it?
> > > > > > > > > >
> > > > > > > > > > Hyperglycemia.
> > > > > > > > >
> > > > > > > > > Hyperglycemia is not glycation but it is a reason to glycation? How
> > > > > > > > > extravascular glycations can effect us?
> > > > > > > >
> > > > > > > > As per earlier discussions.
> > > >
> > > > > I think those were for intravascular glycations?
> > > >
> > > > No.
> >
> > > Soory but if you can tell me again?
> >
> > The glycosylations are happening everywhere and not just
> > intravascularly.
>
> In case of vasoconstriction or reduced blood flow?
Yes.
> > > > > > > Can we combine VAT(please clear, is it VAT--excessive visceral adipose
> > > > > > > tissues or excessive fat store in VAT--greter visceral adiposity),
> > > > > > > glycated substances and absorbed fats which may encourage inflammatory
> > > > > > > responses( as per other topics) as causes to systemic inflammatory
> > > > > > > responses in diabetics?
> > > > > >
> > > > > > It is the inflammatory cytokines produced by the visceral adipose
> > > > > > tissue (VAT) that promote vascular inflammation thereby causing
> > > > > > increased insulin resistance.
> > > >
> > > > > Can't above other conditions/factors add to such inflamation?
> > > >
> > > > It starts with the VAT.
> > >
> > > Whether VAT as you mention, is increase of visceral adipose tissues or
> > > greater adiposity/fat stores in visceral adipose tissues?
> >
> > VAT is simply visceral adipose tissue which can be quantified by
> > measuring waist circumference.
> Can't they store more or less fats? Whether such store is in form of
> triglyciride or fatty acids?
They are present to store fat **and** to release inflammatory cytokines
to cause metabolic syndrome (MetS).
> > > > > > > Btw, whether sugar, carbs, triglycirides(glyceral part)(other are Mg
> > > > > > > and NaCl) attract and absorb water in intestines relieving from
> > > > > > > constipation and resulting into clear motions and increase motility?
> > > > > >
> > > > > > Not clinically seen.
> > > > >
> > > > > They are hygroscopic..somewhat may act as MOM acts?
> > > >
> > > > Still not clinically seen.
> > >
> > > Whether soluble or not solubles fibers are also hygroscopic or releases
> > > some chemicals in intestines?
> >
> > Soluble fiber in the diet comes with its own water so that it is not
> > hygroscopic.
> >
> > > > > Whether bile is hygroscopic? Can bile's abnormal secretion(less) in
> > > > > intestine make a person constipated, lazy esp. in morning, sleeping
> > > > > more, waking late etc.? Two aspects may be related, abnormal bile
> > > > > comong from liver and abnormal secretion from gall bladder(can't say
> > > > > about abnormal re-absorption)?
> > > >
> > > > Clinically not seen.
> >
> > > Can abnormal or obstructed flow of bile from liver to intestine make a
> > > person lazy, more sleepy, waking late etc.?
> >
> > Not clinically seen.
> How bile secretion is triggered?
By CCK released by the physical distension of the stomach.
> Can there be different type or
> quantity of bile secreted in different conditions? > > I feel
> abnormalities in bile flow are related to many conditions
Not clinically seen.
> > > couldn't yet evalued?
> >
> > You would be wise to inform your doctor(s) about your symptoms rather
> > than attributing them to bile flow.
> I was just evaulating. In ancient systems bile was of great importance,
> constitutional(personality type), but I think it is not given much
> importance?
It is not clinically important.
> > > Which out of bile and gastric acid ir related to either unclear or
> > > clear motions? Billary diahrria is thought?
> >
> > No clinical association.
> Diahrria can occur when medication for resisting bile absorption is
> given?
Not clinically seen except as an adverse effect for some.
> Anyway, what increased RBC Dist Width(MCV normal) indicates?
It suggests there may have been some blood loss.
Kumar
Andrew B. Chung, MD/PhD wrote:
> Kumar wrote:
> > Andrew B. Chung, MD/PhD wrote:
> > > Kumar wrote:
> > snip> > Can gum diseases in diabetic be due to cell's swelling
> > interfering in
> > > > blood flow and supply resulting into necrosis?
> > >
> > > In the type 2 diabetic, it is due in part to the inflammatory cytokines
> > > from viseral adipose tissue (VAT).
>
> > How inflammatory cytokines causes gum diseases?
>
> By promoting inflammation there.
brought at the site of inflammation making environment hypotonic and
swelling of cells in that area?
snip> > > The glycosylations are happening everywhere and not just
> > > intravascularly.
> >
> > In case of vasoconstriction or reduced blood flow?
>
> Yes.
intravascularily on vasoconstriction or on decreased blood flow?
Can above feature is valid for all substances including water which are
filtered from capillaries? I am not considering filtring capacity but
quantity of blood flow?
> > > > > > > > Can we combine VAT(please clear, is it VAT--excessive visceral adipose
> > > > > > > > tissues or excessive fat store in VAT--greter visceral adiposity),
> > > > > > > > glycated substances and absorbed fats which may encourage inflammatory
> > > > > > > > responses( as per other topics) as causes to systemic inflammatory
> > > > > > > > responses in diabetics?
> > > > > > >
> > > > > > > It is the inflammatory cytokines produced by the visceral adipose
> > > > > > > tissue (VAT) that promote vascular inflammation thereby causing
> > > > > > > increased insulin resistance.
> > > > >
> > > > > > Can't above other conditions/factors add to such inflamation?
> > > > >
> > > > > It starts with the VAT.
> > > >
> > > > Whether VAT as you mention, is increase of visceral adipose tissues or
> > > > greater adiposity/fat stores in visceral adipose tissues?
> > >
> > > VAT is simply visceral adipose tissue which can be quantified by
> > > measuring waist circumference.
>
> > Can't they store more or less fats? Whether such store is in form of
> > triglyciride or fatty acids?
>
> They are present to store fat **and** to release inflammatory cytokines
> to cause metabolic syndrome (MetS).
Sorry pls clear; "They are present to store fat"?
> > > > > > > > Btw, whether sugar, carbs, triglycirides(glyceral part)(other are Mg
> > > > > > > > and NaCl) attract and absorb water in intestines relieving from
> > > > > > > > constipation and resulting into clear motions and increase motility?
> > > > > > >
> > > > > > > Not clinically seen.
> > > > > >
> > > > > > They are hygroscopic..somewhat may act as MOM acts?
> > > > >
> > > > > Still not clinically seen.
> > > >
> > > > Whether soluble or not solubles fibers are also hygroscopic or releases
> > > > some chemicals in intestines?
> > >
> > > Soluble fiber in the diet comes with its own water so that it is not
> > > hygroscopic.
Whether Non-soluble fibers are hygroscopic? Can isabgol husk absorb
bile and cause more excretion of bile with stool?
> > > > > > Whether bile is hygroscopic? Can bile's abnormal secretion(less) in
> > > > > > intestine make a person constipated, lazy esp. in morning, sleeping
> > > > > > more, waking late etc.? Two aspects may be related, abnormal bile
> > > > > > comong from liver and abnormal secretion from gall bladder(can't say
> > > > > > about abnormal re-absorption)?
> > > > >
> > > > > Clinically not seen.
> > >
> > > > Can abnormal or obstructed flow of bile from liver to intestine make a
> > > > person lazy, more sleepy, waking late etc.?
> > >
> > > Not clinically seen.
>
> > How bile secretion is triggered?
>
> By CCK released by the physical distension of the stomach.
Caningested fats promote bile secretion from in intestine?
> > Can there be different type or
> > quantity of bile secreted in different conditions? > > I feel
> > abnormalities in bile flow are related to many conditions
>
> Not clinically seen.
>
> > > > couldn't yet evalued?
> > >
> > > You would be wise to inform your doctor(s) about your symptoms rather
> > > than attributing them to bile flow.
>
> > I was just evaulating. In ancient systems bile was of great importance,
> > constitutional(personality type), but I think it is not given much
> > importance?
>
> It is not clinically important.
Whether composition of bile can be changed in different conditions?
Whether bile detoxfy blood?
> > > > Which out of bile and gastric acid ir related to either unclear or
> > > > clear motions? Billary diahrria is thought?
> > >
> > > No clinical association.
>
> > Diahrria can occur when medication for resisting bile absorption is
> > given?
>
> Not clinically seen except as an adverse effect for some.
>
> > Anyway, what increase RBC Dist Width(MCV normal) indicates?
>
> It suggests there may have been some blood loss.
How? Is there any relation between increase in RBC dist width,
decreased hematocrit and increased neutrophills?
Whether blood loss is compensated by water to maintain blood volume?
Can you define RBC dist width for me pls?
Andrew B. Chung, MD/PhD
> Andrew B. Chung, MD/PhD wrote:
> > Kumar wrote:
> > > Andrew B. Chung, MD/PhD wrote:
> > > > Kumar wrote:
> > > snip> > Can gum diseases in diabetic be due to cell's swelling
> > > interfering in
> > > > > blood flow and supply resulting into necrosis?
> > > >
> > > > In the type 2 diabetic, it is due in part to the inflammatory cytokines
> > > > from viseral adipose tissue (VAT).
> >
> > > How inflammatory cytokines causes gum diseases?
> >
> > By promoting inflammation there.
> Swelling is one feature of inflammation. Whether more water/fluid is
> brought at the site of inflammation making environment hypotonic and
> swelling of cells in that area?
No.
> snip> > > The glycosylations are happening everywhere and not just
> > > > intravascularly.
> > >
> > > In case of vasoconstriction or reduced blood flow?
> >
> > Yes.
> Will such glycosylation be reduced extravascularily but increased
> intravascularily on vasoconstriction or on decreased blood flow?
No.
> Can above feature is valid for all substances including water which are
> filtered from capillaries? I am not considering filtring capacity but
> quantity of blood flow?
It is relevant to all substances that are able to freely cross through
the capillary basement membrane.
> > > > > > > > > Can we combine VAT(please clear, is it VAT--excessive visceral adipose
> > > > > > > > > tissues or excessive fat store in VAT--greter visceral adiposity),
> > > > > > > > > glycated substances and absorbed fats which may encourage inflammatory
> > > > > > > > > responses( as per other topics) as causes to systemic inflammatory
> > > > > > > > > responses in diabetics?
> > > > > > > >
> > > > > > > > It is the inflammatory cytokines produced by the visceral adipose
> > > > > > > > tissue (VAT) that promote vascular inflammation thereby causing
> > > > > > > > increased insulin resistance.
> > > > > >
> > > > > > > Can't above other conditions/factors add to such inflamation?
> > > > > >
> > > > > > It starts with the VAT.
> > > > >
> > > > > Whether VAT as you mention, is increase of visceral adipose tissues or
> > > > > greater adiposity/fat stores in visceral adipose tissues?
> > > >
> > > > VAT is simply visceral adipose tissue which can be quantified by
> > > > measuring waist circumference.
> >
> > > Can't they store more or less fats? Whether such store is in form of
> > > triglyciride or fatty acids?
> >
> > They are present to store fat **and** to release inflammatory cytokines
> > to cause metabolic syndrome (MetS).
> Sorry pls clear; "They are present to store fat"?
Yes. Such is the nature of adipose tissue even if pathological as in
the case of VAT.
> > > > > > > > > Btw, whether sugar, carbs, triglycirides(glyceral part)(other are Mg
> > > > > > > > > and NaCl) attract and absorb water in intestines relieving from
> > > > > > > > > constipation and resulting into clear motions and increase motility?
> > > > > > > >
> > > > > > > > Not clinically seen.
> > > > > > >
> > > > > > > They are hygroscopic..somewhat may act as MOM acts?
> > > > > >
> > > > > > Still not clinically seen.
> > > > >
> > > > > Whether soluble or not solubles fibers are also hygroscopic or releases
> > > > > some chemicals in intestines?
> > > >
> > > > Soluble fiber in the diet comes with its own water so that it is not
> > > > hygroscopic.
>
> Whether Non-soluble fibers are hygroscopic? Can isabgol husk absorb
> bile and cause more excretion of bile with stool?
Not familiar with isabgol husk.
> > > > > > > Whether bile is hygroscopic? Can bile's abnormal secretion(less) in
> > > > > > > intestine make a person constipated, lazy esp. in morning, sleeping
> > > > > > > more, waking late etc.? Two aspects may be related, abnormal bile
> > > > > > > comong from liver and abnormal secretion from gall bladder(can't say
> > > > > > > about abnormal re-absorption)?
> > > > > >
> > > > > > Clinically not seen.
> > > >
> > > > > Can abnormal or obstructed flow of bile from liver to intestine make a
> > > > > person lazy, more sleepy, waking late etc.?
> > > >
> > > > Not clinically seen.
> >
> > > How bile secretion is triggered?
> >
> > By CCK released by the physical distension of the stomach.
> Caningested fats promote bile secretion from in intestine?
Bile is not secreted from the intestine but rather comes from the
liver.
> > > Can there be different type or
> > > quantity of bile secreted in different conditions? > > I feel
> > > abnormalities in bile flow are related to many conditions
> >
> > Not clinically seen.
> >
> > > > > couldn't yet evalued?
> > > >
> > > > You would be wise to inform your doctor(s) about your symptoms rather
> > > > than attributing them to bile flow.
> >
> > > I was just evaulating. In ancient systems bile was of great importance,
> > > constitutional(personality type), but I think it is not given much
> > > importance?
> >
> > It is not clinically important.
>
> Whether composition of bile can be changed in different conditions?
> Whether bile detoxfy blood?
Bile is used to emulsify ingested fats to aid in their digestion.
> > > > > Which out of bile and gastric acid ir related to either unclear or
> > > > > clear motions? Billary diahrria is thought?
> > > >
> > > > No clinical association.
> >
> > > Diahrria can occur when medication for resisting bile absorption is
> > > given?
> >
> > Not clinically seen except as an adverse effect for some.
> >
> > > Anyway, what increase RBC Dist Width(MCV normal) indicates?
> >
> > It suggests there may have been some blood loss.
> How? Is there any relation between increase in RBC dist width,
> decreased hematocrit and increased neutrophills?
Would suggest that there is infection **and** blood loss.
> Whether blood loss is compensated by water to maintain blood volume?
It is if blood pressure remains normal even upon standing without
abnormally elevated heart rate.
> Can you define RBC dist width for me pls?
It is the range in RBC widths that would include 90% of all red bloods
cells (RBCs).
Kumar
Andrew B. Chung, MD/PhD wrote:
> Kumar wrote:
> > Andrew B. Chung, MD/PhD wrote:
> > > Kumar wrote:
> > > > Andrew B. Chung, MD/PhD wrote:
> > > > > Kumar wrote:
> > > > snip> > Can gum diseases in diabetic be due to cell's swelling
> > > > interfering in
> > > > > > blood flow and supply resulting into necrosis?
> > > > >
> > > > > In the type 2 diabetic, it is due in part to the inflammatory cytokines
> > > > > from viseral adipose tissue (VAT).
> > >
> > > > How inflammatory cytokines causes gum diseases?
> > >
> > > By promoting inflammation there.
>
> > Swelling is one feature of inflammation. Whether more water/fluid is
> > brought at the site of inflammation making environment hypotonic and
> > swelling of cells in that area?
>
> No.
Why it can't be possible?
> > snip> > > The glycosylations are happening everywhere and not just
> > > > > intravascularly.
> > > >
> > > > In case of vasoconstriction or reduced blood flow?
> > >
> > > Yes.
>
> > Will such glycosylation be reduced extravascularily but increased
> > intravascularily on vasoconstriction or on decreased blood flow?
>
> No.
Will glucose or even water be filtered lesser in tatal quantity from
capalliries, if blood flow is reduced due to vasocontriction?
> > Can above feature is valid for all substances including water which are
> > filtered from capillaries? I am not considering filtring capacity but
> > quantity of blood flow?
