Rhinoconjunctivitis And Meat Eating

[ironjustice]

"18 percent of health care workers have some level of latex allergy"

Dietary meat and fat intake and prevalence of rhinoconjunctivitis in
pregnant Japanese women: baseline data from the Kyushu Okinawa
Maternal and Child Health Study.
Nutr J. 2012 Mar 27;11(1):19.
Miyake Y, Tanaka K, Okubo H, Sasaki S, Arakawa M.
Abstract
ABSTRACT:

BACKGROUND: Dietary fat exerts numerous complex effects on
proinflammatory and immunologic pathways. Several epidemiological
studies have examined the relationships between intake of fatty acids
and/or foods high in fat and allergic rhinitis, but have provided
conflicting findings. The current cross-sectional study investigated
such relationships in Japan.

METHODS: Study subjects were 1745 pregnant women. The definition of
rhinoconjunctivitis was based on criteria from the International Study
of Asthma and Allergies in Childhood. Information on dietary factors
was collected using a validated self-administered diet history
questionnaire. Adjustment was made for age; gestation; region of
residence; number of older siblings; number of children; smoking;
secondhand smoke exposure at home and at work; family history of
asthma, atopic eczema, and allergic rhinitis; household income;
education; and body mass index.

RESULTS: The prevalence of rhinoconjunctivitis in the past 12 months
was 25.9%. Higher meat intake was significantly associated with an
increased prevalence of rhinoconjunctivitis: the adjusted odds ratio
between extreme quartiles was 1.71 (95% confidence interval:
1.25-2.35, P for trend = 0.002). No measurable association was found
between fish intake and rhinoconjunctivitis. Intake of total fat,
saturated fatty acids, monounsaturated fatty acids, n-3
polyunsaturated fatty acids, alpha-linolenic acid, eicosapentaenoic
acid, docosahexaenoic acid, n-6 polyunsaturated fatty acids, linoleic
acid, arachidonic acid, and cholesterol and the ratio of n-3 to n-6
polyunsaturated fatty acid intake were not evidently related to the
prevalence of rhinoconjunctivitis.

CONCLUSIONS: The current results suggest that meat intake may be
positively associated with the prevalence of rhinoconjunctivitis in
young adult Japanese women.

PMID:22449171

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http://www.mechdir.com/press/catalog/1079/index.html

"18 percent of health care workers have some level of latex allergy"

------------------

Excess dietary iron is the root cause for increase in childhood
Autism
and allergies
Medical Hypotheses, Volume 61, Issue 2, Pages 220-222
Medical Hypotheses
Abstract
Autism is a profoundly and poorly understood developmental disorder
that impairs a person’s social and communication abilities.
I propose a hypothesis that the excessive dietary iron consumed by
today’s infants is the root cause of increased cases of Autism,
allergies and other childhood diseases. Iron is a powerful immune
system modulator.
Excess iron causes hyperactive immune system.
This hyperactive immune system attacks undigested food peptides.
The chemicals released during these intense allergic reactions can
damage surrounding tissue. Neurodegeneration is caused by combination
of, oxidative stress induced by free iron radicals and intense immune
reactions.
Iron chelators have shown beneficial results in Autism and allergies.


doi:10.1016/S0306-9877(03)00126-9

-------------------------------

"Loading cells with Fe-D increased their response to latex"

Iron uptake and release by macrophages is sensitive to propranolol.
Mol Cell Biochem. 2006 Aug;288(1-2):213-7. Epub 2006 May 23.
Komarov AM, Hall JM, Chmielinska JJ, Weglicki WB.
Division of Experimental Medicine, Departments of Biochemistry &
Molecular Biology, The George Washington University Medical Center,
2300 Eye St., NW, Ross Hall, Rm. 441A, Washington, DC 20037,
USA. phy...@gwumc.edu


In this study we have tested the effects of d-propranolol (D-Pro) on
the iron uptake, iron release and oxidative response of iron-loaded
cells
in a cellular model of iron-overload using isolated rat peritoneal
macrophages incubated with iron-dextran (Fe-D).
Pretreatment of macrophages with D-Pro (5-200 microM) prior to Fe-D
exposure decreased the cellular iron content and partially prevented
iron release from latex-activated macrophages.
Release of reactive oxygen species from activated cells was detected
by dichlorodihydrofluorescein (DCDHF, 5 microM) oxidation.
We found that loading cells with Fe-D increased their response to
latex, which was prevented by the lysosomotropic antioxidant agent D-
Pro
(10 microM).