>
> It is relevant to all substances that are able to freely cross through
> the capillary basement membrane.
Yes but total blood flow can be reduced due to vsoconstriction? Whether
total filteration can't be also dependant and proportionate to total
blood flow in any area?
snip> > > > Can't they store more or less fats? Whether such store is
in form of
> > > > triglyciride or fatty acids?
> > >
> > > They are present to store fat **and** to release inflammatory cytokines
> > > to cause metabolic syndrome (MetS).
>
> > Sorry pls clear; "They are present to store fat"?
>
> Yes. Such is the nature of adipose tissue even if pathological as in
> the case of VAT.
Can they store more or less fats in quantity? Can they be substancially
increased in numbers?
> > > > > > > > > > Btw, whether sugar, carbs, triglycirides(glyceral part)(other are Mg
> > > > > > > > > > and NaCl) attract and absorb water in intestines relieving from
> > > > > > > > > > constipation and resulting into clear motions and increase motility?
> > > > > > > > >
> > > > > > > > > Not clinically seen.
> > > > > > > >
> > > > > > > > They are hygroscopic..somewhat may act as MOM acts?
> > > > > > >
> > > > > > > Still not clinically seen.
> > > > > >
> > > > > > Whether soluble or not solubles fibers are also hygroscopic or releases
> > > > > > some chemicals in intestines?
> > > > >
> > > > > Soluble fiber in the diet comes with its own water so that it is not
> > > > > hygroscopic.
> >
> > Whether Non-soluble fibers are hygroscopic? Can isabgol husk absorb
> > bile and cause more excretion of bile with stool?
>
> Not familiar with isabgol husk.
It is well known. Anyway,
"Isabgol (also Sat-Isabgol or Isapgol), is the Indian term for psyllium
seed husks (not a trademark). Isabgol is used for constipation and
irritable bowel syndrome. A few brand names are "B. G. Telephone Brand
Sat-Isabgol", "Deer Brand Sat Isabgol" and "Dabur Sat Isabgol".
Isabgol is also avaiable internationally in Indian shops and markets,
but as it is typically sold as isabgol rather than psyllium, many
people are not aware of that. Being a generic product, it is
significantly cheaper than similar products sold in pharmacies. Isabgol
(psyllium) is the active ingredient of the American product Metamucil.
http://en.wikipedia.org/wiki/Isabgol "
> > > > > > > > Whether bile is hygroscopic? Can bile's abnormal secretion(less) in
> > > > > > > > intestine make a person constipated, lazy esp. in morning, sleeping
> > > > > > > > more, waking late etc.? Two aspects may be related, abnormal bile
> > > > > > > > comong from liver and abnormal secretion from gall bladder(can't say
> > > > > > > > about abnormal re-absorption)?
> > > > > > >
> > > > > > > Clinically not seen.
> > > > >
> > > > > > Can abnormal or obstructed flow of bile from liver to intestine make a
> > > > > > person lazy, more sleepy, waking late etc.?
> > > > >
> > > > > Not clinically seen.
> > >
> > > > How bile secretion is triggered?
> > >
> > > By CCK released by the physical distension of the stomach.
>
> > Can ingested fats promote bile secretion [from] in intestine?
>
> Bile is not secreted from the intestine but rather comes from the
> liver.
Sorry, I meant; Can ingested fats promote bile secretion in intestine?
> > > > Can there be different type or
> > > > quantity of bile secreted in different conditions? > > I feel
> > > > abnormalities in bile flow are related to many conditions
> > >
> > > Not clinically seen.
> > >
> > > > > > couldn't yet evalued?
> > > > >
> > > > > You would be wise to inform your doctor(s) about your symptoms rather
> > > > > than attributing them to bile flow.
> > >
> > > > I was just evaulating. In ancient systems bile was of great importance,
> > > > constitutional(personality type), but I think it is not given much
> > > > importance?
> > >
> > > It is not clinically important.
> >
> > Whether composition of bile can be changed in different conditions?
> > Whether bile detoxfy blood?
>
> Bile is used to emulsify ingested fats to aid in their digestion.
Yes but if its composition and secreted quantity can be variable due to
different type of fats/foods?
> > > > > > Which out of bile and gastric acid ir related to either unclear or
> > > > > > clear motions? Billary diahrria is thought?
> > > > >
> > > > > No clinical association.
> > >
> > > > Diahrria can occur when medication for resisting bile absorption is
> > > > given?
> > >
> > > Not clinically seen except as an adverse effect for some.
> > >
> > > > Anyway, what increase RBC Dist Width(MCV normal) indicates?
> > >
> > > It suggests there may have been some blood loss.
>
> > How? Is there any relation between increase in RBC dist width,
> > decreased hematocrit and increased neutrophills?
>
> Would suggest that there is infection **and** blood loss.
>
> > Whether blood loss is compensated by water to maintain blood volume?
>
> It is if blood pressure remains normal even upon standing without
> abnormally elevated heart rate.
>
> > Can you define RBC dist width for me pls?
>
> It is the range in RBC widths that would include 90% of all red bloods
> cells (RBCs).
Whether its higher reading tells abnormal macrocytic size and shape of
RBCs?
Whether some higher reading of RDW, low Hct and some higher neutrophils
readin(may be subclinically higher but still within commonly indicated
normal range)gs can be common in people with persisting hyperglycemia
and IR?
Andrew B. Chung, MD/PhD
> Andrew B. Chung, MD/PhD wrote:
> > Kumar wrote:
> > > Andrew B. Chung, MD/PhD wrote:
> > > > Kumar wrote:
> > > > > Andrew B. Chung, MD/PhD wrote:
> > > > > > Kumar wrote:
> > > > > snip> > Can gum diseases in diabetic be due to cell's swelling
> > > > > interfering in
> > > > > > > blood flow and supply resulting into necrosis?
> > > > > >
> > > > > > In the type 2 diabetic, it is due in part to the inflammatory cytokines
> > > > > > from viseral adipose tissue (VAT).
> > > >
> > > > > How inflammatory cytokines causes gum diseases?
> > > >
> > > > By promoting inflammation there.
> >
> > > Swelling is one feature of inflammation. Whether more water/fluid is
> > > brought at the site of inflammation making environment hypotonic and
> > > swelling of cells in that area?
> >
> > No.
>
> Why it can't be possible?
It simply is not seen.
> > > snip> > > The glycosylations are happening everywhere and not just
> > > > > > intravascularly.
> > > > >
> > > > > In case of vasoconstriction or reduced blood flow?
> > > >
> > > > Yes.
> >
> > > Will such glycosylation be reduced extravascularily but increased
> > > intravascularily on vasoconstriction or on decreased blood flow?
> >
> > No.
> Will glucose or even water be filtered lesser in tatal quantity from
> capalliries, if blood flow is reduced due to vasocontriction?
Not to any clinically meaningful extent.
> > > Can above feature is valid for all substances including water which are
> > > filtered from capillaries? I am not considering filtring capacity but
> > > quantity of blood flow?
> >
> > It is relevant to all substances that are able to freely cross through
> > the capillary basement membrane.
>
> Yes but total blood flow can be reduced due to vsoconstriction? Whether
> total filteration can't be also dependant and proportionate to total
> blood flow in any area?
Transient events are not clinically meaningful in chronic disease such
as type-2 diabetes.
> snip> > > > Can't they store more or less fats? Whether such store is
> in form of
> > > > > triglyciride or fatty acids?
> > > >
> > > > They are present to store fat **and** to release inflammatory cytokines
> > > > to cause metabolic syndrome (MetS).
> >
> > > Sorry pls clear; "They are present to store fat"?
> >
> > Yes. Such is the nature of adipose tissue even if pathological as in
> > the case of VAT.
>
> Can they store more or less fats in quantity? Can they be substancially
> increased in numbers?
Wiser to get rid of the VAT rather than dwell on it.
> > > > > > > > > > > Btw, whether sugar, carbs, triglycirides(glyceral part)(other are Mg
> > > > > > > > > > > and NaCl) attract and absorb water in intestines relieving from
> > > > > > > > > > > constipation and resulting into clear motions and increase motility?
> > > > > > > > > >
> > > > > > > > > > Not clinically seen.
> > > > > > > > >
> > > > > > > > > They are hygroscopic..somewhat may act as MOM acts?
> > > > > > > >
> > > > > > > > Still not clinically seen.
> > > > > > >
> > > > > > > Whether soluble or not solubles fibers are also hygroscopic or releases
> > > > > > > some chemicals in intestines?
> > > > > >
> > > > > > Soluble fiber in the diet comes with its own water so that it is not
> > > > > > hygroscopic.
> > >
> > > Whether Non-soluble fibers are hygroscopic? Can isabgol husk absorb
> > > bile and cause more excretion of bile with stool?
> >
> > Not familiar with isabgol husk.
>
> It is well known. Anyway,
>
> "Isabgol (also Sat-Isabgol or Isapgol), is the Indian term for psyllium
> seed husks (not a trademark). Isabgol is used for constipation and
> irritable bowel syndrome. A few brand names are "B. G. Telephone Brand
> Sat-Isabgol", "Deer Brand Sat Isabgol" and "Dabur Sat Isabgol".
>
> Isabgol is also avaiable internationally in Indian shops and markets,
> but as it is typically sold as isabgol rather than psyllium, many
> people are not aware of that. Being a generic product, it is
> significantly cheaper than similar products sold in pharmacies. Isabgol
> (psyllium) is the active ingredient of the American product Metamucil.
> http://en.wikipedia.org/wiki/Isabgol "
Am familiar with psyllium. And, yes, it can help to lower cholesterol
by slowing down recovery of bile salts increasing losses in stool.
> > > > > > > > > Whether bile is hygroscopic? Can bile's abnormal secretion(less) in
> > > > > > > > > intestine make a person constipated, lazy esp. in morning, sleeping
> > > > > > > > > more, waking late etc.? Two aspects may be related, abnormal bile
> > > > > > > > > comong from liver and abnormal secretion from gall bladder(can't say
> > > > > > > > > about abnormal re-absorption)?
> > > > > > > >
> > > > > > > > Clinically not seen.
> > > > > >
> > > > > > > Can abnormal or obstructed flow of bile from liver to intestine make a
> > > > > > > person lazy, more sleepy, waking late etc.?
> > > > > >
> > > > > > Not clinically seen.
> > > >
> > > > > How bile secretion is triggered?
> > > >
> > > > By CCK released by the physical distension of the stomach.
> >
> > > Can ingested fats promote bile secretion [from] in intestine?
> >
> > Bile is not secreted from the intestine but rather comes from the
> > liver.
>
> Sorry, I meant; Can ingested fats promote bile secretion in intestine?
Bile production and release into the intestine is stimulated by
ingested fats.
> > > > > Can there be different type or
> > > > > quantity of bile secreted in different conditions? > > I feel
> > > > > abnormalities in bile flow are related to many conditions
> > > >
> > > > Not clinically seen.
> > > >
> > > > > > > couldn't yet evalued?
> > > > > >
> > > > > > You would be wise to inform your doctor(s) about your symptoms rather
> > > > > > than attributing them to bile flow.
> > > >
> > > > > I was just evaulating. In ancient systems bile was of great importance,
> > > > > constitutional(personality type), but I think it is not given much
> > > > > importance?
> > > >
> > > > It is not clinically important.
> > >
> > > Whether composition of bile can be changed in different conditions?
> > > Whether bile detoxfy blood?
> >
> > Bile is used to emulsify ingested fats to aid in their digestion.
>
> Yes but if its composition and secreted quantity can be variable due to
> different type of fats/foods?
Compostion, no. Quantity, yes.
> > > > > > > Which out of bile and gastric acid ir related to either unclear or
> > > > > > > clear motions? Billary diahrria is thought?
> > > > > >
> > > > > > No clinical association.
> > > >
> > > > > Diahrria can occur when medication for resisting bile absorption is
> > > > > given?
> > > >
> > > > Not clinically seen except as an adverse effect for some.
> > > >
> > > > > Anyway, what increase RBC Dist Width(MCV normal) indicates?
> > > >
> > > > It suggests there may have been some blood loss.
> >
> > > How? Is there any relation between increase in RBC dist width,
> > > decreased hematocrit and increased neutrophills?
> >
> > Would suggest that there is infection **and** blood loss.
> >
> > > Whether blood loss is compensated by water to maintain blood volume?
> >
> > It is if blood pressure remains normal even upon standing without
> > abnormally elevated heart rate.
> >
> > > Can you define RBC dist width for me pls?
> >
> > It is the range in RBC widths that would include 90% of all red bloods
> > cells (RBCs).
>
> Whether its higher reading tells abnormal macrocytic size and shape of
> RBCs?
Higher RDWs can be associated with abnormal forms/shapes.
> Whether some higher reading of RDW, low Hct and some higher neutrophils
> readin(may be subclinically higher but still within commonly indicated
> normal range)gs can be common in people with persisting hyperglycemia
> and IR?
Not clinically seen.
Kumar
Andrew B. Chung, MD/PhD wrote:
> Kumar wrote:
> > Andrew B. Chung, MD/PhD wrote:
> > > Kumar wrote:
> > > > Andrew B. Chung, MD/PhD wrote:
> > > > > Kumar wrote:
> > > > > > Andrew B. Chung, MD/PhD wrote:
> > > > > > > Kumar wrote:
> > > > > > snip> > Can gum diseases in diabetic be due to cell's swelling
> > > > > > interfering in
> > > > > > > > blood flow and supply resulting into necrosis?
> > > > > > >
> > > > > > > In the type 2 diabetic, it is due in part to the inflammatory cytokines
> > > > > > > from viseral adipose tissue (VAT).
> > > > >
> > > > > > How inflammatory cytokines causes gum diseases?
> > > > >
> > > > > By promoting inflammation there.
> > >
> > > > Swelling is one feature of inflammation. Whether more water/fluid is
> > > > brought at the site of inflammation making environment hypotonic and
> > > > swelling of cells in that area?
> > >
> > > No.
> >
> > Why it can't be possible?
>
> It simply is not seen.
Can variation in osmotic pressure of capaliries effect trans-capillary
movements of insulin?
> > > > snip> > > The glycosylations are happening everywhere and not just
> > > > > > > intravascularly.
> > > > > >
> > > > > > In case of vasoconstriction or reduced blood flow?
> > > > >
> > > > > Yes.
> > >
> > > > Will such glycosylation be reduced extravascularily but increased
> > > > intravascularily on vasoconstriction or on decreased blood flow?
> > >
> > > No.
>
> > Will glucose or even water be filtered lesser in tatal quantity from
> > capalliries, if blood flow is reduced due to vasocontriction?
>
> Not to any clinically meaningful extent.
>
> > > > Can above feature is valid for all substances including water which are
> > > > filtered from capillaries? I am not considering filtring capacity but
> > > > quantity of blood flow?
> > >
> > > It is relevant to all substances that are able to freely cross through
> > > the capillary basement membrane.
> >
> > Yes but total blood flow can be reduced due to vsoconstriction? Whether
> > total filteration can't be also dependant and proportionate to total
> > blood flow in any area?
>
> Transient events are not clinically meaningful in chronic disease such
> as type-2 diabetes.
Yes, but can't there be persistance of such events due to persisting
hyperglycemia?
>> > > > > > > Soluble fiber in the diet comes with its own water so that it is not
> > > > > > > hygroscopic.
> > > >
> > > > Whether Non-soluble fibers are hygroscopic? Can isabgol husk absorb
> > > > bile and cause more excretion of bile with stool?
> > >
> > > Not familiar with isabgol husk.
> >
> > It is well known. Anyway,
> >
> > "Isabgol (also Sat-Isabgol or Isapgol), is the Indian term for psyllium
> > seed husks (not a trademark). Isabgol is used for constipation and
> > irritable bowel syndrome. A few brand names are "B. G. Telephone Brand
> > Sat-Isabgol", "Deer Brand Sat Isabgol" and "Dabur Sat Isabgol".
> >
> > Isabgol is also avaiable internationally in Indian shops and markets,
> > but as it is typically sold as isabgol rather than psyllium, many
> > people are not aware of that. Being a generic product, it is
> > significantly cheaper than similar products sold in pharmacies. Isabgol
> > (psyllium) is the active ingredient of the American product Metamucil.
> > http://en.wikipedia.org/wiki/Isabgol "
>
> Am familiar with psyllium. And, yes, it can help to lower cholesterol
> by slowing down recovery of bile salts increasing losses in stool.
It is indicative that butter fat/pure ghee attract more bile, so while
increasing cholestreol it also excrete more bile resulting into
cholestreol's beneficial roles as well controlling their levels?
> > > > > > > > > > Whether bile is hygroscopic? Can bile's abnormal secretion(less) in
> > > > > > > > > > intestine make a person constipated, lazy esp. in morning, sleeping
> > > > > > > > > > more, waking late etc.? Two aspects may be related, abnormal bile
> > > > > > > > > > comong from liver and abnormal secretion from gall bladder(can't say
> > > > > > > > > > about abnormal re-absorption)?
> > > > > > > > >
> > > > > > > > > Clinically not seen.
> > > > > > >
> > > > > > > > Can abnormal or obstructed flow of bile from liver to intestine make a
> > > > > > > > person lazy, more sleepy, waking late etc.?
> > > > > > >
> > > > > > > Not clinically seen.
> > > > >
> > > > > > How bile secretion is triggered?
> > > > >
> > > > > By CCK released by the physical distension of the stomach.
> > >
> > > > Can ingested fats promote bile secretion [from] in intestine?
> > >
> > > Bile is not secreted from the intestine but rather comes from the
> > > liver.
> >
> > Sorry, I meant; Can ingested fats promote bile secretion in intestine?
>
> Bile production and release into the intestine is stimulated by
> ingested fats.
Whether butterfat/pure ghee stimulate comparatively more bile than
begetable oils?
> > > > > > Can there be different type or
> > > > > > quantity of bile secreted in different conditions? > > I feel
> > > > > > abnormalities in bile flow are related to many conditions
> > > > >
> > > > > Not clinically seen.
> > > > >
> > > > > > > > couldn't yet evalued?
> > > > > > >
> > > > > > > You would be wise to inform your doctor(s) about your symptoms rather
> > > > > > > than attributing them to bile flow.
> > > > >
> > > > > > I was just evaulating. In ancient systems bile was of great importance,
> > > > > > constitutional(personality type), but I think it is not given much
> > > > > > importance?
> > > > >
> > > > > It is not clinically important.
> > > >
> > > > Whether composition of bile can be changed in different conditions?
> > > > Whether bile detoxfy blood?
> > >
> > > Bile is used to emulsify ingested fats to aid in their digestion.
> >
> > Yes but if its composition and secreted quantity can be variable due to
> > different type of fats/foods?
>
> Compostion, no. Quantity, yes.
Can it be secretated in intestine less or more concentrated?
> > > > > > > > Which out of bile and gastric acid ir related to either unclear or
> > > > > > > > clear motions? Billary diahrria is thought?
> > > > > > >
> > > > > > > No clinical association.
> > > > >
> > > > > > Diahrria can occur when medication for resisting bile absorption is
> > > > > > given?
> > > > >
> > > > > Not clinically seen except as an adverse effect for some.
> > > > >
> > > > > > Anyway, what increase RBC Dist Width(MCV normal) indicates?
> > > > >
> > > > > It suggests there may have been some blood loss.
> > >
> > > > How? Is there any relation between increase in RBC dist width,
> > > > decreased hematocrit and increased neutrophills?
> > >
> > > Would suggest that there is infection **and** blood loss.
> > >
> > > > Whether blood loss is compensated by water to maintain blood volume?
> > >
> > > It is if blood pressure remains normal even upon standing without
> > > abnormally elevated heart rate.
> > >
> > > > Can you define RBC dist width for me pls?
> > >
> > > It is the range in RBC widths that would include 90% of all red bloods
> > > cells (RBCs).
> >
> > Whether its higher reading tells abnormal macrocytic size and shape of
> > RBCs?
>
> Higher RDWs can be associated with abnormal forms/shapes.
Will such RBCs be macrocytic/megablastic?
> > Whether some higher reading of RDW, low Hct and some higher neutrophils
> > reading (may be subclinically higher but still within commonly indicated
> > normal range)gs can be common in people with persisting hyperglycemia
> > and IR?
>
> Not clinically seen.
Can low Hct or hypotonicity of blood increase RTWs?
Whether swelled cells in vascular wall or circulating attracts
inflammatory response or oxidative mechanism?
> > > > > May GOD continue to heal your heart by curing your diabetes, dear
> > > > > neighbor Kumar whom I love unconditionally.
Oblized.
Andrew B. Chung, MD/PhD
> Andrew B. Chung, MD/PhD wrote:
> > Kumar wrote:
> > > Andrew B. Chung, MD/PhD wrote:
> > > > Kumar wrote:
> > > > > Andrew B. Chung, MD/PhD wrote:
> > > > > > Kumar wrote:
> > > > > > > Andrew B. Chung, MD/PhD wrote:
> > > > > > > > Kumar wrote:
> > > > > > > snip> > Can gum diseases in diabetic be due to cell's swelling
> > > > > > > interfering in
> > > > > > > > > blood flow and supply resulting into necrosis?
> > > > > > > >
> > > > > > > > In the type 2 diabetic, it is due in part to the inflammatory cytokines
> > > > > > > > from viseral adipose tissue (VAT).
> > > > > >
> > > > > > > How inflammatory cytokines causes gum diseases?
> > > > > >
> > > > > > By promoting inflammation there.
> > > >
> > > > > Swelling is one feature of inflammation. Whether more water/fluid is
> > > > > brought at the site of inflammation making environment hypotonic and
> > > > > swelling of cells in that area?
> > > >
> > > > No.
> > >
> > > Why it can't be possible?
> >
> > It simply is not seen.
>
> Can variation in osmotic pressure of capaliries effect trans-capillary
> movements of insulin?
Not clinically seen.
> > > > > snip> > > The glycosylations are happening everywhere and not just
> > > > > > > > intravascularly.
> > > > > > >
> > > > > > > In case of vasoconstriction or reduced blood flow?
> > > > > >
> > > > > > Yes.
> > > >
> > > > > Will such glycosylation be reduced extravascularily but increased
> > > > > intravascularily on vasoconstriction or on decreased blood flow?
> > > >
> > > > No.
> >
> > > Will glucose or even water be filtered lesser in tatal quantity from
> > > capalliries, if blood flow is reduced due to vasocontriction?
> >
> > Not to any clinically meaningful extent.
> >
> > > > > Can above feature is valid for all substances including water which are
> > > > > filtered from capillaries? I am not considering filtring capacity but
> > > > > quantity of blood flow?
> > > >
> > > > It is relevant to all substances that are able to freely cross through
> > > > the capillary basement membrane.
> > >
> > > Yes but total blood flow can be reduced due to vsoconstriction? Whether
> > > total filteration can't be also dependant and proportionate to total
> > > blood flow in any area?
> >
> > Transient events are not clinically meaningful in chronic disease such
> > as type-2 diabetes.
>
> Yes, but can't there be persistance of such events due to persisting
> hyperglycemia?
Hyperglycemia does not cause vasoconstriction.
> >> > > > > > > Soluble fiber in the diet comes with its own water so that it is not
> > > > > > > > hygroscopic.
> > > > >
> > > > > Whether Non-soluble fibers are hygroscopic? Can isabgol husk absorb
> > > > > bile and cause more excretion of bile with stool?
> > > >
> > > > Not familiar with isabgol husk.
> > >
> > > It is well known. Anyway,
> > >
> > > "Isabgol (also Sat-Isabgol or Isapgol), is the Indian term for psyllium
> > > seed husks (not a trademark). Isabgol is used for constipation and
> > > irritable bowel syndrome. A few brand names are "B. G. Telephone Brand
> > > Sat-Isabgol", "Deer Brand Sat Isabgol" and "Dabur Sat Isabgol".
> > >
> > > Isabgol is also avaiable internationally in Indian shops and markets,
> > > but as it is typically sold as isabgol rather than psyllium, many
> > > people are not aware of that. Being a generic product, it is
> > > significantly cheaper than similar products sold in pharmacies. Isabgol
> > > (psyllium) is the active ingredient of the American product Metamucil.
> > > http://en.wikipedia.org/wiki/Isabgol "
> >
> > Am familiar with psyllium. And, yes, it can help to lower cholesterol
> > by slowing down recovery of bile salts increasing losses in stool.
>
> It is indicative that butter fat/pure ghee attract more bile, so while
> increasing cholestreol it also excrete more bile resulting into
> cholestreol's beneficial roles as well controlling their levels?
Not clinically seen.
> > > > > > > > > > > Whether bile is hygroscopic? Can bile's abnormal secretion(less) in
> > > > > > > > > > > intestine make a person constipated, lazy esp. in morning, sleeping
> > > > > > > > > > > more, waking late etc.? Two aspects may be related, abnormal bile
> > > > > > > > > > > comong from liver and abnormal secretion from gall bladder(can't say
> > > > > > > > > > > about abnormal re-absorption)?
> > > > > > > > > >
> > > > > > > > > > Clinically not seen.
> > > > > > > >
> > > > > > > > > Can abnormal or obstructed flow of bile from liver to intestine make a
> > > > > > > > > person lazy, more sleepy, waking late etc.?
> > > > > > > >
> > > > > > > > Not clinically seen.
> > > > > >
> > > > > > > How bile secretion is triggered?
> > > > > >
> > > > > > By CCK released by the physical distension of the stomach.
> > > >
> > > > > Can ingested fats promote bile secretion [from] in intestine?
> > > >
> > > > Bile is not secreted from the intestine but rather comes from the
> > > > liver.
> > >
> > > Sorry, I meant; Can ingested fats promote bile secretion in intestine?
> >
> > Bile production and release into the intestine is stimulated by
> > ingested fats.
> Whether butterfat/pure ghee stimulate comparatively more bile than
> begetable oils?
Not clinically seen.
> > > > > > > Can there be different type or
> > > > > > > quantity of bile secreted in different conditions? > > I feel
> > > > > > > abnormalities in bile flow are related to many conditions
> > > > > >
> > > > > > Not clinically seen.
> > > > > >
> > > > > > > > > couldn't yet evalued?
> > > > > > > >
> > > > > > > > You would be wise to inform your doctor(s) about your symptoms rather
> > > > > > > > than attributing them to bile flow.
> > > > > >
> > > > > > > I was just evaulating. In ancient systems bile was of great importance,
> > > > > > > constitutional(personality type), but I think it is not given much
> > > > > > > importance?
> > > > > >
> > > > > > It is not clinically important.
> > > > >
> > > > > Whether composition of bile can be changed in different conditions?
> > > > > Whether bile detoxfy blood?
> > > >
> > > > Bile is used to emulsify ingested fats to aid in their digestion.
> > >
> > > Yes but if its composition and secreted quantity can be variable due to
> > > different type of fats/foods?
> >
> > Composition, no. Quantity, yes.
>
> Can it be secretated in intestine less or more concentrated?
The latter would be the purpose of the gall bladder.
> > > > > > > > > Which out of bile and gastric acid ir related to either unclear or
> > > > > > > > > clear motions? Billary diahrria is thought?
> > > > > > > >
> > > > > > > > No clinical association.
> > > > > >
> > > > > > > Diahrria can occur when medication for resisting bile absorption is
> > > > > > > given?
> > > > > >
> > > > > > Not clinically seen except as an adverse effect for some.
> > > > > >
> > > > > > > Anyway, what increase RBC Dist Width(MCV normal) indicates?
> > > > > >
> > > > > > It suggests there may have been some blood loss.
> > > >
> > > > > How? Is there any relation between increase in RBC dist width,
> > > > > decreased hematocrit and increased neutrophills?
> > > >
> > > > Would suggest that there is infection **and** blood loss.
> > > >
> > > > > Whether blood loss is compensated by water to maintain blood volume?
> > > >
> > > > It is if blood pressure remains normal even upon standing without
> > > > abnormally elevated heart rate.
> > > >
> > > > > Can you define RBC dist width for me pls?
> > > >
> > > > It is the range in RBC widths that would include 90% of all red bloods
> > > > cells (RBCs).
> > >
> > > Whether its higher reading tells abnormal macrocytic size and shape of
> > > RBCs?
> >
> > Higher RDWs can be associated with abnormal forms/shapes.
>
> Will such RBCs be macrocytic/megablastic?
They are often known as schizocytes.
> > > Whether some higher reading of RDW, low Hct and some higher neutrophils
> > > reading (may be subclinically higher but still within commonly indicated
> > > normal range)gs can be common in people with persisting hyperglycemia
> > > and IR?
> >
> > Not clinically seen.
> Can low Hct or hypotonicity of blood increase RTWs?
Blood loss with normal production of red cells by the bone marrow to
bring the Hct back up would typically increase RDW.
> Whether swelled cells in vascular wall or circulating attracts
> inflammatory response or oxidative mechanism?
Not clinically seen.
> > > > > > May GOD continue to heal your heart by curing your diabetes, dear
> > > > > > neighbor Kumar whom I love unconditionally.
Prayerfully in Christ's amazing love,
kumar
trans-capillary
movements of insulin?
> > > > > > snip> > > The glycosylations are happening everywhere and not just
> > > > > > > > > intravascularly.
> > > > > > > >
> > > > > > > > In case of vasoconstriction or reduced blood flow?
> > > > > > >
> > > > > > > Yes.
> > > > >
> > > > > > Will such glycosylation be reduced extravascularily but increased
> > > > > > intravascularily on vasoconstriction or on decreased blood flow?
> > > > >
> > > > > No.
> > >
> > > > Will glucose or even water be filtered lesser in tatal quantity from
> > > > capalliries, if blood flow is reduced due to vasocontriction?
> > >
> > > Not to any clinically meaningful extent.
> > >
> > > > > > Can above feature is valid for all substances including water which are
> > > > > > filtered from capillaries? I am not considering filtring capacity but
> > > > > > quantity of blood flow?
> > > > >
> > > > > It is relevant to all substances that are able to freely cross through
> > > > > the capillary basement membrane.
> > > >
> > > > Yes but total blood flow can be reduced due to vsoconstriction? Whether
> > > > total filteration can't be also dependant and proportionate to total
> > > > blood flow in any area?
> > >
> > > Transient events are not clinically meaningful in chronic disease such
> > > as type-2 diabetes.
> >
> > Yes, but can't there be persistance of such events due to persisting
> > hyperglycemia?
>
> Hyperglycemia does not cause vasoconstriction.
When increased O2 levels can effect size and numbers of blood vessels
supplying to tissues as local blood flow control, hyperglycemia may
also do that--by reduced blood flow?
> > >> > > > > > > Soluble fiber in the diet comes with its own water so that it is not
> > > > > > > > > hygroscopic.
> > > > > >
> > > > > > Whether Non-soluble fibers are hygroscopic? Can isabgol husk absorb
> > > > > > bile and cause more excretion of bile with stool?
> > > > >
> > > > > Not familiar with isabgol husk.
> > > >
> > > > It is well known. Anyway,
> > > >
> > > > "Isabgol (also Sat-Isabgol or Isapgol), is the Indian term for psyllium
> > > > seed husks (not a trademark). Isabgol is used for constipation and
> > > > irritable bowel syndrome. A few brand names are "B. G. Telephone Brand
> > > > Sat-Isabgol", "Deer Brand Sat Isabgol" and "Dabur Sat Isabgol".