PMID: 16718379

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Latex allergy is a double-edged sword, affecting not only
patients, which include people of all ages who have undergone
multiple surgical / medical procedures, atopic or allergic
individuals and rubber industry workers, but the very people who
are responsible for providing their health care. This is a
monumental problem, but not insurmountable.

There are a few basic changes that can be made to reduce the
exposure to both populations, thereby reducing the risk of
life-threatening reactions and increasing the sensitivity in
those already allergic, and significantly reducing the risk of
becoming sensitized in those who are not yet allergic.

Eliminate powdered gloves. Dr. Dennis Ownby, Dr. Loren Hunt, Dr.
Kevin Kelly and many, many other leading specialists have stated
that exposure to latex through inhalation is a significant
route of exposure. Wherever possible, use latex alternative
gloves. There are plenty of cost-effective, non-latex gloves
available. If latex gloves must be used, use only powder-free
and low latex protein. Latex glove use should be as limited as
possible.

Educate, educate and educate. Identify high-risk populations and
refer those symptomatic individuals to an allergist, well-versed
in latex allergy, for consultation and testing. Conduct
inservices pertaining to appropriate glove use, barrier
protection is essential. These programs should include the risk
of using latex gloves along with signs and symptoms of the
various types of adverse glove reactions. Early detection is
essential!

There are other steps that will provide a latex-safe environment
for both patient and health care worker, we must encourage
awareness so that the appropriate measures can be taken. Future
generations are continuing to be sensitized to latex through
indiscriminate latex glove use.

Latex is everywhere, once you are sensitized, the only treatment
is avoidance. The implications are overwhelming, imagine trying
to practice safe-sex with a latex allergy! The next time you are
in a restaurant, day-care center, grocery store or hair salon,
just to name some common locations, ask if latex gloves are used
on the premise. You may be surprised at the places where these
products are being used.

Those of us who are already sensitized are merely the tip of the
iceberg. Medical costs, workers' compensation costs, disability
costs and the cost of litigation should alert everyone to the
potential expense of ignoring this issue and the continued,
sweeping use of powdered latex gloves. Who can assign a monetary
value to a life? I was always taught that a life is a precious,
priceless commodity.

I have lost my career as a dentist due to becoming anaphylactic
to latex and developing severe occupational asthma that has left
me virtually disabled. Even though I have been out of practice
for a year and a half, the asthma continues to worsen. I am one
of an alarmingly increasing number of highly educated, frustrated
professionals who have not only lost their ability to practice,
but have lost their quality of life. Some have even lost their
lives. The time is now to stop the sensitization process.

ELASTIC, Education for Latex Allergy \ Support Team & Information
Coalition, is a national networking organization, with a monthly
newsletter, Latex Allergy News.

For more information regarding ELASTIC, please contact:

Lise Borel 610-436-4801 ~~or~~ Debi Adkins 203-482-6869

Elizabeth C. Borel DMD


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The following is an excerpt from an article published in Patient
Care, Feb. 15, 1996 v30 n3 p32(3).


Latex allergy: potentially disabling.
Nancy Walsh D'Epiro

"Latex is the allergen of the '90s. We simply can't afford to
sensitize and potentially lose a large segment of our highly
trained, well-paid health care professionals," says Kevin J.
Kelly, MD, Director of Pediatric Allergy and Immunology at
Children's Hospital of Wisconsin in Milwaukee. "I've seen
physicians, surgeons, nurses, laboratory technicians, and
researchers develop such severe occupational asthma that they
become totally disabled professionally."

Latex allergy is an unexpected result of the adoption of
universal precautions against infectious diseases in recent
years. Because products manufactured from the milky sap of Hevea
brasiliensis are impermeable to transmissible viruses -especially
HIV and hepatitis-the demand for them has increased
exponentially. Latex now is ubiquitous in the health care
environment and is used in dressings, catheters, tapes, and, most
notably, protective gloves. Before the adoption of universal
precautions, an estimated 300 million gloves were used annually.
This figure has grown to 9.6 billion per year.

The initial manifestation of latex allergy is an urticarial
condition that develops on the hands, often after an initial
contact dermatitis that may break down the skin barrier. In up
to 50% of exposed persons, however, the condition also affects
the respiratory tract. This occurs because high concentrations
of powder on latex gloves become air, inhaled by workers in
hospitals and clinics. The result can be severe
rhinoconjunctivitis, asthma, and anaphylaxis. Fatalities have
occurred.

Approximately 15-17% of health care workers are now thought to be
sensitized to latex, and many of them manifesting occupational
asthma may have permanent respiratory sequelae that force them to
leave their jobs.

Who loves ya.
Tom


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