> > > >
> > > > Isabgol is also avaiable internationally in Indian shops and markets,
> > > > but as it is typically sold as isabgol rather than psyllium, many
> > > > people are not aware of that. Being a generic product, it is
> > > > significantly cheaper than similar products sold in pharmacies. Isabgol
> > > > (psyllium) is the active ingredient of the American product Metamucil.
> > > > http://en.wikipedia.org/wiki/Isabgol "
> > >
> > > Am familiar with psyllium. And, yes, it can help to lower cholesterol
> > > by slowing down recovery of bile salts increasing losses in stool.
> >
> > It is indicative that butter fat/pure ghee attract more bile, so while
> > increasing cholestreol it also excrete more bile resulting into
> > cholestreol's beneficial roles as well controlling their levels?
>
> Not clinically seen.
Can't any fiber, which can absorb bile and enable more of its excretion
do that?
> > > > > > > > > > > > Whether bile is hygroscopic? Can bile's abnormal secretion(less) in
> > > > > > > > > > > > intestine make a person constipated, lazy esp. in morning, sleeping
> > > > > > > > > > > > more, waking late etc.? Two aspects may be related, abnormal bile
> > > > > > > > > > > > comong from liver and abnormal secretion from gall bladder(can't say
> > > > > > > > > > > > about abnormal re-absorption)?
> > > > > > > > > > >
> > > > > > > > > > > Clinically not seen.
> > > > > > > > >
> > > > > > > > > > Can abnormal or obstructed flow of bile from liver to intestine make a
> > > > > > > > > > person lazy, more sleepy, waking late etc.?
> > > > > > > > >
> > > > > > > > > Not clinically seen.
> > > > > > >
> > > > > > > > How bile secretion is triggered?
> > > > > > >
> > > > > > > By CCK released by the physical distension of the stomach.
> > > > >
> > > > > > Can ingested fats promote bile secretion [from] in intestine?
> > > > >
> > > > > Bile is not secreted from the intestine but rather comes from the
> > > > > liver.
> > > >
> > > > Sorry, I meant; Can ingested fats promote bile secretion in intestine?
> > >
> > > Bile production and release into the intestine is stimulated by
> > > ingested fats.
>
> > Whether butterfat/pure ghee stimulate comparatively more bile than
> > begetable oils?
>
> Not clinically seen.
How cholestrol stimulate bile secretion in intestines?
> > > > > > > > Can there be different type or
> > > > > > > > quantity of bile secreted in different conditions? > > I feel
> > > > > > > > abnormalities in bile flow are related to many conditions
> > > > > > >
> > > > > > > Not clinically seen.
> > > > > > >
> > > > > > > > > > couldn't yet evalued?
> > > > > > > > >
> > > > > > > > > You would be wise to inform your doctor(s) about your symptoms rather
> > > > > > > > > than attributing them to bile flow.
> > > > > > >
> > > > > > > > I was just evaulating. In ancient systems bile was of great importance,
> > > > > > > > constitutional(personality type), but I think it is not given much
> > > > > > > > importance?
> > > > > > >
> > > > > > > It is not clinically important.
> > > > > >
> > > > > > Whether composition of bile can be changed in different conditions?
> > > > > > Whether bile detoxfy blood?
> > > > >
> > > > > Bile is used to emulsify ingested fats to aid in their digestion.
> > > >
> > > > Yes but if its composition and secreted quantity can be variable due to
> > > > different type of fats/foods?
> > >
> > > Composition, no. Quantity, yes.
> >
> > Can it be secretated in intestine less or more concentrated?
>
> The latter would be the purpose of the gall bladder.
What is the purpose of more or less bile's concentration secretions?
> > > > > > > > > > Which out of bile and gastric acid ir related to either unclear or
> > > > > > > > > > clear motions? Billary diahrria is thought?
> > > > > > > > >
> > > > > > > > > No clinical association.
> > > > > > >
> > > > > > > > Diahrria can occur when medication for resisting bile absorption is
> > > > > > > > given?
> > > > > > >
> > > > > > > Not clinically seen except as an adverse effect for some.
> > > > > > >
> > > > > > > > Anyway, what increase RBC Dist Width(MCV normal) indicates?
> > > > > > >
> > > > > > > It suggests there may have been some blood loss.
> > > > >
> > > > > > How? Is there any relation between increase in RBC dist width,
> > > > > > decreased hematocrit and increased neutrophills?
> > > > >
> > > > > Would suggest that there is infection **and** blood loss.
> > > > >
> > > > > > Whether blood loss is compensated by water to maintain blood volume?
> > > > >
> > > > > It is if blood pressure remains normal even upon standing without
> > > > > abnormally elevated heart rate.
> > > > >
> > > > > > Can you define RBC dist width for me pls?
> > > > >
> > > > > It is the range in RBC widths that would include 90% of all red bloods
> > > > > cells (RBCs).
> > > >
> > > > Whether its higher reading tells abnormal macrocytic size and shape of
> > > > RBCs?
> > >
> > > Higher RDWs can be associated with abnormal forms/shapes.
> >
> > Will such RBCs be macrocytic/megablastic?
>
> They are often known as schizocytes.
A fat/puffy person can look of different shape?
Btw, can you tell differenciating factors between puffy,flaby and obese
person?
> > > > Whether some higher reading of RDW, low Hct and some higher neutrophils
> > > > reading (may be subclinically higher but still within commonly indicated
> > > > normal range)gs can be common in people with persisting hyperglycemia
> > > > and IR?
> > >
> > > Not clinically seen.
>
> > Can low Hct or hypotonicity of blood increase RTWs?
>
> Blood loss with normal production of red cells by the bone marrow to
> bring the Hct back up would typically increase RDW.
Wether RDW and Hct's variations(may be sub-clinical but near to
abnormal) are common in diabetics?
> > Whether swelled cells in vascular wall or circulating attracts
> > inflammatory response or oxidative mechanism?
>
> Not clinically seen.
Whether glysylated hemoglobin and elevated circulating lipid levels can
attract inflammatory responses?
Whether alkalizers can oppose inflammatory responses in view of acidity
is linked to control bacterias as in intestines?
Andrew B. Chung, MD/PhD
Not clinically seen.
Not clinically seen.
> > > >> > > > > > > Soluble fiber in the diet comes with its own water so that it is not
> > > > > > > > > > hygroscopic.
> > > > > > >
> > > > > > > Whether Non-soluble fibers are hygroscopic? Can isabgol husk absorb
> > > > > > > bile and cause more excretion of bile with stool?
> > > > > >
> > > > > > Not familiar with isabgol husk.
> > > > >
> > > > > It is well known. Anyway,
> > > > >
> > > > > "Isabgol (also Sat-Isabgol or Isapgol), is the Indian term for psyllium
> > > > > seed husks (not a trademark). Isabgol is used for constipation and
> > > > > irritable bowel syndrome. A few brand names are "B. G. Telephone Brand
> > > > > Sat-Isabgol", "Deer Brand Sat Isabgol" and "Dabur Sat Isabgol".
> > > > >
> > > > > Isabgol is also avaiable internationally in Indian shops and markets,
> > > > > but as it is typically sold as isabgol rather than psyllium, many
> > > > > people are not aware of that. Being a generic product, it is
> > > > > significantly cheaper than similar products sold in pharmacies. Isabgol
> > > > > (psyllium) is the active ingredient of the American product Metamucil.
> > > > > http://en.wikipedia.org/wiki/Isabgol "
> > > >
> > > > Am familiar with psyllium. And, yes, it can help to lower cholesterol
> > > > by slowing down recovery of bile salts increasing losses in stool.
> > >
> > > It is indicative that butter fat/pure ghee attract more bile, so while
> > > increasing cholestreol it also excrete more bile resulting into
> > > cholestreol's beneficial roles as well controlling their levels?
> >
> > Not clinically seen.
> Can't any fiber, which can absorb bile and enable more of its excretion
> do that?
Butter fat is not fiber.
> > > > > > > > > > > > > Whether bile is hygroscopic? Can bile's abnormal secretion(less) in
> > > > > > > > > > > > > intestine make a person constipated, lazy esp. in morning, sleeping
> > > > > > > > > > > > > more, waking late etc.? Two aspects may be related, abnormal bile
> > > > > > > > > > > > > comong from liver and abnormal secretion from gall bladder(can't say
> > > > > > > > > > > > > about abnormal re-absorption)?
> > > > > > > > > > > >
> > > > > > > > > > > > Clinically not seen.
> > > > > > > > > >
> > > > > > > > > > > Can abnormal or obstructed flow of bile from liver to intestine make a
> > > > > > > > > > > person lazy, more sleepy, waking late etc.?
> > > > > > > > > >
> > > > > > > > > > Not clinically seen.
> > > > > > > >
> > > > > > > > > How bile secretion is triggered?
> > > > > > > >
> > > > > > > > By CCK released by the physical distension of the stomach.
> > > > > >
> > > > > > > Can ingested fats promote bile secretion [from] in intestine?
> > > > > >
> > > > > > Bile is not secreted from the intestine but rather comes from the
> > > > > > liver.
> > > > >
> > > > > Sorry, I meant; Can ingested fats promote bile secretion in intestine?
> > > >
> > > > Bile production and release into the intestine is stimulated by
> > > > ingested fats.
> >
> > > Whether butterfat/pure ghee stimulate comparatively more bile than
> > > begetable oils?
> >
> > Not clinically seen.
>
> How cholestrol stimulate bile secretion in intestines?
It does not directly. Indeed, bile is largely comprised of
cholesterol.
> > > > > > > > > Can there be different type or
> > > > > > > > > quantity of bile secreted in different conditions? > > I feel
> > > > > > > > > abnormalities in bile flow are related to many conditions
> > > > > > > >
> > > > > > > > Not clinically seen.
> > > > > > > >
> > > > > > > > > > > couldn't yet evalued?
> > > > > > > > > >
> > > > > > > > > > You would be wise to inform your doctor(s) about your symptoms rather
> > > > > > > > > > than attributing them to bile flow.
> > > > > > > >
> > > > > > > > > I was just evaulating. In ancient systems bile was of great importance,
> > > > > > > > > constitutional(personality type), but I think it is not given much
> > > > > > > > > importance?
> > > > > > > >
> > > > > > > > It is not clinically important.
> > > > > > >
> > > > > > > Whether composition of bile can be changed in different conditions?
> > > > > > > Whether bile detoxfy blood?
> > > > > >
> > > > > > Bile is used to emulsify ingested fats to aid in their digestion.
> > > > >
> > > > > Yes but if its composition and secreted quantity can be variable due to
> > > > > different type of fats/foods?
> > > >
> > > > Composition, no. Quantity, yes.
> > >
> > > Can it be secretated in intestine less or more concentrated?
> >
> > The latter would be the purpose of the gall bladder.
>
> What is the purpose of more or less bile's concentration secretions?
To aid in the emulsification and digestion of fats.
> > > > > > > > > > > Which out of bile and gastric acid ir related to either unclear or
> > > > > > > > > > > clear motions? Billary diahrria is thought?
> > > > > > > > > >
> > > > > > > > > > No clinical association.
> > > > > > > >
> > > > > > > > > Diahrria can occur when medication for resisting bile absorption is
> > > > > > > > > given?
> > > > > > > >
> > > > > > > > Not clinically seen except as an adverse effect for some.
> > > > > > > >
> > > > > > > > > Anyway, what increase RBC Dist Width(MCV normal) indicates?
> > > > > > > >
> > > > > > > > It suggests there may have been some blood loss.
> > > > > >
> > > > > > > How? Is there any relation between increase in RBC dist width,
> > > > > > > decreased hematocrit and increased neutrophills?
> > > > > >
> > > > > > Would suggest that there is infection **and** blood loss.
> > > > > >
> > > > > > > Whether blood loss is compensated by water to maintain blood volume?
> > > > > >
> > > > > > It is if blood pressure remains normal even upon standing without
> > > > > > abnormally elevated heart rate.
> > > > > >
> > > > > > > Can you define RBC dist width for me pls?
> > > > > >
> > > > > > It is the range in RBC widths that would include 90% of all red bloods
> > > > > > cells (RBCs).
> > > > >
> > > > > Whether its higher reading tells abnormal macrocytic size and shape of
> > > > > RBCs?
> > > >
> > > > Higher RDWs can be associated with abnormal forms/shapes.
> > >
> > > Will such RBCs be macrocytic/megablastic?
> >
> > They are often known as schizocytes.
>
> A fat/puffy person can look of different shape?
Not clinically seen.
> Btw, can you tell differenciating factors between puffy,flaby and obese
> person?
They are heavier than optimal.
> > > > > Whether some higher reading of RDW, low Hct and some higher neutrophils
> > > > > reading (may be subclinically higher but still within commonly indicated
> > > > > normal range)gs can be common in people with persisting hyperglycemia
> > > > > and IR?
> > > >
> > > > Not clinically seen.
> >
> > > Can low Hct or hypotonicity of blood increase RTWs?
> >
> > Blood loss with normal production of red cells by the bone marrow to
> > bring the Hct back up would typically increase RDW.
>
> Wether RDW and Hct's variations(may be sub-clinical but near to
> abnormal) are common in diabetics?
Not clinically seen.
> > > Whether swelled cells in vascular wall or circulating attracts
> > > inflammatory response or oxidative mechanism?
> >
> > Not clinically seen.
>
> Whether glysylated hemoglobin and elevated circulating lipid levels can
> attract inflammatory responses?
Not clinically seen.
> Whether alkalizers can oppose inflammatory responses in view of acidity
> is linked to control bacterias as in intestines?
Not clinically seen.
Kumar
Yes, but I asked about fiber.
> > > > > > > > > > > > > > Whether bile is hygroscopic? Can bile's abnormal secretion(less) in
> > > > > > > > > > > > > > intestine make a person constipated, lazy esp. in morning, sleeping
> > > > > > > > > > > > > > more, waking late etc.? Two aspects may be related, abnormal bile
> > > > > > > > > > > > > > comong from liver and abnormal secretion from gall bladder(can't say
> > > > > > > > > > > > > > about abnormal re-absorption)?
> > > > > > > > > > > > >
> > > > > > > > > > > > > Clinically not seen.
> > > > > > > > > > >
> > > > > > > > > > > > Can abnormal or obstructed flow of bile from liver to intestine make a
> > > > > > > > > > > > person lazy, more sleepy, waking late etc.?
> > > > > > > > > > >
> > > > > > > > > > > Not clinically seen.
> > > > > > > > >
> > > > > > > > > > How bile secretion is triggered?
> > > > > > > > >
> > > > > > > > > By CCK released by the physical distension of the stomach.
> > > > > > >
> > > > > > > > Can ingested fats promote bile secretion [from] in intestine?
> > > > > > >
> > > > > > > Bile is not secreted from the intestine but rather comes from the
> > > > > > > liver.
> > > > > >
> > > > > > Sorry, I meant; Can ingested fats promote bile secretion in intestine?
> > > > >
> > > > > Bile production and release into the intestine is stimulated by
> > > > > ingested fats.
> > >
> > > > Whether butterfat/pure ghee stimulate comparatively more bile than
> > > > begetable oils?
> > >
> > > Not clinically seen.
> >
> > How cholestrol stimulate bile secretion in intestines?
>
> It does not directly. Indeed, bile is largely comprised of
> cholesterol.
http://groups.google.com/group/sci.med.cardiology/browse_frm/thread/f0d692953b7eb961/#
What is the fate of cholesterol in bile?
What causes a person to look either puffy or flaby or obese?
> > > > > > Whether some higher reading of RDW, low Hct and some higher neutrophils
> > > > > > reading (may be subclinically higher but still within commonly indicated
> > > > > > normal range)gs can be common in people with persisting hyperglycemia
> > > > > > and IR?
> > > > >
> > > > > Not clinically seen.
> > >
> > > > Can low Hct or hypotonicity of blood increase RTWs?
> > >
> > > Blood loss with normal production of red cells by the bone marrow to
> > > bring the Hct back up would typically increase RDW.
> >
> > Wether RDW and Hct's variations(may be sub-clinical but near to
> > abnormal) are common in diabetics?
>
> Not clinically seen.
>
> > > > Whether swelled cells in vascular wall or circulating attracts
> > > > inflammatory response or oxidative mechanism?
> > >
> > > Not clinically seen.
> >
> > Whether glysylated hemoglobin and elevated circulating lipid levels can
> > attract inflammatory responses?
>
> Not clinically seen.
>
> > Whether alkalizers can oppose inflammatory responses in view of acidity
> > is linked to control bacterias as in intestines?
>
> Not clinically seen.
Other than its tissue damaging effects, Whether inflammatory responses
can keep infections under control?
Andrew B. Chung, MD/PhD
> > > > > increasing cholestreol it also excrete more bile resulting into
> > > > > cholestreol's beneficial roles as well controlling their levels?
**emphasis** added.
> > > >
> > > > Not clinically seen.
> >
> > > Can't any fiber, which can absorb bile and enable more of its excretion
> > > do that?
> >
> > Butter fat is not fiber.
>
> Yes, but I asked about fiber.
See **emphasis** above.
It is reabsorbed.
Overeating.
> > > > > > > Whether some higher reading of RDW, low Hct and some higher neutrophils
> > > > > > > reading (may be subclinically higher but still within commonly indicated
> > > > > > > normal range)gs can be common in people with persisting hyperglycemia
> > > > > > > and IR?
> > > > > >
> > > > > > Not clinically seen.
> > > >
> > > > > Can low Hct or hypotonicity of blood increase RTWs?
> > > >
> > > > Blood loss with normal production of red cells by the bone marrow to
> > > > bring the Hct back up would typically increase RDW.
> > >
> > > Wether RDW and Hct's variations(may be sub-clinical but near to
> > > abnormal) are common in diabetics?
> >
> > Not clinically seen.
> >
> > > > > Whether swelled cells in vascular wall or circulating attracts
> > > > > inflammatory response or oxidative mechanism?
> > > >
> > > > Not clinically seen.
> > >
> > > Whether glysylated hemoglobin and elevated circulating lipid levels can
> > > attract inflammatory responses?
> >
> > Not clinically seen.
> >
> > > Whether alkalizers can oppose inflammatory responses in view of acidity
> > > is linked to control bacterias as in intestines?
> >
> > Not clinically seen.
>
> Other than its tissue damaging effects, Whether inflammatory responses
> can keep infections under control?
Without a specific immune response, the answer would be "no."
Gege
> Without a specific immune response, the answer would be "no."
No
Kumar
Andrew B. Chung, MD/PhD wrote:
> Kumar wrote:
> > Andrew B. Chung, MD/PhD wrote:
> > > > > > > Am familiar with psyllium. And, yes, it can help to lower cholesterol
> > > > > > > by slowing down recovery of bile salts increasing losses in stool.
> > > > > >
> > > > > > It is indicative that **butter fat/pure ghee** attract more bile, so while
> > > > > > increasing cholestreol it also excrete more bile resulting into
> > > > > > cholestreol's beneficial roles as well controlling their levels?
>
> **emphasis** added.
Sorry, your awnser is not clear to me. I meant buuer fat/pure ghee
attract more bile secretions in intestines not bile absorption?
Possibily, more the bile synthesis in liver and secretion in
intestines, comparatively more can be its excretion esp. when blood
cholesterol levels are higher?
>snip> > > > How cholestrol stimulate bile secretion in intestines?
> > >
> > > It does not directly. Indeed, bile is largely comprised of
> > > cholesterol.
> >
> > http://groups.google.com/group/sci.med.cardiology/browse_frm/thread/f0d692953b7eb961/#
> >
> > What is the fate of cholesterol in bile?
>
> It is reabsorbed.
All or as per blood levels?
snip> > > > A fat/puffy person can look of different shape?
> > >
> > > Not clinically seen.
> > >
> > > > Btw, can you tell differenciating factors between puffy,flaby and obese
> > > > person?
> > >
> > > They are heavier than optimal.
Pls tell differenciating factors in between puffy,flaby and obese
people
> > What causes a person to look either puffy or flaby or obese?
>
> Overeating.
Can one be swelled with water other with fats?
> > > > > > > > Whether some higher reading of RDW, low Hct and some higher neutrophils
> > > > > > > > reading (may be subclinically higher but still within commonly indicated
> > > > > > > > normal range)gs can be common in people with persisting hyperglycemia
> > > > > > > > and IR?
> > > > > > >
> > > > > > > Not clinically seen.
> > > > >
> > > > > > Can low Hct or hypotonicity of blood increase RTWs?
> > > > >
> > > > > Blood loss with normal production of red cells by the bone marrow to
> > > > > bring the Hct back up would typically increase RDW.
> > > >
> > > > Wether RDW and Hct's variations(may be sub-clinical but near to
> > > > abnormal) are common in diabetics?
> > >
> > > Not clinically seen.
> > >
> > > > > > Whether swelled cells in vascular wall or circulating attracts
> > > > > > inflammatory response or oxidative mechanism?
> > > > >
> > > > > Not clinically seen.
> > > >
> > > > Whether glysylated hemoglobin and elevated circulating lipid levels can
> > > > attract inflammatory responses?
> > >
> > > Not clinically seen.
> > >
> > > > Whether alkalizers can oppose inflammatory responses in view of acidity
> > > > is linked to control bacterias as in intestines?
> > >
> > > Not clinically seen.
> >
> > Other than its tissue damaging effects, Whether inflammatory responses
> > can keep infections under control?
>
> Without a specific immune response, the answer would be "no."
Whether inflammatory responces preceed infectious immune response?
Btw whether occasional or excessive eaters in other species as
honeu-bees are diabetic?
Andrew B. Chung, MD/PhD
> Andrew B. Chung, MD/PhD wrote:
> > Kumar wrote:
> > > Andrew B. Chung, MD/PhD wrote:
> > > > > > > > Am familiar with psyllium. And, yes, it can help to lower cholesterol
> > > > > > > > by slowing down recovery of bile salts increasing losses in stool.
> > > > > > >
> > > > > > > It is indicative that **butter fat/pure ghee** attract more bile, so while
> > > > > > > increasing cholestreol it also excrete more bile resulting into
> > > > > > > cholestreol's beneficial roles as well controlling their levels?
> >
> > **emphasis** added.
>
> Sorry, your awnser is not clear to me.
It served to help you understand why I wrote to comment how you had
confused butter fat with dietary fiber. The former does not result in
the excretion of more cholesterol for any cholesterol-lower benefit as
compared to the latter.
Kumar
Andrew B. Chung, MD/PhD wrote:
> Kumar wrote:
> > Andrew B. Chung, MD/PhD wrote:
> > > Kumar wrote:
> > > > Andrew B. Chung, MD/PhD wrote:
> > > > > > > > > Am familiar with psyllium. And, yes, it can help to lower cholesterol
> > > > > > > > > by slowing down recovery of bile salts increasing losses in stool.
> > > > > > > >
> > > > > > > > It is indicative that **butter fat/pure ghee** attract more bile, so while
> > > > > > > > increasing cholestreol it also excrete more bile resulting into
> > > > > > > > cholestreol's beneficial roles as well controlling their levels?
> > >
> > > **emphasis** added.
> >
> > Sorry, your awnser is not clear to me.
>
> It served to help you understand why I wrote to comment how you had
> confused butter fat with dietary fiber. The former does not result in
> the excretion of more cholesterol for any cholesterol-lower benefit as
> compared to the latter.
I have not confused about butter with fibers. These are two different
things. I thought, Butter may promote more bile secretion in intestines
so more of its excretion whereas fiber can absorb more bile resulting
more of its excretion in stool.
Andrew B. Chung, MD/PhD
> Andrew B. Chung, MD/PhD wrote:
> > Kumar wrote:
> > > Andrew B. Chung, MD/PhD wrote:
> > > > Kumar wrote:
> > > > > Andrew B. Chung, MD/PhD wrote:
> > > > > > > > > > Am familiar with psyllium. And, yes, it can help to lower cholesterol
> > > > > > > > > > by slowing down recovery of bile salts increasing losses in stool.
> > > > > > > > >
> > > > > > > > > It is indicative that **butter fat/pure ghee** attract more bile, so while
> > > > > > > > > increasing cholestreol it also excrete more bile resulting into
> > > > > > > > > cholestreol's beneficial roles as well controlling their levels?
> > > >
> > > > **emphasis** added.
> > >
> > > Sorry, your awnser is not clear to me.
> >
> > It served to help you understand why I wrote to comment how you had
> > confused butter fat with dietary fiber. The former does not result in
> > compared to the latter.
>
> I have not confused about butter with fibers. These are two different
> things. I thought, Butter may promote more bile secretion in intestines
> so more of its excretion whereas fiber can absorb more bile resulting
> more of its excretion in stool.
Bile secretion occurs **before** digestion and absorption of fats such
as butter.
Kumar
Andrew B. Chung, MD/PhD wrote:
> Kumar wrote:
> > Andrew B. Chung, MD/PhD wrote:
> > > Kumar wrote:
> > > > Andrew B. Chung, MD/PhD wrote:
> > > > > Kumar wrote:
> > > > > > Andrew B. Chung, MD/PhD wrote:
> > > > > > > > > > > Am familiar with psyllium. And, yes, it can help to lower cholesterol
> > > > > > > > > > > by slowing down recovery of bile salts increasing losses in stool.
> > > > > > > > > >
> > > > > > > > > > It is indicative that **butter fat/pure ghee** attract more bile, so while
> > > > > > > > > > increasing cholestreol it also excrete more bile resulting into
> > > > > > > > > > cholestreol's beneficial roles as well controlling their levels?
> > > > >
> > > > > **emphasis** added.
> > > >
> > > > Sorry, your awnser is not clear to me.
> > >
> > > It served to help you understand why I wrote to comment how you had
> > > confused butter fat with dietary fiber. The former does not result in
> > > the excretion of more cholesterol for any cholesterol-lowering benefit as
> > > compared to the latter.
> >
> > I have not confused about butter with fibers. These are two different
> > things. I thought, Butter may promote more bile secretion in intestines
> > so more of its excretion whereas fiber can absorb more bile resulting
> > more of its excretion in stool.
>
> Bile secretion occurs **before** digestion and absorption of fats such
> as butter.
cholesterol intake or on their intake?
Do our body benefits from natural cycle of cholesterol or any other
food form intake to excretion or by less intake oand lesser
excretion/reabsorption?
To deviate from naturally followed practices may be sometimes improper
as we are not understanding everything absolutely, clearly and
completely. As noone can thing any such deviations--right or wrong?
Andrew B. Chung, MD/PhD
The trigger is simply food reaching the gut.
> Do our body benefits from natural cycle of cholesterol or any other
> food form intake to excretion or by less intake oand lesser
> excretion/reabsorption?
Our body benefits from an optimal intake amount which is by GOD's
design and which HE calls an omer:
http://HeartMDPhD.com/HolySpirit/overweight.asp
> To deviate from naturally followed practices may be sometimes improper
> as we are not understanding everything absolutely, clearly and
> completely. As noone can thing any such deviations--right or wrong?
It remains wise to look to LORD GOD Almighty, Creator of heaven and
earth, for knowledge and wisdom.
Kumar
Whether type of food trigger different type of secretions?
> > Do our body benefits from natural cycle of cholesterol or any other
> > food form intake to excretion or by less intake oand lesser
> > excretion/reabsorption?
>
> Our body benefits from an optimal intake amount which is by GOD's
> design and which HE calls an omer:
>
> http://HeartMDPhD.com/HolySpirit/overweight.asp
Yes, but we may be now attuned/brainased to eat less or no
cholesterols?
> > To deviate from naturally followed practices may be sometimes improper
> > as we are not understanding everything absolutely, clearly and
> > completely. As no one can think any such deviations--right or wrong?
>
> It remains wise to look to LORD GOD Almighty, Creator of heaven and
> earth, for knowledge and wisdom.
The other thought;
Can excess glucose in blood cause any blood substances sticky,
aggregate, or club, resuting into some restriction in transcapillary
movement of insulin etc.?
Can hyprglycemia effect changes in endothelliul cells junctution in
capillaries which may restrict normal movement of insulin into tissues?
Andrew B. Chung, MD/PhD
Not clinically seen.
> > > Do our body benefits from natural cycle of cholesterol or any other
> > > food form intake to excretion or by less intake oand lesser
> > > excretion/reabsorption?
> >
> > Our body benefits from an optimal intake amount which is by GOD's
> > design and which HE calls an omer:
> >
> > http://HeartMDPhD.com/HolySpirit/overweight.asp
>
> Yes, but we may be now attuned/brainased to eat less or no
> cholesterols?
Cholesterol is not an essential nutrient.
> > > To deviate from naturally followed practices may be sometimes improper
> > > as we are not understanding everything absolutely, clearly and
> > > completely. As no one can think any such deviations--right or wrong?
> >
> > It remains wise to look to LORD GOD Almighty, Creator of heaven and
> > earth, for knowledge and wisdom.
>
> The other thought;
>
> Can excess glucose in blood cause any blood substances sticky,
> aggregate, or club, resuting into some restriction in transcapillary
> movement of insulin etc.?
Not clinically seen.
> Can hyprglycemia effect changes in endothelliul cells junctution in
> capillaries which may restrict normal movement of insulin into tissues?
Not clinically seen.
Kumar
> > > > Do our body benefits from natural cycle of cholesterol or any other
> > > > food form intake to excretion or by less intake oand lesser
> > > > excretion/reabsorption?
> > >
> > > Our body benefits from an optimal intake amount which is by GOD's
> > > design and which HE calls an omer:
> > >
> > > http://HeartMDPhD.com/HolySpirit/overweight.asp
> >
> > Yes, but we may be now attuned/brainwashed to eat less or no
> > cholesterols?
>
> Cholesterol is not an essential nutrient.
Is it not essential to body?
"Function
Cholesterol is required to build and maintain cell membranes; it makes
the membrane's fluidity - degree of viscosity - stable over wider
temperature intervals (the hydroxyl group on cholesterol interacts with
the phosphate head of the membrane, and the bulky steroid and the
hydrocarbon chain is embedded in the membrane). Some research indicates
that cholesterol may act as an antioxidant.[1] Cholesterol also aids in
the manufacture of bile (which helps digest fats), and is also
important for the metabolism of fat-soluble vitamins, including
vitamins A, D, E and K. It is the major precursor for the synthesis of
vitamin D and of the various steroid hormones (which include cortisol
and aldosterone in the adrenal glands, and the sex hormones
progesterone, the various estrogens, testosterone, and derivatives ).
Recently, cholesterol has also been implicated in cell signalling
processes, where it has been suggested that it forms lipid rafts in the
plasma membrane. It also reduces the permeability of the plasma
membrane to hydrogen ions (protons) and sodium ions.[2]
http://en.wikipedia.org/wiki/Cholesterol "
Kumar
> > > > Do our body benefits from natural cycle of cholesterol or any other
> > > > food form intake to excretion or by less intake oand lesser
> > > > excretion/reabsorption?
> > >
> > > Our body benefits from an optimal intake amount which is by GOD's
> > > design and which HE calls an omer:
> > >
> > > http://HeartMDPhD.com/HolySpirit/overweight.asp
> >
> > Yes, but we may be now attuned/brainwashed to eat less or no
> > cholesterols?
>
> Cholesterol is not an essential nutrient.
Is it not essential to body?
"Function
Cholesterol is required to build and maintain cell membranes; it makes
the membrane's fluidity - degree of viscosity - stable over wider
temperature intervals (the hydroxyl group on cholesterol interacts with
the phosphate head of the membrane, and the bulky steroid and the
hydrocarbon chain is embedded in the membrane). Some research indicates
that cholesterol may act as an antioxidant.[1] Cholesterol also aids in
the manufacture of bile (which helps digest fats), and is also
important for the metabolism of fat-soluble vitamins, including
vitamins A, D, E and K. It is the major precursor for the synthesis of
vitamin D and of the various steroid hormones (which include cortisol
and aldosterone in the adrenal glands, and the sex hormones
progesterone, the various estrogens, testosterone, and derivatives ).
Recently, cholesterol has also been implicated in cell signalling
processes, where it has been suggested that it forms lipid rafts in the
plasma membrane. It also reduces the permeability of the plasma
membrane to hydrogen ions (protons) and sodium ions.[2]
http://en.wikipedia.org/wiki/Cholesterol "
> > > > To deviate from naturally followed practices may be sometimes improper
> > > > as we are not understanding everything absolutely, clearly and
> > > > completely. As no one can think any such deviations--right or wrong?
> > >
> > > It remains wise to look to LORD GOD Almighty, Creator of heaven and
> > > earth, for knowledge and wisdom.
> >
> > The other thought;
> >
> > Can excess glucose in blood cause any blood substances sticky,
> > aggregate, or club, resuting into some restriction in transcapillary
> > movement of insulin etc.?
>
> Not clinically seen.
"Glycosylation Purpose
The polysaccharide chains attached to the target proteins serve various
functions. For instance, some proteins do not fold correctly unless
they are glycosylated first. Also, polysaccharides linked at the amide
nitrogen of asparagine in the protein confer stability on some secreted
glycoproteins. Experiments have shown that glycosylation in this case
is not a strict requirement for proper folding, but the unglycosylated
protein degrades quickly. Glycosylation may play a role in *cell-cell
adhesion* (a mechanism employed by cells of the immune system), as
well.
http://en.wikipedia.org/wiki/Glycosylation "
Whether above quote indicate something as I thought?
> > Can hyprglycemia effect changes in endothelliul cells junctution in
> > capillaries which may restrict normal movement of insulin into tissues?
>
> Not clinically seen.
What can effect Capillaries endothelliul cells junctions to make them
ess or more leaky?
Andrew B. Chung, MD/PhD
No. Simply a conditioned response.
> > > > > Do our body benefits from natural cycle of cholesterol or any other
> > > > > food form intake to excretion or by less intake oand lesser
> > > > > excretion/reabsorption?
> > > >
> > > > Our body benefits from an optimal intake amount which is by GOD's
> > > > design and which HE calls an omer:
> > > >
> > > > http://HeartMDPhD.com/HolySpirit/overweight.asp
> > >
> > > Yes, but we may be now attuned/brainwashed to eat less or no
> > > cholesterols?
> >
> > Cholesterol is not an essential nutrient.
>
> Is it not essential to body?
That which is readily made in the body far more in excess than is
needed for vital functions is not an essential nutrient.
> "Function
> Cholesterol is required to build and maintain cell membranes; it makes
> the membrane's fluidity - degree of viscosity - stable over wider
> temperature intervals (the hydroxyl group on cholesterol interacts with
> the phosphate head of the membrane, and the bulky steroid and the
> hydrocarbon chain is embedded in the membrane). Some research indicates
> that cholesterol may act as an antioxidant.[1] Cholesterol also aids in
> the manufacture of bile (which helps digest fats), and is also
> important for the metabolism of fat-soluble vitamins, including
> vitamins A, D, E and K. It is the major precursor for the synthesis of
> vitamin D and of the various steroid hormones (which include cortisol
> and aldosterone in the adrenal glands, and the sex hormones
> progesterone, the various estrogens, testosterone, and derivatives ).
>
> Recently, cholesterol has also been implicated in cell signalling
> processes, where it has been suggested that it forms lipid rafts in the
> plasma membrane. It also reduces the permeability of the plasma
> membrane to hydrogen ions (protons) and sodium ions.[2]
> http://en.wikipedia.org/wiki/Cholesterol "
See above.
P.S.
Simply here on this blessed LORD's day doing the work that GOD has
prepared in advance for me to do.
"These who do the will of MY Father are MY family." -- LORD Jesus
Christ
Amen !
Laus Deo ! !
Marana tha ! ! !
Kumar
>
> No. Simply a conditioned response.
Fats triggering bile secretion in intestines?
Can prolonged reduced fats intake cause some impairement in bile
synthesis, travel from liver and secretion from GB?
> > > > > > Do our body benefits from natural cycle of cholesterol or any other
> > > > > > food form intake to excretion or by less intake oand lesser
> > > > > > excretion/reabsorption?
> > > > >
> > > > > Our body benefits from an optimal intake amount which is by GOD's
> > > > > design and which HE calls an omer:
> > > > >
> > > > > http://HeartMDPhD.com/HolySpirit/overweight.asp
> > > >
> > > > Yes, but we may be now attuned/brainwashed to eat less or no
> > > > cholesterols?
> > >
> > > Cholesterol is not an essential nutrient.
> >
> > Is it not essential to body?
>
> That which is readily made in the body far more in excess than is
> needed for vital functions is not an essential nutrient.
Can we maintain normally without intake of Cholesterols with food?
> > "Function
> > Cholesterol is required to build and maintain cell membranes; it makes
> > the membrane's fluidity - degree of viscosity - stable over wider
> > temperature intervals (the hydroxyl group on cholesterol interacts with
> > the phosphate head of the membrane, and the bulky steroid and the
> > hydrocarbon chain is embedded in the membrane). Some research indicates
> > that cholesterol may act as an antioxidant.[1] Cholesterol also aids in
> > the manufacture of bile (which helps digest fats), and is also
> > important for the metabolism of fat-soluble vitamins, including
> > vitamins A, D, E and K. It is the major precursor for the synthesis of
> > vitamin D and of the various steroid hormones (which include cortisol
> > and aldosterone in the adrenal glands, and the sex hormones
> > progesterone, the various estrogens, testosterone, and derivatives ).
> >
> > Recently, cholesterol has also been implicated in cell signalling
> > processes, where it has been suggested that it forms lipid rafts in the
> > plasma membrane. It also reduces the permeability of the plasma
> > membrane to hydrogen ions (protons) and sodium ions.[2]
> > http://en.wikipedia.org/wiki/Cholesterol "
>
> See above.
> > > > Can excess glucose in blood cause any blood substances sticky,
> > > > aggregate, or club, resuting into some restriction in transcapillary
> > > > movement of insulin etc.?
> > >
> > > Not clinically seen.
> > >
> > > > Can hyprglycemia effect changes in endothelliul cells junctution in
> > > > capillaries which may restrict normal movement of insulin into tissues?
> > >
> > > Not clinically seen.
How persisted hyperglycemia causes diabetic nephropathy?
Whether DN is just either leaky kidneys or their decreased
performance--filteration and reabsorption?
Andrew B. Chung, MD/PhD
Occurs with the stretching of the stomach.
> Can prolonged reduced fats intake cause some impairement in bile
> synthesis, travel from liver and secretion from GB?
Not clinically seen.
> > > > > > > Do our body benefits from natural cycle of cholesterol or any other
> > > > > > > food form intake to excretion or by less intake oand lesser
> > > > > > > excretion/reabsorption?
> > > > > >
> > > > > > Our body benefits from an optimal intake amount which is by GOD's
> > > > > > design and which HE calls an omer:
> > > > > >
> > > > > > http://HeartMDPhD.com/HolySpirit/overweight.asp
> > > > >
> > > > > Yes, but we may be now attuned/brainwashed to eat less or no
> > > > > cholesterols?
> > > >
> > > > Cholesterol is not an essential nutrient.
> > >
> > > Is it not essential to body?
> >
> > That which is readily made in the body far more in excess than is
> > needed for vital functions is not an essential nutrient.
>
> Can we maintain normally without intake of Cholesterols with food?
Yes.
Persistent hyperglycemia is deleterious.
> Whether DN is just either leaky kidneys or their decreased
> performance--filteration and reabsorption?
Dying kidneys.
May GOD continue to heal our hearts with HIS living water curing our
diabetes, depression, anxiety, and panic so that we can love our
neighbors a little more and LORD Jesus Christ a lot more, dear neighbor
Kumar
of foods including non-fat foods?
> > Can prolonged reduced fats intake cause some impairement in bile
> > synthesis, travel from liver and secretion from GB?
>
> Not clinically seen.
> > > > > > > > Do our body benefits from natural cycle of cholesterol or any other
> > > > > > > > food form intake to excretion or by less intake oand lesser
> > > > > > > > excretion/reabsorption?
> > > > > > >
> > > > > > > Our body benefits from an optimal intake amount which is by GOD's
> > > > > > > design and which HE calls an omer:
> > > > > > >
> > > > > > > http://HeartMDPhD.com/HolySpirit/overweight.asp
> > > > > >
> > > > > > Yes, but we may be now attuned/brainwashed to eat less or no
> > > > > > cholesterols?
> > > > >
> > > > > Cholesterol is not an essential nutrient.
> > > >
> > > > Is it not essential to body?
> > >
> > > That which is readily made in the body far more in excess than is
> > > needed for vital functions is not an essential nutrient.
> >
> > Can we maintain normally without intake of Cholesterols with food?
>
> Yes.
Yes but how it specifically effect kidneys?
> > Whether DN is just either leaky kidneys or their decreased
> > performance--filteration and reabsorption?
>
> Dying kidneys.
Pls tell me types of kindney impairements?
How one can get protienuria still creatnine and BUN level normal?
Stephen Knight
<lo...@thetruth.com> wrote:
>May GOD continue to heal our hearts with HIS living water curing our
>diabetes, depression, anxiety, and panic so that we can love our
>neighbors a little more and LORD Jesus Christ a lot more
That is so sweet and christian. The feeling of the LORD is
overwhelming! I'm ready to join Fred Phelps congregation, and hold a
'Jesus Hates Faggots' banner at a funeral for a gay AIDS person.
I'm floating! I now feel the purest, wonderful LOVE of JESUS.
No more 666 on MY forehead.
Now... if I can find my crayons, I can be a marcher, no.. a soldier
for the LORD.
HOWLAYLOOYA!
MA RANTA HOY! YA! YA! GIDDIUP!
Warlord Steve
BAAWA
Andrew B. Chung, MD/PhD
It is the same.
> > > Can prolonged reduced fats intake cause some impairement in bile
> > > synthesis, travel from liver and secretion from GB?
> >
> > Not clinically seen.
>
> Do you mean fats have no particular role in promoting bile secretions?
No.
By causing glomerulosclerosis.
> > > Whether DN is just either leaky kidneys or their decreased
> > > performance--filteration and reabsorption?
> >
> > Dying kidneys.
>
> Pls tell me types of kindney impairements?
Glomerulosclerosis.
> How one can get protienuria still creatnine and BUN level normal?
Early glomerular basement membrane problems are isolated to the
glomerulus and not the rest of the nephron.
kumar
> > How one can get protienuria still creatnine and BUN level normal?
>
> Early glomerular basement membrane problems are isolated to the
> glomerulus and not the rest of the nephron.
intake or improper metabolism or vasodialation/relaxed vessels or
increased p[ressure or otherwise?
Andrew B. Chung, MD/PhD
Persistently high glucose concentrations is toxic for kidney cells.
> > > How one can get protienuria still creatnine and BUN level normal?
> >
> > Early glomerular basement membrane problems are isolated to the
> > glomerulus and not the rest of the nephron.
>
> Can proteinuria be due to any non damaging reason, say high protien
> intake or improper metabolism or vasodialation/relaxed vessels or
> increased p[ressure or otherwise?
Not clinically seen.
Father Haskell
> Kumar wrote:
> > Do you mean fats have no particular role in promoting bile secretions?
>
> No.
Of course not. That's what religion is for.
kumar
Why then persistently high glucose concentrations can't be toxic to
other target cells?
Andrew B. Chung, MD/PhD
Persistently high glucose concentrations are toxic to target cells as a
rule simply by definition.
May GOD continue to heal our hearts with HIS living water curing our
diabetes, depression, anxiety or panic so that we can love our
Kumar
Andrew B. Chung, MD/PhD wrote:
> kumar wrote:
> > > > > > > > > > > > Can hyprglycemia effect changes in endothelliul cells junctution in
> > > > > > > > > > > > capillaries which may restrict normal movement of insulin into tissues?
> > > > > > > > > > >
> > > > > > > > > > > Not clinically seen.
> > > > > > > >
> > > > > > > > How persisted hyperglycemia causes diabetic nephropathy?
> > > > > > >
> > > > > > > Persistent hyperglycemia is deleterious.
> > > > > >
> > > > > > Yes but how it specifically effect kidneys?
> > > > >
> > > > > By causing glomerulosclerosis.
> > > > >
> > > > > > > > Whether DN is just either leaky kidneys or their decreased
> > > > > > > > performance--filteration and reabsorption?
> > > > > > >
> > > > > > > Dying kidneys.
> > > > > >
> > > > > > Pls tell me types of kindney impairements?
> > > > >
> > > > > Glomerulosclerosis.
> > >
> > > > How high glucose levels causes it?
> > >
> > > Persistently high glucose concentrations is toxic for kidney cells.
> >
> > Why then persistently high glucose concentrations can't be toxic to
> > other target cells?
>
> Persistently high glucose concentrations are toxic to target cells as a
> rule simply by definition.
concentrations to target cells and succeed in it?
Andrew B. Chung, MD/PhD
> Andrew, in the Holy Spirit, boldly wrote:
> > Convicted neighbor kumar wrote:
http://HeartMDPhD.com/Convicts
> > > > > > > > > > > > > Can hyprglycemia effect changes in endothelliul cells junctution in
> > > > > > > > > > > > > capillaries which may restrict normal movement of insulin into tissues?
> > > > > > > > > > > >
> > > > > > > > > > > > Not clinically seen.
> > > > > > > > >
> > > > > > > > > How persisted hyperglycemia causes diabetic nephropathy?
> > > > > > > >
> > > > > > > > Persistent hyperglycemia is deleterious.
> > > > > > >
> > > > > > > Yes but how it specifically effect kidneys?
> > > > > >
> > > > > > By causing glomerulosclerosis.
> > > > > >
> > > > > > > > > Whether DN is just either leaky kidneys or their decreased
> > > > > > > > > performance--filteration and reabsorption?
> > > > > > > >
> > > > > > > > Dying kidneys.
> > > > > > >
> > > > > > > Pls tell me types of kindney impairements?
> > > > > >
> > > > > > Glomerulosclerosis.
> > > >
> > > > > How high glucose levels causes it?
> > > >
> > > > Persistently high glucose concentrations is toxic for kidney cells.
> > >
> > > Why then persistently high glucose concentrations can't be toxic to
> > > other target cells?
> >
> > Persistently high glucose concentrations are toxic to target cells as a
> > rule simply by definition.
> Whether body systems try to avoid such high/toxic glucose
> concentrations to target cells and succeed in it?
Until there is the continued choice to overeat leading to the formation
of visceral adipose tissue (VAT) which is the source of inflammatory
cytokines that fuel the cascading systemic inflammation that leads to
metabolic syndrome (MetS) that is the prerequisite for the development
of type-2 diabetes:
http://HeartMDPhD.com/HolySpirit/overweight.asp
Here is the way to reverse the process:
http://HeartMDPhD.com/wtloss.asp
Which now includes a million dollar guarantee:
http://MabletonGA.OurLittle.net/Guarantee
The existence of VAT concurrently with the existence of subcutaneous
adipose tissue (SAT) can not be explained by Darwin's defunct theory of
evolution but only by GOD, Creator of heaven and earth.
May GOD continue to heal our hearts with HIS living water curing our
diabetes, depression, anxiety or panic so that we can love our
neighbors a little more and LORD Jesus Christ a lot more, dear neighbor
Kumar whom I love unconditionally.
Prayerfully in Christ's amazing love,
Andrew <><
--
Andrew B. Chung
Cardiologist, Atlanta, Georgia, USA
http://HeartMDPhD.com/HolySpirit
As for knowing who are the very elect, these you will know by the
unconditional love they have for everyone including their enemies
(Matthew 5:44-45, 1 Corinthians 13:3, James 2:14-17).
http://HeartMDPhD.com/Love
Happy Birthday Jesus ! ! ! ! ! ! ! !
(((( hugs )))))
Kumar
Andrew B. Chung, MD/PhD wrote:
Whether such systemic inflammation is meant to trigger IR to avoid
high/toxic glucose
concentrations to target cells?
Andrew B. Chung, MD/PhD
IR ultimately causes the high levels of blood glucose that damages
target cells.
Kumar
How high levels of blood glucose damages target cells extravascularily?
Whether damages due to diabetes is related to damages due to
hyperglycemia within blood or/and due to toxicity of glucose to target
extravasular cells?
Andrew B. Chung, MD/PhD
By the post-translational modification (glycosylation) of structural
proteins.
> Whether damages due to diabetes is related to damages due to
> hyperglycemia within blood or/and due to toxicity of glucose to target
> extravasular cells?
Target cells include leukocytes which are intravascular and endothelial
cells which are vascular.
Kumar
Yes but what about damages due to hyperglycemia to target cells which
are extravascular?
Andrew B. Chung, MD/PhD
Also significant.
> > > > > > http://HeartMDPhD.com/HolySpirit/overweight.asp
> > > > > >
> > > > > > Here is the way to reverse the process:
> > > > > >
> > > > > > http://HeartMDPhD.com/wtloss.asp
> > > > > >
> > > > > > Which now includes a million dollar guarantee:
> > > > > >
> > > > > > http://MabletonGA.OurLittle.net/Guarantee
> > > > > >
> > > > > > The existence of VAT concurrently with the existence of subcutaneous
> > > > > > adipose tissue (SAT) can not be explained by Darwin's defunct theory of
> > > > > > evolution but only by GOD, Creator of heaven and earth.
> > > > > >
> > > > > > May GOD continue to heal our hearts with HIS living water curing our
> > > > > > diabetes, depression, anxiety or panic so that we can love our
> > > > > > neighbors a little more and LORD Jesus Christ a lot more, dear neighbor
> > > > > > Kumar whom I love unconditionally.
Prayerfully in Christ's amazing love,
Andrew <><
--
Andrew B. Chung
Cardiologist, Atlanta, Georgia, USA
http://HeartMDPhD.com/HolySpirit
As for knowing who are the very elect, these you will know by the
unconditional love they have for everyone including their enemies
(Matthew 5:44-45, 1 Corinthians 13:3, James 2:14-17).
http://HeartMDPhD.com/Love
Meanwhile, HIS brethren have been blessed:
http://MabletonGA.OurLittle.net/DreadNought
... and continue to be blessed:
http://MabletonGA.OurLittle.net/Guarantee
(note: Only those who are blessed by GOD will have access to these and
other related OurLittle.net articles per a secure IP database. All
others will have to make charitable $30.00 donations to
TheWellnessFoundation.com first to gain access.)
Kumar
Andrew B. Chung, MD/PhD
It can't handle it.
http://MabletonGA.OurLittle.net/DreadNought
http://MabletonGA.OurLittle.net/Guarantee
(note: Only those who are blessed by LORD GOD Almighty, Creator of
heaven and earth, will have access to these and other related
OurLittle.net articles per a secure IP database maintained by
TheWellnessFoundation.com)
Art Deco
>It can't handle it.
Can usenet handle your spamming?
Kumar
Why can't it be by decreased blood flow to target cell's areas?
Andrew B. Chung, MD/PhD
Target cells need the blood just as we need the blood of LORD Jesus
Christ for eternal life, which is granted when HIS blood washes our
robes white so that we would be presentable before GOD, Creator of
Kumar
How decrease in size or number of blood vessels occur as a long term
local blood flow regulation due to O2 concentration is excessive
opposite on low concentration(as on high altitude)? Does it not variate
blood flow?
Whether blood vessels are constricted or dilated on hypertension?
Andrew B. Chung, MD/PhD
It is occurring because of the systemic vascular inflammation fueled by
the inflammatory cytokines coming from the visceral adipose tissue
(VAT).
Kumar
Will it not effect quantity of glucose delivery to extravascular target
tissues?
Andrew B. Chung, MD/PhD
The glucotoxic effect of hyperglycemia is concentration and not
flow-dependent.
In the Holy Spirit,
Andrew <><
--
Andrew B. Chung, MD/PhD
http://EmoryCardiology.com
Kumar
Decreased blood flw can also mean decreased concenrated glucose
delivery?
> In the Holy Spirit,
Pls define Holy sprit and soul. Whether all living beings species
posess soul or sprit?
Andrew B. Chung, MD/PhD
> Andrew, in the Holy Spirit, boldly wrote:
> > Convicted neighbor "Kumar" wrote:
> > > > > Will it not effect quantity of glucose delivery to extravascular target
> > > > > tissues?
> > >
> > > > The glucotoxic effect of hyperglycemia is concentration and not
> > > > flow-dependent.
> >
> > > Decreased blood flw can also mean decreased
> > > concenrated glucose delivery?
> >
>
> Not from obstruction but just reduced.
There is no reduction in blood flow if there is no obstruction.
> "Ischemia: Reduced blood flow to an area of the body due to an
> obstructed vessel."
>
> Btw, can excessive phelgum/mucus discharge be due to relaxed muscular
> tone of respiratory tract?
Not clinically seen.
Would suggest you inform your doctor(s) about your having excessive
phlegm.
Andrew <><
--
Andrew B. Chung, MD/PhD
http://EmoryCardiology.com
> > > > In the Holy Spirit,
> >
> > > Pls define Holy sprit and soul.
> >
> > The former is the Spirit of GOD and the latter is the spirit of others
> > who have received GOD's generous gift of free will **and** remain
> > attached to this world in physical form.
> >
> > > Whether all living beings species
> > > posess soul or sprit?
> >
> > That which is able to freely choose whether to obey GOD's commands have
> > soul.
Art Deco
>Not clinically seen.
Neither are you, Chung, because you don't have a medical practice.
Kumar
Andrew B. Chung, MD/PhD wrote:
> Convicted neighbor Kumar wrote:
> > Andrew, in the Holy Spirit, boldly wrote:
> > > Convicted neighbor "Kumar" wrote:
> > >snip> >
> > > > > > Will it not effect quantity of glucose delivery to extravascular target
> > > > > > tissues?
> > > >
> > > > > The glucotoxic effect of hyperglycemia is concentration and not
> > > > > flow-dependent.
> > >
> > > > Decreased blood flw can also mean decreased
> > > > concenrated glucose delivery?
> > >
> > > Decrease blood flow means ischemia.
> >
> > Not from obstruction but just reduced.
>
> There is no reduction in blood flow if there is no obstruction.
Local blood flow regulation can be without atherosclerosis?
> > "Ischemia: Reduced blood flow to an area of the body due to an
> > obstructed vessel."
> >
> > Btw, can excessive phelgum/mucus discharge be due to relaxed muscular
> > tone of respiratory tract?
>
> Not clinically seen.
How/why phelgum is more or less salty & thin or thick?
> Would suggest you inform your doctor(s) about your having excessive
> phlegm.
It is temporary. However how it is linked to diabitics?
Andrew B. Chung, MD/PhD
> Andrew, in the Holy Spirit, boldly wrote:
> > Convicted neighbor Kumar wrote:
> > > Andrew, in the Holy Spirit, boldly wrote:
> > > > Convicted neighbor "Kumar" wrote:
> > > >snip> >
> > > > > > > Will it not effect quantity of glucose delivery to extravascular target
> > > > > > > tissues?
> > > > >
> > > > > > The glucotoxic effect of hyperglycemia is concentration and not
> > > > > > flow-dependent.
> > > >
> > > > > Decreased blood flw can also mean decreased
> > > > > concenrated glucose delivery?
> > > >
> > > > Decrease blood flow means ischemia.
> > >
> > > Not from obstruction but just reduced.
> >
> > There is no reduction in blood flow if there is no obstruction.
>
> Local blood flow regulation can be without atherosclerosis?
Local blood flow regulation is without ischemia.
> > > "Ischemia: Reduced blood flow to an area of the body due to an
> > > obstructed vessel."
> > >
> > > Btw, can excessive phelgum/mucus discharge be due to relaxed muscular
> > > tone of respiratory tract?
> >
> > Not clinically seen.
>
> How/why phelgum is more or less salty & thin or thick?
Evaporation.
> > Would suggest you inform your doctor(s) about your having excessive
> > phlegm.
>
> It is temporary. However how it is linked to diabitics?
By the predisposition toward infection because of weakened immune
system.
Kumar
Andrew B. Chung, MD/PhD wrote:
> Convicted neighbor Kumar wrote:
> > Andrew, in the Holy Spirit, boldly wrote:
> > > Convicted neighbor Kumar wrote:
> > > > Andrew, in the Holy Spirit, boldly wrote:
> > > > > Convicted neighbor "Kumar" wrote:
> > > > >snip> >
> > > > > > > > Will it not effect quantity of glucose delivery to extravascular target
> > > > > > > > tissues?
> > > > > >
> > > > > > > The glucotoxic effect of hyperglycemia is concentration and not
> > > > > > > flow-dependent.
> > > > >
> > > > > > Decreased blood flw can also mean decreased
> > > > > > concenrated glucose delivery?
> > > > >
> > > > > Decrease blood flow means ischemia.
> > > >
> > > > Not from obstruction but just reduced.
> > >
> > > There is no reduction in blood flow if there is no obstruction.
> >
> > Local blood flow regulation can be without atherosclerosis?
>
> Local blood flow regulation is without ischemia.
> > > > "Ischemia: Reduced blood flow to an area of the body due to an
> > > > obstructed vessel."
> > > >
> > > > Btw, can excessive phelgum/mucus discharge be due to relaxed muscular
> > > > tone of respiratory tract?
> > >
> > > Not clinically seen.
> >
> > How/why phelgum is more or less salty & thin or thick?
>
mechanism of body? If yes,what?
> > > Would suggest you inform your doctor(s) about your having excessive
> > > phlegm.
> >
> > It is temporary. However how it is linked to diabitics?
>
> By the predisposition toward infection because of weakened immune
> system.
Can we get more mucus/phelgum discharge, Na/Ca or othr mineral is
instable or excess?
" Mucin
Mucus is produced by submucosal cells as well as goblet cells in the
respiratory system. It consists of mucin, a highly glycolsilated
peptide. Upon stimulation, MARPKs (myrastine-alanine rich protein
kinases) signal the binding of mucin filled vesciles to the plasma
membrane. **The fusion of the vesciles causes the release of the mucin,
which as it exchanges Ca2+ for Na+ expands up to 600 fold. The result
is a viscoelastic product of interwoven molecules called mucus.**
http://en.wikipedia.org/wiki/Mucus "
Pls simplify; "The fusion of the vesciles causes the release of the
mucin, which as it exchanges Ca2+ for Na+ expands up to 600 fold."
Andrew B. Chung, MD/PhD
> Andrew, in the Holy Spirit, boldly wrote:
> > Convicted neighbor Kumar wrote:
> > > Andrew, in the Holy Spirit, boldly wrote:
> > > > Convicted neighbor Kumar wrote:
> > > > > Andrew, in the Holy Spirit, boldly wrote:
> > > > > > Convicted neighbor "Kumar" wrote:
> > > > > >snip> >
> > > > > > > > > Will it not effect quantity of glucose delivery to extravascular target
> > > > > > > > > tissues?
> > > > > > >
> > > > > > > > The glucotoxic effect of hyperglycemia is concentration and not
> > > > > > > > flow-dependent.
> > > > > >
> > > > > > > Decreased blood flw can also mean decreased
> > > > > > > concenrated glucose delivery?
> > > > > >
> > > > > > Decrease blood flow means ischemia.
> > > > >
> > > > > Not from obstruction but just reduced.
> > > >
> > > > There is no reduction in blood flow if there is no obstruction.
> > >
> > > Local blood flow regulation can be without atherosclerosis?
> >
> > Local blood flow regulation is without ischemia.
>
> yes, therefore decreased blood flow/supply without obstruction,
Regulated blood flow is not overall decreased blood flow.
> > > > > "Ischemia: Reduced blood flow to an area of the body due to an
> > > > > obstructed vessel."
> > > > >
> > > > > Btw, can excessive phelgum/mucus discharge be due to relaxed muscular
> > > > > tone of respiratory tract?
> > > >
> > > > Not clinically seen.
> > >
> > > How/why phelgum is more or less salty & thin or thick?
> >
> Can such variations in phelgum/mucus meant for some regulatory routine
> mechanism of body? If yes,what?
No.
> > > > Would suggest you inform your doctor(s) about your having excessive
> > > > phlegm.
> > >
> > > It is temporary. However how it is linked to diabitics?
> >
> > By the predisposition toward infection because of weakened immune
> > system.
>
> Can we get more mucus/phelgum discharge, Na/Ca or othr mineral is
> instable or excess?
> " Mucin
> Mucus is produced by submucosal cells as well as goblet cells in the
> respiratory system. It consists of mucin, a highly glycolsilated
> peptide. Upon stimulation, MARPKs (myrastine-alanine rich protein
> kinases) signal the binding of mucin filled vesciles to the plasma
> membrane. **The fusion of the vesciles causes the release of the mucin,
> which as it exchanges Ca2+ for Na+ expands up to 600 fold. The result
> is a viscoelastic product of interwoven molecules called mucus.**
> http://en.wikipedia.org/wiki/Mucus "
>
> Pls simplify; "The fusion of the vesciles causes the release of the
> mucin, which as it exchanges Ca2+ for Na+ expands up to 600 fold."
Changes in sputum color and consistency in a diabetic will be more
likely from infection rather than from change in mucin content.
Kumar
Andrew B. Chung, MD/PhD wrote:
> Convicted neighbor Kumar wrote:
> > Andrew, in the Holy Spirit, boldly wrote:
> > > Convicted neighbor Kumar wrote:
> > > > Andrew, in the Holy Spirit, boldly wrote:
> > > > > Convicted neighbor Kumar wrote:
> > > > > > Andrew, in the Holy Spirit, boldly wrote:
> > > > > > > Convicted neighbor "Kumar" wrote:
> > > > > > >snip> >
> > > > > > > > > > Will it not effect quantity of glucose delivery to extravascular target
> > > > > > > > > > tissues?
> > > > > > > >
> > > > > > > > > The glucotoxic effect of hyperglycemia is concentration and not
> > > > > > > > > flow-dependent.
> > > > > > >
> > > > > > > > Decreased blood flw can also mean decreased
> > > > > > > > concenrated glucose delivery?
> > > > > > >
> > > > > > > Decrease blood flow means ischemia.
> > > > > >
> > > > > > Not from obstruction but just reduced.
> > > > >
> > > > > There is no reduction in blood flow if there is no obstruction.
> > > >
> > > > Local blood flow regulation can be without atherosclerosis?
> > >
> > > Local blood flow regulation is without ischemia.
> >
> > yes, therefore decreased blood flow/supply without obstruction,
>
> Regulated blood flow is not overall decreased blood flow.
Decreased blood flow to specific part?
In non-diabetics and in excessive quanity?
However, I was willing to understand above mecanism of Ca/Na exchange?
Andrew B. Chung, MD/PhD
> Andrew, in the Holy Spirit, boldly wrote:
> > Convicted neighbor Kumar wrote:
> > > Andrew, in the Holy Spirit, boldly wrote:
> > > > Convicted neighbor Kumar wrote:
> > > > > Andrew, in the Holy Spirit, boldly wrote:
> > > > > > Convicted neighbor Kumar wrote:
> > > > > > > Andrew, in the Holy Spirit, boldly wrote:
> > > > > > > > Convicted neighbor "Kumar" wrote:
> > > > > > > >snip> >
> > > > > > > > > > > Will it not effect quantity of glucose delivery to extravascular target
> > > > > > > > > > > tissues?
> > > > > > > > >
> > > > > > > > > > The glucotoxic effect of hyperglycemia is concentration and not
> > > > > > > > > > flow-dependent.
> > > > > > > >
> > > > > > > > > Decreased blood flw can also mean decreased
> > > > > > > > > concenrated glucose delivery?
> > > > > > > >
> > > > > > > > Decrease blood flow means ischemia.
> > > > > > >
> > > > > > > Not from obstruction but just reduced.
> > > > > >
> > > > > > There is no reduction in blood flow if there is no obstruction.
> > > > >
> > > > > Local blood flow regulation can be without atherosclerosis?
> > > >
> > > > Local blood flow regulation is without ischemia.
> > >
> > > yes, therefore decreased blood flow/supply without obstruction,
> >
> > Regulated blood flow is not overall decreased blood flow.
>
> Decreased blood flow to specific part?
Would generally be from an obstruction if there is ischemia of that
specific part.
Infection still needs to be considered.
> However, I was willing to understand above mecanism of Ca/Na exchange?
Seems like a preoccupation to me.
Kumar
Andrew B. Chung, MD/PhD wrote:
How ischemia and decreased blood flow as a regulation of local long
term blood flow control are differenent?
Can phelgum be injurious to resiratory tract due to changes in its
type?
Whether changes in quality and quantity of mucus is the main reason of
getting asthama?
> > However, I was willing to understand above mecanism of Ca/Na exchange?
>
> Seems like a preoccupation to me.
Pls make it more clear to me.
Andrew B. Chung, MD/PhD
> Andrew, in the Holy Spirit, boldly wrote:
> > convicted neighbor Kumar wrote:
> > > Andrew, in the Holy Spirit, boldly wrote:
> > > > Regulated blood flow is not overall decreased blood flow.
> > >
> > > Decreased blood flow to specific part?
> >
> > Would generally be from an obstruction if there is ischemia of that
> > specific part.
>
> How ischemia and decreased blood flow as a regulation of local long
> term blood flow control are differenent?
Ischemia is pathological.
Changes in either color or consistency of phlegm can be a marker of
underlying infection which would be injurious.
> Whether changes in quality and quantity of mucus is the main reason of
> getting asthama?
Folks with asthma are more likely to be afflicted with the airway
infections that would cause changes in either the color or consistency
of their phlegm.
> > > However, I was willing to understand above mecanism of Ca/Na exchange?
> >
> > Seems like a preoccupation to me.
> Pls make it more clear to me.
An understanding of Ca++/Na+ exchange in phlegm would not help you
manage your condition.
It would be wiser for you to inform your doctor(s) about the changes in
color, consistency, and amount of your sputum.
Kumar
Andrew B. Chung, MD/PhD wrote:
> convicted neighbor Kumar wrote:
> > Andrew, in the Holy Spirit, boldly wrote:
> > > convicted neighbor Kumar wrote:
> > > > Andrew, in the Holy Spirit, boldly wrote:
> <snip>
> > > > > Regulated blood flow is not overall decreased blood flow.
> > > >
> > > > Decreased blood flow to specific part?
> > >
> > > Would generally be from an obstruction if there is ischemia of that
> > > specific part.
> >
> > How ischemia and decreased blood flow as a regulation of local long
> > term blood flow control are differenent?
>
> Ischemia is pathological.
Local long term blood flow control?
Can Local long term blood flow control become pathological?
How post nasal drip can be irritating to upper resiratory tract leading
to sore throat? Can phelgum directly irritate mucus wall of resp. tract
due to its changed composition?
> > Whether changes in quality and quantity of mucus is the main reason of
> > getting asthama?
>
> Folks with asthma are more likely to be afflicted with the airway
> infections that would cause changes in either the color or consistency
> of their phlegm.
Asthma can be triggered by non-infectious allergens. What is the main
reason to etting asthma?
Is it related to diabetes?
> > > > However, I was willing to understand above mecanism of Ca/Na exchange?
> > >
> > > Seems like a preoccupation to me.
>
> > Pls make it more clear to me.
>
> An understanding of Ca++/Na+ exchange in phlegm would not help you
> manage your condition.
Still I want to understand pls? In one understading manipulating
balance of Na and bio-availabilty of Ca control excessive mucus
discharge. Probably by vasoconstriction?
> It would be wiser for you to inform your doctor(s) about the changes in
> color, consistency, and amount of your sputum.
Thanks but I want to understand for different reason.
Can infection be without fever?
Andrew B. Chung, MD/PhD
> Andrew, in the Holy Spirit, boldly wrote:
> > convicted neighbor Kumar wrote:
> > > Andrew, in the Holy Spirit, boldly wrote:
> > > > convicted neighbor Kumar wrote:
> > > > > Andrew, in the Holy Spirit, boldly wrote:
> > <snip>
> > > > > > Regulated blood flow is not overall decreased blood flow.
> > > > >
> > > > > Decreased blood flow to specific part?
> > > >
> > > > Would generally be from an obstruction if there is ischemia of that
> > > > specific part.
> > >
> > > How ischemia and decreased blood flow as a regulation of local long
> > > term blood flow control are differenent?
> >
> > Ischemia is pathological.
>
> Local long term blood flow control?
Would not cause ischemia.
> Can Local long term blood flow control become pathological?
That which is pathological by definition is without control.
A sore throat is a symptom of infection.
> > > Whether changes in quality and quantity of mucus is the main reason of
> > > getting asthama?
> >
> > Folks with asthma are more likely to be afflicted with the airway
> > infections that would cause changes in either the color or consistency
> > of their phlegm.
>
> Asthma can be triggered by non-infectious allergens. What is the main
> reason to etting asthma?
Airway inflammation.
> Is it related to diabetes?
Type 2 diabetes is a pro-inflammatory condition.
> > > > > However, I was willing to understand above mecanism of Ca/Na exchange?
> > > >
> > > > Seems like a preoccupation to me.
> >
> > > Pls make it more clear to me.
> >
> > An understanding of Ca++/Na+ exchange in phlegm would not help you
> > manage your condition.
>
> Still I want to understand pls? In one understading manipulating
> balance of Na and bio-availabilty of Ca control excessive mucus
> discharge. Probably by vasoconstriction?
Not clinically observed.
> > It would be wiser for you to inform your doctor(s) about the changes in
> > color, consistency, and amount of your sputum.
>
> Thanks but I want to understand for different reason.
>
> Can infection be without fever?
Yes.
Kumar
Andrew B. Chung, MD/PhD wrote:
> convicted neighbor Kumar wrote:
> > Andrew, in the Holy Spirit, boldly wrote:
> > > convicted neighbor Kumar wrote:
> > > > Andrew, in the Holy Spirit, boldly wrote:
> > > > > convicted neighbor Kumar wrote:
> > > > > > Andrew, in the Holy Spirit, boldly wrote:
> > > <snip>
> > > > > > > Regulated blood flow is not overall decreased blood flow.
> > > > > >
> > > > > > Decreased blood flow to specific part?
> > > > >
> > > > > Would generally be from an obstruction if there is ischemia of that
> > > > > specific part.
> > > >
> > > > How ischemia and decreased blood flow as a regulation of local long
> > > > term blood flow control are differenent?
> > >
> > > Ischemia is pathological.
> >
> > Local long term blood flow control?
>
> Would not cause ischemia.
>
> > Can Local long term blood flow control become pathological?
>
> That which is pathological by definition is without control.
pathology..say by decrease/increase in size and numbers of capalliries
supply to tissues?
or in its secretions, which can be real cause of getting infection?
> > > > Whether changes in quality and quantity of mucus is the main reason of
> > > > getting asthama?
> > >
> > > Folks with asthma are more likely to be afflicted with the airway
> > > infections that would cause changes in either the color or consistency
> > > of their phlegm.
> >
> > Asthma can be triggered by non-infectious allergens. What is the main
> > reason to etting asthma?
>
> Airway inflammation.
> > Is it related to diabetes?
>
> Type 2 diabetes is a pro-inflammatory condition.
body?
> > > > > > However, I was willing to understand above mecanism of Ca/Na exchange?
> > > > >
> > > > > Seems like a preoccupation to me.
> > >
> > > > Pls make it more clear to me.
> > >
> > > An understanding of Ca++/Na+ exchange in phlegm would not help you
> > > manage your condition.
> >
> > Still I want to understand pls? In one understading manipulating
> > balance of Na and bio-availabilty of Ca control excessive mucus
> > discharge. Probably by vasoconstriction?
>
> Not clinically observed.
How taking steam or drinking hot water can promote productive cough or
expectoration?
> > > It would be wiser for you to inform your doctor(s) about the changes in
> > > color, consistency, and amount of your sputum.
> >
> > Thanks but I want to understand for different reason.
> >
> > Can infection be without fever?
>
> Yes.
Pls tell me more about infection with or without fever?
Andrew B. Chung, MD/PhD
> Andrew, in the Holy Spirit, boldly wrote:
> > convicted neighbor Kumar wrote:
<snip>
> > > Can Local long term blood flow control become pathological?
> >
> > That which is pathological by definition is without control.
>
> Yes but can Local long term blood flow control result into some
> pathology..say by decrease/increase in size and numbers of capalliries
> supply to tissues?
No.
Kumar
Andrew B. Chung, MD/PhD wrote:
> > Andrew, in the Holy Spirit, boldly wrote:
> > > convicted neighbor Kumar wrote:
>
> <snip>
>
> > > > Can Local long term blood flow control become pathological?
> > >
> > > That which is pathological by definition is without control.
> >
> > Yes but can Local long term blood flow control result into some
> > pathology..say by decrease/increase in size and numbers of capalliries
> > supply to tissues?
>
> No.
supplying the tissues can't be pathological?
Can't such blood flow variations be pathological?
Andrew B. Chung, MD/PhD
> Andrew, in the Holy Spirit, boldly wrote:
> > Convicted neigbor Kumar wrote:
> > > Andrew, in the Holy Spirit, boldly wrote:
> > > > convicted neighbor Kumar wrote:
> >
> > <snip>
> >
> > > > > Can Local long term blood flow control become pathological?
> > > >
> > > > That which is pathological by definition is without control.
> > >
> > > Yes but can Local long term blood flow control result into some
> > > pathology..say by decrease/increase in size and numbers of capalliries
> > > supply to tissues?
> >
> > No.
>
> Why such decrease/increase in size and numbers of blood vessels
> supplying the tissues can't be pathological?
Local blood flow control is normal physiology.
> Can't such blood flow variations be pathological?
Again, it is normal physiology.
Kumar
Andrew B. Chung, MD/PhD wrote:
> > Andrew, in the Holy Spirit, boldly wrote:
> > > convicted neighbor Kumar wrote:
>
> <snip>
>
> > > > Can Local long term blood flow control become pathological?
> > >
> > > That which is pathological by definition is without control.
> >
> > Yes but can Local long term blood flow control result into some
> > pathology..say by decrease/increase in size and numbers of capalliries
> > supply to tissues?
>
> No.
regulation and present somewhat withdrawl symptoms?
Whether Rosacea or reddened face of people living on high altitude is
as a result of long term local blood flow regulation?
Andrew B. Chung, MD/PhD
> Andrew, in the Holy Spirit, boldly wrote:
> > Convicted neigbor Kumar wrote:
> > > Andrew, in the Holy Spirit, boldly wrote:
> > > > convicted neighbor Kumar wrote:
> >
> > <snip>
> >
> > > > > Can Local long term blood flow control become pathological?
> > > >
> > > > That which is pathological by definition is without control.
> > >
> > > Yes but can Local long term blood flow control result into some
> > > pathology..say by decrease/increase in size and numbers of capalliries
> > > supply to tissues?
> >
> > No.
>
> Can such changes cause a person becoming habitual of changed long term
> regulation and present somewhat withdrawl symptoms?
No.
> Whether Rosacea or reddened face of people living on high altitude is
> as a result of long term local blood flow regulation?
This likely has more to do with UV radiation exposure.
Kumar
Andrew B. Chung, MD/PhD wrote:
> > Andrew, in the Holy Spirit, boldly wrote:
> > > Convicted neigbor Kumar wrote:
> > > > Andrew, in the Holy Spirit, boldly wrote:
> > > > > convicted neighbor Kumar wrote:
> > >
> > > <snip>
> > >
> > > > > > Can Local long term blood flow control become pathological?
> > > > >
> > > > > That which is pathological by definition is without control.
> > > >
> > > > Yes but can Local long term blood flow control result into some
> > > > pathology..say by decrease/increase in size and numbers of capalliries
> > > > supply to tissues?
> > >
> > > No.
> >
> > Can such changes cause a person becoming habitual of changed long term
> > regulation and present somewhat withdrawl symptoms?
>
> No.
flow regulation, say by coming to plains from high altitude?
> > Whether Rosacea or reddened face of people living on high altitude is
> > as a result of long term local blood flow regulation?
>
> This likely has more to do with UV radiation exposure.
numbers of arteries supplying to tissues to enhance normal O2 to
tissues. I given impact on reversing such effect previously from a
quote from Textbook of Medical Physiology b guyton.
Andrew B. Chung, MD/PhD
> Andrew, in the Holy Spirit, boldly wrote:
> > Convicted neighbor Kumar wrote:
> > > Andrew, in the Holy Spirit, boldly wrote:
> > > > Convicted neigbor Kumar wrote:
> > > > > Andrew, in the Holy Spirit, boldly wrote:
> > > > > > convicted neighbor Kumar wrote:
> > > >
> > > > <snip>
> > > >
> > > > > > > Can Local long term blood flow control become pathological?
> > > > > >
> > > > > > That which is pathological by definition is without control.
> > > > >
> > > > > Yes but can Local long term blood flow control result into some
> > > > > pathology..say by decrease/increase in size and numbers of capalliries
> > > > > supply to tissues?
> > > >
> > > > No.
> > >
> > > Can such changes cause a person becoming habitual of changed long term
> > > regulation and present somewhat withdrawl symptoms?
> >
> > No.
> How a person can react to sudden reversal of changed long term blood
> flow regulation, say by coming to plains from high altitude?
Our bodies are wonderfully made by GOD, Creator of heaven and earth.
> > > Whether Rosacea or reddened face of people living on high altitude is
> > > as a result of long term local blood flow regulation?
> >
> > This likely has more to do with UV radiation exposure.
>
> I think low O2 levels at higher altitude can cause increase in size and
> numbers of arteries supplying to tissues to enhance normal O2 to
> tissues. I given impact on reversing such effect previously from a
> quote from Textbook of Medical Physiology b guyton.
Reddening of the face would not represent an increase in the size and
number of arteries supplying tissues.
Kumar
Mu
>>> I think low O2 levels at higher altitude can cause increase in size and
>>> numbers of arteries supplying to tissues to enhance normal O2 to
>>> tissues. I given impact on reversing such effect previously from a
>>> quote from Textbook of Medical Physiology b guyton.
>>
>> Reddening of the face would not represent an increase in the size and
>> number of arteries supplying tissues.
> Relaxed blood vessels?
More transluscent skin, vessels rising closer to the epidermis, etc.
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Andrew B. Chung, MD/PhD
Depends on the underlying cause. If UV radiation, there is tissue
damage.